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Cell Metabolism, ISSN 1550-4131, 2005, Volume 2, Issue 6, pp. 373 - 384
Defective glucose-stimulated insulin secretion is the main cause of hyperglycemia in type 2 diabetes mellitus. Mutations in HNF-1α cause a monogenic form of... 
DIABETES-MELLITUS | MEMBRANE-FUSION | HEPATOCYTE NUCLEAR FACTOR-1-ALPHA | ENDOCRINOLOGY & METABOLISM | PANCREATIC BETA-CELLS | FACTOR-I | MUTATIONS | SECRETION | TRANSCRIPTION FACTOR | MOLECULAR-MECHANISMS | ALPHA-GENE | CELL BIOLOGY | Immunohistochemistry | Immunoprecipitation | Membrane Glycoproteins - metabolism | Oligonucleotide Array Sequence Analysis | Calcium - metabolism | Humans | Insulinoma | Molecular Sequence Data | Immunoblotting | Diabetes Mellitus, Type 2 - metabolism | Exocytosis | RNA, Messenger - metabolism | Microscopy, Immunoelectron | Tissue Distribution | Insulin-Secreting Cells - metabolism | Time Factors | Base Sequence | Islets of Langerhans - cytology | Cloning, Molecular | Transcription, Genetic | Insulin Secretion | Genes, Reporter | Disease Models, Animal | Cell Line | Nucleic Acid Hybridization | Growth Hormone - metabolism | Hepatocyte Nuclear Factor 1 - metabolism | Glutathione Transferase - metabolism | Rats | Mice, Transgenic | Photons | Pancreas - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Two-Hybrid System Techniques | Blotting, Northern | Insulin - metabolism | Animals | Glucose - chemistry | Glucose - metabolism | Protein Binding | Mice | Models, Genetic | DNA, Complementary - metabolism | SNARE Proteins - metabolism | Microscopy, Fluorescence | RNA, Small Interfering - metabolism | Type 2 diabetes | Medical colleges | Pancreatic beta cells | Hyperglycemia | Molecular genetics | Insulin | Protein binding | Diabetes therapy | Index Medicus
Journal Article
Experimental Cell Research, ISSN 0014-4827, 2007, Volume 313, Issue 14, pp. 2993 - 3004
In RIN m5F rat insulinoma β-cells, agonists at cannabinoid CB receptors modulate insulin release. Here we investigated in these cells the effect of the... 
Bombesin | Anandamide, TRPV1 | Cannabinoid receptor | Calcium, insulinoma β-cells | SIGNAL-TRANSDUCTION PATHWAYS | VANILLOID VR1 RECEPTORS | BOMBESIN-LIKE PEPTIDES | PHOSPHOLIPASE-C | G-PROTEINS | ENDOCANNABINOID SYSTEM | CA2+ SIGNALS | CELL BIOLOGY | insulinoma beta-cells | ENERGY-BALANCE | ONCOLOGY | anandamide | TRPV1cannabinoid receptor bombesin calcium | CB1 RECEPTORS | Receptor, Cannabinoid, CB2 - agonists | Calcium - metabolism | Capsaicin - metabolism | Cell Line, Tumor - drug effects | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Receptor, Cannabinoid, CB2 - genetics | Neuroprotective Agents - metabolism | Dose-Response Relationship, Drug | TRPV Cation Channels - metabolism | Arachidonic Acids - metabolism | Receptor, Cannabinoid, CB1 - agonists | Colforsin - metabolism | TRPV Cation Channels - antagonists & inhibitors | Dronabinol - analogs & derivatives | Pyrrolidinones - metabolism | Estrenes - metabolism | Phosphoinositide Phospholipase C - metabolism | Sensory System Agents - metabolism | Cannabinoids - metabolism | Cell Line, Tumor - metabolism | Phosphoinositide Phospholipase C - antagonists & inhibitors | Enzyme Inhibitors - metabolism | TRPV Cation Channels - agonists | Neurotransmitter Agents - metabolism | Pertussis Toxin - metabolism | Rats | TRPV Cation Channels - genetics | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Insulinoma - metabolism | Thapsigargin - metabolism | Signal Transduction - physiology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Dronabinol - metabolism | Bombesin - metabolism | Index Medicus
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 2010, Volume 1802, Issue 3, pp. 363 - 372
Farnesoid X Receptor plays an important role in maintaining bile acid, cholesterol homeostasis and glucose metabolism. Here we investigated whether FXR is... 
