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PROTEOMICS, ISSN 1615-9853, 05/2013, Volume 13, Issue 10-11, pp. 1672 - 1686
Exosomes are small extracellular 40–100 nm diameter membrane vesicles of late endosomal origin that can mediate intercellular transfer of RNA s and proteins to... 
Cell biology | Protein complex | Secretome | Metastasis | Exosomes | Cancer | STEM-CELLS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOCHEMICAL RESEARCH METHODS | COLON-CANCER | PREMETASTATIC NICHE | LIPID RAFTS | GROWTH-FACTOR | PROTEINS | CARCINOMA | TUMOR MICROENVIRONMENT | PROGRESSION | Ephrin-B2 - secretion | Calgranulin B - metabolism | Cell Proliferation | Calgranulin A - metabolism | Membrane Microdomains - metabolism | Protein-Serine-Threonine Kinases - secretion | Receptor, Epidermal Growth Factor - secretion | Humans | Exosomes - pathology | Tenascin - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neoplasm Metastasis | Receptor, Epidermal Growth Factor - metabolism | Serrate-Jagged Proteins | Exosomes - physiology | src-Family Kinases - metabolism | Colorectal Neoplasms | Membrane Proteins - metabolism | Intercellular Signaling Peptides and Proteins - secretion | src-Family Kinases - secretion | Endothelial Cells - physiology | Protein-Serine-Threonine Kinases - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Membrane Proteins - secretion | Signal Transduction | Ephrin-B2 - metabolism | Animals | Proteome - secretion | Calcium-Binding Proteins - secretion | Cell Line, Tumor | Tenascin - secretion | Mice | Proteome - metabolism | Proteins | Development and progression | Electron microscopy | Cells | Colorectal cancer | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 08/2011, Volume 71, Issue 15, pp. 5296 - 5306
The switch of tumor cells from an epithelial to a mesenchymal-like phenotype [designated as epithelial-to-mesenchymal transition (EMT)] is known to induce... 
BREAST-CARCINOMA | STEM-CELLS | IN-VITRO | ONCOLOGY | SERUM INTERLEUKIN-8 | PROSTATE-CANCER | METASTATIC PHENOTYPE | MALIGNANT-MELANOMA | TUMOR-GROWTH | EXPRESSION | PROGRESSION | Receptors, Interleukin-8 - genetics | T-Box Domain Proteins - physiology | Breast Neoplasms - secretion | Fibronectins - biosynthesis | Carcinoma - secretion | T-Box Domain Proteins - antagonists & inhibitors | Epithelial-Mesenchymal Transition - physiology | Humans | Neoplasm Proteins - physiology | T-Box Domain Proteins - biosynthesis | Culture Media, Conditioned - pharmacology | Culture Media, Serum-Free | Interleukin-8 - physiology | Neoplasm Proteins - antagonists & inhibitors | Bystander Effect | Tumor Microenvironment - physiology | Recombinant Fusion Proteins - antagonists & inhibitors | Cell Line, Tumor - pathology | Receptors, Interleukin-8 - biosynthesis | Cell Line, Tumor - secretion | Gene Expression Regulation, Neoplastic - drug effects | Intercellular Signaling Peptides and Proteins - secretion | Carcinoma - pathology | Neoplasm Proteins - genetics | Pancreatic Neoplasms - secretion | Promoter Regions, Genetic | Chemokines - secretion | Neoplasm Invasiveness | Neoplasm Proteins - biosynthesis | Pancreatic Neoplasms - pathology | RNA, Small Interfering - pharmacology | Cytokines - secretion | Fetal Proteins - physiology | Fetal Proteins - antagonists & inhibitors | Fetal Proteins - biosynthesis | T-Box Domain Proteins - genetics | Breast Neoplasms - pathology | Fetal Proteins - genetics | Fibronectins - genetics | Cell Movement | Index Medicus | Brachyury | tumor microenvironment | IL-8 | metastasis | EMT
Journal Article
Nature, ISSN 0028-0836, 07/2011, Volume 475, Issue 7356, pp. 381 - 387
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 12/2017, Volume 47, Issue 6, pp. 1100 - 1113.e6
Natural killer (NK) cells are present in large populations at the maternal-fetal interface during early pregnancy. However, the role of NK cells in fetal... 
