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Cancer Research, ISSN 0008-5472, 02/2011, Volume 71, Issue 3, pp. 1029 - 1040
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 02/2014, Volume 13, Issue 2, pp. 399 - 409
Insulin-like growth factor (IGF) signaling is thought to play a role in the development and progression of multiple cancer types. To date, therapeutic... 
CANCER-CELLS | GROWTH-FACTOR RECEPTOR | INSULIN-RECEPTOR | ADVANCED SOLID TUMORS | INHIBITION | ONCOLOGY | COLORECTAL-CANCER | RESISTANCE | MONOCLONAL-ANTIBODY | FACTOR-I | PHASE-I | Neoplasms - metabolism | Antibodies, Neutralizing - administration & dosage | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Male | Insulin-Like Growth Factor II - immunology | Insulin-Like Growth Factor I - immunology | Signal Transduction - immunology | Antibodies, Neutralizing - immunology | Female | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Antibodies, Monoclonal - immunology | Cell Line | Antibodies, Monoclonal - pharmacology | Antibodies, Neutralizing - pharmacology | Treatment Outcome | Mice, Inbred Strains | Blotting, Western | Insulin-Like Growth Factor II - metabolism | Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Sirolimus - administration & dosage | Insulin-Like Growth Factor II - antagonists & inhibitors | Animals | Signal Transduction - drug effects | Antibodies, Monoclonal - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Mice, Nude | Neoplasms - immunology | Receptor, IGF Type 1 - immunology | Cell Line, Tumor | Cell Proliferation - drug effects | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism
Journal Article
Journal Article
Endocrinology, ISSN 0013-7227, 02/2007, Volume 148, Issue 2, pp. 903 - 911
Rosiglitazone (Rosi) belongs to the class of thiazolidinediones (TZDs) that are ligands for peroxisome proliferator-activated receptor γ (PPARγ). Stimulation... 
QUANTITATIVE TRAIT LOCI | OSTEOBLAST DIFFERENTIATION | BONE-MINERAL DENSITY | CANCER CELLS | SIGNALING PATHWAY | INBRED STRAINS | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | PPAR-GAMMA-2 | WNT | CONGENIC MICE | Receptor, IGF Type 1 - antagonists & inhibitors | PPAR gamma - drug effects | Humans | Insulin-Like Growth Factor Binding Protein 3 - blood | Insulin-Like Growth Factor I - genetics | PPAR gamma - metabolism | RNA, Messenger - metabolism | Thiazolidinediones - administration & dosage | Insulin-Like Growth Factor I - secretion | Adipose Tissue - metabolism | Transfection | Microarray Analysis | Stromal Cells - drug effects | Female | Thiazolidinediones - pharmacology | Insulin-Like Growth Factor Binding Protein 4 - antagonists & inhibitors | Somatomedins - metabolism | PPAR gamma - genetics | Cell Line | Drug Administration Schedule | Ovariectomy | Down-Regulation | Liver - metabolism | Mice, Inbred C57BL | Osteoblasts - physiology | Stromal Cells - metabolism | Insulin-Like Growth Factor Binding Protein 4 - genetics | Receptor, IGF Type 1 - genetics | Insulin-Like Growth Factor II - metabolism | Insulin-Like Growth Factor II - antagonists & inhibitors | Animals | Liver - cytology | Mice | Insulin-Like Growth Factor Binding Protein 2 - blood | Bone Marrow Cells - metabolism | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism
Journal Article
The Journal of Nutritional Biochemistry, ISSN 0955-2863, 07/2016, Volume 33, pp. 103 - 110
Maternal folic acid supplementation can alter DNA methylation and gene expression in the developing fetus, which may confer disease susceptibility later in... 
DNA methylation | Gene expression | Folate | Folic acid | Maternal nutrition | UNITED-STATES | PROTEIN RESTRICTION | MESSENGER-RNA EXPRESSION | PROLIFERATOR-ACTIVATED RECEPTORS | NEURAL-TUBE DEFECTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CORD BLOOD | COLON-CANCER CELLS | NUTRITION & DIETETICS | COLORECTAL-CANCER | DIHYDROFOLATE-REDUCTASE | DIETARY-FOLATE | Brain - embryology | Receptors, Glucocorticoid - antagonists & inhibitors | Folic Acid - therapeutic use | Maternal Nutritional Physiological Phenomena | Neural Tube Defects - metabolism | Neurons - cytology | Receptors, Glucocorticoid - metabolism | Brain - growth & development | Folic Acid - administration & dosage | Receptors, Glucocorticoid - agonists | Brain - metabolism | DNA Methylation | Estrogen Receptor alpha - agonists | Insulin-Like Growth Factor II - agonists | Insulin-Like Growth Factor II - genetics | Gene Expression Regulation, Developmental | Estrogen Receptor alpha - metabolism | Female | Neurons - metabolism | Animals, Newborn | Peroxisome Proliferator-Activated Receptors - agonists | Peroxisome Proliferator-Activated Receptors - genetics | Nerve Tissue Proteins - antagonists & inhibitors | Peroxisome Proliferator-Activated Receptors - antagonists & inhibitors | Liver - metabolism | Nerve Tissue Proteins - agonists | Fetal Development | Estrogen Receptor alpha - antagonists & inhibitors | Neural Tube Defects - prevention & control | Random Allocation | Organ Specificity | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Insulin-Like Growth Factor II - metabolism | Nerve Tissue Proteins - metabolism | Pregnancy | Insulin-Like Growth Factor II - antagonists & inhibitors | Animals | Estrogen Receptor alpha - genetics | Folic Acid - blood | Peroxisome Proliferator-Activated Receptors - metabolism | Receptors, Glucocorticoid - genetics | Folic Acid - metabolism | Neural Tube Defects - blood | Dietary Supplements | Genes | Liver | DNA | Genetic research | Disease susceptibility | Methylation
Journal Article
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 09/2011, Volume 10, Issue 9, pp. 1677 - 1685
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 09/2010, Volume 9, Issue 9, pp. 2593 - 2604
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 01/2013, Volume 365, Issue 1, pp. 17 - 24
► A cell-based IR-B assay to measure total insulin-like activity in human serum. ► A cell-based IR-A assay to measure total insulin-like activity in human... 
