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PLoS pathogens, ISSN 1553-7374, 2013, Volume 9, Issue 11, p. e1003773
...) with antiviral function. Type I IFN has been known for more than half a century. However, more recently, type III IFN... 
CELLS | DEFENSE | VIRUS-INFECTION | DISTINCT | RIG-I | MICROBIOLOGY | IRF-7 | ANTIVIRAL RESPONSE | VIROLOGY | GENE | SIGNALING PATHWAY | ADAPTER PROTEIN | PARASITOLOGY | Epithelial Cells - metabolism | Influenza A virus - genetics | Respiratory Mucosa - virology | Interferon Regulatory Factor-7 - genetics | Interferon Type I - immunology | Interferon Regulatory Factor-3 - genetics | Interleukins - metabolism | Orthomyxoviridae Infections - genetics | Respiratory Mucosa - pathology | Interleukins - genetics | Interleukins - immunology | Respiratory Mucosa - immunology | Adaptor Proteins, Signal Transducing - immunology | Interferon Type I - metabolism | Influenza A virus - immunology | Membrane Proteins - metabolism | Interferon Regulatory Factor-3 - immunology | Nerve Tissue Proteins - immunology | Membrane Proteins - genetics | Orthomyxoviridae Infections - metabolism | Epithelial Cells - pathology | Membrane Proteins - immunology | Interferon Regulatory Factor-7 - immunology | Interferon Regulatory Factor-7 - metabolism | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Influenza A virus - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | Adaptor Proteins, Signal Transducing - genetics | Interferon Regulatory Factor-3 - metabolism | Interferon Type I - genetics | Mice | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Orthomyxoviridae Infections - immunology | Epithelial cells | Influenza | Physiological aspects | Host-parasite relationships | Interferon | Genetic aspects | Genetic transcription | Research | Health aspects | Airway (Medicine) | Interleukins/metabolism | Nerve Tissue Proteins/immunology | Orthomyxoviridae Infections/genetics | Orthomyxoviridae Infections/metabolism | Membrane Proteins/genetics | Adaptor Proteins, Signal Transducing/genetics | Interferon Regulatory Factor-7/genetics | Interleukins/immunology | Membrane Proteins/immunology | Life Sciences | Orthomyxoviridae Infections/immunology | Influenza A virus/genetics | Nerve Tissue Proteins/metabolism | Respiratory Mucosa/immunology | Immunology | Interferon Type I/immunology | Epithelial Cells/immunology | Epithelial Cells/virology | Interferon Regulatory Factor-7/immunology | Epithelial Cells/metabolism | Interferon Regulatory Factor-3/immunology | Respiratory Mucosa/pathology | Influenza A virus/immunology | Interleukins/genetics | Interferon Regulatory Factor-3/genetics | Influenza A virus/metabolism | Adaptor Proteins, Signal Transducing/metabolism | Epithelial Cells/pathology | Adaptor Proteins, Signal Transducing/immunology | Interferon Type I/genetics | Interferon Regulatory Factor-3/metabolism | Membrane Proteins/metabolism | Nerve Tissue Proteins/genetics | Interferon Regulatory Factor-7/metabolism | Interferon Type I/metabolism | Respiratory Mucosa/metabolism | Respiratory Mucosa/virology | Cytokines | Genes | Rodents | Genomics | Genomes | Kinases | Experiments | Viral infections
Journal Article
Pediatric rheumatology online journal, ISSN 1546-0096, 2016, Volume 14, Issue 1, p. 35
Defective regulation of type I interferon response is associated with severe inflammatory phenotypes and autoimmunity... 
