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Journal of Immunology, ISSN 0022-1767, 01/2014, Volume 192, Issue 2, pp. 623 - 629
.... Although the precise origin of the unchecked inflammatory response in obesity is unclear, it is known that overproduction of proinflammatory cytokines by innate immune cells affects metabolism... 
IMMUNE-SYSTEM | INFLAMMATION | DISEASE | RESISTANCE | FAT | AKT | IMMUNOLOGY | DIET-INDUCED OBESITY | KEY PLAYERS | ADIPOSE-TISSUE | CUTTING EDGE | Tumor Necrosis Factor-alpha - metabolism | Antigens, CD - immunology | T-Lymphocytes, Regulatory - metabolism | Epithelial Cells - metabolism | TOR Serine-Threonine Kinases - metabolism | Obesity - immunology | Insulin - immunology | Interferon-gamma - metabolism | Antigens, CD - metabolism | T-Lymphocytes, Regulatory - immunology | Epithelium - immunology | Signal Transduction - immunology | Inflammation - metabolism | Tumor Necrosis Factor-alpha - immunology | Interleukin-10 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Receptor, Insulin - immunology | Macrophages - immunology | Hyperinsulinism - metabolism | Intra-Abdominal Fat - immunology | Transforming Growth Factor beta - immunology | Epithelium - metabolism | Mice, Inbred C57BL | Apyrase - metabolism | Cells, Cultured | Apyrase - immunology | CTLA-4 Antigen - metabolism | Inflammation - immunology | CTLA-4 Antigen - immunology | Intra-Abdominal Fat - metabolism | Obesity - metabolism | TOR Serine-Threonine Kinases - immunology | Insulin - metabolism | Macrophages - metabolism | Animals | Hyperinsulinism - immunology | Proto-Oncogene Proteins c-akt - immunology | Epithelial Cells - immunology | Interferon-gamma - immunology | Interleukin-10 - antagonists & inhibitors | Receptor, Insulin - metabolism | Mice | Interleukin-10 - immunology | Transforming Growth Factor beta - metabolism
Journal Article
Diabetes care, ISSN 1935-5548, 2010, Volume 33, Issue 4, pp. 861 - 868
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2013, Volume 190, Issue 9, pp. 4640 - 4649
Dendritic cells (DC) are professional APCs that regulate innate and adaptive immunity. The role of fatty-acid synthesis in DC development and function is... 
CANCER-CELLS | APOPTOSIS | KAPPA-B ACTIVATION | UNFOLDED PROTEIN RESPONSE | CYTOKINE PRODUCTION | SYNTHASE INHIBITION | ER STRESS | MAP KINASE | MACROPHAGES | ENDOPLASMIC-RETICULUM STRESS | IMMUNOLOGY | Fatty Acids - immunology | Leukocytes, Mononuclear - metabolism | Chemokine CCL2 - immunology | Dendritic Cells - immunology | Humans | Caspase 3 - metabolism | Male | Cyclin B1 - metabolism | Interferon-gamma - metabolism | PPAR gamma - metabolism | CD4-Positive T-Lymphocytes - immunology | Mitogen-Activated Protein Kinase Kinases - metabolism | Liver - immunology | CD8-Positive T-Lymphocytes - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Liver - metabolism | CD4-Positive T-Lymphocytes - metabolism | Interleukin-12 - metabolism | B7-2 Antigen - metabolism | Cell Differentiation - immunology | Intercellular Adhesion Molecule-1 - metabolism | Proto-Oncogene Proteins c-akt - immunology | T-Lymphocytes, Cytotoxic - metabolism | Endoplasmic Reticulum - immunology | Interleukin-12 - immunology | Mice | Killer Cells, Natural - metabolism | CD8-Positive T-Lymphocytes - immunology | bcl-X Protein - metabolism | Fatty Acids - biosynthesis | Mitogen-Activated Protein Kinase Kinases - immunology | Caspase 3 - immunology | Endoplasmic Reticulum - metabolism | Genes, MHC Class II - immunology | PPAR gamma - immunology | Cyclin B1 - immunology | Leukocytes, Mononuclear - immunology | bcl-X Protein - immunology | Killer Cells, Natural - immunology | Chemokine CCL2 - metabolism | B7-2 Antigen - immunology | Fatty Acids - metabolism | Dendritic Cells - metabolism | T-Lymphocytes, Cytotoxic - immunology | B7-1 Antigen - immunology | Intercellular Adhesion Molecule-1 - immunology | Mice, Inbred C57BL | B7-1 Antigen - metabolism | Animals | Apoptosis - immunology | Interferon-gamma - immunology | Dendritic Cells - cytology | Fatty-acid synthesis | ER stress | NK cells | T cells | Bone marrow dendritic cells
Journal Article
Arthritis research & therapy, ISSN 1478-6354, 2013, Volume 15, Issue 5, pp. R151 - R151
Introduction: T helper (Th)-17 cells are increased in systemic sclerosis (SSc). We therefore assessed whether Th17 cells could modulate the inflammatory and... 
