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Nature Medicine, ISSN 1078-8956, 05/2016, Volume 22, Issue 5, pp. 516 - 523
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 135, Issue 6, pp. 1476 - 1485.e7
Background Chronic rhinosinusitis (CRS) with nasal polyps (NPs) in Western populations is associated with TH 2 cytokine polarization. IL-25, an IL-17 family... 
Allergy and Immunology | sinusitis | allergy | animal models | IL-25 | Nasal polyp | SKIN BARRIER DYSFUNCTION | MAST-CELLS | CYTOKINE | INNATE LYMPHOID-CELLS | IMMUNOLOGY | ATOPIC-DERMATITIS | RESPONSES | INTERLEUKIN-17 FAMILY | INFLAMMATION | MOUSE MODEL | IN-VIVO | Eosinophil Cationic Protein - genetics | Eosinophils - drug effects | Humans | Middle Aged | Male | Receptors, Interleukin-17 - genetics | Eosinophils - immunology | Rhinitis - drug therapy | Eosinophil Cationic Protein - immunology | Transforming Growth Factor beta2 - immunology | Sinusitis - genetics | Gene Expression - immunology | Eosinophils - pathology | Disease Models, Animal | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Antibodies, Monoclonal - pharmacology | Neutrophils - drug effects | Interleukin-17 - genetics | Rhinitis - complications | Mice | Chronic Disease | Sinusitis - drug therapy | GATA3 Transcription Factor - genetics | Cell Adhesion Molecules - genetics | Gene Expression - drug effects | GATA3 Transcription Factor - immunology | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | T-Box Domain Proteins - immunology | Cell Adhesion Molecules - immunology | Case-Control Studies | Sinusitis - complications | Transforming Growth Factor beta1 - immunology | Sinusitis - immunology | Nasal Polyps - genetics | Adult | Female | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Neutrophils - pathology | Nasal Polyps - immunology | Rhinitis - immunology | Neutrophils - immunology | Transforming Growth Factor beta1 - genetics | Nasal Polyps - drug therapy | T-Box Domain Proteins - genetics | Rhinitis - genetics | Animals | Nasal Polyps - complications | Interleukin-17 - antagonists & inhibitors | Transforming Growth Factor beta2 - genetics | Medical colleges | Nurse practitioners | Nasal polyps | Atopic dermatitis | Health aspects | Proteins | Immunoglobulins | Pulmonary fibrosis | Disease | Laboratories | Rodents | Surgery | Family medical history | Endoscopy | Quality of life | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, pp. e0175153 - e0175153
IL-1 beta is a potent player in cutaneous inflammation and central for the development of a Th17 micro-milieu in autoinflammatory diseases including psoriasis.... 
VITAMIN-D | SYSTEMIC-LUPUS-ERYTHEMATOSUS | INTERFERON-GAMMA | MULTIDISCIPLINARY SCIENCES | PSORIASIN S100A7 | INFLAMMATORY CASPASES | KERATINOCYTES | 1,25-DIHYDROXYVITAMIN D-3 | MOLECULAR-CLONING | EXPRESSION | T-CELLS | Caspases - immunology | Humans | Interleukin-17 - immunology | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | S100 Proteins - immunology | Psoriasis - genetics | S100 Proteins - genetics | Th17 Cells - drug effects | Transfection | Inflammasomes - drug effects | Psoriasis - pathology | Adaptor Proteins, Signal Transducing - immunology | Apoptosis Regulatory Proteins - genetics | Skin - pathology | Skin - immunology | Th17 Cells - pathology | Interleukin-1beta - secretion | Psoriasis - immunology | S100 Calcium Binding Protein A7 | Apoptosis Regulatory Proteins - immunology | DNA - immunology | Signal Transduction | Caspases - genetics | Cells, Cultured | Gene Expression Regulation | Interleukin-17 - genetics | Interleukin-1beta - immunology | Vitamin D - pharmacology | Keratinocytes - cytology | Keratinocytes - immunology | DNA - genetics | Interleukin-17 - antagonists & inhibitors | Keratinocytes - drug effects | Inflammasomes - immunology | Adaptor Proteins, Signal Transducing - genetics | Th17 Cells - immunology | Interferon-gamma - pharmacology | Interleukins | Physiological aspects | Development and progression | Skin diseases | Genetic aspects | Inflammation | Research | Cell culture | Transcription | Interleukin | Helper cells | Lymphocytes T | Inflammatory diseases | Vitamin D | Lymphocytes | Interleukin 1 | Lesions | Deoxyribonucleic acid--DNA | Psoriasis | Cytokines | Dermatology | Keratinocytes | Caspase | Epidermis | Hazards | Gene expression | Caspase-5 | Hospitals | Biopsy | Interferon | Autoimmune diseases | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Immunity, ISSN 1074-7613, 2008, Volume 29, Issue 1, pp. 138 - 149
After activation, CD4 helper T (Th) cells differentiate into distinct effector subsets. Although chemokine (C-X-C motif) receptor 5-expressing T follicular... 
