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Hypertension, ISSN 0194-911X, 09/2004, Volume 44, Issue 3, pp. 264 - 270
Angiotensin II (Ang II) upregulates vascular endothelial growth factor (VEGF) and activates vascular inflammation. However, the decisive role of VEGF in Ang... 
Growth substances | Endothelial growth factors | Angiotensin II | Remodeling | Arteriosclerosis | MACROPHAGE INFILTRATION | FACTOR VEGF | remodeling | NITRIC-OXIDE SYNTHESIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | endothelial growth factors | FACTOR-KAPPA-B | TUMOR ANGIOGENESIS | CHRONIC BLOCKADE | TYPE-1 RECEPTOR | arteriosclerosis | growth substances | angiotensin II | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | TISSUE ANGIOTENSIN | Genetic Therapy | Tetrazoles - pharmacology | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Male | Gene Expression Profiling | Vasculitis - prevention & control | Vascular Endothelial Growth Factor A - genetics | Angiotensin II Type 1 Receptor Blockers - pharmacology | Transforming Growth Factor beta - biosynthesis | Hypoxia-Inducible Factor 1, alpha Subunit | Interleukin-1 - biosynthesis | Vascular Endothelial Growth Factor Receptor-2 - genetics | Imidazoles - therapeutic use | Receptors, Chemokine - genetics | Vasculitis - chemically induced | Coronary Vessels - pathology | Natriuretic Peptide, Brain - genetics | Transforming Growth Factor beta1 | Hypoxia-Inducible Factor 1 | Interleukin-6 - genetics | Extracellular Matrix Proteins - genetics | Imidazoles - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Aorta - pathology | Macrophages - metabolism | Intercellular Adhesion Molecule-1 - genetics | Chemokine CCL2 - biosynthesis | Nonmuscle Myosin Type IIB | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Vascular Endothelial Growth Factor A - physiology | Hypertrophy | Interleukin-1 - genetics | Tunica Media - pathology | Vascular Endothelial Growth Factor A - biosynthesis | Extracellular Matrix Proteins - biosynthesis | Tunica Media - drug effects | Vasculitis - physiopathology | Recombinant Fusion Proteins - physiology | Ventricular Remodeling | Nuclear Proteins - biosynthesis | Cell Division | Intercellular Adhesion Molecule-1 - biosynthesis | Receptors, Chemokine - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Vascular Endothelial Growth Factor Receptor-2 - biosynthesis | Angiotensin II - toxicity | Nuclear Proteins - genetics | Olmesartan Medoxomil | Hypertrophy, Left Ventricular - etiology | Mice, Inbred C57BL | Extracellular Matrix Proteins - physiology | Natriuretic Peptide, Brain - biosynthesis | Chemokine CCL2 - genetics | Renin-Angiotensin System - physiology | Transcription Factors - biosynthesis | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Animals | Transforming Growth Factor beta - genetics | Interleukin-6 - biosynthesis | Myosin Heavy Chains | Tetrazoles - therapeutic use | DNA-Binding Proteins - biosynthesis | Receptors, CCR2
Journal Article
Nature Communications, ISSN 2041-1723, 03/2011, Volume 2, Issue 1, p. 240
The immune system can both promote and suppress cancer. Chronic inflammation and proinflammatory cytokines such as interleukin (IL)-1 and IL-6 are considered... 
