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Endocrine Reviews, ISSN 1945-7189, 2016, Volume 37, Issue 1, pp. 62 - 110
.... These factors can impair growth by their effects on the GH-IGF axis and also directly at the level of the growth plate via alterations in chondrogenesis and local growth factor signaling... 
LINEAR BONE-GROWTH | PEDIATRIC CROHNS-DISEASE | EXCLUSIVE ENTERAL NUTRITION | I IGF-I | JUVENILE IDIOPATHIC ARTHRITIS | ENDOCRINOLOGY & METABOLISM | PLATE CHONDROCYTE PROLIFERATION | CATCH-UP GROWTH | NECROSIS-FACTOR-ALPHA | RANDOMIZED CONTROLLED-TRIAL | GLUCOCORTICOID-RECEPTOR MODULATOR | Cystic Fibrosis - therapy | Growth Plate - pathology | Recombinant Proteins - therapeutic use | Growth Disorders - pathology | Cystic Fibrosis - physiopathology | Humans | Inflammatory Bowel Diseases - immunology | Cystic Fibrosis - pathology | Growth Plate - immunology | Growth Disorders - etiology | Inflammatory Bowel Diseases - physiopathology | Insulin-Like Growth Factor I - genetics | Cystic Fibrosis - immunology | Human Growth Hormone - therapeutic use | Growth Substances - genetics | Human Growth Hormone - metabolism | Arthritis, Juvenile - therapy | Inflammatory Bowel Diseases - pathology | Growth Plate - drug effects | Puberty, Delayed - etiology | Drug Therapy, Combination | Insulin-Like Growth Factor I - therapeutic use | Child | Recombinant Proteins - metabolism | Human Growth Hormone - genetics | Growth Disorders - immunology | Growth Disorders - prevention & control | Combined Modality Therapy | Puberty, Delayed - immunology | Evidence-Based Medicine | Growth Plate - metabolism | Arthritis, Juvenile - physiopathology | Puberty, Delayed - pathology | Animals | Arthritis, Juvenile - immunology | Growth Substances - therapeutic use | Adolescent | Puberty, Delayed - prevention & control | Arthritis, Juvenile - pathology | Inflammatory Bowel Diseases - therapy | Insulin-Like Growth Factor I - metabolism | Growth Substances - metabolism | Practice Guidelines as Topic
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e76133
Journal Article
Nature medicine, ISSN 1546-170X, 2012, Volume 18, Issue 7, pp. 1095 - 1101
Insulin-like growth factor 1 (IGF-1), the most abundant growth factor in the bone matrix, maintains bone mass in adulthood... 
MEDICINE, RESEARCH & EXPERIMENTAL | LIFE-SPAN | PHYSIOLOGY | RESORPTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | OSTEOPOROSIS | CELL BIOLOGY | INSULIN-RECEPTOR | OSTEOBLAST PROGENITORS | RELEVANCE | FRACTURES | POSTMENOPAUSAL WOMEN | GROWTH-FACTOR-I | Bone Matrix - metabolism | Immunohistochemistry | Mesenchymal Stromal Cells - enzymology | Bone and Bones - pathology | Receptor, IGF Type 1 - metabolism | Insulin-Like Growth Factor I - pharmacology | Femur - pathology | TOR Serine-Threonine Kinases - metabolism | Cell Count | Humans | Aging - drug effects | Insulin-Like Growth Factor Binding Protein 3 - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Femur - diagnostic imaging | Insulin Receptor Substrate Proteins - metabolism | X-Ray Microtomography | Bone and Bones - drug effects | Insulin-Like Growth Factor Binding Protein 3 - metabolism | Bone Matrix - drug effects | Bone and Bones - metabolism | Insulin-Like Growth Factor I - administration & dosage | Bone Resorption - metabolism | Insulin-Like Growth Factor Binding Protein 3 - administration & dosage | Receptor, IGF Type 1 - deficiency | Proto-Oncogene Proteins c-akt - metabolism | Femur - growth & development | Mesenchymal Stromal Cells - drug effects | Bone Resorption - blood | Osteoblasts - enzymology | Osteoblasts - drug effects | Osteogenesis - drug effects | Rats | Enzyme Activation - drug effects | Mice, Knockout | Bone Resorption - diagnostic imaging | Osteoblasts - pathology | Organ Size - drug effects | Animals | Cell Differentiation - drug effects | Bone Resorption - pathology | Mice | Mesenchymal Stromal Cells - pathology | Insulin-Like Growth Factor I - metabolism | Aging - metabolism | Physiological aspects | Research | Insulin-like growth factor 1 | Protein kinases | Stem cells | Bone marrow | Insulin-like growth factors
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 2013, Volume 17, Issue 1, pp. 30 - 54
.... Particularly, the sustained stimulation of epidermal growth factor receptor (EGFR), insulin‐like growth factor‐1 receptor (IGF‐1R), stem cell factor (SCF) receptor KIT, transforming growth factor... 
Hypoxia‐inducible factors | Targeted therapies | Cancer‐initiating cells | Hypoxia | Cancer progression | Metabolic pathways | Cancer stem/progenitor cells | Metastasis‐initiating cells | Metastases | Hypoxia-inducible factors | Cancer-initiating cells | Metastasis-initiating cells | MEDICINE, RESEARCH & EXPERIMENTAL | CLINICALLY RELEVANT SUBTYPES | PANCREATIC-CANCER | Cancer stem | progenitor cells | CHRONIC MYELOID-LEUKEMIA | INHIBITS TUMOR-GROWTH | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | PROSTATE-CANCER | GENE-EXPRESSION | FATTY-ACID SYNTHASE | ENDOTHELIAL GROWTH-FACTOR | Neoplasms - metabolism | Hypoxia - drug therapy | Neoplastic Stem Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Molecular Targeted Therapy | Hypoxia - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neoplasm Metastasis | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Intercellular Signaling Peptides and Proteins - genetics | Transcription Factors - genetics | Neoplasms - drug therapy | Transcription Factors - metabolism | Hypoxia - genetics | Hypoxia - pathology | Cell Transformation, Neoplastic - drug effects | Neoplasms - pathology | Apoptosis | Prevention | Glucose metabolism | Physiological aspects | Development and progression | Genetic transcription | Metastasis | Transforming growth factors | Vascular endothelial growth factor | Cancer | Energy metabolism | Transcription factors | Mesenchyme | AKT protein | Insulin-like growth factors | Kinases | Autophagy | Cancer therapies | Neoplasms | Proteins | Homing | Angiogenesis | Signal transduction | Receptors | Epidermal growth factor | Bone marrow | Tumor necrosis factor-TNF | miRNA | Growth factors | Deprivation | Cell survival | Cytokines | Epidermal growth factor receptors | Breast cancer | Rapamycin | Metabolism | Gene expression | Cell differentiation | Insulin | CXCR4 protein | 1-Phosphatidylinositol 3-kinase | Signaling | Pancreatic cancer | Stem cells | Glycolysis | Regulation | Prostate | Cell migration | Tumors | Reviews
Journal Article