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1. Full Text Cell Junction Pathology of Neural Stem Cells Is Associated With Ventricular Zone Disruption, Hydrocephalus, and Abnormal Neurogenesis
by Montserrat Guerra, Maria and Henzi, Roberto and Ortloff, Alexander and Lichtin, Nicole and Vio, Karin and Jimenez, Antonio J and Dolores Dominguez-Pinos, Maria and Gonzalez, Cesar and Clara Jara, Maria and Hinostroza, Fernando and Roiguez, Sara and Jara, Maryoris and Ortega, Eduardo and Guerra, Francisco and Sival, Deborah A and den Dunnen, Wilfred F. A and Perez-Figares, Jose M and McAllister, James P and Johanson, Conrad E and Roiguez, Esteban M
Journal of Neuropathology & Experimental Neurology, ISSN 0022-3069, 07/2015, Volume 74, Issue 7, pp. 653 - 671
Fetal-onset hydrocephalus affects 1 to 3 per 1,000 live births. It is not only a disorder of cerebrospinal fluid dynamics but also a brain disorder that... 
Human | HUMAN FETAL EPENDYMA | Cerebrospinal fluid | SUBVENTRICULAR ZONE | CEREBROSPINAL-FLUID | CORTICAL DEVELOPMENT | NEUROBLAST MIGRATION | HTx rat | ARNOLD-CHIARI MALFORMATION | Ventricular zone disruption | Neurospheres | Congenital hydrocephalus | BLOOD-VESSELS | Neural stem cells | CENTRAL-NERVOUS-SYSTEM | Junction pathology | ADHERENS JUNCTIONS | EPENDYMAL DENUDATION | SUBCOMMISSURAL ORGAN | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | Age Factors | Embryo, Mammalian | Humans | Hydrocephalus - pathology | Male | Glial Fibrillary Acidic Protein - metabolism | Neural Stem Cells - ultrastructure | Ventricular Outflow Obstruction - pathology | Telencephalon - growth & development | Intercellular Junctions - pathology | Telencephalon - pathology | Female | Fetus | Cell Differentiation | Animals, Newborn | Cells, Cultured | Rats | Telencephalon - embryology | Microscopy, Electron | Gestational Age | Neural Stem Cells - pathology | Animals | Neurogenesis - physiology | Intercellular Junctions - ultrastructure | Telencephalon - ultrastructure | Cell Movement
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2. Full Text Regulation of intestinal epithelial permeability by tight junctions
by Suzuki, Takuya and Suzuki, Takuya
Cellular and Molecular Life Sciences, ISSN 1420-682X, 2/2013, Volume 70, Issue 4, pp. 631 - 659
The gastrointestinal epithelium forms the boundary between the body and external environment. It effectively provides a selective permeable barrier that limits... 
Life Sciences | Biochemistry, general | Life Sciences, general | Cytokine | Tight junction | Nutrient | Intestinal epithelium | Pathogen | Biomedicine general | Cell Biology | ACETALDEHYDE-INDUCED INCREASE | PROTEIN-KINASE-C | KAPPA-B ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLOSTRIDIUM-PERFRINGENS ENTEROTOXIN | NECROSIS-FACTOR-ALPHA | CELL BIOLOGY | INFLAMMATORY-BOWEL-DISEASE | ENTEROPATHOGENIC ESCHERICHIA-COLI | CHAIN FATTY-ACIDS | EPIDERMAL-GROWTH-FACTOR | CACO-2 CELL MONOLAYERS | Tight Junctions - metabolism | Intestinal Mucosa - metabolism | Tight Junctions - microbiology | Cytokines - metabolism | Humans | Nutritional Physiological Phenomena | Intestinal Mucosa - microbiology | Permeability | Intercellular Signaling Peptides and Proteins - metabolism | Animals | Tight Junctions - immunology | Intestinal Mucosa - immunology | Intercellular Signaling Peptides and Proteins - immunology | Tight Junctions - pathology | Cytokines - immunology | Intestinal Mucosa - pathology | Analysis | Pathology | Immunology | Cytokines | Cellular biology | Digestive system | Nutrients
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3. Full Text A cell junction pathology of neural stem cells leads to abnormal neurogenesis and hydrocephalus
by Roíguez, Esteban M and Guerra, María M and Vío, Karin and González, César and Ortloff, Alexander and Bátiz, Luis F and Roíguez, Sara and Jara, María C and Muñoz, Rosa I and Ortega, Eduardo and Jaque, Jaime and Guerra, Francisco and Sival, Deborah A and den Dunnen, Wilfred F A and Jiménez, Antonio J and Domínguez-Pinos, María D and Pérez-Fígares, José M and McAllister, James P and Johanson, Conrad
Biological research, ISSN 0716-9760, 2012, Volume 45, Issue 3, pp. 231 - 242
Most cells of the developing mammalian brain derive from the ventricular (VZ) and the subventricular (SVZ) zones. The VZ is formed by the multipotent radial... 