MOLIMMUNO | CELLIMMUNO | CXC CHEMOKINE RECEPTOR-5 | B-CELLS | RESPONSES | AUTOIMMUNITY | ROR-GAMMA | T(H)17 | GENE-EXPRESSION | DIFFERENTIATION | IMMUNOLOGY | IL-21 | BETA | Immunohistochemistry | T-Lymphocyte Subsets - immunology | Oligonucleotide Array Sequence Analysis | T-Lymphocyte Subsets - cytology | Germinal Center - immunology | Interleukin-17 - immunology | Gene Expression Profiling | Adoptive Transfer | Interleukins - metabolism | Signal Transduction - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Mice, Mutant Strains | T-Lymphocytes, Helper-Inducer - immunology | Interleukins - immunology | T-Lymphocytes, Helper-Inducer - cytology | Germinal Center - cytology | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Transforming Growth Factor beta - immunology | Gene Expression | Reverse Transcriptase Polymerase Chain Reaction | Proteins - immunology | Cell Differentiation - immunology | Interleukin-17 - metabolism | Animals | B-Lymphocytes - immunology | Proteins - metabolism | Inducible T-Cell Co-Stimulator Ligand | Interleukin-6 - immunology | Mice | STAT3 Transcription Factor - immunology | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | Universities and colleges | Interleukins | Transforming growth factors | T cells | Gene expression | Medical research | Lymphocytes | Rodents | Chemokines | Immune system | Cancer | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 07/2008, Volume 205, Issue 7, pp. 1517 - 1522
Some immunologists have characterized T helper (Th)17 T cells as the master mediators of tissue damage in a variety of pathological conditions. New data now... 
MEDICINE, RESEARCH & EXPERIMENTAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | GROWTH-FACTOR-BETA | ROR-GAMMA-T | MULTIPLE-SCLEROSIS | IFN-GAMMA | EFFECTOR RESPONSE | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | BIOLOGICAL-ACTIVITIES | IMMUNOLOGY | T-H-17 CELLS | T-HELPER-CELLS | Dendritic Cells - immunology | Humans | Central Nervous System - pathology | Encephalomyelitis, Autoimmune, Experimental - immunology | Interleukin-23 - genetics | Encephalomyelitis, Autoimmune, Experimental - genetics | Disease Susceptibility | Macrophages - pathology | Interleukin-17 - genetics | Interleukin-12 Subunit p35 - genetics | Interleukin-23 - immunology | Mice, Knockout | Models, Immunological | Up-Regulation - immunology | Uveitis - immunology | Mice | Uveitis - genetics | Proteins - antagonists & inhibitors | Eye - pathology | Chemokines, CXC - immunology | Interleukin-17 - immunology | Autoimmunity - genetics | Neutralization Tests | Adoptive Transfer | Paralysis - pathology | Th1 Cells - immunology | Central Nervous System - immunology | Eye - immunology | Nitric Oxide Synthase Type II - immunology | Female | Interferon-gamma - genetics | Macrophages - immunology | Neutrophils - pathology | Inflammation Mediators - immunology | Cell Line | Encephalomyelitis, Autoimmune, Experimental - pathology | Myelin Sheath - immunology | Myelin Sheath - pathology | Gene Expression Regulation - immunology | Uveitis - prevention & control | Mice, Inbred C57BL | Neutrophils - immunology | Paralysis - immunology | Up-Regulation - genetics | Proteins - immunology | Antibodies - pharmacology | Gene Expression Regulation - drug effects | Animals | Interleukin-12 Subunit p35 - immunology | Interleukin-17 - antagonists & inhibitors | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Antigens - immunology | Autoimmunity - drug effects | Uveitis - pathology | Index Medicus | Commentaries
Journal Article
Immunity, ISSN 1074-7613, 09/2015, Volume 43, Issue 3, pp. 475 - 487
Interleukin-17 (IL-17) induces pathology in autoimmunity and infections; therefore, constraint of this pathway is an essential component of its regulation. We... 
