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PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0175153
IL-1 beta is a potent player in cutaneous inflammation and central for the development of a Th17 micro-milieu in autoinflammatory diseases including psoriasis.... 
VITAMIN-D | SYSTEMIC-LUPUS-ERYTHEMATOSUS | INTERFERON-GAMMA | MULTIDISCIPLINARY SCIENCES | PSORIASIN S100A7 | INFLAMMATORY CASPASES | KERATINOCYTES | 1,25-DIHYDROXYVITAMIN D-3 | MOLECULAR-CLONING | EXPRESSION | T-CELLS | Caspases - immunology | Humans | Interleukin-17 - immunology | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | S100 Proteins - immunology | Psoriasis - genetics | S100 Proteins - genetics | Th17 Cells - drug effects | Transfection | Inflammasomes - drug effects | Psoriasis - pathology | Adaptor Proteins, Signal Transducing - immunology | Apoptosis Regulatory Proteins - genetics | Skin - pathology | Skin - immunology | Th17 Cells - pathology | Interleukin-1beta - secretion | Psoriasis - immunology | S100 Calcium Binding Protein A7 | Apoptosis Regulatory Proteins - immunology | DNA - immunology | Signal Transduction | Caspases - genetics | Cells, Cultured | Gene Expression Regulation | Interleukin-17 - genetics | Interleukin-1beta - immunology | Vitamin D - pharmacology | Keratinocytes - cytology | Keratinocytes - immunology | DNA - genetics | Interleukin-17 - antagonists & inhibitors | Keratinocytes - drug effects | Inflammasomes - immunology | Adaptor Proteins, Signal Transducing - genetics | Th17 Cells - immunology | Interferon-gamma - pharmacology | Interleukins | Physiological aspects | Development and progression | Skin diseases | Genetic aspects | Inflammation | Research | Cell culture | Transcription | Interleukin | Helper cells | Lymphocytes T | Inflammatory diseases | Vitamin D | Lymphocytes | Interleukin 1 | Lesions | Deoxyribonucleic acid--DNA | Psoriasis | Cytokines | Dermatology | Keratinocytes | Caspase | Epidermis | Hazards | Gene expression | Caspase-5 | Hospitals | Biopsy | Interferon | Autoimmune diseases | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Experimental Dermatology, ISSN 0906-6705, 02/2012, Volume 21, Issue 2, pp. 104 - 110
:  Atopic eczema and psoriasis are common skin diseases. While it is well established that the pathogenesis of these diseases varies, both are characterized by... 
keratinocytes | IL‐17 | atopic eczema | epidermal barrier | filaggrin | Keratinocytes | Epidermal barrier | Filaggrin | Atopic eczema | IL-17 | EPICUTANEOUS SENSITIZATION | OF-FUNCTION MUTATIONS | SKIN BARRIER FUNCTION | FUNCTION VARIANTS | POLYETHYLENE-GLYCOLS | CULTURED KERATINOCYTES | DERMATOLOGY | ATOPIC-DERMATITIS | SODIUM LAURYL SULFATE | TIGHT JUNCTION PROTEINS | TH17 CELLS | Cathepsin L - genetics | Cell Adhesion Molecules - genetics | Cadherins - metabolism | Gene Expression - drug effects | Oligonucleotide Array Sequence Analysis | Cytoskeletal Proteins - genetics | Gene Expression - genetics | Cathepsin B - genetics | Humans | Interleukin-17 - pharmacology | Calpain - genetics | Phosphoproteins - metabolism | Cell Differentiation - genetics | S100 Proteins - genetics | Intermediate Filament Proteins - genetics | Membrane Proteins - metabolism | Cadherins - genetics | Cell Line | Claudins - genetics | Membrane Proteins - genetics | Cells, Cultured | gamma Catenin - genetics | gamma Catenin - metabolism | Up-Regulation - genetics | Phosphoproteins - genetics | Down-Regulation - genetics | Zonula Occludens-1 Protein | Keratinocytes - drug effects | Keratinocytes - metabolism | Cathepsin D - genetics | Intermediate Filament Proteins - metabolism | Zonula Occludens-2 Protein | Enzymes | Messenger RNA | Psoriasis | Interleukins | Analysis | Genes | Genetic research | Genetic aspects | Skin
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2015, Volume 135, Issue 6, pp. 1476 - 1485.e7
Background Chronic rhinosinusitis (CRS) with nasal polyps (NPs) in Western populations is associated with TH 2 cytokine polarization. IL-25, an IL-17 family... 