growth-promoting factors | fetal growth restriction | maternal-fetal interface | aged pregnancy | tissue-resident NK | decidual NK cells | fetal development | INTERFACE | RESIDENT NK CELLS | INFLAMMATION | SENESCENCE | INNATE LYMPHOID-CELLS | DIFFERENTIATION | IMMUNOLOGY | IMMUNE TOLERANCE | HLA-G | EXPRESSION | PREGNANCY | Abortion, Habitual - pathology | Antigens, CD - immunology | Carrier Proteins - secretion | Leukocyte Immunoglobulin-like Receptor B1 - genetics | Humans | Abortion, Habitual - immunology | Basic-Leucine Zipper Transcription Factors - immunology | Fetal Growth Retardation - genetics | T-Box Domain Proteins - immunology | Adoptive Transfer | Antigens, CD - genetics | Leukocyte Immunoglobulin-like Receptor B1 - immunology | Gene Expression Regulation, Developmental | Abortion, Habitual - genetics | Killer Cells, Natural - immunology | Adult | Female | Fetus | HLA-G Antigens - immunology | Intercellular Signaling Peptides and Proteins - secretion | HLA-G Antigens - genetics | Cytokines - genetics | Carrier Proteins - immunology | Cytokines - immunology | Killer Cells, Natural - transplantation | Decidua - immunology | Signal Transduction | Mice, Inbred C57BL | Cytokines - secretion | Intercellular Signaling Peptides and Proteins - genetics | Cellular Microenvironment | Fetal Growth Retardation - prevention & control | Decidua - pathology | Basic-Leucine Zipper Transcription Factors - genetics | T-Box Domain Proteins - genetics | Fetal Growth Retardation - pathology | Fetal Growth Retardation - immunology | Pregnancy | Carrier Proteins - genetics | Killer Cells, Natural - cytology | Animals | Fetal Development - immunology | Integrin alpha1 - genetics | Integrin alpha1 - immunology | Mice | Intercellular Signaling Peptides and Proteins - immunology | Killer cells | Histocompatibility antigens | Growth | HLA histocompatibility antigens | Osteoglycin | Secretion | Crosstalk | Fetuses | Pleiotrophin | Decidua | Adoptive transfer | Cells | Interfaces | Biomedical materials | Uterus | Biocompatibility | Histocompatibility antigen HLA | Natural killer cells | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, pp. e16899 - e16899
Immune evasion from NK surveillance related to inadequate NK-cell function has been suggested as an explanation of the high incidence of relapse and fatal... 
MHC CLASS-I | IMMUNORECEPTOR | NK CELLS | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | DOWN-REGULATION | RECEPTOR | LEUKEMIA | TUMOR-DERIVED EXOSOMES | EXPRESSION | CYTOTOXICITY | Exosomes - drug effects | Exosomes - metabolism | Leukemia, B-Cell - metabolism | Temperature | Immune Tolerance - physiology | Humans | Oxidative Stress - physiology | Intracellular Signaling Peptides and Proteins - metabolism | Histocompatibility Antigens Class I - metabolism | Lymphoma - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Immune Tolerance - immunology | Leukemia, T-Cell - metabolism | GPI-Linked Proteins - secretion | Intracellular Signaling Peptides and Proteins - secretion | Lymphoma - pathology | Stress, Physiological - drug effects | Intercellular Signaling Peptides and Proteins - secretion | Tumor Cells, Cultured | Stress, Physiological - physiology | Jurkat Cells | Hydrogen Peroxide - pharmacology | Leukemia, B-Cell - pathology | GPI-Linked Proteins - metabolism | Leukemia, B-Cell - genetics | Leukemia, T-Cell - genetics | Immune Tolerance - genetics | Ligands | Oxidative Stress - drug effects | Leukemia, T-Cell - pathology | Oxidative stress | B cells | RNA | Analysis | Toxicity | Leukemia | Cytotoxicity | mRNA | Exosomes | Proteins | Immunology | Humidity | Immune system | Stresses | Antigens | Cytokines | Secretion | Cloning | T cell receptors | Electron microscopy | Lymphoma | Stress | Polymerase chain reaction | Cytometry | Immunosuppression | Surveillance | Lymphocytes B | Cell lines | Lymphomas | Cancer | Apoptosis | Tumors | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1620 - 1636
Kidney injury molecule 1 (KIM-1, also known as TIM-1) is markedly upregulated in the proximal tubule after injury and is maladaptive when chronically... 
ISCHEMIA-REPERFUSION INJURY | B-CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | ISCHEMIA/REPERFUSION INJURY | MOLECULE-1 KIM-1 | EPITHELIAL-CELLS | APOPTOTIC CELLS | INNATE IMMUNE-RESPONSE | T-CELL IG | ACUTE-RENAL-FAILURE | URINARY BIOMARKER | Epithelial Cells - metabolism | Kidney - blood supply | Macrophage Activation - genetics | Apoptosis - drug effects | Intercellular Signaling Peptides and Proteins - biosynthesis | Male | NF-kappa B - metabolism | LLC-PK1 Cells | Epithelial Cells - secretion | Cisplatin - toxicity | Extracellular Matrix Proteins | Membrane Proteins - physiology | Swine | Kidney Tubules, Proximal - secretion | Acute Kidney Injury - chemically induced | Intercellular Signaling Peptides and Proteins - secretion | Acute Kidney Injury - etiology | Macrophage Activation - physiology | Protein Structure, Tertiary | Membrane Proteins - genetics | Mice, Inbred C57BL | Cytokines - secretion | Gene Expression Regulation | Phagocytosis - physiology | Inflammation | Radiation Chimera | Hepatitis A Virus Cellular Receptor 1 | Immunity, Innate | Acute Kidney Injury - prevention & control | Homeodomain Proteins - genetics | Mice, Knockout | Animals | Reperfusion Injury - prevention & control | Membrane Proteins - chemistry | Kidney Tubules, Proximal - metabolism | Phosphatidylinositol 3-Kinases - physiology | Mice | Apoptosis - physiology | Cytokines - biosynthesis | Kidneys | Immune response | Genetic research | Genetic aspects | Research | Gene expression | Injuries | Proteins | Phosphorylation | Hospitals | Laboratories | Mortality | Rodents | Acidification | Kinases | Apoptosis | Index Medicus | Abridged Index Medicus | Nephrology | Cell Biology
Journal Article