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Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, pp. e47463 - e47463
Human genetic background strongly influences susceptibility to malaria infection and progression to severe disease and death. Classical genetic studies... 
GLUCOSE-6-PHOSPHATE DEHYDROGENASE | TUMOR-NECROSIS-FACTOR | WEST-AFRICA | G6PD DEFICIENCY | AFRICAN CHILDREN | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | IL-10 PRODUCTION | INFECTION | PLASMODIUM-FALCIPARUM MALARIA | ASSOCIATION | Malaria, Falciparum - genetics | Genetic Predisposition to Disease - genetics | Genes, X-Linked - genetics | Membrane Proteins - genetics | Humans | Risk Factors | Tanzania | Child, Preschool | Genotype | Infant | Logistic Models | Male | Nerve Tissue Proteins - genetics | Carrier Proteins - genetics | Interleukin-13 - genetics | CD40 Ligand - genetics | Sex Factors | Female | Interleukin-1alpha - genetics | Polymorphism, Single Nucleotide | Glucosephosphate Dehydrogenase - genetics | Adenylyl Cyclases - genetics | Child | Medical research | Malaria | Genes | Genetic research | Medicine, Experimental | Development and progression | Disease susceptibility | Genetic aspects | Single nucleotide polymorphisms | Haplotypes | Disease | Genomics | Risk | Single-nucleotide polymorphism | Males | Gene polymorphism | Epidemiology | Population genetics | CD40L protein | Tropical diseases | Red blood cells | Interleukin 1 | Population | Tumor necrosis factor-TNF | Children | Glucosephosphate dehydrogenase | Blood groups | Vector-borne diseases | Cytokines | Mortality | Health risks | Mass spectroscopy | Interleukin 13 | Gene expression | Heterozygosity | Medicine | Genotyping | Lactic acid | Mutation | Females | Acidosis | Mass spectrometry | Hygiene | Polymorphism | Index Medicus
Journal Article
Infection and Immunity, ISSN 0019-9567, 2015, Volume 83, Issue 12, pp. 4673 - 4681
The human skin exerts many functions in order to maintain its barrier integrity and protect the host from invading microorganisms. One such pathogen is... 
TOLL-LIKE-RECEPTORS | INFECTIOUS DISEASES | GROUP-A STREPTOCOCCUS | C-FOS | PHOSPHORYLATION | PYOGENES TRIGGERS | SURFACE-PROTEINS | GENE-EXPRESSION | IMMUNOLOGY | NF-KAPPA-B | CYSTEINE PROTEINASE | INNATE IMMUNITY | Interleukin-1 Receptor-Like 1 Protein | Interleukin-8 - genetics | Intramolecular Oxidoreductases - immunology | Interleukin-1alpha - immunology | Toll-Like Receptor 2 - genetics | Transcription Factor AP-1 - genetics | Humans | Macrophage Migration-Inhibitory Factors - immunology | Antigens, Bacterial - immunology | NF-kappa B - immunology | Transcription Factor AP-1 - immunology | Interleukin 1 Receptor Antagonist Protein - genetics | Intracellular Signaling Peptides and Proteins - immunology | Chemokine CXCL1 - genetics | Antigens, Bacterial - genetics | Signal Transduction - immunology | Macrophage Migration-Inhibitory Factors - genetics | Basigin - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Basigin - immunology | Interleukin-1alpha - genetics | Intracellular Signaling Peptides and Proteins - genetics | Keratinocytes - microbiology | Bacterial Outer Membrane Proteins - genetics | Carrier Proteins - immunology | Mitogen-Activated Protein Kinase 3 - immunology | Mitogen-Activated Protein Kinase 1 - immunology | Bacterial Outer Membrane Proteins - immunology | Interleukin 1 Receptor Antagonist Protein - immunology | Mitogen-Activated Protein Kinase 3 - genetics | Streptococcus pyogenes - immunology | Gene Expression Regulation | p38 Mitogen-Activated Protein Kinases - genetics | p38 Mitogen-Activated Protein Kinases - immunology | Receptors, Cell Surface - immunology | Keratinocytes - immunology | Host-Pathogen Interactions | Carrier Proteins - genetics | Chemokine CXCL1 - immunology | Intramolecular Oxidoreductases - genetics | NF-kappa B - genetics | Toll-Like Receptor 2 - immunology | Interleukin-8 - immunology | Cell Line, Transformed | Receptors, Cell Surface - genetics | Index Medicus | Host Response and Inflammation
Journal Article
Science, ISSN 0036-8075, 09/2015, Volume 349, Issue 6255, pp. aaa5612 - aaa5612
Journal Article
Molecular Human Reproduction, ISSN 1360-9947, 09/2017, Volume 23, Issue 9, pp. 628 - 645
STUDY QUESTION: Does A20 regulate mediators involved in the terminal processes of human labour in primary myometrial and amnion cells? SUMMARY ANSWER: A20 is a... 
