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Journal Article
Science, ISSN 0036-8075, 05/2016, Volume 352, Issue 6288, pp. aad3018 - aad3018
Macrophages accumulate with glioblastoma multiforme (GBM) progression and can be targeted via inhibition of colony-stimulating factor-1 receptor (CSF-1R) to... 
B-CELLS | INSULIN | MACROPHAGE PLASTICITY | GLIOBLASTOMA | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GROWTH | RECEPTOR | CENTRAL-NERVOUS-SYSTEM | CANCER | PROGRESSION | Human Umbilical Vein Endothelial Cells | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Drug Resistance, Neoplasm | Phosphatidylinositol 3-Kinases - metabolism | Pyrazines - therapeutic use | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Neoplasms, Experimental - immunology | Imidazoles - therapeutic use | Macrophages - immunology | Picolinic Acids - pharmacology | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - antagonists & inhibitors | Neoplasm Recurrence, Local - metabolism | Signal Transduction | Benzothiazoles - pharmacology | NFATC Transcription Factors - metabolism | Neoplasms, Experimental - therapy | Imidazoles - pharmacology | STAT6 Transcription Factor - metabolism | Mice, Inbred Strains | Tumor Microenvironment - immunology | Animals | Glioblastoma - immunology | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Picolinic Acids - therapeutic use | Macrophages - drug effects | Benzothiazoles - therapeutic use | Mice | Glioblastoma - drug therapy | Pyrazines - pharmacology | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Cancer therapies | Brain cancer | Tumors | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 09/2018, Volume 115, Issue 38, pp. E8948 - E8957
Angiogenesis is essential in the early stage of solid tumor recurrence, but how a suspensive tumor is reactivated before angiogenesis is mostly unknown.... 
Cancer stem cell | Angiogenesis | Recurrence | Self-renewal | IGF-1 | TUMOR DORMANCY OFFER | IGF-I | angiogenesis | MULTIDISCIPLINARY SCIENCES | RISK | self-renewal | recurrence | INHIBITION | cancer stem cell | WNT/BETA-CATENIN | PLASMA-LEVELS | PROSTATE-CANCER | LOW-FAT DIET | MATRIGEL | GROWTH-FACTOR-I | Cell Proliferation | Placenta Growth Factor - antagonists & inhibitors | Humans | Chemokine CXCL1 - antagonists & inhibitors | Placenta Growth Factor - metabolism | Lung Neoplasms - pathology | Phosphatidylinositol 3-Kinases - metabolism | Neovascularization, Pathologic - blood | Neovascularization, Pathologic - pathology | Neoplasm Recurrence, Local - pathology | Neoplastic Stem Cells - pathology | Female | Chemokine CXCL1 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Neoplasm Recurrence, Local - blood | Carcinoma, Non-Small-Cell Lung - pathology | Signal Transduction | Insulin-Like Growth Factor I - analysis | beta Catenin - metabolism | Xenograft Model Antitumor Assays | Animals | Mice, Nude | AC133 Antigen - metabolism | Cell Line, Tumor | Carcinoma, Non-Small-Cell Lung - blood | Lung Neoplasms - blood | Mice | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Relapse | Lung cancer | Cancer cells | Stem cells | Physiological aspects | Development and progression | Neovascularization | Health aspects | Cancer | Insulin-like growth factor I | Xenotransplantation | Lung | Antibodies | Stem cell transplantation | AKT protein | Activation | β-catenin | Expansion | Growth factors | Symmetry | Level (quantity) | Insulin | Patients | Cells | 1-Phosphatidylinositol 3-kinase | Studies | Asymmetry | Solid tumors | Tumors | Apoptosis | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
FEBS Journal, ISSN 1742-464X, 05/2013, Volume 280, Issue 10, pp. 2248 - 2259
Journal Article
Cancer Research, ISSN 0008-5472, 1994, Volume 54, Issue 20, pp. 5474 - 5478
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2013, Volume 110, Issue 37, pp. 15043 - 15048
MicroRNAs (miRNAs) are small 19-to 24-nt noncoding RNAs that have the capacity to regulate fundamental biological processes essential for cancer initiation and... 