RHEUMATOID-ARTHRITIS | MATRIX METALLOPROTEINASES | SYNOVIAL FIBROBLASTS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | GENE-EXPRESSION | SKIN FIBROSIS | NECROSIS-FACTOR-ALPHA | RHEUMATOLOGY | NF-KAPPA-B | T-CELLS | PULMONARY-FIBROSIS | Interleukin-8 - genetics | Gene Expression - drug effects | Skin - metabolism | Humans | Scleroderma, Systemic - pathology | Culture Media, Conditioned - pharmacology | Male | Interleukin-17 - pharmacology | Dose-Response Relationship, Drug | Collagen Type I - genetics | Th17 Cells - metabolism | Radioimmunoassay | Inflammation Mediators - metabolism | Female | Chemokine CCL2 - metabolism | Interleukin-8 - metabolism | Matrix Metalloproteinase 1 - genetics | Skin - pathology | Fibroblasts - metabolism | Collagen Type I - metabolism | Enzyme-Linked Immunosorbent Assay | Scleroderma, Systemic - metabolism | Cells, Cultured | Scleroderma, Systemic - genetics | Chemokine CCL2 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Tumor Necrosis Factor-alpha - pharmacology | Fibroblasts - drug effects | Matrix Metalloproteinase 1 - metabolism | Interferon-gamma - pharmacology | Culture Media, Conditioned - metabolism | Proteins | Medical equipment and supplies industry | Interleukins | Systemic scleroderma | Medical test kit industry | Scleroderma (Disease) | High-definition television | Skin | Biological response modifiers | Enzyme-linked immunosorbent assay
Journal Article
Gastroenterology, ISSN 0016-5085, 2007, Volume 133, Issue 2, pp. 559 - 573
Background & Aims: Recent studies have revealed that murine intestinal mucosa contains several kinds of lineage markers (lin)– c-kit+ immune precursor cells.... 
Gastroenterology and Hepatology | INFLAMMATORY-BOWEL-DISEASE | GUT CRYPTOPATCHES | NK CELLS | DENDRITIC CELLS | PEYERS-PATCHES | ALPHA-BETA | LYMPH-NODES | DELTA-T-CELLS | GASTROENTEROLOGY & HEPATOLOGY | LYMPHOCYTES-T | FETAL THYMUS | Membrane Glycoproteins - metabolism | Humans | Colitis, Ulcerative - genetics | Crohn Disease - metabolism | Hematopoietic Stem Cells - pathology | Lymphotoxin-alpha - metabolism | Interferon-gamma - metabolism | RNA, Messenger - metabolism | Colitis, Ulcerative - immunology | Antigens, CD - metabolism | Immunoglobulins - metabolism | Lymphotoxin-alpha - genetics | Time Factors | Intestinal Mucosa - immunology | Inhibitor of Differentiation Protein 2 - genetics | Cell Differentiation | Gene Expression | Colitis, Ulcerative - metabolism | Colitis, Ulcerative - pathology | Inhibitor of Differentiation Protein 2 - metabolism | Cell Lineage | Adult Stem Cells - metabolism | Interleukin-2 Receptor alpha Subunit - metabolism | Crohn Disease - pathology | ADP-ribosyl Cyclase 1 - metabolism | Killer Cells, Natural - metabolism | Intestinal Mucosa - pathology | Crohn Disease - genetics | Intestinal Mucosa - metabolism | Antigens, CD34 - metabolism | Killer Cells, Natural - pathology | Proto-Oncogene Proteins c-kit - metabolism | Adult Stem Cells - immunology | DNA-Binding Proteins - metabolism | Hematopoietic Stem Cells - immunology | Trans-Activators - genetics | Killer Cells, Natural - immunology | Adult Stem Cells - pathology | Sialic Acid Binding Ig-like Lectin 3 | Proto-Oncogene Proteins - metabolism | CD56 Antigen - metabolism | Receptors, Interleukin-7 - metabolism | Cells, Cultured | Immunophenotyping | Proto-Oncogene Proteins - genetics | Hematopoietic Stem Cells - metabolism | Integrin alpha Chains - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Crohn Disease - immunology | Transcription Factors - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Trans-Activators - metabolism
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2009, Volume 106, Issue 32, pp. 13463 - 13468
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 11, p. e112945
The kynurenine pathway (KP) is the principal route of L-tryptophan (TRP) catabolism leading to the production of kynurenine (KYN), the neuroprotectants,... 
