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Journal of Neuroimmunology, ISSN 0165-5728, 2007, Volume 194, Issue 1, pp. 54 - 61
Abstract Interleukin (IL)-17-producing helper T cells may play a pivotal role in the pathogenesis of multiple sclerosis. Here, we examined the effects of IL-17... 
Neurology | Allergy and Immunology | EAE | Cytokine | IL-17 | Microglia | cytokine | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MULTIPLE-SCLEROSIS LESIONS | MESSENGER-RNA EXPRESSION | NEUROTROPHIC FACTOR | IMMUNOLOGY | NEUROSCIENCES | NERVE GROWTH-FACTOR | INTERLEUKIN-17 | NEURONAL DEATH | NITRIC-OXIDE | TNF-ALPHA | microglia | T-CELLS | Brain-Derived Neurotrophic Factor - genetics | Microglia - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | Interleukin-17 - physiology | Receptors, Interleukin-17 - genetics | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | Interleukin-23 - genetics | RNA, Messenger - biosynthesis | Chemokine CXCL2 - genetics | Brain-Derived Neurotrophic Factor - biosynthesis | Interleukin-1beta - biosynthesis | Multiple Sclerosis - metabolism | Autocrine Communication | Nitric Oxide - biosynthesis | Cells, Cultured - drug effects | Interleukin-6 - genetics | Microglia - drug effects | Receptors, Interleukin-17 - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - genetics | Interleukin-17 - biosynthesis | Recombinant Proteins - pharmacology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Interleukin-23 - biosynthesis | Interleukin-6 - biosynthesis | Cells, Cultured - metabolism | Chemokine CXCL2 - biosynthesis | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Multiple sclerosis | Interleukins | T cells | Nitric oxide | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 03/2011, Volume 2, Issue 1, pp. 240 - 240
The immune system can both promote and suppress cancer. Chronic inflammation and proinflammatory cytokines such as interleukin (IL)-1 and IL-6 are considered... 
LUNG-CANCER | INTERFERON-INDUCIBLE PROTEIN-10 | NECROSIS IN-VIVO | PROGNOSTIC-FACTORS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | CD4(+) T-CELLS | ADJUVANT THERAPY | CARCINOMA | EXPRESSION | Angiostatic Proteins - immunology | Immunohistochemistry | Neoplasms - metabolism | Interleukin-1alpha - immunology | Chemokine CXCL9 - biosynthesis | Tumor Microenvironment | Multiple Myeloma - immunology | Th2 Cells - immunology | Th1 Cells - immunology | CD4-Positive T-Lymphocytes - immunology | Chemokine CXCL10 - biosynthesis | Inflammation - metabolism | Interleukin-2 - immunology | Chemokine CXCL10 - immunology | Lymphoma, B-Cell - immunology | Interleukin-1beta - biosynthesis | Macrophages - immunology | Interleukin-12 - biosynthesis | Lymphoma, B-Cell - metabolism | Signal Transduction | Angiostatic Proteins - biosynthesis | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Interleukin-1beta - immunology | Chemokine CXCL9 - immunology | Inflammation - immunology | Macrophages - cytology | Mice, SCID | Multiple Myeloma - metabolism | Macrophages - metabolism | Multiple Myeloma - pathology | Animals | Neoplasms - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Interleukin-6 - immunology | Lymphoma, B-Cell - pathology | Cell Line, Tumor | Interleukin-6 - biosynthesis | Interleukin-1alpha - biosynthesis | Mice | Interleukin-2 - biosynthesis | Neoplasms - pathology | Interferon-gamma - biosynthesis | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2011, Volume 300, Issue 4, pp. 1418 - 1426
Post-myocardial infarction (MI), chemokine homing of inflammatory cells into the injured left ventricle (LV) regulates ventricular remodeling, in part by... 
CC chemokines | Inflammation | Matrix metalloproteinases | RECRUITMENT | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ANGIOGENESIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INCREASES | ISLET XENOGRAFTS | HEART | inflammation | PERIPHERAL VASCULAR DISEASE | MICE | matrix metalloproteinases | EXPRESSION | LEFT-VENTRICULAR FUNCTION | Myocardial Infarction - genetics | Macrophage Activation - genetics | Antigens, CD - biosynthesis | Mannose-Binding Lectins - biosynthesis | Receptors, CCR5 - genetics | Arginase - biosynthesis | Male | Lectins, C-Type - biosynthesis | Gene Deletion | Myocardial Infarction - pathology | Ventricular Remodeling - genetics | Female | Interleukin-1beta - biosynthesis | Receptors, Cell Surface - biosynthesis | Receptors, CCR5 - physiology | Procollagen - biosynthesis | Transforming Growth Factor beta1 - biosynthesis | HSP47 Heat-Shock Proteins - biosynthesis | Antigens, Differentiation, Myelomonocytic - biosynthesis | Macrophages - pathology | Ventricular Remodeling - physiology | Collagen Type I - biosynthesis | Macrophages - metabolism | Animals | Interleukin-6 - biosynthesis | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Gene mutations | Physiological aspects | Genetic aspects | Research | Macrophages | Health aspects | Chemokine receptors | Heart attack | Risk factors | Proteins | Heart attacks | Collagen | Rodents | Metabolism | Cells | Tumors | Index Medicus | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2016, Volume 196, Issue 8, pp. 3421 - 3428
Journal Article
Journal Article
Biochemical Journal, ISSN 0264-6021, 08/2010, Volume 430, Issue 1, pp. 141 - 149
A paradoxical but common finding in the obesity clinic is the identification of individuals who can be considered 'inappropriately' healthy for their degree of... 