LUNG-CANCER | INTERFERON-INDUCIBLE PROTEIN-10 | NECROSIS IN-VIVO | PROGNOSTIC-FACTORS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | CD4(+) T-CELLS | ADJUVANT THERAPY | CARCINOMA | EXPRESSION | Angiostatic Proteins - immunology | Immunohistochemistry | Neoplasms - metabolism | Interleukin-1alpha - immunology | Chemokine CXCL9 - biosynthesis | Tumor Microenvironment | Multiple Myeloma - immunology | Th2 Cells - immunology | Th1 Cells - immunology | CD4-Positive T-Lymphocytes - immunology | Chemokine CXCL10 - biosynthesis | Inflammation - metabolism | Interleukin-2 - immunology | Chemokine CXCL10 - immunology | Lymphoma, B-Cell - immunology | Interleukin-1beta - biosynthesis | Macrophages - immunology | Interleukin-12 - biosynthesis | Lymphoma, B-Cell - metabolism | Signal Transduction | Angiostatic Proteins - biosynthesis | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Interleukin-1beta - immunology | Chemokine CXCL9 - immunology | Inflammation - immunology | Macrophages - cytology | Mice, SCID | Multiple Myeloma - metabolism | Macrophages - metabolism | Multiple Myeloma - pathology | Animals | Neoplasms - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Interleukin-6 - immunology | Lymphoma, B-Cell - pathology | Cell Line, Tumor | Interleukin-6 - biosynthesis | Interleukin-1alpha - biosynthesis | Mice | Interleukin-2 - biosynthesis | Neoplasms - pathology | Interferon-gamma - biosynthesis
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 12/2015, Volume 52, Issue 3, pp. 1547 - 1560
In this study, we tried to explore the molecular mechanism that Corilagin protected against herpes simplex virus-1 encephalitis through inhibiting the TLR2... 
Signaling pathways | Neurology | Neurosciences | Biomedicine | Toll-like receptor 2 | Neurobiology | Corilagin | Inflammation | Herpes simplex virus-1 | Cell Biology | ACTIVATION | INNATE IMMUNE-SYSTEM | MICROGLIAL CELLS | NEUROSCIENCES | ETHANOL EXTRACT | RESPONSES | HERPES-SIMPLEX-VIRUS | TOLL-LIKE RECEPTORS | INFECTION | TNF-ALPHA | PATTERN-RECOGNITION RECEPTORS | Microglia - metabolism | RNA, Small Interfering - genetics | TNF Receptor-Associated Factor 6 - biosynthesis | Toll-Like Receptor 2 - antagonists & inhibitors | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - biosynthesis | Receptors, Interleukin-1 - genetics | Tumor Necrosis Factor-alpha - genetics | Male | Brain - metabolism | RNA, Messenger - biosynthesis | Protein Processing, Post-Translational - drug effects | RNA Interference | Nerve Tissue Proteins - biosynthesis | Hydrolyzable Tannins - pharmacology | Encephalitis, Herpes Simplex - prevention & control | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | TNF Receptor-Associated Factor 6 - genetics | Intracellular Signaling Peptides and Proteins - genetics | Lipopeptides - toxicity | Antiviral Agents - pharmacology | Glucosides - pharmacology | Interleukin-6 - genetics | Microglia - drug effects | RNA, Messenger - genetics | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Receptors, Interleukin-1 - biosynthesis | Down-Regulation - drug effects | Intracellular Signaling Peptides and Proteins - biosynthesis | Nerve Tissue Proteins - genetics | Toll-Like Receptor 2 - biosynthesis | Herpesvirus 1, Human | Membrane Glycoproteins - genetics | Animals | NF-kappa B - genetics | Signal Transduction - drug effects | NF-kappa B - biosynthesis | Brain - pathology | Interleukin-6 - biosynthesis | Mice | Mice, Inbred BALB C | Myeloid Differentiation Factor 88 - biosynthesis | Tumor Necrosis Factor-alpha - biosynthesis | RNA | Yuan (China) | Encephalitis | Signaling | Tumor necrosis factor-TNF | Molecular biology | Neurons
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 06/2006, Volume 79, Issue 6, pp. 1279 - 1285
Dendritic cells (DCs) play an important role in innate and adaptive immune responses. In addition to their phagocytic activity, DCs present foreign antigens to... 