Myometrium | Pro-labour mediators | Inflammation | Preterm birth | Foetal membranes | A20 | preterm birth | TOLL-LIKE RECEPTOR | foetal membranes | NECROSIS-FACTOR-ALPHA | DEVELOPMENTAL BIOLOGY | PRETERM LABOR | TERM LABOR | OBSTETRICS & GYNECOLOGY | myometrium | PRO-LABOR MEDIATORS | IN-VITRO | REPRODUCTIVE BIOLOGY | EPITHELIAL-CELLS | inflammation | pro-labour mediators | SMOOTH-MUSCLE-CELLS | NF-KAPPA-B | EXPRESSION | Interleukin-8 - genetics | Myometrium - drug effects | RNA, Small Interfering - genetics | Interleukin-1beta - pharmacology | Premature Birth - genetics | Humans | NF-kappa B - metabolism | Tumor Necrosis Factor alpha-Induced Protein 3 - metabolism | Chemokine CXCL1 - genetics | Dinoprost - metabolism | Flagellin - pharmacology | Matrix Metalloproteinase 9 - metabolism | Term Birth - metabolism | Premature Birth - physiopathology | Cyclooxygenase 2 - genetics | Matrix Metalloproteinase 9 - genetics | Myometrium - metabolism | Interleukin-1alpha - genetics | Interleukin-8 - metabolism | Chemokine CXCL1 - metabolism | Interleukin-6 - metabolism | Poly I-C - pharmacology | Term Birth - genetics | Interleukin-6 - genetics | Signal Transduction | Amnion - cytology | Labor, Obstetric - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Dinoprost - genetics | Intercellular Adhesion Molecule-1 - metabolism | Intercellular Adhesion Molecule-1 - genetics | Vascular Cell Adhesion Molecule-1 - metabolism | RNA, Small Interfering - metabolism | Interleukin-1alpha - metabolism | Receptors, Prostaglandin - metabolism | Amnion - metabolism | Receptors, Prostaglandin - genetics | Gene Expression Regulation, Developmental | Female | Vascular Cell Adhesion Molecule-1 - genetics | Amnion - drug effects | Chemokine CCL2 - metabolism | Premature Birth - metabolism | Chemokine CCL2 - genetics | Myometrium - cytology | Pregnancy | Tumor Necrosis Factor alpha-Induced Protein 3 - genetics | NF-kappa B - genetics | Cyclooxygenase 2 - metabolism | Labor, Obstetric - genetics | Primary Cell Culture | Tumor Necrosis Factor alpha-Induced Protein 3 - antagonists & inhibitors | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2013, Volume 288, Issue 35, pp. 25025 - 25037
The expression of N-myc downstream-regulated gene 1 (NDRG1) was significantly correlated with tumor angiogenesis and malignant progression together with poor... 