GAP-JUNCTION | NEURONAL MIGRATION | hydrocephalus | EMBRYONIC CEREBROSPINAL-FLUID | INJURED SPINAL-CORD | neural stem cells | H-TX RAT | stem cell transplantation | PERIVENTRICULAR NODULAR HETEROTOPIA | CONGENITAL HYDROCEPHALUS | SUBVENTRICULAR ZONE | UMCG Approved | CEREBRAL-CORTEX | abnormal neurogenesis | ALPHA-SNAP | Stem cell transplantation | Abnormal neurogenesis | Neural stem cells | Hydrocephalus | EPENDYMAL DENUDATION | POINT MUTATION | SPINAL-CORD | CEREBROSPINAL-FLUID | CORTICAL DEVELOPMENT | BIOLOGY | Cerebral Ventricles - pathology | Cell Proliferation | Humans | Rats | Hydrocephalus - pathology | Hydrocephalus - therapy | Neural Stem Cells - pathology | Neurogenesis | Cerebral Aqueduct - pathology | Stem Cell Transplantation - methods | Animals | Intercellular Junctions - pathology | Neural Stem Cells - transplantation | Cell Differentiation | Cerebral Ventricles - embryology
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4. Full Text Effects of angiopoietin-2-blocking antibody on endothelial cell-cell junctions and lung metastasis
by Holopainen, Tanja and Saharinen, Pipsa and D'Amico, Gabriela and Lampinen, Anita and Eklund, Lauri and Sormunen, Raija and Anisimov, Andrey and Zarkada, Georgia and Lohela, Marja and Heloterä, Hanna and Tammela, Tuomas and Benjamin, Laura E and Ylä-Herttuala, Seppo and Leow, Ching Ching and Koh, Gou Young and Alitalo, Kari
Journal of the National Cancer Institute, ISSN 0027-8874, 03/2012, Volume 104, Issue 6, pp. 461 - 475
Background Angiopoietin-2 (Ang2), a ligand for endothelial TEK (Tie2) tyrosine kinase receptor, is induced in hypoxic endothelial cells of tumors, where it... 
