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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2007, Volume 104, Issue 12, pp. 5115 - 5120
Journal Article
Journal Article
Journal Article
Diabetologia, ISSN 0012-186X, 1/2013, Volume 56, Issue 1, pp. 185 - 193
Immunohistochemical staining reveals that the enteroviral capsid protein VP1 is present at higher frequency in the insulin-containing islets of patients with... 
Antiviral response | Medicine & Public Health | Human Physiology | Immunocytochemistry | Metabolic Diseases | Islet autoimmunity | Internal Medicine | Enterovirus | Apoptosis | COMPLEX | VIRUS | RNA | TISSUE | CYTOKINE | MELLITUS | PANCREATIC-ISLETS | REPLICATION | ENDOCRINOLOGY & METABOLISM | INFECTION | DELETIONS | Antigens, Viral - metabolism | Enterovirus Infections - metabolism | United States | eIF-2 Kinase - metabolism | Humans | Child, Preschool | Diabetes Mellitus, Type 1 - metabolism | Infant | Proto-Oncogene Proteins c-bcl-2 - metabolism | Insulin-Secreting Cells - metabolism | Enterovirus Infections - immunology | Islets of Langerhans - metabolism | Adult | Diabetes Mellitus, Type 1 - immunology | Child | Enterovirus - metabolism | Biomarkers - metabolism | Diabetes Mellitus, Type 1 - virology | Islets of Langerhans - pathology | Capsid Proteins - metabolism | Down-Regulation | eIF-2 Kinase - biosynthesis | Diabetes Mellitus, Type 1 - pathology | Enzyme Induction | Islets of Langerhans - immunology | United Kingdom | Enterovirus - isolation & purification | Insulin-Secreting Cells - immunology | Insulin-Secreting Cells - virology | Myeloid Cell Leukemia Sequence 1 Protein | Adolescent | Enterovirus - immunology | Islets of Langerhans - virology | Enterovirus Infections - microbiology | Enterovirus Infections - pathology | Insulin-Secreting Cells - pathology | Cohort Studies | Virus diseases | Autoimmunity | Antiviral agents | Viral proteins | Type 1 diabetes | Leukemia | Drugstores | Protein kinases | Formaldehyde | Index Medicus
Journal Article
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 03/2010, Volume 159, Issue 3, pp. 338 - 343
Summary Type 1 diabetes results from a T cell-mediated destruction of insulin-producing pancreatic β cells. Little is known on local factors contributing to... 
CXCL10 | type 1 diabetes | IP-10 | CXCR3 | autoreactive T cells | Autoreactive T cells | Type 1 diabetes | IMMUNE-RESPONSE | NOD MICE | AUTOANTIGENS | ALLOGRAFT | BETA-CELLS | RISK | MELLITUS | IMMUNOLOGY | INFILTRATION | T-CELL RESPONSES | INSULIN EPITOPE | Lymph Nodes - pathology | Humans | Child, Preschool | Male | Diabetes Mellitus, Type 1 - therapy | Enterovirus Infections - immunology | Chemokine CXCL10 - immunology | Adult | Enterovirus Infections - therapy | Female | Retrospective Studies | Diabetes Mellitus, Type 1 - immunology | T-Lymphocytes - pathology | Diabetes Mellitus, Type 1 - virology | Gene Expression Regulation - immunology | Diabetes Mellitus, Type 1 - pathology | Inflammation - virology | Inflammation - immunology | Lymph Nodes - immunology | Insulin-Secreting Cells - immunology | Inflammation - therapy | Insulin-Secreting Cells - virology | Interferon-gamma - immunology | Adolescent | Receptors, CXCR3 - immunology | Enterovirus - immunology | T-Lymphocytes - immunology | Enterovirus Infections - pathology | Insulin-Secreting Cells - pathology | Immunity, Cellular | Interleukin-10 - immunology | Autoimmunity | Diabetics | Pancreatic beta cells | Autoantigens | Inflammation | Biological response modifiers | T cells | Infections | Diabetes | Patients | Viral infections | Chemokines | Immune system | Index Medicus | Insulitis | Leukocyte migration | Diabetes mellitus | Data processing | Lymphocytes T | CXCR3 protein | a-Interferon | Chemokine receptors | Lymph nodes | Beta cells | Infection | Interleukins | CXCL10 protein | Remission | Islets of Langerhans | Pancreas | Translational Studies
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, pp. e38522 - e38522
Epidemiological and experimental studies have suggested that Hepatitis C virus (HCV) infection is associated with the development of type 2 diabetes.... 
TUMOR-NECROSIS-FACTOR | REPLICATION | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | INFECTION | ENDOPLASMIC-RETICULUM STRESS | EXTRAHEPATIC MANIFESTATIONS | PREVALENCE | TYPE-2 DIABETES-MELLITUS | ASSOCIATION | FAILURE | Signal Transduction | Cell Survival | Humans | Caspase 3 - metabolism | Hepatitis - pathology | Mitochondria - metabolism | Animals | Insulin-Secreting Cells - virology | Cell Shape | Endoplasmic Reticulum Stress | Hepatitis - virology | Cell Line, Tumor | Hepacivirus - physiology | Insulinoma - pathology | Insulinoma - virology | Mice | Insulin-Secreting Cells - pathology | Virus Replication - physiology | Apoptosis | Insulin-Secreting Cells - enzymology | Type 2 diabetes | Infection | Virus diseases | Medical research | Pancreatic beta cells | Medicine, Experimental | Development and progression | Hepatitis C virus | Health aspects | Epidemiology | Investigations | Cell proliferation | Laboratories | Pathogenesis | Genomics | Insulinoma | Science | Viruses | Genomes | Infections | ADP | Caspase-3 | Cell surface | Nuclei | Proteins | Hepatitis | Mitochondria | Ribose | DNA fragmentation | Membrane potential | Pancreas | Phosphatidylserine | Deoxyribonucleic acid--DNA | Liver diseases | Diabetes mellitus | Mortality | Caspase | Gene expression | Ribonucleic acid--RNA | Virology | Beta cells | Studies | Polymerase | Progeny | Hypotheses | Cell death | Death | Insulin resistance | Nuclei (cytology) | Diabetes | Mutation | Hepatitis C | Endoplasmic reticulum | Index Medicus | RNA |