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1. Full Text Dll4 and Notch signalling couples sprouting angiogenesis and artery formation
by Pitulescu, Mara E and Schmidt, Inga and Giaimo, Benedetto Daniele and Antoine, Tobiah and Berkenfeld, Frank and Ferrante, Francesca and Park, Hongryeol and Ehling, Manuel and Biljes, Daniel and Rocha, Susana F and Langen, Urs H and Stehling, Martin and Nagasawa, Takashi and Ferrara, Napoleone and Borggrefe, Tilman and Adams, Ralf H
Nature cell biology, ISSN 1465-7392, 08/2017, Volume 19, Issue 8, pp. 915 - 927
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cell Proliferation | Jagged-1 Protein - metabolism | Male | Intracellular Signaling Peptides and Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Time Factors | Female | Receptors, CXCR4 - genetics | Cell Differentiation | Membrane Proteins - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Retinal Artery - metabolism | Signal Transduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Cell Communication | Genotype | Mice, Transgenic | Receptor, Notch1 - metabolism | Jagged-1 Protein - genetics | Receptors, CXCR4 - metabolism | Cell Lineage | Phenotype | Animals | Retinal Artery - cytology | Receptor, Notch1 - genetics | Neovascularization, Physiologic | Cell Movement | Angiogenesis | Intercellular signalling | Blood vessels | Notch protein | Cell interactions | Vascular endothelial growth factor | Arteries | Endothelial cells | CXCR4 protein | Veins & arteries | Index Medicus
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2. Full Text Endothelial Wnt/β-catenin signaling inhibits glioma angiogenesis and normalizes tumor blood vessels by inducing PDGF-B expression
by Reis, Marco and Czupalla, Cathrin J and Ziegler, Nicole and Devraj, Kavi and Zinke, Jenny and Seidel, Sascha and Heck, Rosario and Thom, Sonja and Macas, Jadranka and Bockamp, Ernesto and Fruttiger, Marcus and Taketo, Makoto M and Dimmeler, Stefanie and Plate, Karl H and Liebner, Stefan
The Journal of experimental medicine, ISSN 0022-1007, 08/2012, Volume 209, Issue 9, pp. 1611 - 1627
Immunology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Wnt1 Protein - genetics | Humans | Brain Neoplasms - pathology | Neovascularization, Pathologic | Brain Neoplasms - blood supply | Intracellular Signaling Peptides and Proteins - metabolism | Wnt1 Protein - metabolism | Glioma - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neoplasm Grading | Glioma - pathology | Female | Membrane Proteins - metabolism | Wnt Signaling Pathway | Central Nervous System Neoplasms - metabolism | Proto-Oncogene Proteins c-sis - genetics | Proto-Oncogene Proteins c-sis - metabolism | Brain Neoplasms - genetics | Forkhead Transcription Factors - genetics | beta Catenin - metabolism | Blood-Brain Barrier - metabolism | Glioma - blood supply | Xenograft Model Antitumor Assays | Central Nervous System Neoplasms - pathology | Animals | Endothelium, Vascular - metabolism | Mice, Nude | Mice | Central Nervous System Neoplasms - blood supply | Index Medicus
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3. Full Text Normal glucagon signaling and β-cell function after near-total α-cell ablation in adult mice
by Thorel, Fabrizio and Damond, Nicolas and Chera, Simona and Wiederkehr, Andreas and Thorens, Bernard and Meda, Paolo and Wollheim, Claes B and Herrera, Pedro L
Diabetes (New York, N.Y.), ISSN 0012-1797, 11/2011, Volume 60, Issue 11, pp. 2872 - 2882
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Insulin-Secreting Cells - secretion | Apoptosis - drug effects | Cell Count | Glucagon - genetics | Male | Diphtheria Toxin - toxicity | Insulin - blood | Glucagon - blood | Diabetes Mellitus, Experimental - blood | Hypoglycemia - prevention & control | Intercellular Signaling Peptides and Proteins - metabolism | Glucagon-Secreting Cells - drug effects | Insulin-Secreting Cells - metabolism | Hyperglycemia - chemically induced | Glucagon-Secreting Cells - metabolism | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Glucagon-Secreting Cells - secretion | Hyperglycemia - prevention & control | Promoter Regions, Genetic | Signal Transduction | Glucagon-Secreting Cells - pathology | Intercellular Signaling Peptides and Proteins - genetics | Pancreas - drug effects | Pancreas - pathology | Receptors, Glucagon - metabolism | Mice, Transgenic | Pancreas - metabolism | Heparin-binding EGF-like Growth Factor | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Tamoxifen - pharmacology | Diabetes Mellitus, Experimental - pathology | Glucagon - metabolism | Mice | Streptozocin - toxicity | Insulin-Secreting Cells - pathology | Selective Estrogen Receptor Modulators - pharmacology | Index Medicus | Abridged Index Medicus | Islet Studies
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4. Full Text APJ acts as a dual receptor in cardiac hypertrophy