negative regulation | Regnase-1 | fungal immunity | autoimmunity | signal transduction | IL-17 | Autoimmunity | Signal transduction | Fungal immunity | Negative regulation | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | IL-17 RECEPTOR | TARGET GENES | CANDIDA-ALBICANS | C/EBP-BETA | GENE-EXPRESSION | HOST-DEFENSE | NECROSIS-FACTOR-ALPHA | MESSENGER-RNA STABILITY | T-CELL | IMMUNOLOGY | Candidiasis - genetics | Oncogene Proteins - genetics | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Immunoblotting | Male | Receptors, Interleukin-17 - genetics | Ribonucleases - metabolism | Host-Pathogen Interactions - immunology | Inflammation - metabolism | Lipocalins - genetics | Adaptor Proteins, Signal Transducing - immunology | Pneumonia - immunology | Candida albicans - physiology | Encephalomyelitis, Autoimmune, Experimental - genetics | Receptors, Interleukin-17 - metabolism | Interleukin-6 - metabolism | Pneumonia - genetics | Interleukin-6 - genetics | Oncogene Proteins - metabolism | Signal Transduction - genetics | Candida albicans - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Interleukin-17 - metabolism | Lipocalin-2 | Lipocalins - immunology | Interleukin-6 - immunology | Pneumonia - metabolism | Ribonucleases - genetics | Encephalomyelitis, Autoimmune, Experimental - metabolism | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | Acute-Phase Proteins - genetics | Candidiasis - microbiology | Signal Transduction - immunology | HEK293 Cells | Female | Candidiasis - immunology | Nuclear Proteins - genetics | Oncogene Proteins - immunology | Lipocalins - metabolism | Cell Line | Acute-Phase Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Nuclear Proteins - metabolism | Inflammation - immunology | Nuclear Proteins - immunology | Animals | Ribonucleases - immunology | Adaptor Proteins, Signal Transducing - genetics | Inflammation - genetics | Adaptor Proteins, Signal Transducing - metabolism | Acute-Phase Proteins - immunology | Medical colleges | Nucleases | Interleukins | RNA | Cellular signal transduction | Inflammation | Proteins | Cytokines | Rodents | Fibroblasts | Infections | Kinases | Laboratory animals | Gene expression | Experiments | Index Medicus
Journal Article
Science, ISSN 0036-8075, 03/2016, Volume 351, Issue 6279, pp. 1329 - 1333
Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2009, Volume 183, Issue 8, pp. 5418 - 5427
IFN-beta-1a has been used over the past 15 years as a primary therapy for relapsing-remitting multiple sclerosis (MS). However, the immunomodulatory mechanisms... 
EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | GROWTH-FACTOR-BETA | T(H)17 CELLS | MULTIPLE-SCLEROSIS | DENDRITIC CELLS | MESSENGER-RNA EXPRESSION | CENTRAL-NERVOUS-SYSTEM | INTERFERON-BETA | IMMUNOLOGY | UP-REGULATION | T-CELLS | Receptors, CCR6 - immunology | Interleukin-12 Subunit p35 - metabolism | Interleukin-23 Subunit p19 - antagonists & inhibitors | Dendritic Cells - immunology | Humans | Receptors, CCR6 - antagonists & inhibitors | CD4-Positive T-Lymphocytes - immunology | Receptors, Interleukin - metabolism | Interleukin-23 Subunit p19 - metabolism | STAT1 Transcription Factor - metabolism | Interleukin-1beta - metabolism | Receptors, Retinoic Acid - immunology | Phosphorylation - immunology | Phosphorylation - drug effects | Receptors, Thyroid Hormone - immunology | Interleukin-1beta - antagonists & inhibitors | Nuclear Receptor Subfamily 1, Group F, Member 3 | Receptors, Interleukin - immunology | Receptors, Interleukin - antagonists & inhibitors | Receptors, Retinoic Acid - metabolism | Cell Differentiation - immunology | Interferon beta-1a | Up-Regulation - immunology | Interleukin-23 Subunit p19 - immunology | STAT3 Transcription Factor - immunology | Interleukin-10 - immunology | Interleukin-17 - immunology | STAT1 Transcription Factor - drug effects | T-Lymphocytes, Helper-Inducer - drug effects | T-Lymphocytes, Helper-Inducer - immunology | Receptors, CCR6 - metabolism | Interleukin-10 - metabolism | Dendritic Cells - drug effects | Interleukin-10 - agonists | STAT3 Transcription Factor - metabolism | Cells, Cultured | Interleukin-1beta - immunology | Interleukin-12 Subunit p35 - agonists | Down-Regulation - drug effects | STAT1 Transcription Factor - immunology | Receptors, Thyroid Hormone - metabolism | Up-Regulation - drug effects | Interferon-beta - pharmacology | Interleukin-12 Subunit p35 - immunology | Cell Differentiation - drug effects | Down-Regulation - immunology | STAT3 Transcription Factor - drug effects | Multiple Sclerosis - immunology | CD4-Positive T-Lymphocytes - drug effects | Index Medicus | Abridged Index Medicus
Journal Article
The Journal of Immunology, ISSN 0022-1767, 07/2002, Volume 169, Issue 1, pp. 443 - 453
We have biologically characterized two new members of the IL-17 cytokine family: IL-17F and IL-25. In contrast to conventional in vitro screening approaches,... 
