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Archives of Oral Biology, ISSN 0003-9969, 2013, Volume 58, Issue 8, pp. 912 - 929
Abstract Objective An analysis of the genes involved in both osteoporosis and modifications of the jawbone, through text mining, using a web search tool, of... 
Advanced Basic Science | Dentistry | Bone resorption | Osteoporosis | Mandible | Bone remodelling | Genomics | Bone repair | Maxilla | Computational biology | BONE-MINERAL DENSITY | HUMAN OSTEOBLASTS | TOMOGRAPHIC FOLLOW-UP | SECONDARY HYPERPARATHYROIDISM | FACTOR-I | DENTISTRY, ORAL SURGERY & MEDICINE | CELL-CYCLE | HUMAN T-LYMPHOCYTES | EXPRESSION | ALVEOLAR RIDGE AUGMENTATION | GENOME-WIDE ASSOCIATION | Osteopontin - genetics | Bone Regeneration - genetics | Bone Morphogenetic Protein 7 - genetics | Genes, jun - genetics | Humans | Data Mining | Insulin-Like Growth Factor I - genetics | Interleukin-1beta - genetics | NF-kappa B p50 Subunit - genetics | Vascular Endothelial Growth Factor A - genetics | Collagen Type I - genetics | Bone Resorption - genetics | Jaw Diseases - genetics | Matrix Metalloproteinase 9 - genetics | Osteoporosis - genetics | Multigene Family - genetics | Bone Morphogenetic Protein 2 - genetics | Interleukin-6 - genetics | Computational Biology | Transforming Growth Factor beta1 - genetics | beta Catenin - genetics | RANK Ligand - genetics | Intercellular Adhesion Molecule-1 - genetics | Interleukin-10 - genetics | Bone Remodeling - genetics | Core Binding Factor Alpha 1 Subunit - genetics | Medical colleges | Analysis | Genes | Online searching | Genetic research | Internet/Web search services | Database searching | Data mining
Journal Article
PLoS genetics, ISSN 1553-7404, 2013, Volume 9, Issue 10, p. e1003880
Journal Article
Journal of immunology research, ISSN 2314-7156, 2014, Volume 2014, pp. 1 - 19
Acute inflammation is a response to an alteration induced by a pathogen or a physical or chemical insult, which functions to eliminate the source of the damage... 
EPITHELIAL-MESENCHYMAL TRANSITION | ACTIVATED PROTEIN-KINASE | CARCINOMA-CELL-LINES | TGF-BETA | NITRIC-OXIDE SYNTHASE | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | TNF-ALPHA | IMMUNOLOGY | ENDOTHELIAL GROWTH-FACTOR | PRIMARY SCLEROSING CHOLANGITIS | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Oxidative Stress | Colitis - genetics | Colorectal Neoplasms - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | Colitis - complications | Colitis - pathology | Cholangiocarcinoma - metabolism | Inflammation - complications | Colorectal Neoplasms - etiology | Inflammation - metabolism | Bile Ducts, Intrahepatic - metabolism | Tumor Microenvironment - genetics | Interleukin-10 - metabolism | Bile Duct Neoplasms - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Bile Ducts, Intrahepatic - pathology | Gene Expression | Bile Duct Neoplasms - metabolism | Interleukin-6 - genetics | Carcinogenesis - genetics | Cholangiocarcinoma - pathology | Transforming Growth Factor beta - genetics | Interleukin-10 - genetics | Colitis - metabolism | Cholangiocarcinoma - genetics | Inflammation - genetics | Bile Duct Neoplasms - pathology | Colorectal Neoplasms - pathology | Chronic Disease | Transforming Growth Factor beta - metabolism | Free radicals | Cytokines | Disease | Mortality | Clinical trials | Metastasis | Cancer therapies | Proteins | Angiogenesis | Medical prognosis | Tumorigenesis | Nonsteroidal anti-inflammatory drugs | Chemokines | Immune system | Tumors | Cancer | Apoptosis | Review
Journal Article
Annals of the New York Academy of Sciences, ISSN 0077-8923, 05/2006, Volume 1067, Issue 1, pp. 252 - 263
:  Aging is due to a complex interaction of genetic, epigenetic, and environmental factors, but a strong genetic component appears to have an impact on survival to extreme ages... 