by Scimia, Maria Cecilia and Hurtado, Cecilia and Ray, Saugata and Metzler, Scott and Wei, Ke and Wang, Jianming and Woods, Chris E and Purcell, Nicole H and Catalucci, Daniele and Akasaka, Takeshi and Bueno, Orlando F and Vlasuk, George P and Kaliman, Perla and Bodmer, Rolf and Smith, Layton H and Ashley, Euan and Mercola, Mark and Brown, Joan Heller and Ruiz-Lozano, Pilar
Nature (London), ISSN 0028-0836, 08/2012, Volume 488, Issue 7411, pp. 394 - 398
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cardiology. Vascular system | Heart | Biological and medical sciences | Medical sciences | Heart failure, cardiogenic pulmonary edema, cardiac enlargement | Blood Pressure | Mechanotransduction, Cellular - drug effects | Receptors, G-Protein-Coupled - metabolism | Arrestins - genetics | Cardiomegaly - pathology | Male | Receptors, G-Protein-Coupled - agonists | Mechanotransduction, Cellular - physiology | Arrestins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Female | Intercellular Signaling Peptides and Proteins - deficiency | Mechanoreceptors - metabolism | Apelin | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Cardiomegaly - physiopathology | Adipokines | Mice, Knockout | Aorta - pathology | Myocytes, Cardiac - pathology | Animals | Cardiomegaly - prevention & control | Myocytes, Cardiac - drug effects | Receptors, G-Protein-Coupled - deficiency | Signal Transduction - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | beta-Arrestins | Apelin Receptors | Mice | Receptors, G-Protein-Coupled - genetics | Arrestins - deficiency | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Cardiomegaly - metabolism | Genetically modified mice | Physiological aspects | Development and progression | Research | Heart enlargement | Proteins | Heart failure | Cardiomyocytes | Kinases | Laboratory animals | Rodents | Index Medicus
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5. Full Text The Notch Ligands Dll4 and Jagged1 Have Opposing Effects on Angiogenesis
by Benedito, Rui and Roca, Cristina and Sörensen, Inga and Adams, Susanne and Gossler, Achim and Fruttiger, Marcus and Adams, Ralf H
Cell (Cambridge), ISSN 0092-8674, 06/2009, Volume 137, Issue 6, pp. 1124 - 1135
CELLBIO | DEVBIO | SIGNALING | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Calcium-Binding Proteins - metabolism | Endothelial Cells - metabolism | Membrane Proteins - genetics | Receptors, Notch - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Male | Mice, Transgenic | Intracellular Signaling Peptides and Proteins - metabolism | Retina - embryology | Blood Vessels - cytology | Embryo, Mammalian - metabolism | Blood Vessels - embryology | Intercellular Signaling Peptides and Proteins - metabolism | Animals | Endothelium, Vascular - metabolism | Serrate-Jagged Proteins | Female | Membrane Proteins - metabolism | Mice | Mutation | Neovascularization, Physiologic | Calcium-Binding Proteins - genetics | Jagged-1 Protein | Research | Universities and colleges | Neovascularization | Transferases | Oncology, Experimental | Cancer | Index Medicus
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6. Full Text Regulation of monocyte cell fate by blood vessels mediated by Notch signalling
by Gamrekelashvili, Jaba and Giagnorio, Roberto and Jussofie, Jasmin and Soehnlein, Oliver and Duchene, Johan and Briseño, Carlos G and Ramasamy, Saravana K and Krishnasamy, Kashyap and Limbourg, Anne and Kapanadze, Tamar and Ishifune, Chieko and Hinkel, Rabea and Radtke, Freddy and Strobl, Lothar J and Zimber-Strobl, Ursula and Napp, L. Christian and Bauersachs, Johann and Haller, Hermann and Yasutomo, Koji and Kupatt, Christian and Murphy, Kenneth M and Adams, Ralf H and Weber, Christian and Limbourg, Florian P
Nature communications, ISSN 2041-1723, 2016, Volume 7, Issue 1, pp. 12597 - 12597
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Recombinant Proteins - metabolism | Calcium-Binding Proteins | Endothelial Cells - metabolism | Humans | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Receptor, Notch2 - metabolism | Male | Spleen - cytology | Adoptive Transfer | Healthy Volunteers | GPI-Linked Proteins - metabolism | Receptors, IgG - metabolism | Mice, Knockout | Intercellular Signaling Peptides and Proteins - metabolism | Animals | Antigens, Ly - metabolism | Signal Transduction - physiology | Cell Differentiation | Mice | Monocytes - physiology | Bone Marrow Cells - metabolism | Index Medicus
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7. Full Text Obesity induced a leptin‐Notch signaling axis in breast cancer
by Battle, Monica and Gillespie, Corey and Quarshie, Alexander and Lanier, Viola and Harmon, Tia and Wilson, Kaamilah and Torroella‐Kouri, Marta and Gonzalez‐Perez, Ruben R