RECEPTOR-GAMMA CHAIN | LYMPHOID DEVELOPMENT | ANTIBODY | ASTHMA | GENE-THERAPY | AIRWAY HYPERRESPONSIVENESS | DEFICIENT MICE | IMMUNOLOGY | IDENTIFICATION | CELL | EXPRESSION | Interleukin-13 - physiology | Bronchial Hyperreactivity - immunology | Humans | Pulmonary Eosinophilia - genetics | Pulmonary Eosinophilia - virology | Interleukin-17 - physiology | Male | Th2 Cells - immunology | Lymphocyte Subsets - immunology | Leukocytosis - virology | Cell Movement - immunology | Th1 Cells - metabolism | RNA, Messenger - biosynthesis | Growth Substances - genetics | Interleukins | Adenoviridae - immunology | Growth Substances - administration & dosage | Amino Acid Sequence | Leukocytosis - immunology | Interleukin-17 - genetics | Neutrophils - virology | Intestinal Diseases, Parasitic - immunology | Mice, Knockout | Mice | Mice, Inbred BALB C | Amino Acid Motifs - genetics | Strongylida Infections - genetics | Molecular Sequence Data | Interleukin-5 - biosynthesis | Th1 Cells - immunology | Hematopoietic Stem Cells - immunology | Interleukin-17 - chemistry | Intestinal Diseases, Parasitic - genetics | Growth Substances - chemistry | Strongylida Infections - immunology | Basophils - immunology | Adenoviridae - genetics | Killer Cells, Natural - immunology | Female | Mucus - metabolism | Interleukin-5 - physiology | Lung - metabolism | Pulmonary Eosinophilia - immunology | Mast Cells - immunology | Lung - pathology | Mice, Inbred C57BL | Aspergillosis - genetics | Neutrophils - immunology | Hematopoietic Stem Cells - metabolism | Inflammation - immunology | Interleukin-17 - biosynthesis | Administration, Intranasal | Th2 Cells - metabolism | Sequence Homology, Amino Acid | Animals | Amino Acid Motifs - immunology | Growth Substances - physiology | Inflammation - genetics | Genetic Vectors | Leukocytosis - genetics | Granulocytes - immunology | Lung - immunology | Aspergillosis - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 10/2016, Volume 68, Issue 10, pp. 2492 - 2502
ObjectiveAntinuclear antibodies (ANAs) are detected in approximate to 18% of females, yet autoimmune disease develops in only 5-8%. Immunologic differences... 
SYSTEMIC-LUPUS-ERYTHEMATOSUS | INTERFERON | RISK-FACTORS | CYTOKINE PRODUCTION | DENDRITIC CELLS | ANTINUCLEAR ANTIBODIES | RHEUMATOLOGY | EXPRESSION | BLOOD MONONUCLEAR-CELLS | IG-LIKE RECEPTOR-1 | T-CELLS | Interferon-alpha - pharmacology | Multivariate Analysis | Phosphoproteins - drug effects | Interferon-beta - immunology | Antibodies, Antinuclear - immunology | Humans | Middle Aged | Interleukin-17 - immunology | STAT1 Transcription Factor - drug effects | Centromere Protein B - immunology | Male | Monocytes - immunology | Ribosomal Proteins - immunology | Healthy Volunteers | Case-Control Studies | Leukocytes - immunology | Young Adult | Flow Cytometry | Autoimmunity - immunology | Lupus Erythematosus, Systemic - immunology | Ribonucleoproteins - immunology | Tumor Necrosis Factor-alpha - immunology | Aged, 80 and over | Adult | B-Lymphocyte Subsets - immunology | Female | Interleukin-12 Subunit p40 - immunology | Cytokines - immunology | Interleukin 1 Receptor Antagonist Protein - immunology | Immunophenotyping | Logistic Models | Nuclear Proteins - immunology | STAT1 Transcription Factor - immunology | Stem Cell Factor - immunology | Interferon-alpha - immunology | Autoantibodies - immunology | Interferon-gamma - immunology | STAT3 Transcription Factor - drug effects | Sex Factors | Aged | Granulocyte Colony-Stimulating Factor - immunology | STAT3 Transcription Factor - immunology | Plasma Cells - immunology | Index Medicus | Abridged Index Medicus | Systemic Lupus Erythematosus
Journal Article