CETP | TLR-4 | apolipoproteins | insulin/IGF-1 | PON1 | longevity genes | IL-6 | IL-10 | PPARγ | IL-1 cluster | p53, p66shc | TNF-α | TGF-β | insulin/IGF‐1 | TNF‐α | IL‐6 | TGF‐β | IL‐10 | TLR‐4 | IL‐1 cluster | Apolipoproteins | Longevity genes | p53, p66 | LIFE-SPAN | RISK-FACTORS | AGED PEOPLE | TNF-alpha | MULTIDISCIPLINARY SCIENCES | SPORADIC ALZHEIMERS-DISEASE | PROMOTER POLYMORPHISM | NECROSIS-FACTOR-ALPHA | PPAR gamma | p53, p66(shc) | OLDER PERSONS | TGF-beta | HEALTHY CENTENARIANS | ITALIAN CENTENARIANS | GROWTH-FACTOR-I | Interleukin-1 - genetics | Multigene Family | Oxidative Stress | Humans | Tumor Necrosis Factor-alpha - genetics | Insulin-Like Growth Factor I - genetics | Tumor Suppressor Protein p53 - genetics | Aging - genetics | Apolipoproteins - genetics | Aged, 80 and over | Lipid Metabolism - genetics | Insulin - genetics | PPAR gamma - genetics | Shc Signaling Adaptor Proteins | Glycoproteins - genetics | Interleukin-6 - genetics | Signal Transduction | Toll-Like Receptor 4 - genetics | Longevity - genetics | Aryldialkylphosphatase - genetics | Polymorphism, Genetic | Carrier Proteins - genetics | Src Homology 2 Domain-Containing, Transforming Protein 1 | Aging - physiology | Interleukin-10 - genetics | Adaptor Proteins, Signal Transducing - genetics | Inflammation - genetics | Cholesterol Ester Transfer Proteins | Longevity - physiology
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2010, Volume 126, Issue 2, pp. 338 - 346.e10
Background Regulatory T–cell dysfunction is associated with development of the complex genetic conditions atopy and asthma... 
Allergy and Immunology | T regulatory cells | birth cohort | IgE | atopy | interaction | asthma | MDR | polymorphism | SERUM IGE | AIRWAY INFLAMMATION | UMCG Approved | CHILDREN | MULTIFACTOR-DIMENSIONALITY REDUCTION | PROMOTER POLYMORPHISMS | TRANSFORMING GROWTH-FACTOR-BETA-1 | ALLERGIC DISEASE | ASSOCIATION | IMMUNOGLOBULIN-E | Interaction | Birth cohort | Asthma | Atopy | Polymorphism | IMMUNOLOGY | ALLERGY | Forkhead Transcription Factors - immunology | Allergens - adverse effects | Receptors, Transforming Growth Factor beta - genetics | Prospective Studies | Receptors, Transforming Growth Factor beta - immunology | Toll-Like Receptor 2 - genetics | Humans | Genetic Loci - genetics | Child, Preschool | Infant | Male | Genetic Loci - immunology | Heme Oxygenase-1 - genetics | Transforming Growth Factor beta1 - immunology | Immunoglobulin E - immunology | Interleukin-2 - immunology | Interleukin-2 Receptor alpha Subunit - genetics | Asthma - immunology | Female | Receptors, Interleukin-6 - immunology | T-Lymphocytes - pathology | Child | Interleukin-2 Receptor alpha Subunit - immunology | Genetic Predisposition to Disease | Interleukin-6 - genetics | Receptors, Interleukin-6 - genetics | Protein-Serine-Threonine Kinases - genetics | Logistic Models | Transforming Growth Factor beta1 - genetics | Forkhead Transcription Factors - genetics | Asthma - genetics | Polymorphism, Genetic | Interleukin-2 - genetics | Interleukin-10 - genetics | Epistasis, Genetic - immunology | Heme Oxygenase-1 - immunology | Interleukin-6 - immunology | Toll-Like Receptor 2 - immunology | Protein-Serine-Threonine Kinases - immunology | T-Lymphocytes - immunology | Models, Genetic | Asthma - pathology | Interleukin-10 - immunology | Allergy | Genetic disorders | Child development | Asthma in children | Immunoglobulin E | Family medicine | Children | Transforming growth factors | T cells | Health aspects | Allergic reaction | Public health | Studies | Food allergies | Genes | Statistical methods | Allergies | Deoxyribonucleic acid--DNA | Milk
Journal Article
PLoS medicine, ISSN 1549-1676, 2007, Volume 4, Issue 4, pp. e124 - 689
In multiple sclerosis, inflammation can successfully be prevented, while promoting repair is still a major challenge. Microglial cells, the resident phagocytes... 