CANDLE | Type I interferonopathies | Familial lupus | Type I interferon | Aicardi-Goutières syndrome | SAVI | ALPHA MONOCLONAL-ANTIBODY | SYSTEMIC-LUPUS-ERYTHEMATOSUS | HYPER-GAMMA-GLOBULINEMIA | RHEUMATOLOGY | MUSCULAR-ATROPHY | AICARDI-GOUTIERES-SYNDROME | FAMILIAL CHILBLAIN LUPUS | Aicardi-Goutieres syndrome | DNA EXONUCLEASE TREX1 | PROTEASOME SUBUNIT | PEDIATRICS | PHASE-I | IFN-ALPHA | Proteome - genetics | Nervous System Malformations - immunology | Rare Diseases - immunology | Humans | Vascular Calcification - genetics | Rare Diseases - diagnosis | Arthritis, Juvenile - diagnosis | Metacarpus - abnormalities | Interferon Type I - immunology | Mutation - immunology | Autoimmune Diseases - genetics | Osteochondrodysplasias - genetics | Proteome - immunology | Lupus Erythematosus, Systemic - immunology | Osteoporosis - genetics | Autoimmune Diseases of the Nervous System - diagnosis | Metacarpus - immunology | Osteochondrodysplasias - immunology | Osteoporosis - immunology | Signal Transduction | Vascular Calcification - immunology | Aortic Diseases - immunology | Autoimmune Diseases - immunology | Autoimmune Diseases - diagnosis | Mutation - genetics | Odontodysplasia - immunology | Lupus Erythematosus, Systemic - diagnosis | Autoimmune Diseases of the Nervous System - immunology | Dental Enamel Hypoplasia - immunology | Homozygote | Aortic Diseases - genetics | Muscular Diseases - immunology | Rare Diseases - therapy | Arthritis, Juvenile - immunology | Lupus Erythematosus, Systemic - genetics | Nervous System Malformations - diagnosis | Autoimmune Diseases - therapy | Interferon Type I - genetics | Odontodysplasia - genetics | Muscular Diseases - genetics | Dental Enamel Hypoplasia - genetics | Systemic lupus erythematosus | Rheumatic diseases | Practice | Interferon | Research | Children | Rheumatologists | Health aspects | Risk factors
Journal Article
PLoS medicine, ISSN 1549-1676, 2006, Volume 3, Issue 7, pp. e289 - 1164
.... In autoimmune polyendocrinopathy syndrome type 1 (APS1; OMIM 240300), recessive AIRE mutations lead to autoimmunity targetting endocrine and other epithelial tissues, although chronic candidiasis usually appears first... 
DIABETES-MELLITUS | MYASTHENIA-GRAVIS PATIENTS | MEDICINE, GENERAL & INTERNAL | INTERFERON-ALPHA | SYNDROME TYPE-I | NEUTRALIZING ANTIBODIES | DENDRITIC CELLS | IFN-GAMMA | DISEASE | EXPRESSION | T-CELLS | Antibody Specificity | Polyendocrinopathies, Autoimmune - diagnosis | Alopecia - immunology | Autoantibodies - blood | Dendritic Cells - immunology | Humans | Middle Aged | Child, Preschool | Male | Interferon Type I - immunology | Interferons - immunology | Lymphocyte Subsets - immunology | Palatine Tonsil - immunology | Autoimmunity - immunology | Polyendocrinopathies, Autoimmune - genetics | Thymoma - immunology | HLA Antigens - immunology | Genotype | Polyendocrinopathies, Autoimmune - immunology | Lymphoid Tissue - pathology | Thymus Gland - ultrastructure | Norway - epidemiology | Adolescent | Cell Line, Tumor | Candidiasis, Chronic Mucocutaneous - etiology | Interferons - physiology | Finland - epidemiology | Self Tolerance - immunology | Transcription Factors - deficiency | Alopecia - etiology | Neutralization Tests | Keratoconjunctivitis - immunology | Keratoconjunctivitis - etiology | Myasthenia Gravis - etiology | Lymphoid Tissue - immunology | Thymus Neoplasms - immunology | Antigen Presentation | Immunoglobulin G - immunology | Self Tolerance - genetics | Interleukins - immunology | Adult | Female | Candidiasis, Chronic Mucocutaneous - immunology | Transcription Factors - genetics | Polyendocrinopathies, Autoimmune - epidemiology | Interferon-alpha - immunology | Thymus Neoplasms - complications | Autoantibodies - immunology | Interferons - classification | Thymus Gland - immunology | Myasthenia Gravis - immunology | Thymoma - complications | Development and progression | Genetic aspects | Research | Autoimmune diseases | Allergy | Immunology | Gene Therapy | Genomics | Autoimmune Diseases | Genetics | Immunology and Allergy
Journal Article
The Journal of experimental medicine, ISSN 0022-1007, 07/2009, Volume 206, Issue 7, pp. 1589 - 1602
Journal Article
Journal Article
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2013, Volume 39, Issue 1, pp. 111 - 122
Immune cells sense microbial products through Toll-like receptors (TLR), which trigger host defense responses including type 1 interferons (IFNs) secretion... 
