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Nature Medicine, ISSN 1078-8956, 04/2008, Volume 14, Issue 4, pp. 421 - 428
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2016, Volume 126, Issue 6, pp. 2049 - 2063
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2015, Volume 212, Issue 5, pp. 619 - 631
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2014, Volume 289, Issue 24, pp. 16773 - 16789
Lipocalin-2 (LCN2) plays an important role in cellular processes as diverse as cell growth, migration/invasion, differentiation, and death/survival.... 
PATHOGENESIS | MULTIPLE-SCLEROSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | NEURON DEATH | REACTIVE ASTROCYTES | CENTRAL-NERVOUS-SYSTEM | GELATINASE-ASSOCIATED LIPOCALIN | CHEMOKINE RECEPTORS | EXPRESSION | CD8(+) T-CELLS | Oncogene Proteins - genetics | Spinal Cord - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Acute-Phase Proteins - genetics | Interferon-gamma - metabolism | Myelin Sheath - metabolism | Matrix Metalloproteinase 9 - metabolism | T-Lymphocytes - metabolism | Lipocalins - genetics | Matrix Metalloproteinase 9 - genetics | Spinal Cord - pathology | Gene Deletion | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Interferon-gamma - genetics | Neutrophils - metabolism | Dendritic Cells - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Lipocalins - metabolism | Acute-Phase Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Oncogene Proteins - metabolism | Interleukin-17 - genetics | Lymphoid Tissue - metabolism | Receptors, Cell Surface - metabolism | Interleukin-17 - metabolism | Lymphoid Tissue - pathology | Animals | Lipocalin-2 | Myelin Sheath - genetics | Mice | Astrocytes - metabolism | Receptors, Cell Surface - genetics | Index Medicus | Multiple Sclerosis | Autoimmune Disease | Central Nervous System | Neurobiology | EAE | LCN2 | Neuroinflammation | Astrocyte | Microglia
Journal Article
Journal Article
FASEB Journal, ISSN 0892-6638, 2017, Volume 31, Issue 12, pp. 5543 - 5556
Interleukin 17A (IL-17A) and complement (C') activation have each been implicated in the pathogenesis of idiopathic pulmonary fibrosis (IPF). We have reported... 
C3a | DAF | ER stress | C5b-9 | C5a | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | ENDOPLASMIC-RETICULUM STRESS | TRANSPLANTATION | CELL BIOLOGY | CIRCULATING KL-6 | EPITHELIAL-CELLS | INTERLEUKIN-17 | BIOLOGY | INFECTION | IDIOPATHIC PULMONARY-FIBROSIS | OBLITERATIVE BRONCHIOLITIS | ACUTE EXACERBATION | Caspase 7 - metabolism | Lung Diseases - metabolism | Humans | Middle Aged | Caspase 3 - metabolism | Male | Fibrosis - metabolism | Female | Real-Time Polymerase Chain Reaction | Complement Activation - drug effects | Hemolysis - genetics | Hemolysis - physiology | Fibrosis - genetics | Enzyme-Linked Immunosorbent Assay | Cells, Cultured | Interleukin-17 - genetics | Interleukin-17 - deficiency | Blotting, Western | Interleukin-17 - metabolism | Lung Diseases - genetics | Animals | Fluorescent Antibody Technique | Bleomycin - pharmacology | Aged | Mice | G protein-coupled receptors | Pathogenesis | Epithelial cells | Complement component C5a | Lung | Bronchopneumonia | Interleukin | Activation | Complement | mRNA | Caspase-3 | Respiratory tract | Receptors | Bleomycin | Rodents | Transcription activation | Interleukin 1 | Bronchiolitis obliterans | Alveoli | Deposition | Interleukin 8 | Injuries | Cytokines | RNA-mediated interference | Lung diseases | Caspase | Gene silencing | Signaling | Complement activation | DNA microarrays | Lungs | Decay-accelerating factor | Complement component C3 | Collagen | Fibrosis | Neutralization | Apoptosis | Index Medicus | Research
Journal Article
Immunity, ISSN 1074-7613, 08/2013, Volume 39, Issue 2, pp. 372 - 385
Endogenous tryptophan (Trp) metabolites have an important role in mammalian gut immune homeostasis, yet the potential contribution of Trp metabolites from... 