International journal of cancer, ISSN 0020-7136, 04/2014, Volume 134, Issue 7, pp. 1605 - 1616
dominant negative RBP‐Jk | high‐fat diet | breast cancer, obesity, leptin, notch | E0771 cells | PEG‐LPrA2 | PEG-LPrA2 | dominant negative RBP-Jk | high-fat diet | Life Sciences & Biomedicine | Oncology | Science & Technology | Obesity | Biological and medical sciences | Metabolic diseases | Multiple tumors. Solid tumors. Tumors in childhood (general aspects) | Medical sciences | Tumors | Receptors, Notch - blood | Inhibitor of Apoptosis Proteins - genetics | Humans | Cell Growth Processes - physiology | Receptors, Notch - genetics | Obesity - blood | Obesity - genetics | Cell Movement - physiology | Intercellular Signaling Peptides and Proteins - metabolism | Leptin - blood | MCF-7 Cells | Serrate-Jagged Proteins | Female | Inhibitor of Apoptosis Proteins - metabolism | Membrane Proteins - metabolism | Repressor Proteins - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Signal Transduction | Membrane Proteins - genetics | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Repressor Proteins - genetics | Leptin - genetics | Obesity - pathology | Animals | Breast Neoplasms - genetics | Breast Neoplasms - blood | Cell Line, Tumor | Mice, Obese | Mice | Body Weight - physiology | Calcium-Binding Proteins - genetics | Leptin | Breast cancer | Hormones | Rodents | Risk factors | Index Medicus | Notch
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8. Full Text Classification and prediction of clinical Alzheimer's diagnosis based on plasma signaling proteins
by Galasko, Douglas R and Robinson, William H and Leszek, Jerzy and Karydas, Anna and Quinn, Joseph F and Tibshirani, Robert and Britschgi, Markus and Kaye, Jeffrey A and Rabinovici, Gil D and Yesavage, Jerome A and Herbert, Charles and Jutel, Marek and Minthon, Lennart and Wyss-Coray, Tony and Takeda-Uchimura, Yoshiko and Miller, Bruce L and Tinklenberg, Jared and Sabbagh, Marwan N and Boxer, Adam and Tabaton, Massimo and Ray, Sandip and Friedman, Leah F and So, Yuen T and Sparks, D Larry and Blennow, Kaj and Sahlgrenska akademin and Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi and Göteborgs universitet and Gothenburg University and Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry and Sahlgrenska Academy
Nature medicine, ISSN 1078-8956, 11/2007, Volume 13, Issue 11, pp. 1359 - 1362
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Alzheimer Disease - diagnosis | Cognition Disorders - diagnosis | Predictive Value of Tests | Phenotype | Alzheimer Disease - classification | Cognition Disorders - blood | Humans | Cognition Disorders - classification | Biomarkers - blood | Alzheimer Disease - blood | Intercellular Signaling Peptides and Proteins - blood | Proteins | Plasma | Signal transduction | Cellular biology | Classification | Alzheimers disease | Medical diagnosis | Index Medicus | disease | immune | Biokemi och molekylärbiologi | Cell and Molecular Biology | Biochemistry and Molecular Biology | responses | mild cognitive impairment | gene-expression | MEDICIN OCH HÄLSOVETENSKAP | Cell- och molekylärbiologi | MEDICAL AND HEALTH SCIENCES
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9. Full Text Stability and function of adult vasculature is sustained by Akt/Jagged1 signalling axis in endothelium
by Kerr, Bethany A and West, Xiaoxia Z and Kim, Young-Woong and Zhao, Yongzhong and Tischenko, Miroslava and Cull, Rebecca M and Phares, Timothy W and Peng, Xiao-Ding and Bernier-Latmani, Jeremiah and Petrova, Tatiana V and Adams, Ralf H and Hay, Nissim and Naga Prasad, Sathyamangla V and Byzova, Tatiana V
Nature communications, ISSN 2041-1723, 03/2016, Volume 7, Issue 1, pp. 10960 - 10960
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Heart | Human Umbilical Vein Endothelial Cells | Angiography | Eye - blood supply | Blood Vessels - metabolism | Humans | Immunoblotting | Retina | X-Ray Microtomography | Proto-Oncogene Proteins c-akt - genetics | Coronary Vessels - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Laminin | Serrate-Jagged Proteins | Membrane Proteins - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Echocardiography | Proteoglycans | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Membrane Proteins - genetics | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Signal Transduction - genetics | Muscle, Smooth, Vascular - cytology | Reverse Transcriptase Polymerase Chain Reaction | Blood-Brain Barrier - metabolism | Mice, Knockout | Animals | Myocytes, Smooth Muscle | Endothelium - metabolism | Collagen | Fluorescent Antibody Technique | Pericytes | Biocompatible Materials | Mice | Lung - blood supply | Drug Combinations | Homeostasis - genetics | Calcium-Binding Proteins - genetics | Index Medicus
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