MEDICINE, GENERAL & INTERNAL | Genetic Therapy | Neurons - pathology | Nitric Oxide Synthase Type II - biosynthesis | Receptors, Immunologic - physiology | Humans | Myeloid Cells - physiology | Cells, Cultured - transplantation | Genetic Vectors - therapeutic use | Multiple Sclerosis | Membrane Glycoproteins - physiology | Flow Cytometry | Microglia - physiology | Female | Lentivirus - genetics | Myeloid Cells - drug effects | Monocytes - physiology | Cytokines - genetics | Genes, Reporter | Disease Models, Animal | Encephalomyelitis, Autoimmune, Experimental - surgery | Promoter Regions, Genetic | Encephalomyelitis, Autoimmune, Experimental - pathology | Transduction, Genetic | Mice, Inbred C57BL | Gene Expression Regulation | Genes, Synthetic | Cells, Cultured - physiology | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Hematopoietic Stem Cell Transplantation | Genetic Vectors - genetics | Membrane Glycoproteins - genetics | Cell Lineage | Encephalomyelitis, Autoimmune, Experimental - therapy | Animals | Nitric Oxide Synthase Type II - genetics | Cell Differentiation - drug effects | Interleukin-10 - genetics | Interleukin-10 - biosynthesis | Myeloid Cells - transplantation | Mice | Receptors, Immunologic - genetics | Cytokines - biosynthesis | Phagocytosis | Apoptosis | Cell Movement | Multiple sclerosis | Biochemical genetics | Inflammation | Research | Neuroglia | Central nervous system | Neurological Disorders | Neurology | Neuroscience | Immunology | Immunology and Allergy
Journal Article
Aging cell, ISSN 1474-9718, 08/2017, Volume 16, Issue 4, pp. 859 - 869
Summary During aging, decreases in energy expenditure and locomotor activity lead to body weight and fat gain. Aging is also associated with decreases in... 
frailty | Sarcopenia | wasting | growth hormone | growth hormone secretagogue receptor | inflammation | ACTIVATED PROTEIN-KINASE | FIBER TYPE | ELDERLY-WOMEN | AGE-RELATED LOSS | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | GROWTH-HORMONE SECRETAGOGUE | TISSUE DISTRIBUTION | SARCOPENIC OBESITY | BODY-MASS | PHYSICAL-ACTIVITY | MICE | Tumor Necrosis Factor-alpha - metabolism | Sarcopenia - genetics | AMP-Activated Protein Kinases - metabolism | Body Weight | Interleukin-12 - genetics | Tumor Necrosis Factor-alpha - genetics | Male | Insulin-Like Growth Factor I - genetics | Interferon-gamma - metabolism | Interleukin-1beta - genetics | Obesity - genetics | Receptors, Ghrelin - genetics | Interleukin-1beta - metabolism | Interleukin-10 - metabolism | Receptors, Ghrelin - metabolism | Interferon-gamma - genetics | Energy Metabolism - genetics | Interleukin-6 - metabolism | Sarcopenia - metabolism | Interleukin-6 - genetics | Growth Hormone - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Ghrelin - deficiency | Ghrelin - genetics | Interleukin-12 - metabolism | Longevity - genetics | Eating - genetics | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Growth Hormone - genetics | Animals | Sarcopenia - prevention & control | Interleukin-10 - genetics | Obesity - prevention & control | Sarcopenia - pathology | Mice | AMP-Activated Protein Kinases - genetics | Insulin-Like Growth Factor I - metabolism | Prevention | Obesity | Somatotropin | Adenylic acid | Protein kinases | Body weight | AMP | Energy expenditure | Fatigue | Longevity | Kinases | Body weight gain | Protein kinase | Adenosine kinase | Ghrelin | Food intake | Rodents | Aging | Endurance | Muscular strength | Locomotor activity | Original
Journal Article