INTESTINAL INFLAMMATION | LACTOBACILLUS-PLANTARUM | CANDIDA-ALBICANS | GASTROINTESTINAL-TRACT | GUT | REGULATORY T-CELLS | MICE | COLONIZATION | JOHNSONII STRAIN NCC533 | INNATE LYMPHOID-CELLS | IMMUNOLOGY | Indoleamine-Pyrrole 2,3,-Dioxygenase - genetics | Lactobacillus reuteri - metabolism | Receptors, Aryl Hydrocarbon - deficiency | Toll-Like Receptor 2 - genetics | Tryptophan - chemistry | Indoleamine-Pyrrole 2,3,-Dioxygenase - deficiency | Lactobacillus reuteri - immunology | Tryptophan - metabolism | Interleukins - metabolism | Gastrointestinal Tract - immunology | Toll-Like Receptor 2 - deficiency | Indoles - metabolism | Receptors, Aryl Hydrocarbon - metabolism | Female | Candidiasis - immunology | Basic Helix-Loop-Helix Transcription Factors - deficiency | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Inbred C57BL | Myeloid Differentiation Factor 88 - genetics | Receptors, Aryl Hydrocarbon - genetics | Gastrointestinal Tract - microbiology | Interleukin-17 - genetics | Interleukin-17 - deficiency | Candida albicans - immunology | Mice, SCID | Gastrointestinal Tract - metabolism | Mice, Inbred C3H | Mice, Knockout | Probiotics | Animals | Myeloid Differentiation Factor 88 - deficiency | Energy Metabolism | Lactobacillus reuteri - growth & development | Mice | Mice, Inbred BALB C | Metagenome | Tryptophan | Metabolites | Interleukins | Aldehydes | Medical research | Stomach | Colleges & universities | Homeostasis | Ligands | Software | Infections | Metabolism | Experiments | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 151, Issue 6, pp. 1100 - 1104
Interleukin 10 receptor (IL10R)−deficient mice develop spontaneous colitis and, similarly, patients with loss-of-function mutations in IL10R develop severe... 
Gastroenterology and Hepatology | VEOIBD | Ubiquitination | Treg Cell | Caspase-1 | CELLS | ACTIVATION | MACROPHAGES | SPONTANEOUS COLITIS | GASTROENTEROLOGY & HEPATOLOGY | BOWEL-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Inflammasomes - metabolism | Colitis - genetics | Humans | Caspase 8 - metabolism | Child, Preschool | Interferon-gamma - metabolism | Interleukin-1beta - genetics | Macrophages | Inflammatory Bowel Diseases - metabolism | Adenosine Triphosphate - pharmacology | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Inflammasomes - drug effects | Interleukin-1beta - metabolism | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Interleukin-10 - metabolism | Adult | CD4-Positive T-Lymphocytes | Colitis - immunology | Antirheumatic Agents - therapeutic use | Inflammatory Bowel Diseases - drug therapy | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Cells, Cultured | Immunity, Innate | Homeodomain Proteins - genetics | Mice, Knockout | Gene Expression Regulation - drug effects | Interleukin-17 - metabolism | Animals | Interleukin 1 Receptor Antagonist Protein - therapeutic use | Interleukin-10 Receptor alpha Subunit - genetics | Colitis - metabolism | Lipopolysaccharides - pharmacology | Protein Biosynthesis - drug effects | Mice | Mutation | Receptors, Interleukin-10 - deficiency | Interleukin-10 - pharmacology | Receptors, Interleukin-10 - genetics | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 137, Issue 4, pp. 1189 - 1196.e2
Background Data on patients affected by chronic mucocutaneous candidiasis underscore the preponderant role of IL-17 receptor A (IL-17RA) in preserving... 