human | cytokines | cell activation | Human | Cytokines | Cell activation | SIGNAL-TRANSDUCTION PATHWAYS | ACTIVATION | RECEPTOR | ANTIGEN PRESENTATION | IMMUNOLOGY | CUTTING EDGE | CELL BIOLOGY | B-CELLS | HUMAN NK | COMMON GAMMA-CHAIN | HEMATOLOGY | T-CELLS | IFN-ALPHA | Dendritic Cells - immunology | Humans | Membrane Glycoproteins - biosynthesis | Receptors, Interleukin-1 - genetics | Tumor Necrosis Factor-alpha - genetics | Suppressor of Cytokine Signaling 1 Protein | Gene Expression Profiling | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - biosynthesis | Interleukins - physiology | T-Lymphocytes - metabolism | Chemokines, CC - biosynthesis | Cells, Cultured - immunology | Chemokines, CXC - biosynthesis | Cytokines - genetics | Intracellular Signaling Peptides and Proteins - genetics | Interleukin-12 - biosynthesis | Chemokine CCL5 | Cells, Cultured - drug effects | Receptors, Interleukin-21 | Lymphocyte Activation | Repressor Proteins - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Toll-Like Receptor 4 - genetics | Receptors, Interleukin-1 - biosynthesis | Receptors, Interleukin - genetics | Dendritic Cells - secretion | Feedback, Physiological | Macrophages - metabolism | Repressor Proteins - biosynthesis | T-Lymphocytes - immunology | Cells, Cultured - metabolism | HLA-DR Antigens - biosynthesis | Suppressor of Cytokine Signaling Proteins - biosynthesis | Tumor Necrosis Factor-alpha - biosynthesis | Receptors, Interleukin - biosynthesis | Toll-Like Receptor 4 - biosynthesis | Interleukin-12 - genetics | Dendritic Cells - drug effects | Interleukins - pharmacology | Receptors, Interferon - genetics | Dendritic Cells - metabolism | RNA, Messenger - genetics | Cell Communication | Recombinant Proteins - pharmacology | Chemokines, CXC - genetics | Membrane Glycoproteins - genetics | Gene Expression Regulation - drug effects | Suppressor of Cytokine Signaling 3 Protein | Chemokines, CC - genetics | Macrophages - drug effects | Interleukin-21 Receptor alpha Subunit | Receptors, Interferon - biosynthesis | Cytokines - biosynthesis | Interferon-gamma - pharmacology
Journal Article
Blood, ISSN 0006-4971, 04/2001, Volume 97, Issue 7, pp. 2121 - 2129
Leukostasis and tissue infiltration by leukemic cells are poorly understood life-threatening complications of acute leukemia. This study has tested the... 
SELECTIN GLYCOPROTEIN LIGAND-1 | INTERCELLULAR-ADHESION MOLECULE-1 | COLONY-STIMULATING FACTOR | IN-VIVO | P-SELECTIN | ACUTE MYELOID-LEUKEMIA | LEUKOCYTE ADHESION | HEMATOLOGY | NF-KAPPA-B | GRANULOCYTE-MACROPHAGE | HUMAN T-CELLS | Neoplastic Stem Cells - cytology | Lymphocyte Function-Associated Antigen-1 - biosynthesis | Coculture Techniques | Humans | Leukemia, Myeloid - genetics | Antigens, CD - genetics | Neoplastic Stem Cells - metabolism | E-Selectin - genetics | E-Selectin - biosynthesis | Leukostasis - genetics | Neoplasm Proteins - genetics | CD18 Antigens - genetics | Gene Expression Regulation, Leukemic | Cell Adhesion | Leukostasis - metabolism | Endothelium, Vascular - metabolism | Lymphocyte Function-Associated Antigen-1 - genetics | Macrophage-1 Antigen - genetics | Intercellular Adhesion Molecule-1 - genetics | Endothelium, Vascular - pathology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Integrin beta1 - genetics | Macrophage-1 Antigen - biosynthesis | Receptors, Lymphocyte Homing - genetics | Tumor Necrosis Factor-alpha - metabolism | Cell Adhesion Molecules - genetics | Leukemic Infiltration - genetics | Lewis X Antigen - genetics | Antigens, CD - biosynthesis | Integrin alphaXbeta2 - biosynthesis | L-Selectin - genetics | Neoplasm Proteins - metabolism | L-Selectin - biosynthesis | Leukemia, Myeloid - pathology | Integrin alphaXbeta2 - genetics | Intercellular Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Tumor Cells, Cultured | Integrin alpha6 | Leukemic Infiltration - metabolism | Interleukin-1 - metabolism | Integrin alpha4 | Integrin alpha5 | Cell Adhesion Molecules - biosynthesis | Neoplasm Proteins - biosynthesis | Cells, Cultured | Integrin beta1 - biosynthesis | Receptors, Lymphocyte Homing - biosynthesis | Leukemia, Myeloid - metabolism | Lewis X Antigen - biosynthesis | CD18 Antigens - biosynthesis
Journal Article
European Journal of Immunology, ISSN 0014-2980, 01/2018, Volume 48, Issue 1, pp. 168 - 179
IL‐22 induces STAT3 phosphorylation and mediates psoriasis‐related gene expression. However, the signaling mechanism leading from pSTAT3 to the expression of... 
