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Journal Article
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2005, Volume 25, Issue 2, pp. 334 - 340
OBJECTIVE—Expression of adhesion molecules on endothelial cells and subsequent leukocyte recruitment are critical early events in the development of... 
Superoxide dismutase | Endothelial cell | Adhesion molecule | Atherosclerosis | MAPKs | atherosclerosis | ACTIVATION | INDUCED APOPTOSIS | endothelial cell | adhesion molecule | MAP KINASES | INDUCTION | AP-1 | SIGNAL-TRANSDUCTION | IN-VITRO | superoxide dismutase | VCAM-1 EXPRESSION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | NF-KAPPA-B | Endothelium, Vascular - cytology | Neutrophils - cytology | Transcription, Genetic - drug effects | MAP Kinase Signaling System - physiology | Phosphorylation | Superoxide Dismutase - genetics | Humans | Recombinant Fusion Proteins - physiology | Depression, Chemical | Superoxide Dismutase - physiology | Intercellular Adhesion Molecule-1 - biosynthesis | Superoxides - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Flavonoids - pharmacology | NF-kappa B - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Transduction, Genetic | p38 Mitogen-Activated Protein Kinases - physiology | Endothelial Cells - metabolism | NADPH Oxidases - antagonists & inhibitors | Imidazoles - pharmacology | Inflammation | Cell Adhesion - drug effects | Anthracenes - pharmacology | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Arteriosclerosis - prevention & control | Transcription Factor AP-1 - antagonists & inhibitors | MAP Kinase Signaling System - drug effects | Cell Adhesion - physiology | Endothelium, Vascular - metabolism | Intercellular Adhesion Molecule-1 - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - physiology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Protein Processing, Post-Translational | Pyridines - pharmacology | Aorta - cytology | Superoxide Dismutase-1
Journal Article
Atherosclerosis, ISSN 0021-9150, 2005, Volume 183, Issue 2, pp. 259 - 267
In this study the effects of stable and intermittent high glucose concentrations on ICAM-1, VCAM-1 and E-selectin production, PKC activity and PKCβI, βII and δ... 
Adhesion molecules | PKC | Mitochondrial superoxide | Intermittent glucose | Endothelium | endothelium | OXIDATIVE STRESS | ACTIVATION | CIRCULATING ADHESION MOLECULES | BLOOD-GLUCOSE | ATHEROSCLEROSIS | MECHANISMS | intermittent glucose | DIABETES-MELLITUS | mitochondrial superoxide | HYPERGLYCEMIA | adhesion molecules | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | COMPLICATIONS | Endothelium, Vascular - cytology | Mitochondria - enzymology | Vascular Cell Adhesion Molecule-1 - drug effects | Gene Expression - drug effects | Humans | Glucose - administration & dosage | Dose-Response Relationship, Drug | RNA, Messenger - biosynthesis | E-Selectin - genetics | Protein Kinase C - metabolism | Intercellular Adhesion Molecule-1 - biosynthesis | E-Selectin - biosynthesis | Superoxides - metabolism | Intercellular Adhesion Molecule-1 - drug effects | Vascular Cell Adhesion Molecule-1 - genetics | E-Selectin - drug effects | Umbilical Veins - cytology | Deoxyguanosine - analogs & derivatives | Infant, Newborn | Enzyme-Linked Immunosorbent Assay | Protein Kinase C - drug effects | RNA, Messenger - genetics | Cells, Cultured | Blotting, Western | Blotting, Northern | Endothelium, Vascular - metabolism | Intercellular Adhesion Molecule-1 - genetics | Deoxyguanosine - metabolism | Vascular Cell Adhesion Molecule-1 - biosynthesis | Oxidative Stress - drug effects | In Vitro Techniques | Sweetening Agents - administration & dosage | Umbilical Veins - metabolism | Messenger RNA | Atherosclerosis | Superoxide | Glucose | Protein kinases | Dextrose | Porphyrins
Journal Article
