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Biochemical Journal, ISSN 0264-6021, 2010, Volume 432, Issue 1, pp. 199 - 205
Journal Article
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 04/2013, Volume 32, Issue 15, pp. 1910 - 1920
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, p. e106249
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e86459
Accumulating evidence indicates that a small population of cancer stem cells (CSCs) is involved in intrinsic resistance to cancer treatment. The hypoxic... 
TYROSINE KINASE INHIBITORS | MUTANT | MET AMPLIFICATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | ACQUIRED-RESISTANCE | PHASE-II | MUTATIONS | MESENCHYMAL TRANSITION | FACTOR-I RECEPTOR | CHEMOTHERAPY | Lung Neoplasms - drug therapy | Neoplasm Transplantation | Receptor, Epidermal Growth Factor - genetics | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Lung Neoplasms - metabolism | Glycoproteins - metabolism | Spheroids, Cellular - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Lung Neoplasms - pathology | Insulin-Like Growth Factor I - genetics | Antigens, CD - metabolism | Gene Knockdown Techniques | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neoplastic Stem Cells - pathology | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Cell Hypoxia - drug effects | Carcinoma, Non-Small-Cell Lung - genetics | Cell Separation | Carcinogenesis - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Mutation - genetics | AC133 Antigen | Receptor, IGF Type 1 - genetics | Carcinogenesis - drug effects | Carcinogenesis - pathology | Up-Regulation - drug effects | Drug Resistance, Neoplasm - genetics | Animals | Quinazolines - therapeutic use | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Mice, Inbred NOD | Carcinoma, Non-Small-Cell Lung - drug therapy | Cell Hypoxia - genetics | Quinazolines - pharmacology | Insulin-Like Growth Factor I - metabolism | Drug Resistance, Neoplasm - drug effects | Care and treatment | Epidermal growth factor | Genes | Stem cells | Lung cancer, Non-small cell | Gefitinib | Cancer | Insulin-like growth factor I | Oct-4 protein | Lung cancer | Insulin-like growth factors | Activation | Kinases | Drug resistance | Cancer therapies | Clonal deletion | Deletion | Hypoxia-inducible factors | Parasitology | Epidermal growth factor receptors | Non-small cell lung carcinoma | Exposure | Tumorigenicity | Insulin | CXCR4 protein | Medicine | Chemotherapy | Medical prognosis | Cell lines | Hypoxia | Mutation | Tumors | Pharmaceuticals
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e97016
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2008, Volume 105, Issue 24, pp. 8387 - 8392
A subset of gastrointestinal stromal tumors (GISTs) lack gain-of-function mutations in c-KIT and PDGFRα. These so-called wild-type (WT) GISTs tend to be less... 
Pediatrics | Receptors | Gastrointestinal stromal tumors | Somatomedins | Exons | Cell lines | Genetic mutation | Summarization | Tumors | Cancer | Imatinib mesylase | Adult wild-type GIST | Pediatric GIST | NYP-AEW541 | Tyrosine kinase inhibitors | CARCINOMA CELLS | MULTIDISCIPLINARY SCIENCES | adult wild-type GIST | C-KIT | FACTOR-I | ANTITUMOR-ACTIVITY | pediatric GIST | tyrosine kinase inhibitors | BREAST-CANCER | GROWTH-FACTOR RECEPTOR | imatinib mesylase | COPY NUMBER CHANGES | TYROSINE KINASE INHIBITOR | EXPRESSION | IMATINIB MESYLATE | Gastrointestinal Stromal Tumors - enzymology | RNA, Small Interfering - genetics | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Gene Expression Regulation, Neoplastic | Antineoplastic Agents - therapeutic use | Mitogen-Activated Protein Kinase Kinases - metabolism | DNA Mutational Analysis | Proto-Oncogene Proteins c-kit - genetics | Antineoplastic Agents - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Pyrroles - therapeutic use | Gastrointestinal Stromal Tumors - genetics | Signal Transduction | Gene Silencing | Piperazines - therapeutic use | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Receptor, IGF Type 1 - genetics | Pyrroles - pharmacology | Gene Amplification | Gastrointestinal Stromal Tumors - drug therapy | Pyrimidines - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Cell Line, Tumor | Cell Proliferation - drug effects | Benzamides | Mutation | Apoptosis | Immunohistochemistry | Prevention | Usage | Care and treatment | Gastrointestinal tumors | Genetic aspects | Research | Insulin-like growth factor 1 | Health aspects | Methods | Risk factors | Biological Sciences
Journal Article
Cancer, ISSN 0008-543X, 10/2014, Volume 120, Issue 19, pp. 2980 - 2985
BACKGROUND Targeting a single pathway in pancreatic adenocarcinoma (PC) is unlikely to affect its natural history. We tested the hypothesis that simulataneous... 
pancreatic cancer | erlotinib signaling | randomized phase II | IGF‐1R | cixutumumab | EGFR | targeted treatment | Erlotinib signaling | Targeted treatment | Randomized phase II | Pancreatic cancer | Cixutumumab | IGF-1R | CARCINOMA-CELLS | DUCTAL ADENOCARCINOMA | MONOCLONAL-ANTIBODY | SINGLE-AGENT CETUXIMAB | BREAST-CANCER | INHIBITION | K-RAS | ONCOLOGY | THERAPEUTIC TARGET | C-MET | RESISTANCE | Erlotinib Hydrochloride | Pancreatic Neoplasms - metabolism | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Receptor, Epidermal Growth Factor - drug effects | Male | Insulin-Like Growth Factor I - drug effects | Pancreatic Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - metabolism | Adenocarcinoma - metabolism | Adult | Deoxycytidine - adverse effects | Female | Quinazolines - administration & dosage | Drug Administration Schedule | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Treatment Outcome | Adenocarcinoma - drug therapy | Adenocarcinoma - secondary | Disease-Free Survival | Signal Transduction - drug effects | Antibodies, Monoclonal - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Quinazolines - adverse effects | Aged | Deoxycytidine - analogs & derivatives | Insulin-Like Growth Factor I - metabolism | Index Medicus | Abridged Index Medicus
Journal Article