IL‐22 | Bcl‐3 | p50 | Psoriasis | STAT3 | Bcl-3 | IL-22 | ENCODES | CELLS | IMMUNITY | TRANSCRIPTION | RECEPTOR EXPRESSION | INTERLEUKIN-22 | IMMUNOLOGY | IN-VITRO | SKIN | NF-KAPPA-B | CANDIDATE PROTOONCOGENE BCL-3 | Interleukin-8 - genetics | RNA, Small Interfering - genetics | Phosphorylation | Active Transport, Cell Nucleus - physiology | Humans | Proto-Oncogene Proteins - biosynthesis | Chemokine CCL20 - biosynthesis | Interleukins - metabolism | Interleukin-1 - biosynthesis | RNA, Messenger - biosynthesis | Psoriasis - genetics | S100 Proteins - genetics | Psoriasis - pathology | RNA Interference | Interleukins - biosynthesis | STAT3 Transcription Factor - metabolism | Skin - pathology | beta-Defensins - genetics | NF-kappa B p50 Subunit - metabolism | Gene Expression Regulation - genetics | Interleukin-8 - biosynthesis | RNA, Messenger - genetics | Cells, Cultured | Proto-Oncogene Proteins - genetics | Transcription Factors - biosynthesis | Interleukin-17 - biosynthesis | Transcription Factors - genetics | Interleukin-17 - metabolism | Keratinocytes - metabolism | Enzyme Activation | beta-Defensins - biosynthesis | Skin | Gene expression | Genes | Interleukin 19 | Pathogenesis | Stat3 protein | Keratinocytes | Interleukin 22 | Activation | siRNA | Kinases | Nuclei | Bcl-3 protein | Signaling | CCL20 protein | Skin diseases | Lesions
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2002, Volume 99, Issue 9, pp. 6198 - 6203
Endothelial cells (EC) play a central role in inflammatory immune responses and efficiently induce effector functions in T cells, despite lacking the classical... 
T lymphocytes | Cytometry | Molecules | Biological Sciences | Human umbilical vein endothelial cells | Isotypes | Cytokines | Secretion | Coculture techniques | Ligands | Cultured cells | RESPONSES | HELPER | PROTEIN | SPECIFICITY | MULTIDISCIPLINARY SCIENCES | CO-STIMULATION | RECEPTOR | ANTIGEN-PRESENTING CELLS | MONOCLONAL-ANTIBODY | IDENTIFICATION | MOLECULE ICOS | Endothelium, Vascular - cytology | Inducible T-Cell Co-Stimulator Protein | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Protein Biosynthesis | Alternative Splicing | Antigens, CD - biosynthesis | Humans | Membrane Glycoproteins - biosynthesis | Lipopolysaccharides - metabolism | Interleukin-13 - biosynthesis | Th1 Cells - metabolism | B7-2 Antigen | Interleukin-4 - biosynthesis | Flow Cytometry | T-Lymphocytes - metabolism | Time Factors | Antigens, Differentiation, T-Lymphocyte - physiology | Cell Division | B7-1 Antigen - biosynthesis | Interleukin-1 - metabolism | Proteins - physiology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Reverse Transcriptase Polymerase Chain Reaction | Th2 Cells - metabolism | Precipitin Tests | Animals | Inducible T-Cell Co-Stimulator Ligand | Protein Binding | Interleukin-10 - biosynthesis | Mice | Mice, Inbred BALB C | Antibodies, Monoclonal - metabolism | Antigens, Differentiation, T-Lymphocyte - biosynthesis | Interleukin-2 - biosynthesis | Cytokines - biosynthesis | Interferon-gamma - biosynthesis
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 2010, Volume 647, Issue 1, pp. 31 - 36
Degenerative joint diseases are related to the excessive degradation of collagen and proteoglycans in cartilage. One of the potent inflammatory mediators of... 