KLF11 | Akt signaling | GLUT2 | Farnesoid X Receptor | Insulin gene transcription | Insulin secretion | DEPENDENT DIABETES-MELLITUS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MESSENGER-RNA STABILITY | NUCLEAR RECEPTOR | NOD MOUSE | BIOPHYSICS | GLUCOSE | FARNESOID-X-RECEPTOR | PANCREATIC BETA-CELLS | CARBOHYDRATE-METABOLISM | GENE-TRANSCRIPTION | Glucose Transporter Type 2 - genetics | RNA-Binding Proteins - genetics | Phosphorylation | Humans | Insulinoma - genetics | RNA, Messenger - metabolism | Glucose Transporter Type 2 - metabolism | Immunoenzyme Techniques | Proto-Oncogene Proteins c-akt - genetics | Repressor Proteins - antagonists & inhibitors | Cell Cycle Proteins - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Liver Neoplasms, Experimental - metabolism | Islets of Langerhans - metabolism | Carcinoma, Hepatocellular - genetics | Islets of Langerhans - cytology | Cell Cycle Proteins - genetics | Transcription, Genetic | Liver Neoplasms, Experimental - genetics | Insulin - genetics | Liver Neoplasms, Experimental - pathology | Proto-Oncogene Proteins c-akt - metabolism | Insulin Secretion | Repressor Proteins - metabolism | RNA-Binding Proteins - antagonists & inhibitors | DNA-Binding Proteins - antagonists & inhibitors | Glycemic Index | RNA, Messenger - genetics | Cell Cycle Proteins - metabolism | Cells, Cultured | Repressor Proteins - genetics | Transcription Factors - antagonists & inhibitors | Glucose - pharmacology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Transcription Factors - metabolism | Insulin - metabolism | Animals | Insulinoma - metabolism | Carcinoma, Hepatocellular - pathology | Fluorescent Antibody Technique | Insulinoma - pathology | Mice, Inbred NOD | Mice | RNA-Binding Proteins - metabolism | Carcinoma, Hepatocellular - metabolism
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2006, Volume 116, Issue 10, pp. 2610 - 2621
Inhibitors of VEGF signaling can block angiogenesis and reduce tumor vascularity, but little is known about the reversibility of these changes after treatment... 
MEDICINE, RESEARCH & EXPERIMENTAL | NERVE REGENERATION | BASAL LAMINA | ANGIOGENESIS INHIBITORS | BLOOD-VESSELS | EXTRACELLULAR-MATRIX | MUSCLE ACTIN EXPRESSION | ENDOTHELIAL SPROUTS | BASEMENT-MEMBRANES | GROWTH IN-VIVO | ESTABLISHED TUMORS | Basement Membrane - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Actins - metabolism | Endothelium, Vascular - drug effects | Vascular Endothelial Growth Factor A - metabolism | Basement Membrane - pathology | Carcinoma, Lewis Lung - blood supply | Neovascularization, Pathologic - pathology | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Insulinoma - blood supply | Pericytes - pathology | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Organic Chemicals - pharmacology | Angiogenesis Inhibitors - therapeutic use | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Imidazoles - therapeutic use | Collagen Type IV - immunology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Collagen Type IV - metabolism | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Pericytes - metabolism | Insulinoma - drug therapy | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Mice, Transgenic | Neoplasms - blood supply | Treatment Outcome | Imidazoles - pharmacology | Basement Membrane - metabolism | Carcinoma, Lewis Lung - drug therapy | Neoplasms - drug therapy | Indazoles - pharmacology | Animals | Endothelium, Vascular - metabolism | Neovascularization, Pathologic - drug therapy | Blood Vessels - drug effects | Carcinoma, Lewis Lung - pathology | Matrix Metalloproteinase Inhibitors | Endothelium, Vascular - pathology | Insulinoma - pathology | Mice | Neovascularization, Pathologic - metabolism | Neoplasms - pathology | Indazoles - therapeutic use | Care and treatment | Research | Vascular endothelial growth factor | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2015, Volume 290, Issue 7, pp. 4086 - 4096
In pancreatic beta-cells, ATP acts as a signaling molecule initiating plasma membrane electrical activity linked to Ca2+ influx, which triggers insulin... 
