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Clinical Endocrinology, ISSN 0300-0664, 02/2018, Volume 88, Issue 2, pp. 333 - 340
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2007, Volume 104, Issue 25, pp. 10589 - 10594
Vessel loss precipitates many diseases. In particular, vessel loss resulting in hypoxia induces retinal neovascularization in diabetic retinopathy and in... 
Diabetic angiopathies | Diabetic retinopathy | Oxygen | Messenger RNA | Blood vessels | Retina | Regrowth | Hypoxia | Mice | Infants | Angiogenesis | Retinopathy of prematurity | Insulin-like growth factor | Binding protein-3 | Insulin-like growth factor 1 | Stem cells | SURVIVAL | stem cells | FACTOR-BINDING PROTEIN-3 | angiogenesis | MULTIDISCIPLINARY SCIENCES | MOUSE | binding protein-3 | DNA-SYNTHESIS | retinopathy of prematurity | BREAST-CANCER | ENDOTHELIAL-CELLS | DISEASE | LINE | insulin-like growth factor | insulin-like growth factor 1 | PREMATURITY | GROWTH-FACTOR-I | Insulin-Like Growth Factor Binding Protein 3 - genetics | Retinopathy of Prematurity - classification | Humans | Insulin-Like Growth Factor Binding Protein 3 - blood | Insulin-Like Growth Factor Binding Protein 3 - deficiency | Insulin-Like Growth Factor Binding Protein 3 - pharmacology | Retinal Neovascularization - metabolism | RNA, Messenger - metabolism | Retinopathy of Prematurity - physiopathology | Dose-Response Relationship, Drug | Retina - cytology | Hyperoxia - complications | Infant, Newborn | Disease Models, Animal | Retinopathy of Prematurity - etiology | Gene Expression Regulation | Mice, Transgenic | Gestational Age | Mice, Knockout | Animals | Infant, Premature | Retinal Neovascularization - chemically induced | Insulin-Like Growth Factor Binding Protein 1 - blood | Retinopathy of Prematurity - metabolism | Retinopathy of Prematurity - pathology | Angiogenesis inhibitors | Research | Retrolental fibroplasia | Binding proteins | Biological Sciences | like growth factor 1 | insulin | like growth factor binding protein | Premature | Disease Models | RNA | Transgenic | Retinal Neovascularization/chemically induced/metabolism | Infant | MEDICIN OCH HÄLSOVETENSKAP | Insulin-Like Growth Factor Binding Protein 1/blood | Prematurity/classification/etiology/metabolism/pathology/physiopathology | Hyperoxia/complications | MEDICAL AND HEALTH SCIENCES | Drug | Insulin-Like Growth Factor Binding Protein | Retinopathy of | Knockout | Messenger/metabolism | Retina/cytology | 3/blood/deficiency/genetics/pharmacology | Newborn | Animal | Dose-Response Relationship
Journal Article
Journal Article
Cancer Epidemiology Biomarkers & Prevention, ISSN 1055-9965, 05/2009, Volume 18, Issue 5, pp. 1333 - 1340
Journal Article
International Journal of Cancer, ISSN 0020-7136, 07/2007, Volume 121, Issue 2, pp. 368 - 376
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, p. e0125023
Background Protein-energy malnutrition (PEM) can lead to growth hormone (GH) resistance. Leucine supplementation diets have been shown to increase protein... 
SOMATOMEDIN-C | RESTRICTED RATS | CELLS | ACID | REDUCTION | I IGF-I | MULTIDISCIPLINARY SCIENCES | LIVER | BINDING PROTEIN | AXIS | CHILDREN | Insulin-Like Growth Factor Binding Protein 3 - genetics | TOR Serine-Threonine Kinases - metabolism | Insulin-Like Growth Factor Binding Protein 3 - blood | Male | Muscle, Skeletal - metabolism | Insulin-Like Growth Factor I - genetics | RNA, Messenger - metabolism | Disease Models, Animal | Protein-Energy Malnutrition - pathology | Growth Hormone - blood | Signal Transduction | Growth Hormone - metabolism | Liver - metabolism | RNA, Messenger - genetics | Leucine - administration & dosage | Rats | Rats, Sprague-Dawley | Receptors, Somatotropin - genetics | Animals | Protein-Energy Malnutrition - diet therapy | Protein-Energy Malnutrition - metabolism | Insulin-Like Growth Factor Binding Protein 1 - blood | Muscle, Skeletal - pathology | Dietary Supplements | Insulin-Like Growth Factor I - metabolism | Insulin-Like Growth Factor Binding Protein 1 - genetics | Somatotropin | RNA | Protein-energy malnutrition | Muscles | Amino acids | Protein biosynthesis | Cellular signal transduction | Malnutrition | Protein binding | TOR protein | Phosphorylation | Insulin-like growth factor I | Liver | Insulin-like growth factor-binding protein 1 | AKT protein | Insulin-like growth factors | Leucine | Signal transduction | Energy | Supplementation | Dietary supplements | Rapamycin | Ribosomal protein S6 | Gene expression | Insulin | Skeletal muscle | Ribosomal protein S6 kinase | Signaling | Protein synthesis | Effectors | Transduction | Growth hormone
Journal Article
Journal of Clinical Endocrinology and Metabolism, ISSN 0021-972X, 01/2017, Volume 102, Issue 1, pp. 267 - 278
Journal Article
Journal Article