TUMOR-NECROSIS-FACTOR | ACTIVATION | I INTERFERONS | CHORIOMENINGITIS VIRUS | PROTEIN-TYROSINE PHOSPHATASES | HOST-DEFENSE | MURINE LUPUS | INFLAMMATORY ARTHRITIS | IMMUNOLOGY | NF-KAPPA-B | T-CELLS | Lymphocytic choriomeningitis virus - immunology | Colitis - genetics | Humans | Autoimmunity - genetics | Immunoblotting | Arthritis - genetics | Interferon Type I - immunology | Lymphocytic Choriomeningitis - immunology | Host-Pathogen Interactions - immunology | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - immunology | Autoimmunity - immunology | Myeloid Cells - immunology | TNF Receptor-Associated Factor 3 - genetics | Colitis - chemically induced | HEK293 Cells | Arthritis - immunology | Interferon Type I - metabolism | Lymphocytic choriomeningitis virus - physiology | Toll-Like Receptors - metabolism | Colitis - immunology | Lymphocytic Choriomeningitis - genetics | Cell Line | Lymphocytic Choriomeningitis - virology | Mice, Inbred C57BL | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - metabolism | Toll-Like Receptors - immunology | Cells, Cultured | Immunity - immunology | Mice, Transgenic | TNF Receptor-Associated Factor 3 - metabolism | Ubiquitination - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Animals | Dextran Sulfate - immunology | TNF Receptor-Associated Factor 3 - immunology | Toll-Like Receptors - genetics | Interferon Type I - genetics | Myeloid Cells - metabolism | Mice | Immunity - genetics | Ubiquitin | Autoimmunity | Interferon | Biological response modifiers
Journal Article
Virus research, ISSN 0168-1702, 2011, Volume 162, Issue 1, pp. 12 - 18
...–50 years of unpredictable severity. Among the first lines of defense against influenza virus infection, the type I interferon (IFN... 
Influenza A | Review | Type 1 interferon | Influenza B | DOUBLE-STRANDED-RNA | B VIRUS | A-VIRUS | SENSOR RIG-I | PLASMACYTOID DENDRITIC CELLS | NS1 PROTEIN | ANTIVIRAL RESPONSES | VIROLOGY | STRUCTURAL BASIS | TOLL-LIKE RECEPTORS | INFECTED-CELLS | Antigens, CD - immunology | Ubiquitins - genetics | Virus Replication - genetics | Humans | Viral Proteins - immunology | eIF-2 Kinase - immunology | Interferon Type I - immunology | Antigens, CD - genetics | GTP-Binding Proteins - genetics | Viral Proteins - metabolism | Host-Pathogen Interactions - immunology | Orthomyxoviridae - immunology | Interferon Type I - biosynthesis | Myxovirus Resistance Proteins | Influenza, Human - virology | Cytokines - genetics | Cytokines - immunology | eIF-2 Kinase - genetics | DEAD Box Protein 58 | GPI-Linked Proteins - immunology | Ubiquitins - immunology | GTP-Binding Proteins - immunology | Toll-Like Receptors - immunology | Orthomyxoviridae - metabolism | Viral Proteins - genetics | Virus Replication - immunology | Immunity, Innate | Proteins - immunology | 2',5'-Oligoadenylate Synthetase - genetics | Proteins - genetics | DEAD-box RNA Helicases - genetics | Animals | Immune Evasion | 2',5'-Oligoadenylate Synthetase - immunology | Toll-Like Receptors - genetics | DEAD-box RNA Helicases - immunology | Antigens, Differentiation - genetics | Mice | Antigens, Differentiation - immunology | GPI-Linked Proteins - genetics | Influenza, Human - immunology | Influenza viruses | Interferon | Biological response modifiers | Health aspects | Influenza
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2013, Volume 131, Issue 6, pp. 1611 - 1623.e3