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NATURE, ISSN 0028-0836, 02/2011, Volume 470, Issue 7333, pp. 264 - 264
Journal Article
by Tsoi, L.C and Spain, S.L and Knight, J and Ellinghaus, E and Stuart, P.E and Capon, F and Ding, J and Li, J and Tejasvi, T and Gudjonsson, J.E and Kang, H and Allen, M.H and McManus, R and Novelli, G and Samuelsson, L and Schalkwijk, J and Stahle, M and Burden, A.D and Smith, C.H and Cork, M.J and Estivill, X and Bowcock, A and Krueger, G.G and Weger, W and Worthington, J and Tazi-Ahnini, R and Nestle, F.O and Hayday, A and Hoffmann, P and Winkelmann, J and Wijmenga, C and Langford, C and Edkins, S and Anews, R and Blackburn, H and Strange, A and Band, G and Pearson, R and Vukcevic, D and Spencer, C.C and Deloukas, P and Mrowietz, U and Schreiber, S and Weidinger, S and Koks, S and Kingo, K and Esko, T and Metspalu, A and Lim, H.W and Voorhees, J.J and Weichenthal, M and Wichmann, H.E and Chanan, V and Rosen, C.F and Rahman, P and Gladman, D.D and Griffiths, C.E and Reis, A and Kere, J and Duffin, K.C and Helms, C and Goldgar, D and Li, Y and Paschall, J and Malloy, M.J and Pullinger, C.R and Kane, J.P and Gardner, J and Perlmutter, A and Miner, A and Feng, B.J and Hiremagalore, R and Ike, R.W and Christophers, E and Henseler, T and Ruether, A and Schrodi, S.J and Prahalad, S and Guthery, S.L and Hiregalore, R and Donnelly, P and Collaborative Assoc Study Psoriasi and Psoriasis Assoc Genetics Extension and Genetic Anal Psoriasis Consortium and Wellcome Trust Case Control Consor and Psoriasis Association Genetics Extension and Genetic Analysis of Psoriasis Consortium and Collaborative Association Study of Psoriasis (CASP) and Wellcome Trust Case Control Consortium 2 and Sahlgrenska akademin and Institutionen för biomedicin, avdelningen för medicinsk genetik och klinisk genetik and Göteborgs universitet and Gothenburg University and Institute of Biomedicine, Department of Medical and Clinical Genetics and Sahlgrenska Academy
Nature Genetics, ISSN 1061-4036, 2012, Volume 44, Issue 12, pp. 1341 - 1348
To gain further insight into the genetic architecture of psoriasis, we conducted a meta-analysis of 3 genome-wide association studies (GWAS) and 2 independent... 
COMMON VARIANTS | SIGNALING PATHWAYS | MULTIPLE COMMON | INTERFERON-GAMMA | TH17 DIFFERENTIATION | GENETICS & HEREDITY | GENE-EXPRESSION | T-CELL DEVELOPMENT | GENOME-WIDE ASSOCIATION | CELIAC-DISEASE | NEGATIVE REGULATOR | Oligonucleotide Array Sequence Analysis | Humans | Immunity, Innate - genetics | Genetic Loci | CARD Signaling Adaptor Proteins - genetics | Psoriasis - genetics | Core Binding Factor Alpha 3 Subunit - immunology | STAT3 Transcription Factor - genetics | Skin - immunology | Psoriasis - immunology | CARD Signaling Adaptor Proteins - immunology | DEAD Box Protein 58 | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Guanylate Cyclase - immunology | Membrane Proteins - genetics | Membrane Proteins - immunology | GTPase-Activating Proteins - immunology | DEAD-box RNA Helicases - genetics | Core Binding Factor Alpha 3 Subunit - genetics | DEAD-box RNA Helicases - immunology | Guanylate Cyclase - genetics | T-Lymphocytes - immunology | Polymorphism, Single Nucleotide | GTPase-Activating Proteins - genetics | STAT3 Transcription Factor - immunology | Psoriasis | Physiological aspects | Disease susceptibility | Genetic aspects | Research | Autoimmune diseases | Gene expression | Chromosomes | Risk factors | Index Medicus | genetic analysis | innate immunity | Dermatologi och venereologi | Dermatology and Venereal Diseases | psoriasis
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 4, pp. 993 - 1006.e1
Journal Article
Science, ISSN 0036-8075, 2014, Volume 343, Issue 6175, pp. 1118 - 1118
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4190 - 4202
Macrophages in atherosclerotic plaques drive inflammatory responses, degrade lipoproteins, and phagocytose dead cells. MicroRNAs (miRs) control the... 
MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNE-SYSTEM | REVERSE CHOLESTEROL TRANSPORT | ACTIVATED-RECEPTOR-GAMMA | LIPID UPTAKE | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | DEFICIENT MICE | INFLAMMATORY RESPONSE | FOAM CELLS | APOLIPOPROTEIN-E | Vasculitis - metabolism | Plaque, Atherosclerotic - genetics | Atherosclerosis - genetics | Vasculitis - pathology | MicroRNAs - metabolism | NF-kappa B - metabolism | Interferon-gamma - metabolism | Lipoproteins, LDL - genetics | Apolipoproteins E - metabolism | DNA-Binding Proteins - metabolism | Vasculitis - genetics | Chemokine CCL2 - metabolism | Lipoproteins, LDL - metabolism | Interferon-gamma - genetics | Plaque, Atherosclerotic - pathology | Atherosclerosis - pathology | DNA-Binding Proteins - antagonists & inhibitors | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Gene Expression Regulation - genetics | Chemokine CCL2 - genetics | Signal Transduction - genetics | DNA-Binding Proteins - genetics | Atherosclerosis - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | NF-kappa B - genetics | Apolipoproteins E - genetics | Mice | MicroRNAs - genetics | Proto-Oncogene Proteins c-bcl-6 | MicroRNA | Atherosclerosis | Development and progression | Genetic aspects | Research | Properties | Macrophages | Gene expression | Index Medicus | Abridged Index Medicus | Atherogenesis | Lipoproteins (low density) | Transcription factors | Immune response | Data processing | Inflammation | Lipoproteins | Monocytes | NF- Kappa B protein | Arteriosclerosis | gamma -Interferon | miRNA | Differentiation | Chemokines | Monocyte chemoattractant protein 1 | Plaques | Bcl-6 protein
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2012, Volume 131, Issue 11, pp. 2553 - 2561
Estrogen receptor‐beta (ERβ) has been suggested to exert anti‐inflammatory and anti‐tumorigenic effects in the colon, providing a translational potential to... 
dextran sodium sulfate | estrogen receptor‐beta | colon cancer | azoxymethane | inflammatory bowel disease | estrogen receptor-beta | Estradiol - blood | Mucin-2 - genetics | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Colitis - genetics | Colorectal Neoplasms - genetics | Tumor Necrosis Factor-alpha - genetics | Colitis - pathology | NF-kappa B - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Estrogen Receptor beta - metabolism | Cell Differentiation - genetics | Inflammation - metabolism | Estrogen Receptor beta - genetics | Proliferating Cell Nuclear Antigen - genetics | Neoplasms - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Female | Interferon-gamma - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Mucin-2 - metabolism | Mucin-1 - metabolism | Interleukin-6 - genetics | Colon - pathology | Mice, Inbred C57BL | Caveolin 1 - genetics | Interleukin-17 - genetics | beta Catenin - metabolism | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Caveolin 1 - metabolism | Interleukin-17 - metabolism | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Colitis - metabolism | Inflammation - genetics | Mice | Colorectal Neoplasms - pathology | Neoplasms - pathology | Proliferating Cell Nuclear Antigen - metabolism | Mucin-1 - genetics | Nitric Oxide Synthase Type II - metabolism | Index Medicus | Estrogen receptor-beta
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 02/2013, Volume 52, Issue 2, pp. 155 - 166
The Janus kinase (JAK)/signal transducer and activator of transcription (STAT) signaling pathway is involved in immune function and cell growth. We evaluated... 