OXIDATIVE-PHOSPHORYLATION | ACTIVATION | DEFECTS | ESSENTIAL COMPONENT | MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CALCIUM UNIPORTER | BETA-CELL | MICU1 | MCU | EXCHANGER | RNA, Small Interfering - genetics | Cation Transport Proteins - antagonists & inhibitors | Cell Proliferation | Calcium Channels - metabolism | Calcium - metabolism | Calcium-Binding Proteins - antagonists & inhibitors | Insulinoma - genetics | Male | Immunoenzyme Techniques | Insulin-Secreting Cells - metabolism | Cation Transport Proteins - metabolism | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial | Cation Transport Proteins - genetics | Insulin-Secreting Cells - cytology | Calcium Channels - genetics | Insulin Secretion | Real-Time Polymerase Chain Reaction | Calcium-Binding Proteins - metabolism | RNA, Messenger - genetics | Cells, Cultured | Oxidative Phosphorylation | Rats | Mitochondria - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Blotting, Western | Insulin - metabolism | Animals | Energy Metabolism | Insulinoma - metabolism | Calcium Channels - chemistry | Glucose - metabolism | Insulinoma - pathology | Calcium-Binding Proteins - genetics | Hydrogen-Ion Concentration | Index Medicus | mitochondrial respiratory chain complex | Leucine zipper-EF hand-containing transmembrane protein 1 | pancreatic islet | Bioenergetics | mitochondrial transport | mitochondrial membrane potential | INS-1E cells | Mitochondrial pH gradient | Mitochondrial Ca2+ uniporter | insulin secretion | mitochondrial metabolism
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 2015, Volume 309, Issue 8, pp. E715 - E726
Proinflammatory cytokines impact islet beta-cell mass and function by altering the transcriptional activity within pancreatic beta-cells, producing increases... 
Chemokine | Interleukin-1 | Inflammation | Nitric oxide | Insulin secretion | chemokine | nitric oxide | PHYSIOLOGY | interleukin-1 | MECHANISM | IFN-GAMMA | INFLAMMATORY REACTION | insulin secretion | HUMAN ISLETS | inflammation | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | MICE | INHIBITORS | EXPRESSION | LANGERHANS | GENE-TRANSCRIPTION | Chemokine CCL20 - metabolism | Rats, Wistar | Homeodomain Proteins - metabolism | Insulinoma | Immunoblotting | NF-kappa B - metabolism | RNA, Messenger - metabolism | Ribosomal Proteins - metabolism | Insulin-Secreting Cells - metabolism | Interleukin-1beta - metabolism | Chemokine CCL20 - genetics | Chemokine CCL2 - metabolism | Tumor Cells, Cultured | Insulin - genetics | Pancreatic Neoplasms | Insulin Secretion | Electron Spin Resonance Spectroscopy | Enzyme-Linked Immunosorbent Assay | Ribosomal Proteins - genetics | Gene Expression Regulation | Oxygen Consumption | Diabetes Mellitus - metabolism | Rats | Chemokine CCL2 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Chemokines - genetics | Homeodomain Proteins - genetics | Rats, Zucker | Insulin - metabolism | Patch-Clamp Techniques | Animals | Nitric Oxide Synthase Type II - genetics | Chemokines - metabolism | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Index Medicus | Call for Papers
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 04/2018, Volume 217, Issue 4, pp. 1287 - 1301
In mammalian pancreatic beta cells, the IRE1 alpha-XBP1 pathway is constitutively and highly activated under physiological conditions. To elucidate the precise... 
