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Diabetes, obesity & metabolism, ISSN 1462-8902, 2017, Volume 19, Issue Suppl 1, pp. 124 - 136
The progressive loss of pancreatic β‐cell mass that occurs in both type 1 and type 2 diabetes is a primary factor driving efforts to identify strategies for effectively increasing, enhancing or restoring β‐cell mass... 
beta‐cell | islets | beta-cell | Glucagon-Secreting Cells - immunology | Cell Proliferation | Species Specificity | Humans | Somatostatin-Secreting Cells - cytology | Extracellular Matrix - immunology | Diabetes Mellitus, Type 1 - metabolism | Extracellular Matrix - metabolism | Islets of Langerhans - innervation | Diabetes Mellitus, Type 2 - metabolism | Pancreatic Polypeptide-Secreting Cells - metabolism | Pancreatic Polypeptide-Secreting Cells - cytology | Insulin-Secreting Cells - metabolism | Glucagon-Secreting Cells - metabolism | Islets of Langerhans - cytology | Pancreatic Polypeptide-Secreting Cells - immunology | Somatostatin-Secreting Cells - immunology | Insulin-Secreting Cells - cytology | Pancreatic Polypeptide-Secreting Cells - pathology | Macrophages - immunology | Glucagon-Secreting Cells - cytology | Islets of Langerhans - blood supply | Somatostatin-Secreting Cells - pathology | Islets of Langerhans - pathology | Macrophages - pathology | Glucagon-Secreting Cells - pathology | Diabetes Mellitus, Type 1 - pathology | Cell Communication | Cellular Microenvironment | Macrophages - cytology | Insulin-Secreting Cells - immunology | Macrophages - metabolism | Animals | Models, Biological | Diabetes Mellitus, Type 2 - pathology | Insulin-Secreting Cells - pathology | Extracellular Matrix - pathology | Somatostatin-Secreting Cells - metabolism | Apoptosis | beta cell
Journal Article
Diabetes, ISSN 0012-1797, 11/2011, Volume 60, Issue 11, pp. 2872 - 2882
OBJECTIVE-To evaluate whether healthy or diabetic adult mice can tolerate an extreme loss of pancreatic a-cells and how this sudden massive depletion affects beta-cell function and blood glucose homeostasis... 
INSULIN | HYPERGLUCAGONEMIA | ENDOCRINE PANCREAS | GLUCOSE-HOMEOSTASIS | ENDOCRINOLOGY & METABOLISM | RECEPTOR GENE | SECRETION | DIFFERENTIATION | HYPERPLASIA | ISLET CELLS | EXPRESSION | Insulin-Secreting Cells - secretion | Apoptosis - drug effects | Cell Count | Glucagon - genetics | Male | Diphtheria Toxin - toxicity | Insulin - blood | Glucagon - blood | Diabetes Mellitus, Experimental - blood | Hypoglycemia - prevention & control | Intercellular Signaling Peptides and Proteins - metabolism | Glucagon-Secreting Cells - drug effects | Insulin-Secreting Cells - metabolism | Hyperglycemia - chemically induced | Glucagon-Secreting Cells - metabolism | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Glucagon-Secreting Cells - secretion | Hyperglycemia - prevention & control | Promoter Regions, Genetic | Signal Transduction | Glucagon-Secreting Cells - pathology | Intercellular Signaling Peptides and Proteins - genetics | Pancreas - drug effects | Pancreas - pathology | Receptors, Glucagon - metabolism | Mice, Transgenic | Pancreas - metabolism | Heparin-binding EGF-like Growth Factor | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Tamoxifen - pharmacology | Diabetes Mellitus, Experimental - pathology | Glucagon - metabolism | Mice | Streptozocin - toxicity | Insulin-Secreting Cells - pathology | Selective Estrogen Receptor Modulators - pharmacology | Islet Studies
Journal Article
American Journal of Pathology, ISSN 0002-9440, 2010, Volume 176, Issue 2, pp. 861 - 869
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2016, Volume 113, Issue 13, pp. E1826 - E1834
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 138, Issue 5, pp. 1954 - 1965.e8
Journal Article