Obesity | Signal transduction | Insulin resistance | Inflammation | 3T3-L1 ADIPOCYTES | SER | PROINFLAMMATORY STATE | signal transduction | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MONONUCLEAR-CELLS | ALPHA | inflammation | RESISTANCE | insulin resistance | INSULIN-RECEPTOR SUBSTRATE-1 | obesity | Insulin - physiology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Obesity, Morbid - immunology | Male | Interleukin-1beta - genetics | I-kappa B Proteins - biosynthesis | I-kappa B Proteins - genetics | Proto-Oncogene Proteins c-akt - genetics | RNA, Messenger - biosynthesis | Inflammation - metabolism | Insulin Receptor Substrate Proteins - biosynthesis | Adult | Female | Interleukin-1beta - biosynthesis | Insulin Receptor Substrate Proteins - genetics | Obesity, Morbid - metabolism | Macrophages - immunology | Biomarkers - metabolism | Intra-Abdominal Fat - immunology | NF-KappaB Inhibitor alpha | Interleukin-6 - genetics | Insulin Resistance | Mitogen-Activated Protein Kinases - biosynthesis | Inflammation - immunology | Intra-Abdominal Fat - metabolism | Proto-Oncogene Proteins c-akt - biosynthesis | NF-kappa B - genetics | NF-kappa B - biosynthesis | Interleukin-6 - biosynthesis | Mitogen-Activated Protein Kinases - genetics | Tumor Necrosis Factor-alpha - biosynthesis | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 03/2007, Volume 115, Issue 12, pp. 1599 - 1608
Background-Stroke is the second to third leading cause of death. Toll-like receptor 4 (TLR4) is a signaling receptor in innate immunity that is a specific... 
Infection | Cerebral ischemia | Stroke | Nitric oxide synthase | Inflammation | Metalloproteinases | Immune system | infection | CARDIAC & CARDIOVASCULAR SYSTEMS | LIPID-PEROXIDATION | FOCAL CEREBRAL-ISCHEMIA | metalloproteinases | stroke | NITRIC-OXIDE-SYNTHASE | MATRIX METALLOPROTEINASES | TUMOR-NECROSIS-FACTOR | TOLL-LIKE RECEPTOR-4 | MESSENGER-RNA | inflammation | cerebral ischemia | immune system | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | nitric oxide synthase | HEMATOLOGY | PATTERN-RECOGNITION RECEPTORS | INNATE IMMUNITY | Interferons - biosynthesis | Nitric Oxide Synthase Type II - biosynthesis | Oxidative Stress | Tumor Necrosis Factor-alpha - genetics | Nitrobenzenes - therapeutic use | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Cyclooxygenase 2 - biosynthesis | Toll-Like Receptor 4 - deficiency | Cyclooxygenase 2 - genetics | Infarction, Middle Cerebral Artery - complications | Matrix Metalloproteinase 9 - genetics | Cyclooxygenase 2 Inhibitors - therapeutic use | Brain Damage, Chronic - metabolism | Interferons - genetics | Interferon Regulatory Factor-1 - biosynthesis | Encephalitis - pathology | Mice, Knockout | Toll-Like Receptor 4 - physiology | Brain Damage, Chronic - etiology | Cerebral Infarction - etiology | Brain Chemistry | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Brain Damage, Chronic - prevention & control | Encephalitis - etiology | Dinoprostone - analysis | Infarction, Middle Cerebral Artery - metabolism | Cerebral Infarction - metabolism | Encephalitis - drug therapy | Nitric Oxide Synthase Type II - antagonists & inhibitors | Encephalitis - metabolism | Inflammation Mediators - metabolism | Nerve Tissue Proteins - biosynthesis | Interleukin-1beta - biosynthesis | Brain Damage, Chronic - pathology | Cerebral Infarction - pathology | Matrix Metalloproteinase 9 - biosynthesis | Mice, Inbred C57BL | Gene Expression Regulation | Infarction, Middle Cerebral Artery - pathology | Nerve Tissue Proteins - genetics | Mice, Inbred C3H | Interferon Regulatory Factor-1 - genetics | Animals | Nitric Oxide Synthase Type II - genetics | Sulfonamides - therapeutic use | Lipid Peroxidation | Nitric Oxide - metabolism | Stroke (Disease) | Complications and side effects | Patient outcomes | Physiological aspects | Development and progression | Research | Chemokines | Index Medicus | Abridged Index Medicus
Journal Article
American journal of respiratory cell and molecular biology, ISSN 1044-1549, 11/2015, Volume 53, Issue 5, pp. 676 - 688
Macrophages are dynamic cells that mature under the influence of signals from the local microenvironment into either classically (M1) or alternatively (M2)... 
