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The Journal of immunology (1950), ISSN 1550-6606, 01/2004, Volume 172, Issue 1, pp. 567 - 576
The signaling mechanism by which the anti-inflammatory cytokine IL-10 mediates suppression of proinflammatory cytokine synthesis remains largely unknown.... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Protein Binding - genetics | Protein Biosynthesis | Interleukin-6 - antagonists & inhibitors | Humans | Tumor Necrosis Factor-alpha - genetics | Immunoglobulins - genetics | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Repressor Proteins - antagonists & inhibitors | Antigens, CD - metabolism | Trans-Activators - physiology | Protein Tyrosine Phosphatases - antagonists & inhibitors | RNA, Messenger - biosynthesis | Protein Tyrosine Phosphatases - genetics | Inflammation Mediators - physiology | Glycoproteins - genetics | DNA-Binding Proteins - physiology | Protein Tyrosine Phosphatases - biosynthesis | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction - genetics | DNA - metabolism | Down-Regulation - genetics | Macrophages - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 2 | Repressor Proteins - biosynthesis | Up-Regulation - immunology | Interleukin-10 - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Adenoviruses, Human - genetics | Interleukin-10 - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Phosphorylation | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Antigens, CD - biosynthesis | Receptors, Cell Surface | Receptors, IgG - biosynthesis | Receptors, IgG - antagonists & inhibitors | Interleukin-10 - physiology | Signal Transduction - immunology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Signaling Lymphocytic Activation Molecule Family Member 1 | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | RNA, Messenger - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type II | Trans-Activators - genetics | Inflammation Mediators - antagonists & inhibitors | Trans-Activators - biosynthesis | Immunoglobulins - biosynthesis | Macrophages - immunology | Inflammation Mediators - immunology | Receptors, Tumor Necrosis Factor - metabolism | Immune Sera - pharmacology | Proteins - physiology | Cells, Cultured | Glycoproteins - antagonists & inhibitors | Histocompatibility Antigens Class II - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - antagonists & inhibitors | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Up-Regulation - genetics | DNA-Binding Proteins - genetics | DNA - antagonists & inhibitors | Glycoproteins - biosynthesis | Suppressor of Cytokine Signaling 3 Protein | Down-Regulation - immunology | Interleukin-6 - biosynthesis | Receptors, Tumor Necrosis Factor - biosynthesis | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Genetic Vectors | DNA-Binding Proteins - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | SOCS-3 protein | Index Medicus | Abridged Index Medicus
Journal Article
European spine journal, ISSN 0940-6719, 3/2012, Volume 21, Issue 3, pp. 537 - 545
The symptoms of lumbar disc herniation, such as low back pain and sciatica, have been associated with local release of cytokines following the inflammatory... 
Surgical Orthopedics | Medicine & Public Health | Lumbar disc herniation | Radicular pain | Neurosurgery | Dorsal root ganglion | Hyperalgesia | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Orthopedics | Science & Technology | Interleukin-1beta - physiology | Interleukin-6 - antagonists & inhibitors | Rats, Wistar | Chemokine CXCL1 - antagonists & inhibitors | Hyperalgesia - prevention & control | Rats | Male | Antibodies - physiology | Cytokines - physiology | Interleukin-10 - physiology | Hyperalgesia - pathology | Hyperalgesia - immunology | Animals | Time Factors | Chemokine CXCL1 - physiology | Interleukin-10 - antagonists & inhibitors | Interleukin-6 - physiology | Tumor Necrosis Factor-alpha - physiology | Inflammation Mediators - antagonists & inhibitors | Cytokines - antagonists & inhibitors | Intervertebral Disc - pathology | Inflammation Mediators - physiology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Interleukin-1beta - antagonists & inhibitors | Viral antibodies | Cytokines | Animal behavior | Analysis | Antibodies | Inflammation | Backache | Chronic pain | Index Medicus | Spinal nerves | Animal models | Pain perception | Low back pain | Spine (lumbar) | Mechanical properties | Intervertebral discs | Dorsal root ganglia | nucleus pulposus | Interleukin 1 | Tumor necrosis factor- alpha | Original
Journal Article
European journal of immunology, ISSN 0014-2980, 02/2011, Volume 41, Issue 2, pp. 413 - 424
During infection, TLR agonists are released and trigger mature as well as differentiating innate immune cells. Early encounter with TLR agonists (R848; LPS)... 
Tolerance | STAT‐3 | TLR | DC | PD‐L1 | PD-L1 | STAT-3 | Life Sciences & Biomedicine | Immunology | Science & Technology | T-Lymphocyte Subsets - immunology | Antigen-Presenting Cells - cytology | Immune Tolerance - physiology | Monocytes - cytology | Dendritic Cells - immunology | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | STAT Transcription Factors - metabolism | Monocytes - immunology | Antigens, CD - metabolism | Lipopolysaccharide Receptors - metabolism | Lymphocyte Culture Test, Mixed | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Interleukin-4 - pharmacology | Interleukin-10 - metabolism | Histocompatibility Antigens Class II - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Antigen-Presenting Cells - metabolism | B7-H1 Antigen | Gene Expression Regulation - immunology | Antigen-Presenting Cells - drug effects | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Imidazoles - pharmacology | Antigen-Presenting Cells - immunology | Toll-Like Receptors - agonists | Monocytes - drug effects | Cell Differentiation - immunology | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Differentiation - drug effects | Antigens, CD1 - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Dendritic Cells - cytology | Protein Kinase Inhibitors - pharmacology | STAT Transcription Factors - antagonists & inhibitors | STAT3 Transcription Factor - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - metabolism | Medical research | Rodents | Index Medicus
Journal Article
Cell communication and signaling, ISSN 1478-811X, 12/2014, Volume 12, Issue 1, pp. 1 - 13
Journal Article