QUINOLINIC ACID | INHIBITION | METABOLISM | AMINOTRANSFERASE | MULTIDISCIPLINARY SCIENCES | INDOLEAMINE 2,3-DIOXYGENASE | T-CELL | TUMORAL IMMUNE RESISTANCE | TRYPTOPHAN DEGRADATION | IDO EXPRESSION | BRAIN | Immunohistochemistry | Indoleamine-Pyrrole 2,3,-Dioxygenase - genetics | Indoleamine-Pyrrole 2,3,-Dioxygenase - metabolism | Kynurenic Acid - blood | Gene Expression - drug effects | Humans | Kynurenine - biosynthesis | Brain Neoplasms - physiopathology | Picolinic Acids - blood | Quinolinic Acid - blood | Chromatography, High Pressure Liquid | Glial Fibrillary Acidic Protein - metabolism | Kynurenic Acid - metabolism | Carboxy-Lyases - genetics | Tryptophan - metabolism | Brain Neoplasms - metabolism | Picolinic Acids - metabolism | Antigens, CD - metabolism | Glioma - metabolism | Glioma - genetics | Kynurenine - blood | Tryptophan - blood | Carboxy-Lyases - metabolism | Tumor Cells, Cultured | Astrocytes - drug effects | Disaccharides | Cells, Cultured | Brain Neoplasms - genetics | Biosynthetic Pathways | Reverse Transcriptase Polymerase Chain Reaction | Glucuronates | Tryptophan Oxygenase - metabolism | Tryptophan Oxygenase - genetics | Antigens, Differentiation, Myelomonocytic - metabolism | Quinolinic Acid - metabolism | CD11b Antigen - metabolism | Glioma - physiopathology | Interferon-gamma - pharmacology | Astrocytes - metabolism | Tryptophan | Niacinamide | Methyl aspartate | Enzymes | Metabolites | Gliomas | Neuroprotection | Brain | Brain tumors | Brain cancer | Glioblastoma | Tryptophan 2,3-dioxygenase | Immunity | DNA repair | Picolinic acid | Rodents | Glioma cells | L-Tryptophan | Kynurenic acid | Quinolinic acid | Deoxyribonucleic acid--DNA | Pyridines | Medical research | Hydroxylase | Adenine | Kynurenine-oxoglutarate transaminase | NAD | Acids | γ-Interferon | Nicotinamide adenine dinucleotide | Catabolism | Nicotinamide | Interferon | Tumors | Deoxyribonucleic acid | DNA
Journal Article
International journal of cancer, ISSN 0020-7136, 2012, Volume 131, Issue 11, pp. 2553 - 2561
Estrogen receptor‐beta (ERβ) has been suggested to exert anti‐inflammatory and anti‐tumorigenic effects in the colon, providing a translational potential to... 