MicroRNA | Delta cells | Lungs | Cell lines | Cell cycle | Small interfering RNA | Lung neoplasms | Tumors | Cancer | Apoptosis | SURVIVAL | DIAGNOSIS | SIGNATURES | PROGNOSIS | MULTIDISCIPLINARY SCIENCES | RECEPTOR | COMBINATION | EXPRESSION | CARCINOMA | PROGRESSION | ERLOTINIB | RNA, Small Interfering - genetics | Receptor, IGF Type 1 - metabolism | Class Ia Phosphatidylinositol 3-Kinase - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Cell Proliferation | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | MicroRNAs - metabolism | Insulin-Like Growth Factor I - genetics | Genes, p53 | 3' Untranslated Regions | Class Ia Phosphatidylinositol 3-Kinase - genetics | Genes, Tumor Suppressor | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Receptor, IGF Type 1 - genetics | Class Ia Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Cell Cycle Checkpoints | Animals | Mice, Nude | Cell Line, Tumor | Mice | MicroRNAs - genetics | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Cell Movement | Tissue | Insulin-like growth factors | Ribonucleic acid--RNA | Lung cancer | Index Medicus | Biological Sciences
Journal Article
Developmental Cell, ISSN 1534-5807, 04/2015, Volume 33, Issue 1, pp. 36 - 46
Organ wasting, related to changes in nutrition and metabolic activity of cells and tissues, is observed under conditions of starvation and in the context... 
CANCER CACHEXIA | GENE | RNA-SEQ | MUSCLE ATROPHY | IN-VIVO | GROWTH | HIPPO PATHWAY | TUMOR-SUPPRESSOR | STEM-CELL PROLIFERATION | DEVELOPMENTAL BIOLOGY | DROSOPHILA | CELL BIOLOGY | Metabolomics | Wasting Syndrome - metabolism | Oligonucleotide Array Sequence Analysis | Male | Muscle, Skeletal - metabolism | Gene Expression Profiling | Stem Cells - cytology | Drosophila Proteins - metabolism | Muscle, Skeletal - cytology | Ovary - cytology | Stem Cells - metabolism | Drosophila melanogaster - genetics | Drosophila melanogaster - metabolism | Fat Body - cytology | Hyperglycemia - pathology | Trans-Activators - genetics | Female | Nuclear Proteins - genetics | Fat Body - metabolism | Insulin Secretion | Real-Time Polymerase Chain Reaction | Ovary - metabolism | Biomarkers - metabolism | RNA, Messenger - genetics | Cells, Cultured | Hemolymph - metabolism | Nuclear Proteins - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Animals, Genetically Modified - metabolism | Blotting, Western | Gastrointestinal Tract - metabolism | Hyperglycemia - metabolism | Insulin - metabolism | Animals | Animals, Genetically Modified - growth & development | Animals, Genetically Modified - genetics | Insulin - chemistry | Drosophila melanogaster - growth & development | Trans-Activators - metabolism | Gastrointestinal Tract - cytology | Drosophila Proteins - genetics | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Wasting Syndrome - pathology | Enzymes | Starvation | Analysis | Stem cells | Physiological aspects | Development and progression | Cellular signal transduction | Ovarian cancer | Medical colleges | Resveratrol | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 02/2011, Volume 71, Issue 3, pp. 1029 - 1040
Journal Article
by Guo, XF and Zhu, XF and Cao, HY and Zhong, GS and Li, L and Deng, BG and Chen, P and Wang, PZ and Miao, QF and Zhen, YS
ONCOTARGET, ISSN 1949-2553, 04/2017, Volume 8, Issue 16, pp. 27286 - 27299
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 04/2002, Volume 15, Issue 8, pp. 1327 - 1342
A major reason for the insufficient recovery of function after motor nerve injury are the numerous axonal branches which often re‐innervate muscles with... 