DIABETES-MELLITUS | DISULFIDE-ISOMERASE | XBP1 | UNFOLDED PROTEIN RESPONSE | MESSENGER-RNA | ER-STRESS | INSULIN-SECRETION | IRE1 | ENDOPLASMIC-RETICULUM STRESS | TRANSCRIPTION FACTOR | CELL BIOLOGY | Diabetes Mellitus - blood | Diabetes Mellitus - enzymology | Protein-Serine-Threonine Kinases - deficiency | Endoribonucleases - genetics | Phosphorylation | Molecular Chaperones - metabolism | Proinsulin - chemistry | Proinsulin - metabolism | Diabetes Mellitus - genetics | Protein Disulfide-Isomerases - metabolism | eIF-2 Kinase - metabolism | Insulinoma - genetics | Male | Thioredoxins - genetics | Thioredoxins - metabolism | Membrane Proteins - metabolism | Endoribonucleases - deficiency | Insulin - genetics | Binding Sites | Protein-Serine-Threonine Kinases - metabolism | Promoter Regions, Genetic | Endoribonucleases - metabolism | Oxidation-Reduction | Signal Transduction | Membrane Proteins - genetics | Molecular Chaperones - genetics | Pancreatic Neoplasms - enzymology | Protein-Serine-Threonine Kinases - genetics | Pancreatic Neoplasms - genetics | Proinsulin - genetics | Insulinoma - enzymology | Protein Folding | Mice, Knockout | Activating Transcription Factor 6 - metabolism | Insulin - metabolism | Protein Disulfide-Isomerases - genetics | Animals | X-Box Binding Protein 1 - metabolism | Cell Line, Tumor | Blood Glucose - metabolism | X-Box Binding Protein 1 - genetics | Insulin-Secreting Cells - enzymology | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2010, Volume 285, Issue 46, pp. 36199 - 36206
Heterozygous loss-of-function mutations in the hepatocyte nuclear factor 1A (HNF1A) gene result in the pathogenesis of maturity-onset diabetes-of-the-young... 
METFORMIN | STIMULATION | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | INSULIN-SECRETION | PANCREATIC BETA-CELLS | MITOCHONDRIAL DYSFUNCTION | DEATH | DOMINANT-NEGATIVE SUPPRESSION | EXPRESSION | BH3-ONLY PROTEINS | AMP-Activated Protein Kinases - metabolism | Hepatocyte Nuclear Factor 1-alpha - genetics | Apoptosis - drug effects | Insulinoma - genetics | Hepatocyte Nuclear Factor 1-alpha - metabolism | Protein Subunits - metabolism | Aminoimidazole Carboxamide - pharmacology | Ribonucleotides - pharmacology | Flow Cytometry | RNA Interference | Adenosine Triphosphate - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Energy Metabolism - physiology | Protein Subunits - genetics | Mice, Inbred C57BL | Rats | Mice, Transgenic | Enzyme Activation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Hypoglycemic Agents - pharmacology | Animals | Aminoimidazole Carboxamide - analogs & derivatives | Insulinoma - metabolism | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Doxycycline - pharmacology | Insulinoma - pathology | Mice | Apoptosis - physiology | Adaptor Proteins, Signal Transducing - metabolism | AMP-Activated Protein Kinases - genetics | Energy Metabolism - drug effects | Index Medicus | Molecular Bases of Disease | Bcl2-modifying Factor (Bmf) | Bioenergetics | Signal Transduction | Bioenergetic Stress | AMP-activated Kinase (AMPK) | Diabetes | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 06/2018, Volume 115, Issue 25, pp. E5706 - E5715
The stability of organic dyes against photobleaching is critical in single-molecule tracking and localization microscopy. Since oxygen accelerates... 
Oxygen scavenger | Single-molecule live-cell imaging | Metabolite | ATP | Fluorescence microscopy | TO-ADP RATIO | GLYCERALDEHYDE | FLUOROPHORES | STIMULATION | PHOSPHATIDYLINOSITOL 4,5-BISPHOSPHATE | HIT-T15 INSULINOMA CELLS | MULTIDISCIPLINARY SCIENCES | CLATHRIN-COATED PITS | imaging | fluorescence microscopy | NANOPARTICLES | TRANSPORT | metabolite | Single-molecule live-cell | METABOLISM | oxygen scavenger | Cell Line | Glucose Oxidase - metabolism | Humans | Receptor, PAR-2 - metabolism | Fluorescence | Fluorescent Dyes - metabolism | Glutamine - metabolism | Carbocyanines - metabolism | Flavin-Adenine Dinucleotide - analogs & derivatives | Mitochondria - metabolism | Keto Acids - metabolism | Oxygen - metabolism | Flavin-Adenine Dinucleotide - metabolism | Phosphatidylinositol 4,5-Diphosphate - metabolism | Microscopy, Fluorescence - methods | Adenosine Triphosphate - metabolism | HEK293 Cells | NAD - metabolism | Photobleaching | Antioxidants | Physiological aspects | Oxygen | Adenosine triphosphate | Methods | Neuroimaging | Glucose oxidase | Phosphorylation | Dyes | Glyceraldehyde | Transplantation | Phosphatidylinositol 4,5-diphosphate | Receptors | Mitochondria | NADH | Ischemia | Metabolites | Cell cycle | Supplementation | Localization | Scavengers | Deprivation | Heart transplantation | Intermediates | Oxidative phosphorylation | Microscopy | Bleaching | Internalization | Nicotinamide adenine dinucleotide | Brain damage | Intracellular | Brain injury | Glutamine | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
Cellular Signalling, ISSN 0898-6568, 10/2017, Volume 38, pp. 212 - 222
Journal Article
Cancer Cell, ISSN 1535-6108, 2008, Volume 13, Issue 6, pp. 507 - 518
Immune responses may arrest tumor growth by inducing tumor dormancy. The mechanisms leading to either tumor dormancy or promotion of multistage carcinogenesis... 