LYMPHATIC VESSELS | TRANSGENE EXPRESSION | ANGIOGENESIS | ONCOLOGY | IN-VIVO | GENE-THERAPY | VEGF-C | TUMOR-GROWTH | CANCER | RECEPTOR TYROSINE KINASE | TIE2 RECEPTOR | Immunohistochemistry | Antibodies, Blocking - metabolism | Up-Regulation | Immunoprecipitation | Melanoma - blood supply | Coculture Techniques | Endothelium, Vascular - drug effects | Immunoblotting | Transplantation, Heterologous | Antibodies, Blocking - pharmacology | Angiopoietin-2 - metabolism | Cell Hypoxia | Retinal Vessels - pathology | Lung Neoplasms - secondary | Intercellular Junctions - pathology | Female | Neovascularization, Pathologic - prevention & control | Receptor, TIE-2 - metabolism | Lymphangiogenesis | Lung Neoplasms - blood supply | Microscopy, Electron, Transmission | Retinal Vessels - metabolism | Intercellular Junctions - metabolism | Angiogenesis Inhibitors - pharmacology | Mice, Transgenic | Lymphatic Metastasis | Retinal Vessels - drug effects | Mice, SCID | Melanoma - secondary | Animals | Neovascularization, Pathologic - drug therapy | Intercellular Junctions - drug effects | Endothelium, Vascular - pathology | Mice | Mice, Inbred BALB C | Neovascularization, Pathologic - metabolism | Angiopoietin-2 - antagonists & inhibitors | Endothelial Cells - pathology | Endothelial Cells - drug effects | Junctional complexes (Epithelium) | Lung cancer | Cell junctions | Physiological aspects | Development and progression | Models | Metastasis | Research | Growth factor receptors
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5. Full Text Human immunodeficiency virus infection of human astrocytes disrupts blood-brain barrier integrity by a gap junction-dependent mechanism
by Eugenin, Eliseo A and Clements, Janice E and Christine Zink, M and Berman, Joan W
Journal of Neuroscience, ISSN 0270-6474, 06/2011, Volume 31, Issue 26, pp. 9456 - 9465
HIV infection of the CNS is an early event after primary infection, resulting in neurological complications in a significant number of individuals despite... 
GLIOVASCULAR INTERFACE | POLYMERASE-CHAIN-REACTION | INTERCELLULAR COMMUNICATION | ENDOTHELIAL-CELLS | IN-VIVO | HUMAN FETAL ASTROCYTES | CENTRAL-NERVOUS-SYSTEM | CHEMOATTRACTANT PROTEIN-1 | CEREBROSPINAL-FLUID | HIV-1 INFECTION | NEUROSCIENCES | HIV-1 | In Situ Nick-End Labeling | Gap Junctions - pathology | Macaca nemestrina | Cell Count | Humans | Astrocytes - pathology | Cells, Cultured | Brain - virology | Blood-Brain Barrier - pathology | Simian Acquired Immunodeficiency Syndrome - virology | Animals | Fluorescent Antibody Technique | Brain - pathology | Blood-Brain Barrier - virology | Gap Junctions - virology | Endothelial Cells - pathology | Endothelial Cells - virology | Simian Acquired Immunodeficiency Syndrome - pathology | Astrocytes - virology
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6. Full Text Refining the molecular organization of the cardiac intercalated disc
by Vermij, Sarah H and Abriel, Hugues and van Veen, Toon A.B
Cardiovascular Research, ISSN 0008-6363, 03/2017, Volume 113, Issue 3, pp. 259 - 275
This review presents an extensively integrated model of the cardiac intercalated disc (ID), a highly orchestrated structure that connects adjacent... 
Cardiac arrhythmia | Wnt signaling | Arrhythmogenic cardiomyopathy | Brugada syndrome | Physiology | Cardiology and Cardiovascular Medicine | Physiology (medical) | Intercalated disc | ANKYRIN-G | CARDIAC & CARDIOVASCULAR SYSTEMS | AREA-COMPOSITA | HEART-MUSCLE CELLS | SODIUM-CHANNEL NA(V)1.5 | 3-DIMENSIONAL STRUCTURE | GAP-JUNCTIONS | ADENOVIRUS RECEPTOR | ARRHYTHMOGENIC CARDIOMYOPATHIES | RIGHT-VENTRICULAR CARDIOMYOPATHY | Gap Junctions - metabolism | Humans | Adherens Junctions - metabolism | Arrhythmias, Cardiac - physiopathology | Arrhythmias, Cardiac - pathology | Desmosomes - pathology | Intercellular Junctions - pathology | Membrane Proteins - metabolism | Arrhythmias, Cardiac - genetics | Arrhythmias, Cardiac - metabolism | Desmosomes - metabolism | Gap Junctions - pathology | Genetic Predisposition to Disease | Signal Transduction | Membrane Proteins - genetics | Intercellular Junctions - metabolism | Cell Communication | Intercellular Junctions - genetics | Adherens Junctions - pathology | Mechanotransduction, Cellular | Myocytes, Cardiac - pathology | Animals | Ion Channels - metabolism | Myocytes, Cardiac - metabolism | Mutation
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7. Full Text Proinflammatory Cytokines Disrupt Epithelial Barrier Function by Apoptosis-Independent Mechanisms
by Bruewer, Matthias and Luegering, Andreas and Kucharzik, Torsten and Parkos, Charles A and Madara, James L and Hopkins, Ann M and Nusrat, Asma
The Journal of Immunology, ISSN 0022-1767, 12/2003, Volume 171, Issue 11, pp. 6164 - 6172
It is well known that inflammatory conditions of the intestinal mucosa result in compromised barrier function. Inflammation is characterized by an influx into... 