motoneuron | nerve conduit | axotomy | retrograde labelling | rat | facial nerve | Nerve conduit | Facial nerve | Axotomy | Motoneuron | Rat | Retrograde labelling | ROOT GANGLION NEURONS | FIBROBLAST-GROWTH-FACTOR | RAT SCIATIC-NERVE | MOTONEURONS IN-VIVO | NEUROSCIENCES | MESSENGER-RNA | SPINAL MOTOR-NEURONS | ANTI-NGF TREATMENT | FACIAL-NERVE | ADULT-RAT | SCHWANN-CELLS | Brain-Derived Neurotrophic Factor - antagonists & inhibitors | Facial Nerve - immunology | Rats, Wistar | Recovery of Function - drug effects | Facial Nerve - growth & development | Nerve Regeneration - immunology | Neuronal Plasticity - drug effects | Facial Nerve - drug effects | Brain-Derived Neurotrophic Factor - immunology | Schwann Cells - immunology | Facial Nerve Injuries - drug therapy | Insulin-Like Growth Factor I - immunology | Facial Nerve Injuries - immunology | Nerve Growth Factor - antagonists & inhibitors | Glial Cell Line-Derived Neurotrophic Factor | Motor Neurons - cytology | Nerve Growth Factor - immunology | Antibodies - immunology | Female | Growth Cones - immunology | Macrophages - immunology | Motor Neurons - drug effects | Antibodies - therapeutic use | Nerve Tissue Proteins - antagonists & inhibitors | Motor Neurons - immunology | Nerve Tissue Proteins - immunology | Rats | Nerve Growth Factors - immunology | Schwann Cells - metabolism | Antibodies - pharmacology | Cell Differentiation - immunology | Fibroblast Growth Factor 2 - antagonists & inhibitors | Macrophages - metabolism | Recovery of Function - immunology | Animals | Growth Cones - metabolism | Fibroblast Growth Factor 2 - immunology | Cell Differentiation - drug effects | Nerve Regeneration - drug effects | Nerve Growth Factors - antagonists & inhibitors | Growth Cones - drug effects | Neuronal Plasticity - immunology | Fluorescent Dyes | Insulin-Like Growth Factor I - antagonists & inhibitors | Index Medicus
Journal Article
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 02/2014, Volume 13, Issue 2, pp. 399 - 409
Insulin-like growth factor (IGF) signaling is thought to play a role in the development and progression of multiple cancer types. To date, therapeutic... 
CANCER-CELLS | GROWTH-FACTOR RECEPTOR | INSULIN-RECEPTOR | ADVANCED SOLID TUMORS | INHIBITION | ONCOLOGY | COLORECTAL-CANCER | RESISTANCE | MONOCLONAL-ANTIBODY | FACTOR-I | PHASE-I | Neoplasms - metabolism | Antibodies, Neutralizing - administration & dosage | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Male | Insulin-Like Growth Factor II - immunology | Insulin-Like Growth Factor I - immunology | Signal Transduction - immunology | Antibodies, Neutralizing - immunology | Female | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Antibodies, Monoclonal - immunology | Cell Line | Antibodies, Monoclonal - pharmacology | Antibodies, Neutralizing - pharmacology | Treatment Outcome | Mice, Inbred Strains | Blotting, Western | Insulin-Like Growth Factor II - metabolism | Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Sirolimus - administration & dosage | Insulin-Like Growth Factor II - antagonists & inhibitors | Animals | Signal Transduction - drug effects | Antibodies, Monoclonal - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Mice, Nude | Neoplasms - immunology | Receptor, IGF Type 1 - immunology | Cell Line, Tumor | Cell Proliferation - drug effects | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Index Medicus
Journal Article