Insulin-like growth factors | Insulin | Insulin bioactivity | Insulin receptor isoforms | Insulin receptor KIRA assays | CELLS | ASSAY | TISSUE | I BIOACTIVITY | HUMAN SERUM | CELL BIOLOGY | ENDOCRINOLOGY & METABOLISM | IMMUNOASSAY | INTERFERONS | PCR | Immunohistochemistry | Phosphorylation | Humans | Antibodies, Neutralizing - metabolism | Insulin-Like Growth Factor I - genetics | Insulin - blood | RNA, Messenger - metabolism | Antigens, CD - genetics | Protein Subunits - metabolism | Antigens, CD - metabolism | Insulin-Like Growth Factor II - analysis | Kidney - metabolism | Insulin-Like Growth Factor II - genetics | Osmolar Concentration | Protein Isoforms - metabolism | Receptor, Insulin - genetics | HEK293 Cells | Recombinant Proteins - metabolism | Gene Expression | Recombinant Proteins - antagonists & inhibitors | Signal Transduction | Kidney - cytology | Insulin-Like Growth Factor I - analysis | Receptor, Insulin - antagonists & inhibitors | Insulin-Like Growth Factor II - metabolism | Insulin - metabolism | Insulin-Like Growth Factor II - antagonists & inhibitors | Tyrosine - metabolism | Receptor, Insulin - metabolism | Protein Processing, Post-Translational | Protein Subunits - antagonists & inhibitors | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Protein Isoforms - antagonists & inhibitors | Human | Receptors | Infrared radiation | Activation | Assaying | Serums | Signalling
Journal Article
International Journal of Cancer, ISSN 0020-7136, 11/2015, Volume 137, Issue 9, pp. 2243 - 2252
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, p. e100165
Drug resistance is an obstacle to the effective treatment of ovarian cancer. We and others have shown that the insulin-like growth factor (IGF) signaling... 
CELLS | EFFICACY | MULTIDISCIPLINARY SCIENCES | IGF-II | RNA, Small Interfering - genetics | Receptor, IGF Type 1 - metabolism | Paclitaxel - pharmacology | Apoptosis - drug effects | Humans | Ovarian Neoplasms - pathology | Insulin-Like Growth Factor I - genetics | Cystadenocarcinoma, Serous - genetics | Cystadenocarcinoma, Serous - pathology | Cystadenocarcinoma, Serous - drug therapy | Ovarian Neoplasms - genetics | Insulin-Like Growth Factor II - genetics | Female | Phosphorylation - drug effects | Tumor Cells, Cultured | Ovarian Neoplasms - drug therapy | Real-Time Polymerase Chain Reaction | RNA, Messenger - genetics | Reverse Transcriptase Polymerase Chain Reaction | Receptor, IGF Type 1 - genetics | Blotting, Western | Insulin-Like Growth Factor II - metabolism | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Insulin-Like Growth Factor II - antagonists & inhibitors | Animals | Signal Transduction - drug effects | Mice, Nude | Cell Proliferation - drug effects | Mice | Antineoplastic Agents, Phytogenic - pharmacology | Cell Cycle - drug effects | Insulin-Like Growth Factor I - metabolism | Antimitotic agents | RNA | Genomics | Genomes | Genetic aspects | Drug resistance | Drug therapy | Antineoplastic agents | Health aspects | Diabetes therapy | Ovarian cancer | Cancer | Biotechnology | Ovarian carcinoma | Insulin-like growth factor I | Leukemia | Medical services | Oncology | Insulin-like growth factors | Kinases | Experiments | Cancer therapies | Signal transduction | Rodents | Paclitaxel | Xenografts | Insulin-like growth factor II | Taxol | Gynecology | Tumor cell lines | Gene expression | Ribonucleic acid--RNA | Insulin | Obstetrics | Medicine | Signaling | Androgens | Sensitivity | Chemotherapy | Womens health | Cell lines | Monoclonal antibodies | In vivo methods and tests | Laboratory animals | Tumors | Ribonucleic acid
Journal Article