FACTOR TNF RECEPTORS | TIGHT JUNCTIONS | INTERCELLULAR-ADHESION MOLECULE-1 | INFLAMMATORY-BOWEL-DISEASE | INTERFERON-GAMMA | CROHNS-DISEASE | IFN-GAMMA | CD95 (APO-1/FAS)-MEDIATED APOPTOSIS | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | INTESTINAL PERMEABILITY | Inflammation - pathology | Intestinal Mucosa - metabolism | Membrane Microdomains - metabolism | Fluoresceins - metabolism | Humans | Adherens Junctions - immunology | Adherens Junctions - metabolism | Membrane Microdomains - immunology | Inflammation - metabolism | Intestinal Mucosa - immunology | Octoxynol | Dextrans - metabolism | Tight Junctions - metabolism | Solubility | Interferon-gamma - physiology | Inflammation - immunology | Detergents | Adherens Junctions - pathology | Cell Membrane Permeability - immunology | Apoptosis - immunology | Tight Junctions - immunology | Up-Regulation - immunology | Tumor Necrosis Factor-alpha - physiology | Cell Line, Tumor | Membrane Microdomains - pathology | Tight Junctions - pathology | Intestinal Mucosa - pathology
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8. Full Text Spatio-temporal regulation of connexin43 phosphorylation and gap junction dynamics
by Solan, Joell L and Lampe, Paul D
BBA - Biomembranes, ISSN 0005-2736, 01/2018, Volume 1860, Issue 1, pp. 83 - 90
Gap junctions are specialized membrane domains containing tens to thousands of intercellular channels. These channels permit exchange of small molecules (<... 
Phosphorylation | Connexin43 | Kinase | Gap junction | ROUS-SARCOMA VIRUS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARDIAC MYOCYTES | PLASMA-MEMBRANE | PROTEIN CONNEXIN43 | ISCHEMIA-REPERFUSION INJURY | BIOPHYSICS | INTERCELLULAR COMMUNICATION | V-SRC | MOLECULAR-BASIS | ZONULA OCCLUDENS-1 | LIVING CELLS | Arrhythmias, Cardiac - metabolism | Arrhythmias, Cardiac - pathology | Gap Junctions - metabolism | Gap Junctions - pathology | Biological Transport, Active | Animals | Connexin 43 - metabolism | Humans | Heart Failure - metabolism | Heart Failure - pathology | Second Messenger Systems | Physiological aspects | Epidermal growth factor | Metabolites | Cyclic guanylic acid | Protein kinases | Membrane proteins
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9. Full Text The role of differential VE-cadherin dynamics in cell rearrangement during angiogenesis
by Bentley, Katie and Franco, Claudio Areias and Philippides, Andrew and Blanco, Raquel and Dierkes, Martina and Gebala, Véronique and Stanchi, Fabio and Jones, Martin and Aspalter, Irene M and Cagna, Guiseppe and Weström, Simone and Claesson-Welsh, Lena and Vestweber, Dietmar and Gerhardt, Holger and Medicinska fakulteten and Cancer och vaskulärbiologi and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för immunologi, genetik och patologi and Uppsala universitet
Nature Cell Biology, ISSN 1465-7392, 2014, Volume 16, Issue 4, pp. 309 - 321
Endothelial cells show surprising cell rearrangement behaviour during angiogenic sprouting; however, the underlying mechanisms and functional importance remain... 