OVEREXPRESSION | KAPPA-B ACTIVATION | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | PANCREATIC-CANCER | PROSTATE-CANCER | NECROSIS-FACTOR-ALPHA | CAP43/NDRG1/DRG-1 | NDRG1 | ENDOTHELIAL GROWTH-FACTOR | METASTASIS SUPPRESSOR GENE | Inflammation - pathology | Transcription Factor AP-1 - genetics | Humans | Stomach Neoplasms - metabolism | Interleukin-1alpha - metabolism | Male | Stomach Neoplasms - pathology | Intracellular Signaling Peptides and Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Transcription Factor AP-1 - metabolism | Neovascularization, Pathologic - pathology | Neoplasms, Experimental - pathology | Inflammation - metabolism | Phosphorylation - genetics | MAP Kinase Kinase 4 - metabolism | Neoplasms, Experimental - genetics | Cell Cycle Proteins - genetics | Interleukin-1alpha - genetics | Interleukin-4 - genetics | Intracellular Signaling Peptides and Proteins - genetics | Gene Expression Regulation, Neoplastic - genetics | Matrix Metalloproteinase 1 - genetics | Stomach Neoplasms - genetics | Macrophages - pathology | Interleukin-4 - metabolism | Matrix Metalloproteinase 13 - genetics | Cell Cycle Proteins - metabolism | Chemokines, CXC - genetics | Matrix Metalloproteinase 13 - metabolism | Macrophages - metabolism | Animals | Chemokines, CXC - metabolism | Cell Line, Tumor | Inflammation - genetics | MAP Kinase Kinase 4 - genetics | Neovascularization, Pathologic - genetics | Mice | Mice, Inbred BALB C | Neovascularization, Pathologic - metabolism | Neoplasms, Experimental - metabolism | Enzyme Activation - genetics | Matrix Metalloproteinase 1 - metabolism | Index Medicus | Angiogenesis | Interleukin | AP1 Transcription Factor | Gastric Cancer | N-myc Downstream-regulated Gene 1 | Inflammation | Cell Biology
Journal Article
Archives of Oral Biology, ISSN 0003-9969, 2010, Volume 56, Issue 1, pp. 54 - 62
Journal Article
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 06/2009, Volume 296, Issue 6, pp. 1324 - 1331
Interleukin-1 (IL-1) is rapidly expressed in response to tissue damage; however, its role in coordinating the progression from injury to fibrogenesis is not... 
Thioacetamide | Hepatic stellate cell | Matrix metalloproteinase | Bile duct ligation | Carbon tetrachloride | ACTIVATION | PHYSIOLOGY | TGF-BETA | COLLAGEN | carbon tetrachloride | NORMAL RAT-LIVER | thioacetamide | MATRIX METALLOPROTEINASES | bile duct ligation | HEPATIC STELLATE CELLS | MATRIX-METALLOPROTEINASE-9 | matrix metalloproteinase | hepatic stellate cell | EXTRACELLULAR-MATRIX | MICE | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Interleukin-1 - genetics | Thioacetamide - pharmacology | Liver - pathology | Gene Expression - drug effects | Gene Expression - genetics | Receptors, Interleukin-1 - genetics | Actins - metabolism | Hepatic Stellate Cells - metabolism | Male | Interleukin-1beta - genetics | Liver Cirrhosis - chemically induced | Tissue Inhibitor of Metalloproteinase-2 - genetics | Actins - genetics | Matrix Metalloproteinase 9 - metabolism | Collagen Type I - genetics | Liver - drug effects | Matrix Metalloproteinase 9 - genetics | Liver Cirrhosis - metabolism | Interleukin-1alpha - genetics | Hepatic Stellate Cells - drug effects | Liver Cirrhosis - etiology | Matrix Metalloproteinase 2 - metabolism | Liver - metabolism | Matrix Metalloproteinase 13 - genetics | Mice, Inbred C57BL | Collagen Type III - genetics | Mice, Inbred Strains | Mice, Knockout | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Transforming Growth Factor beta - genetics | Liver Cirrhosis - pathology | Mice | Mice, Inbred BALB C | Alanine Transaminase - metabolism | Pathology | Liver diseases | Cytokines | Medical disorders | Hepatology | Index Medicus | Mediators | Inflammation | Immunity
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 55, Issue 5, pp. 1086 - 1094
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e58639 - e58639
miR-181a has been presumed to target the 3'-untranslated regions (3'-UTR) of IL1a based on software predictions. miR-181a and IL1a have opposite expression... 
CELLS | MICRORNA MIR-181A | <