Allergy and Immunology | CECR1 | Adenosine deaminase 2 | whole-genome analyses | vasculitis | immunodeficiency | chronic mucocutaneous candidiasis | chronic inflammation | IL17RA | INTERLEUKIN-1-BETA | CANDIDIASIS | ADA2 | IMMUNOLOGY | T-HELPER-CELLS | FAMILY | ALLERGY | DIFFERENTIATION | VASCULOPATHY | TH17 CELLS | Sequence Deletion | Humans | Receptors, Interleukin-17 - immunology | Child, Preschool | Receptors, Interleukin-17 - genetics | Vasculitis - complications | Vasculitis - immunology | Receptors, Interleukin-17 - deficiency | Inflammation - complications | Vasculitis - genetics | Fatal Outcome | Female | Intercellular Signaling Peptides and Proteins - deficiency | Adenosine Deaminase - deficiency | Child | Siblings | Adenosine Deaminase - genetics | Candidiasis, Chronic Mucocutaneous - genetics | Candidiasis, Chronic Mucocutaneous - immunology | Intercellular Signaling Peptides and Proteins - genetics | Inflammation - immunology | Comparative Genomic Hybridization | Adolescent | Candidiasis, Chronic Mucocutaneous - complications | Inflammation - genetics | Chronic Disease | Adenosine Deaminase - immunology | Intercellular Signaling Peptides and Proteins - immunology | Physiological aspects | Health aspects | Medical genetics | Diseases | Genomics | Analysis | Adenosine | Genealogy | Bacterial infections | Cytokines | Laboratories | Cloning | Neutrophils | Infections | Inflammation | Patients | Dermatitis | Blood | Granulocytes | Lymphocytes | Biopsy | Bone marrow | Mutation | Age | Immune system | Neutropenia | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2013, Volume 190, Issue 3, pp. 997 - 1007
Annexin A1 (AnxA1) is recognized as an endogenous anti-inflammatory molecule. However, its effects on the adaptive immune response and, in particular, on T... 
PERIPHERAL-BLOOD LEUKOCYTES | COLLAGEN-INDUCED ARTHRITIS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | IN-VITRO | ACTIVATION | IMMUNE-RESPONSES | CONTACT HYPERSENSITIVITY | HOST-DEFENSE | IL-17-DEFICIENT MICE | IMMUNOLOGY | EXPRESSION | Annexin A1 - deficiency | Hypersensitivity, Delayed - pathology | Dermatitis, Allergic Contact - immunology | Inflammation - pathology | Lymphokines - secretion | Ovalbumin - immunology | Peptide Fragments - toxicity | Oxazolone - toxicity | Dermatitis, Allergic Contact - pathology | Hypersensitivity, Delayed - immunology | Enzyme Activation - immunology | Arthritis, Experimental - pathology | CD4-Positive T-Lymphocytes - immunology | Oxazolone - immunology | Signal Transduction - immunology | Ovalbumin - toxicity | Lymphokines - genetics | Annexin A1 - immunology | Peptide Fragments - immunology | Lymphokines - biosynthesis | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Specific Pathogen-Free Organisms | Gene Expression Regulation - immunology | Lymphocyte Activation | Mice, Inbred C57BL | Neutrophils - immunology | Interleukin-17 - genetics | Mice, Transgenic | Arthritis, Experimental - immunology | Inflammation - immunology | Interleukin-17 - biosynthesis | Cell Adhesion | Mice, Knockout | Animals | CD4-Positive T-Lymphocytes - secretion | Nuclear Receptor Subfamily 1, Group F, Member 3 - biosynthesis | Mice | CD8-Positive T-Lymphocytes - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2016, Volume 113, Issue 51, pp. E8277 - E8285
Journal Article