Allergy and Immunology | sinusitis | allergy | animal models | IL-25 | Nasal polyp | SKIN BARRIER DYSFUNCTION | MAST-CELLS | CYTOKINE | INNATE LYMPHOID-CELLS | IMMUNOLOGY | ATOPIC-DERMATITIS | RESPONSES | INTERLEUKIN-17 FAMILY | INFLAMMATION | MOUSE MODEL | IN-VIVO | Eosinophil Cationic Protein - genetics | Eosinophils - drug effects | Humans | Middle Aged | Male | Receptors, Interleukin-17 - genetics | Eosinophils - immunology | Rhinitis - drug therapy | Eosinophil Cationic Protein - immunology | Transforming Growth Factor beta2 - immunology | Sinusitis - genetics | Gene Expression - immunology | Eosinophils - pathology | Disease Models, Animal | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Antibodies, Monoclonal - pharmacology | Neutrophils - drug effects | Interleukin-17 - genetics | Rhinitis - complications | Mice | Chronic Disease | Sinusitis - drug therapy | GATA3 Transcription Factor - genetics | Cell Adhesion Molecules - genetics | Gene Expression - drug effects | GATA3 Transcription Factor - immunology | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | T-Box Domain Proteins - immunology | Cell Adhesion Molecules - immunology | Case-Control Studies | Sinusitis - complications | Transforming Growth Factor beta1 - immunology | Sinusitis - immunology | Nasal Polyps - genetics | Adult | Female | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Neutrophils - pathology | Nasal Polyps - immunology | Rhinitis - immunology | Neutrophils - immunology | Transforming Growth Factor beta1 - genetics | Nasal Polyps - drug therapy | T-Box Domain Proteins - genetics | Rhinitis - genetics | Animals | Nasal Polyps - complications | Interleukin-17 - antagonists & inhibitors | Transforming Growth Factor beta2 - genetics | Medical colleges | Nurse practitioners | Nasal polyps | Atopic dermatitis | Health aspects | Proteins | Immunoglobulins | Pulmonary fibrosis | Disease | Laboratories | Rodents | Surgery | Family medical history | Endoscopy | Quality of life
Journal Article
International journal of cancer, ISSN 0020-7136, 2012, Volume 131, Issue 11, pp. 2553 - 2561
... cancers do not have a well‐defined genetic etiology; their genesis appears to be a result of chronic inflammation. 6 Supportive evidence showed that the risk... 
dextran sodium sulfate | estrogen receptor‐beta | colon cancer | azoxymethane | inflammatory bowel disease | estrogen receptor-beta | Estradiol - blood | Mucin-2 - genetics | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Colitis - genetics | Colorectal Neoplasms - genetics | Tumor Necrosis Factor-alpha - genetics | Colitis - pathology | NF-kappa B - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Estrogen Receptor beta - metabolism | Cell Differentiation - genetics | Inflammation - metabolism | Estrogen Receptor beta - genetics | Proliferating Cell Nuclear Antigen - genetics | Neoplasms - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Female | Interferon-gamma - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Mucin-2 - metabolism | Mucin-1 - metabolism | Interleukin-6 - genetics | Colon - pathology | Mice, Inbred C57BL | Caveolin 1 - genetics | Interleukin-17 - genetics | beta Catenin - metabolism | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Caveolin 1 - metabolism | Interleukin-17 - metabolism | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Colitis - metabolism | Inflammation - genetics | Mice | Colorectal Neoplasms - pathology | Neoplasms - pathology | Proliferating Cell Nuclear Antigen - metabolism | Mucin-1 - genetics | Nitric Oxide Synthase Type II - metabolism | Estrogen receptor-beta
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2012, Volume 122, Issue 11, pp. 3965 - 3976
Psoriasis is a chronic inflammatory disorder of the skin affecting approximately 2% of the world's population. Accumulating evidence has revealed that the... 
GENERALIZED PUSTULAR PSORIASIS | MEDICINE, RESEARCH & EXPERIMENTAL | INFLAMMATORY SKIN-DISEASE | FAMILY-MEMBERS | MOUSE MODEL | DELTA-T-CELLS | NF-KAPPA-B | AUTOINFLAMMATORY DISEASE | INTERLEUKIN-12/23 MONOCLONAL-ANTIBODY | CUTTING EDGE | TRANSGENIC MICE | Cell Communication - immunology | Interleukin-1 - genetics | Membrane Glycoproteins - metabolism | Dermatitis - immunology | Dendritic Cells - immunology | Receptors, Interleukin-1 - genetics | Interleukin-17 - immunology | Dendritic Cells - pathology | Interleukins - metabolism | Interleukin-23 - genetics | Interleukins - genetics | Psoriasis - genetics | Interleukin-23 - metabolism | Psoriasis - pathology | Interleukins - immunology | Toll-Like Receptor 7 - immunology | Interleukin-1 - immunology | Psoriasis - metabolism | Membrane Glycoproteins - immunology | Dendritic Cells - metabolism | Psoriasis - immunology | Toll-Like Receptor 7 - genetics | Interleukin-1 - metabolism | Toll-Like Receptor 7 - metabolism | Dermatitis - genetics | Receptors, Interleukin-1 - immunology | Interleukin-17 - genetics | Cell Communication - genetics | Interleukin-23 - immunology | Keratinocytes - immunology | Membrane Glycoproteins - genetics | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Dermatitis - pathology | Interleukin-17 - metabolism | Keratinocytes - pathology | Animals | Keratinocytes - metabolism | Mice | Dermatitis - metabolism | Psoriasis | Interleukins | Keratinocytes | Genetic aspects | Dosage and administration | Diagnosis | Drug therapy
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2008, Volume 205, Issue 7, pp. 1543 - 1550
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2015, Volume 43, Issue 3, pp. 475 - 487
Interleukin-17 (IL-17) induces pathology in autoimmunity and infections; therefore, constraint of this pathway is an essential component of its regulation. We... 