dextran sodium sulfate | estrogen receptor‐beta | colon cancer | azoxymethane | inflammatory bowel disease | estrogen receptor-beta | Estradiol - blood | Mucin-2 - genetics | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Colitis - genetics | Colorectal Neoplasms - genetics | Tumor Necrosis Factor-alpha - genetics | Colitis - pathology | NF-kappa B - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Estrogen Receptor beta - metabolism | Cell Differentiation - genetics | Inflammation - metabolism | Estrogen Receptor beta - genetics | Proliferating Cell Nuclear Antigen - genetics | Neoplasms - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Female | Interferon-gamma - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Mucin-2 - metabolism | Mucin-1 - metabolism | Interleukin-6 - genetics | Colon - pathology | Mice, Inbred C57BL | Caveolin 1 - genetics | Interleukin-17 - genetics | beta Catenin - metabolism | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Caveolin 1 - metabolism | Interleukin-17 - metabolism | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Colitis - metabolism | Inflammation - genetics | Mice | Colorectal Neoplasms - pathology | Neoplasms - pathology | Proliferating Cell Nuclear Antigen - metabolism | Mucin-1 - genetics | Nitric Oxide Synthase Type II - metabolism | Estrogen receptor-beta
Journal Article
Science translational medicine, ISSN 1946-6242, 2015, Volume 7, Issue 311, pp. 311ra173 - 311ra173
Journal Article
Nature communications, ISSN 2041-1723, 2018, Volume 9, Issue 1, pp. 3728 - 11
Anti-microbial signaling pathways are normally triggered by innate immune receptors when detecting pathogenic microbes to provide protective immunity. Here we... 
Inflammasomes | Signal Transduction | Humans | Mice, Inbred C57BL | Gastrointestinal Microbiome | Clostridiales | Interferon-gamma/metabolism | Male | Mice, Transgenic | Adaptor Proteins, Signal Transducing/genetics | Colon/pathology | Animals | Gene Deletion | Intestinal Mucosa/metabolism | T-Lymphocytes/cytology | Vancomycin/pharmacology | Rectum/metabolism | Apoptosis Regulatory Proteins/genetics | Female | Interleukin-18/metabolism | Mice | Colitis/metabolism | Inflammatory Bowel Diseases/drug therapy | Butyrates/metabolism | ACTIVATION | INTERLEUKIN-18 | CROHNS-DISEASE | COLITIS | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | MICROBIOTA | POLYMORPHISM | PYROPTOSIS | SEVERITY | Butyrates - metabolism | Intestinal Mucosa - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Vancomycin - pharmacology | Apoptosis Regulatory Proteins - genetics | Inflammatory Bowel Diseases - drug therapy | Colon - pathology | Rectum - metabolism | Apoptosis Regulatory Proteins - metabolism | T-Lymphocytes - cytology | Adaptor Proteins, Signal Transducing - genetics | Colitis - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Interleukin-18 - metabolism | Inflammatory bowel diseases | Commensals | Mucosa | Vancomycin | Immunity | Antiinfectives and antibacterials | Interleukin 18 | Inflammatory bowel disease | Receptors | Microorganisms | Intestine | γ-Interferon | Rectum | Colon | Supplementation | Ulcerative colitis
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2014, Volume 289, Issue 24, pp. 16773 - 16789
Lipocalin-2 (LCN2) plays an important role in cellular processes as diverse as cell growth, migration/invasion, differentiation, and death/survival.... 
PATHOGENESIS | MULTIPLE-SCLEROSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | NEURON DEATH | REACTIVE ASTROCYTES | CENTRAL-NERVOUS-SYSTEM | GELATINASE-ASSOCIATED LIPOCALIN | CHEMOKINE RECEPTORS | EXPRESSION | CD8(+) T-CELLS | Oncogene Proteins - genetics | Spinal Cord - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Acute-Phase Proteins - genetics | Interferon-gamma - metabolism | Myelin Sheath - metabolism | Matrix Metalloproteinase 9 - metabolism | T-Lymphocytes - metabolism | Lipocalins - genetics | Matrix Metalloproteinase 9 - genetics | Spinal Cord - pathology | Gene Deletion | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Interferon-gamma - genetics | Neutrophils - metabolism | Dendritic Cells - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Lipocalins - metabolism | Acute-Phase Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Oncogene Proteins - metabolism | Interleukin-17 - genetics | Lymphoid Tissue - metabolism | Receptors, Cell Surface - metabolism | Interleukin-17 - metabolism | Lymphoid Tissue - pathology | Animals | Lipocalin-2 | Myelin Sheath - genetics | Mice | Astrocytes - metabolism | Receptors, Cell Surface - genetics | Multiple Sclerosis | Autoimmune Disease | Central Nervous System | Neurobiology | EAE | LCN2 | Neuroinflammation | Astrocyte | Microglia
Journal Article
12.