Peripheral benzodiazepine receptor | Chondrocyte | IL-1β | Ro-54864 | Pk-11195 | RHEUMATOID-ARTHRITIS | INTERLEUKIN-1 | LIGAND-BINDING | IL-1 beta | PROTEOGLYCAN | HYALURONIC-ACID | INSULIN | INHIBITION | ARTICULAR CHONDROCYTES | PHARMACOLOGY & PHARMACY | EXPRESSION | GROWTH-FACTOR-I | Receptor, IGF Type 1 - metabolism | Chondrocytes - cytology | Insulin-Like Growth Factor I - pharmacology | Apoptosis - drug effects | Humans | Protective Agents - metabolism | Extracellular Matrix - metabolism | Osteoarthritis - drug therapy | Osteoarthritis - physiopathology | Interleukin-1 - biosynthesis | Chondrocytes - drug effects | Isoquinolines - pharmacology | Protective Agents - pharmacology | Collagen - biosynthesis | Chondrocytes - metabolism | Isoquinolines - metabolism | Interleukin-1 - metabolism | Benzodiazepinones - pharmacology | Cells, Cultured | Proteoglycans - metabolism | Cartilage - metabolism | Interleukin-1 - pharmacology | Proteoglycans - pharmacology | Signal Transduction - drug effects | Mitogen-Activated Protein Kinases - pharmacology | Receptors, GABA-A - physiology | Convulsants - pharmacology | Receptors, GABA-A - metabolism | Insulin-Like Growth Factor I - metabolism | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Rheumatoid factor | Interleukins | Collagen | Benzodiazepines
Journal Article
The Journal of Immunology, ISSN 0022-1767, 05/2005, Volume 174, Issue 9, pp. 5789 - 5795
The adipokine resistin is suggested to be an important link between obesity and insulin resistance. In the present study, we assessed the impact of resistin as... 
RHEUMATOID-ARTHRITIS | TRIAL | IN-VITRO | PROTEIN | CONCOMITANT METHOTREXATE | INSULIN-RESISTANCE | GENE-EXPRESSION | MONOCLONAL-ANTIBODY | IMMUNOLOGY | ANTITUMOR NECROSIS FACTOR | FACTOR-ALPHA | Hormones, Ectopic - administration & dosage | Inflammation Mediators - administration & dosage | Interleukin-1 - genetics | Intracellular Fluid - immunology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Synovial Membrane - immunology | Synovial Membrane - pathology | Male | NF-kappa B - metabolism | Arthritis, Experimental - metabolism | Interleukin-1 - biosynthesis | Arthritis, Experimental - pathology | Arthritis, Rheumatoid - metabolism | Signal Transduction - immunology | Aged, 80 and over | Adult | Female | Inflammation Mediators - physiology | Adipocytes - immunology | Intracellular Fluid - metabolism | Resistin | Interleukin-6 - genetics | Cells, Cultured | Arthritis, Experimental - immunology | Hormones, Ectopic - physiology | Recombinant Proteins - pharmacology | Synovial Membrane - metabolism | Recombinant Proteins - administration & dosage | Arthritis, Rheumatoid - pathology | Animals | Adipocytes - metabolism | Adolescent | Injections, Intra-Articular | NF-kappa B - physiology | Interleukin-6 - biosynthesis | Aged | Mice | Arthritis, Rheumatoid - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Recombinant Proteins/administration & dosage/pharmacology | Intracellular Fluid/immunology/metabolism | Arthritis | MEDICIN OCH HÄLSOVETENSKAP | Hormones | Experimental/immunology/metabolism/pathology | Inflammation Mediators/administration & dosage/physiology | 80 and over | Interleukin-6/biosynthesis/genetics | Tumor Necrosis Factor-alpha/biosynthesis/genetics | Ectopic/administration & dosage/physiology | Cultured | Adipocytes/immunology/metabolism | Interleukin-1/biosynthesis/genetics | Rheumatoid/immunology/metabolism/pathology | MEDICAL AND HEALTH SCIENCES | Synovial Membrane/immunology/metabolism/pathology | Cells | Intra-Articular | NF-kappa B/metabolism/physiology | Injections | Signal Transduction/immunology
Journal Article
Circulation, ISSN 0009-7322, 11/2004, Volume 110, Issue 18, pp. 2903 - 2909
Background - During systemic inflammation, activation of vascular endothelium by proinflammatory cytokines leads to hypotension, microvascular thrombosis, and... 