negative regulation | Regnase-1 | fungal immunity | autoimmunity | signal transduction | IL-17 | Autoimmunity | Signal transduction | Fungal immunity | Negative regulation | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | IL-17 RECEPTOR | TARGET GENES | CANDIDA-ALBICANS | C/EBP-BETA | GENE-EXPRESSION | HOST-DEFENSE | NECROSIS-FACTOR-ALPHA | MESSENGER-RNA STABILITY | T-CELL | IMMUNOLOGY | Candidiasis - genetics | Oncogene Proteins - genetics | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Immunoblotting | Male | Receptors, Interleukin-17 - genetics | Ribonucleases - metabolism | Host-Pathogen Interactions - immunology | Inflammation - metabolism | Lipocalins - genetics | Adaptor Proteins, Signal Transducing - immunology | Pneumonia - immunology | Candida albicans - physiology | Encephalomyelitis, Autoimmune, Experimental - genetics | Receptors, Interleukin-17 - metabolism | Interleukin-6 - metabolism | Pneumonia - genetics | Interleukin-6 - genetics | Oncogene Proteins - metabolism | Signal Transduction - genetics | Candida albicans - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Interleukin-17 - metabolism | Lipocalin-2 | Lipocalins - immunology | Interleukin-6 - immunology | Pneumonia - metabolism | Ribonucleases - genetics | Encephalomyelitis, Autoimmune, Experimental - metabolism | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | Acute-Phase Proteins - genetics | Candidiasis - microbiology | Signal Transduction - immunology | HEK293 Cells | Female | Candidiasis - immunology | Nuclear Proteins - genetics | Oncogene Proteins - immunology | Lipocalins - metabolism | Cell Line | Acute-Phase Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Nuclear Proteins - metabolism | Inflammation - immunology | Nuclear Proteins - immunology | Animals | Ribonucleases - immunology | Adaptor Proteins, Signal Transducing - genetics | Inflammation - genetics | Adaptor Proteins, Signal Transducing - metabolism | Acute-Phase Proteins - immunology | Medical colleges | Nucleases | Interleukins | RNA | Cellular signal transduction | Inflammation | Proteins | Cytokines | Rodents | Fibroblasts | Infections | Kinases | Laboratory animals | Gene expression | Experiments
Journal Article
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2009, Volume 206, Issue 1, pp. 35 - 41
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2009, Volume 119, Issue 3, pp. 565 - 572
Th17 cells are involved in. the pathogenesis of many autoimmune diseases, but it is not clear whether they play a pathogenic role in type I diabetes. Here we... 
ENCEPHALOMYELITIS | MEDICINE, RESEARCH & EXPERIMENTAL | INTERFERON-GAMMA RECEPTOR | NOD MICE | TGF-BETA | NONOBESE DIABETIC MICE | IN-VIVO | DIFFERENTIATION | T-HELPER TYPE-1 | EXPRESSION | CUTTING EDGE | Gene Expression - drug effects | Xenopus Proteins - genetics | Pancreas - cytology | Interleukin-17 - immunology | Th17 Cells - transplantation | Adoptive Transfer | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - transplantation | Th1 Cells - metabolism | Th17 Cells - drug effects | Th17 Cells - metabolism | Gene Expression - immunology | Receptors, Interleukin-12 - genetics | Th17 Cells - cytology | Diabetes Mellitus, Type 1 - immunology | Interferon-gamma - genetics | Macrophages - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Interleukin-12 - pharmacology | Basic Helix-Loop-Helix Transcription Factors - genetics | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Receptors, Aryl Hydrocarbon - genetics | Interleukin-17 - genetics | Up-Regulation - genetics | Lymph Nodes - immunology | Lymph Nodes - cytology | Mice, SCID | Pancreas - immunology | Receptors, Interleukin - genetics | T-Box Domain Proteins - genetics | Down-Regulation - genetics | Cell Differentiation - immunology | Interleukin-17 - metabolism | Macrophages - metabolism | Animals | Cell Differentiation - drug effects | Diabetes Mellitus, Type 1 - blood | Interferon-gamma - immunology | Th17 Cells - immunology | Macrophages - drug effects | Mice, Inbred NOD | Mice | Th1 Cells - cytology | Blood Glucose - metabolism | Causes of | Physiological aspects | Development and progression | Research | Diabetes | Autoimmune diseases | T cells
Journal Article