MIGRATION | MORPHOGENESIS | REGULATES FORMATION | JUNCTIONS | MOUSE | PERMEABILITY IN-VIVO | ADHESION HYPOTHESIS | MODEL | ADAPTER PROTEIN | VASCULAR ENDOTHELIAL-CADHERIN | CELL BIOLOGY | Cadherins - metabolism | Signal Transduction | Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Male | Mice, Transgenic | Vascular Endothelial Growth Factor A - metabolism | Diabetic Retinopathy - pathology | Cell Movement - physiology | Diabetic Retinopathy - metabolism | Antigens, CD - metabolism | Neovascularization, Pathologic - pathology | Animals | Image Processing, Computer-Assisted | Cell Adhesion - physiology | Computer Simulation | Intercellular Junctions - pathology | Female | Mice | Neovascularization, Pathologic - metabolism | Endothelial Cells - pathology | Cell research | Cellular control mechanisms | Physiological aspects | Cadherins | Neovascularization | Research | Properties | Medical and Health Sciences | Medicin och hälsovetenskap
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10. Full Text Amyloid-β may be released from non-junctional varicosities of axons generated from abnormal tau-containing brainstem nuclei in sporadic Alzheimer's disease: A hypothesis
by Braak, Heiko and Del Tredici, Kelly
Acta Neuropathologica, ISSN 0001-6322, 08/2013, Volume 126, Issue 2, pp. 303 - 306
NATIONAL INSTITUTE | PROTEIN | EVOLUTION | DEPOSITION | NEUROFIBRILLARY DEGENERATION | ASSOCIATION GUIDELINES | PLAQUES | PATHOLOGY | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | CLINICAL NEUROLOGY | NEUROPATHOLOGIC ASSESSMENT | Brain Stem - pathology | Axons - pathology | Humans | Intercellular Junctions - pathology | Alzheimer Disease - metabolism | Amyloid beta-Peptides - metabolism | Brain Stem - metabolism | Intercellular Junctions - metabolism | Axons - metabolism | tau Proteins - metabolism | Alzheimer Disease - pathology
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11. Full Text Regulation of cardiovascular connexins by mechanical forces and junctions
by Meens, Merlijn J and Pfenniger, Anna and Kwak, Brenda R and Delmar, Mario
Cardiovascular Research, ISSN 0008-6363, 07/2013, Volume 99, Issue 2, pp. 304 - 314
Connexins form a family of transmembrane proteins that consists of 20 members in humans and 21 members in mice. Six connexins assemble into a connexon that can... 
Cardiovascular system | Connexins | Mechanotransduction | Gap junctions | CARDIAC & CARDIOVASCULAR SYSTEMS | PULMONARY ARTERIAL-HYPERTENSION | NITRIC-OXIDE SYNTHASE | CARDIAC MYOCYTES | ANGIOTENSIN-II | RAT AORTIC ENDOTHELIUM | ENDOTHELIUM-DEPENDENT RELAXATIONS | SHEAR-STRESS | MYOENDOTHELIAL GAP-JUNCTIONS | HYPERTROPHIED RIGHT VENTRICLES | RIGHT-VENTRICULAR CARDIOMYOPATHY | Humans | Intercellular Junctions - metabolism | Stress, Mechanical | Myocardium - pathology | Arteries - pathology | Regional Blood Flow | Hypertension - pathology | Hypertension - physiopathology | Arteries - metabolism | Connexins - metabolism | Hypertension - metabolism | Cardiovascular System - pathology | Mechanotransduction, Cellular | Biomechanical Phenomena | Cardiovascular System - physiopathology | Animals | Cardiovascular System - metabolism | Arteries - physiopathology | Intercellular Junctions - pathology | Myocardium - metabolism | Hemodynamics | Spotlight Reviews | Spotlight on Biomechanical Factors in Cardiovascular Disease
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