Inflammation | Interleukins | Genes | Cell adhesion molecules | Endothelium | endothelium | cell adhesion molecules | CARDIAC & CARDIOVASCULAR SYSTEMS | genes | MECHANISMS | PROINFLAMMATORY CYTOKINES | KAPPA-B | interleukins | FACTOR-ALPHA | ADHESION MOLECULE-1 | TUMOR-NECROSIS-FACTOR | SYNTHASE MESSENGER-RNA | inflammation | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | TRANSCRIPTION FACTOR | SEVERE SEPSIS | Transcription, Genetic - drug effects | Cell Adhesion Molecules - genetics | Receptors, Thrombin - biosynthesis | Protein C - physiology | Blood Coagulation Factors - biosynthesis | Vasculitis - physiopathology | Humans | Gene Expression Profiling | Nitric Oxide Synthase - genetics | Biopterin - analogs & derivatives | Protein C - pharmacology | RNA, Messenger - biosynthesis | Vasculitis - genetics | Nitric Oxide Synthase - biosynthesis | Receptors, Cell Surface - biosynthesis | Cytokines - genetics | Receptor, PAR-2 - genetics | Cells, Cultured - drug effects | Receptor, PAR-2 - biosynthesis | Cell Adhesion Molecules - biosynthesis | RNA, Messenger - genetics | Cytokines - secretion | Nitric Oxide Synthase Type III | Biopterin - biosynthesis | Transcription Factors - biosynthesis | Blood Coagulation Factors - genetics | Recombinant Proteins - pharmacology | Transcription Factors - genetics | Receptor, PAR-1 - genetics | Interleukin-1 - pharmacology | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Receptor, PAR-1 - biosynthesis | Receptors, Thrombin - genetics | NF-kappa B - genetics | Coronary Vessels - cytology | NF-kappa B - biosynthesis | Cytokines - biosynthesis | Interferon-gamma - pharmacology | Protein C - genetics | Receptors, Cell Surface - genetics
Journal Article
The Journal of Immunology, ISSN 0022-1767, 06/2002, Volume 168, Issue 12, pp. 6199 - 6207
Dendritic cells (DC) derived from plasmacytoid precursors depend on IL-3 for survival and proliferation in culture, and they induce preferentially Th2... 
COLONY-STIMULATING FACTOR | HUMAN PERIPHERAL-BLOOD | GM-CSF | IMMUNE-RESPONSES | MAST-CELLS | DOWN-REGULATION | RECEPTOR | IMMUNOLOGY | AUTOIMMUNE-DISEASE | CLASS-II COMPARTMENT | MHC CLASS-II | Interleukin-3 Receptor alpha Subunit | Coculture Techniques | Dendritic Cells - immunology | Humans | Membrane Glycoproteins - biosynthesis | Monocytes - immunology | Th2 Cells - immunology | Interleukin-1 - biosynthesis | Lymphocyte Culture Test, Mixed | RNA, Messenger - biosynthesis | Toll-Like Receptors | Interleukin-4 - biosynthesis | Interleukin-4 - pharmacology | Endocytosis - immunology | Interleukin-12 - biosynthesis | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - biosynthesis | Adjuvants, Immunologic - physiology | Antigens, CD1 - biosynthesis | T-Lymphocytes, Helper-Inducer - metabolism | Cell Differentiation - immunology | Up-Regulation - immunology | Receptors, Interleukin-3 - biosynthesis | Mice | Receptors, Cell Surface - genetics | CD40 Ligand - physiology | Dose-Response Relationship, Immunologic | Monocytes - cytology | Monocytes - metabolism | Interleukin-5 - biosynthesis | Interleukin-3 - pharmacology | Cell Size - immunology | T-Lymphocytes, Helper-Inducer - immunology | Leukocyte Count | Receptors, Cell Surface - biosynthesis | Tumor Cells, Cultured | Dendritic Cells - metabolism | Drosophila Proteins | Cells, Cultured | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Immunophenotyping | Th2 Cells - metabolism | Membrane Glycoproteins - genetics | Animals | Down-Regulation - immunology | Interleukin-10 - biosynthesis | Dendritic Cells - cytology | Cytokines - biosynthesis | Drug Combinations | Interferon-gamma - biosynthesis
Journal Article