International Journal of Cardiology, ISSN 0167-5273, 2017, Volume 232, pp. 233 - 242
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1217 - 1223
Sepsis, a systemic inflammatory response to infection, commonly progresses to acute lung injury (ALI), an inflammatory lung disease with high morbidity. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ICAM-1 | MODEL | MOLECULES | CELL BIOLOGY | LEUKOCYTES | IN-VIVO | HEPARAN-SULFATE | CELL | UNFRACTIONATED HEPARIN | Intestinal Perforation - complications | Humans | Heparitin Sulfate - antagonists & inhibitors | Male | Adoptive Transfer | Heparitin Sulfate - metabolism | Pulmonary Alveoli - enzymology | Glucuronidase - deficiency | Glucuronidase - analysis | Receptors, Tumor Necrosis Factor, Type I - deficiency | Glucuronidase - physiology | Respiratory Insufficiency - enzymology | Ventilator-Induced Lung Injury - enzymology | Acute Lung Injury - prevention & control | Endotoxemia - physiopathology | Intercellular Adhesion Molecule-1 - biosynthesis | Endothelium - enzymology | Disease Models, Animal | Acute Lung Injury - physiopathology | Lipopolysaccharides - toxicity | Pulmonary Alveoli - pathology | Mice, Inbred C57BL | Neutrophils - physiology | Ventilator-Induced Lung Injury - pathology | Lung - physiopathology | Mice, Knockout | Gene Expression Regulation - drug effects | Respiratory Insufficiency - pathology | Glycocalyx - physiology | Animals | Cell Adhesion - physiology | Endotoxemia - complications | Receptors, Tumor Necrosis Factor, Type I - physiology | Intercellular Adhesion Molecule-1 - genetics | Tumor Necrosis Factor-alpha - physiology | Mice | Enzyme Activation | Acute Lung Injury - etiology | Intestinal Perforation - microbiology | Endothelium - physiology | Care and treatment | Glycocalyx | Cellular control mechanisms | Neutrophils | Cell adhesion | Physiological aspects | Sepsis | Development and progression | Research | Properties | Respiratory physiology | Endothelium | Leucocytes | Glycoproteins | Lung diseases | Cell adhesion & migration | Immune system
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2010, Volume 185, Issue 3, pp. 1786 - 1793
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2010, Volume 185, Issue 1, pp. 302 - 312
Mesenchymal stem cells (MSCs) exert immunomodulatory properties via the inhibition of T cell activation and proliferation. Because of the deleterious role of... 
PROGENITOR CELLS | GROWTH-FACTOR-BETA | TGF-BETA | ROR-GAMMA-T | DENDRITIC CELLS | INTERFERON-GAMMA | SPECIFICITY | NITRIC-OXIDE | IMMUNOLOGY | LYMPHOCYTE-PROLIFERATION | PROSTAGLANDIN-E2 | Cell Communication - immunology | T-Lymphocytes, Regulatory - metabolism | CD18 Antigens - physiology | Coculture Techniques | Humans | Interleukin-17 - physiology | L Cells (Cell Line) | T-Lymphocytes, Regulatory - pathology | Mesenchymal Stromal Cells - immunology | T-Lymphocytes, Regulatory - immunology | CD11a Antigen - physiology | Cell Movement - immunology | T-Lymphocytes, Helper-Inducer - immunology | Bone Marrow Cells - immunology | Intercellular Adhesion Molecule-1 - biosynthesis | Inflammation Mediators - metabolism | Receptors, CCR6 - metabolism | T-Lymphocytes, Helper-Inducer - cytology | Inflammation Mediators - physiology | Receptors, CCR6 - physiology | CD11a Antigen - metabolism | Bone Marrow Cells - cytology | Cells, Cultured | Interferon-gamma - physiology | Immunophenotyping | Interleukin-17 - biosynthesis | CD18 Antigens - metabolism | Cell Adhesion - immunology | Cell Differentiation - immunology | Animals | Interleukin-17 - antagonists & inhibitors | Tumor Necrosis Factor-alpha - physiology | Growth Inhibitors - physiology | Ligands | Mice | Mesenchymal Stromal Cells - pathology | Bone Marrow Cells - metabolism
Journal Article
Experimental Cell Research, ISSN 0014-4827, 02/2015, Volume 331, Issue 2, pp. 292 - 308
Journal Article