CELLCYCLE | METASTATIC MELANOMA | INTERFERON-GAMMA | DENDRITIC CELLS | ONCOLOGY | INTEGRIN EXPRESSION | ANGIOGENIC SWITCH | ENDOTHELIAL-CELLS | SKIN CARCINOGENESIS | PANCREATIC-ISLET TUMORS | NECROSIS-FACTOR-ALPHA | INNATE IMMUNITY | CELL BIOLOGY | Th1 Cells - pathology | Whole-Body Irradiation | Immunotherapy - methods | Cell Proliferation | Pancreatic Neoplasms - metabolism | Pancreatic Neoplasms - blood supply | Insulinoma - genetics | GTPase-Activating Proteins - metabolism | Interferon-gamma - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | CD4-Positive T-Lymphocytes - pathology | Th1 Cells - immunology | Integrin alphaVbeta3 - metabolism | Neovascularization, Pathologic - pathology | CD4-Positive T-Lymphocytes - immunology | Receptors, Tumor Necrosis Factor, Type I - deficiency | Time Factors | Cell Transformation, Neoplastic - genetics | Insulinoma - blood supply | Neovascularization, Pathologic - immunology | Antigens, Viral, Tumor - genetics | Insulinoma - immunology | Signal Transduction | Cell Survival | Pancreatic Neoplasms - pathology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Mice, Transgenic | Pancreatic Neoplasms - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Cell Transformation, Neoplastic - metabolism | Insulinoma - therapy | Mice, Inbred C3H | Mice, Knockout | Antigens, Viral, Tumor - metabolism | Cell Transformation, Neoplastic - immunology | Animals | CD4-Positive T-Lymphocytes - transplantation | Insulinoma - metabolism | Pancreatic Neoplasms - immunology | Insulinoma - pathology | Mice | GTPase-Activating Proteins - genetics | Blood Glucose - metabolism | Cell Transformation, Neoplastic - pathology | Cell Movement | Pancreatic Neoplasms - therapy | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 138, Issue 5, pp. 1954 - 1965.e8
Background & Aims The tumor suppressor menin is recognized as a key regulator of pancreatic islet development, proliferation, and β-cell function, whereas its... 
Gastroenterology and Hepatology | Pancreatic α Cells | Transdifferentiation | Insulinoma | Men1 Ablation | Pancreatic alpha Cells | MOUSE PANCREAS | BETA-CELLS | IDENTIFICATION | TUMORS | GENE | IN-VIVO | MULTIPLE ENDOCRINE NEOPLASIA | MICE | DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | TYPE-1 MEN1 | Cell Proliferation | Pancreatic Neoplasms - metabolism | Age Factors | Gene Expression Regulation, Neoplastic | Insulinoma - genetics | Gene Expression Profiling | Cell Fusion | Insulin-Secreting Cells - metabolism | Cell Transformation, Neoplastic - genetics | Gene Deletion | Glucagon-Secreting Cells - metabolism | Cell Transdifferentiation | Glucagonoma - pathology | Biomarkers - metabolism | Glucagon-Secreting Cells - pathology | Pancreatic Neoplasms - pathology | Genotype | Glucagonoma - metabolism | Pancreatic Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins - deficiency | Cell Transformation, Neoplastic - metabolism | Aging - pathology | Mice, Knockout | Transcription Factors - metabolism | Cell Lineage | Insulin - metabolism | Phenotype | Animals | Insulinoma - metabolism | Glucagon - metabolism | Insulinoma - pathology | Mice | Glucagonoma - genetics | Insulin-Secreting Cells - pathology | Cell Transformation, Neoplastic - pathology | Aging - metabolism | Index Medicus | Abridged Index Medicus
Journal Article