cigarette smoking | STAT | estrogen | JAK | colon cancer | NSAIDs | rectal cancer | SOCS | Colon cancer | Rectal cancer | Estrogen | Cigarette smoking | OXIDATIVE STRESS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | RISK | ENERGY-BALANCE | INHIBITION | ONCOLOGY | JANUS KINASES | COLORECTAL-CANCER | INFLAMMATION | PHYSICAL-ACTIVITY | Smoking - adverse effects | Colonic Neoplasms - genetics | Receptors, Estrogen - metabolism | Colorectal Neoplasms - genetics | Humans | Middle Aged | STAT Transcription Factors - metabolism | STAT4 Transcription Factor - metabolism | TYK2 Kinase - metabolism | Suppressor of Cytokine Signaling 1 Protein | Male | Janus Kinases - metabolism | Interferon-gamma - metabolism | Case-Control Studies | Colonic Neoplasms - metabolism | Janus Kinase 1 - metabolism | STAT1 Transcription Factor - metabolism | Rectal Neoplasms - genetics | Janus Kinase 1 - genetics | Janus Kinase 2 - metabolism | Adult | Female | Interferon-gamma - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Colonic Neoplasms - mortality | Genetic Predisposition to Disease | Interleukin-6 - genetics | Janus Kinases - genetics | Signal Transduction | Janus Kinase 2 - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Survival Rate | STAT1 Transcription Factor - genetics | Rectal Neoplasms - mortality | STAT Transcription Factors - genetics | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | STAT4 Transcription Factor - genetics | Aged | Polymorphism, Single Nucleotide | Rectal Neoplasms - metabolism | Suppressor of Cytokine Signaling Proteins - metabolism | TYK2 Kinase - genetics | Aspirin | Analysis | Oncology, Experimental | Research | Public health | Smoking | Cancer | Genetic diversity | Kinases | Risk factors | Colorectal cancer | Index Medicus
Journal Article
Inflammatory Bowel Diseases, ISSN 1078-0998, 05/2016, Volume 22, Issue 8, pp. 1812 - 1834
Background:Clinical observations or animal studies implicate enteric glial cells in motility disorders, irritable bowel syndrome, inflammatory bowel disease,... 
adenosine | human enteric glial cells | purinergic signaling | signaling | mechanosensitivity | bacterial lipopolysaccharide | ATP | Ca 2 | NERVOUS-SYSTEM | OXIDATIVE STRESS | Ca2+ signaling | CROHNS-DISEASE | ULCERATIVE-COLITIS | PROTECT NEURONS | ASTROCYTES | INTESTINAL INFLAMMATION | INFLAMMATORY-BOWEL-DISEASE | MOUSE MODEL | MICE | GASTROENTEROLOGY & HEPATOLOGY | Enteric Nervous System - cytology | Superoxide Dismutase - genetics | Gene Expression - drug effects | Calcium - metabolism | Humans | Gastrointestinal Motility | Heme Oxygenase-1 - genetics | Inflammation - metabolism | Adenosine Triphosphate - metabolism | Caspase 3 - genetics | Jejunum - cytology | S100 Calcium Binding Protein beta Subunit - metabolism | Calcium Channels - genetics | Cytokines - genetics | Mechanotransduction, Cellular - genetics | Cytokines - metabolism | Vesicular Transport Proteins - genetics | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Signal Transduction - genetics | Transcription Factors - genetics | Receptors, Purinergic - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Receptors, Purinergic - genetics | Tryptophan Hydroxylase - genetics | Up-Regulation - drug effects | Carrier Proteins - genetics | Phenotype | Gastrointestinal Diseases - genetics | Gastrointestinal Diseases - metabolism | Nitric Oxide Synthase Type II - genetics | Lipopolysaccharides - pharmacology | Colon, Sigmoid - cytology | Inflammation - genetics | Neuroglia - metabolism | Interferon-gamma - pharmacology | Index Medicus | Human Enteric Glial Cells
Journal Article