classically activated macrophages (CAM/M1) | Phagocytosis/ endocytosis | phagocytosis/endocytosis | phenotypic stability | alternatively activated macrophages (AAM/M2) | reprogramming of polarization | PHAGOCYTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MONOCYTES | DIFFERENTIAL REGULATION | MURINE | CELL BIOLOGY | GM-CSF | RESPIRATORY SYSTEM | THP-1 CELLS | MANNOSE | RECEPTOR-ACTIVITY | POLARIZATION | EXPRESSION | Chemokines, CC - secretion | Gene Expression - drug effects | Monocytes - cytology | Humans | Monocytes - immunology | Chemokine CCL5 - secretion | Chemokine CCL17 - secretion | Chemokine CXCL10 - biosynthesis | Interleukin-4 - pharmacology | Chemokine CCL17 - biosynthesis | Gene Expression - immunology | Chemokines, CC - biosynthesis | Interleukin-8 - secretion | Cell Differentiation | Interleukin-1beta - biosynthesis | Macrophages - immunology | Endocytosis - immunology | Interleukin-1beta - secretion | Interleukin-8 - biosynthesis | Endocytosis - drug effects | Tumor Necrosis Factor-alpha - secretion | Immunophenotyping | Macrophages - cytology | Interleukin-13 - pharmacology | Monocytes - drug effects | Chemokine CCL5 - biosynthesis | Chemokine CXCL10 - secretion | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Primary Cell Culture | Macrophage Activation - drug effects | Tumor Necrosis Factor-alpha - biosynthesis | Interferon-gamma - pharmacology | Studies | Biology | Immunology | Blood & organ donations | Cytokines | Gene expression | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 12, pp. e116142 - e116142
Rationale: Replication deficient adenoviruses (Ad) vectors are common tools in gene therapy. Since Ad vectors are known to activate innate and adaptive... 
PATHOGENESIS | MATRIX | ACTIVATION | HIPPEL-LINDAU-PROTEIN | MULTIDISCIPLINARY SCIENCES | IN-VIVO | DISEASE | ANIMAL-MODELS | MEDIATED GENE-TRANSFER | IDIOPATHIC PULMONARY-FIBROSIS | EXPRESSION | Male | Interleukin-13 - biosynthesis | Lung Injury - virology | Proliferating Cell Nuclear Antigen - biosynthesis | Pulmonary Fibrosis - virology | Interleukin-16 - biosynthesis | Bronchoalveolar Lavage Fluid - cytology | Interleukin-1beta - biosynthesis | Adenoviridae - immunology | Integrins - biosynthesis | Matrix Metalloproteinase 2 - biosynthesis | Transforming Growth Factor beta1 - biosynthesis | Disease Models, Animal | Matrix Metalloproteinase 9 - biosynthesis | Actins - biosynthesis | Mice, Inbred C57BL | Inflammation - virology | Pulmonary Fibrosis - immunology | Inflammation - immunology | Bleomycin - administration & dosage | Collagen Type I - biosynthesis | Animals | Interleukin-1alpha - biosynthesis | Mice | Bronchoalveolar Lavage Fluid - chemistry | Wnt Proteins - biosynthesis | Lung Injury - immunology | RNA | Collagen | Adenoviruses | Fibrosis | Bone morphogenetic proteins | Transforming growth factors | Gene therapy | Health aspects | Integrins | Pediatrics | Collagen (type I) | Disease | Pathogenesis | Lung | Immunity | Proteins | Bleomycin | Proliferating cell nuclear antigen | Physiology | Chronic obstructive pulmonary disease | Drug dosages | Deoxyribonucleic acid--DNA | Cytokines | Bronchus | Interleukin 13 | Tumor necrosis factor-α | Gene expression | Interleukin 16 | DNA viruses | Vectors (Biology) | Pulmonary fibrosis | Interleukin 10 | Wnt protein | Laboratories | Staining | Critical care | Inflammatory response | Viruses | mRNA | Infections | Kinases | Tissues | Bronchoalveolar lavage | Rodents | Alveoli | Trachea | Injuries | Expression vectors | Lung diseases | Inflammation | Adaptive immunity | Gelatinase B | Gelatinase A | Signaling | Lungs | Molecular biology | Immunofluorescence | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Journal Article
Journal of Virology, ISSN 0022-538X, 10/2012, Volume 86, Issue 20, pp. 10961 - 10978
Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
VIRAL LOAD | TUMOR-NECROSIS-FACTOR | DAI DLM-1/ZBP1 | VIROLOGY | DNA FRAGMENTATION | IMMUNE-RESPONSES | PROGRAMMED NECROSIS | RIBONUCLEOTIDE REDUCTASE HOMOLOG | MURINE CYTOMEGALOVIRUS | FACTOR RECEPTOR | FACTOR-ALPHA | Microglia - metabolism | Murine Acquired Immunodeficiency Syndrome - complications | Caspase 8 - biosynthesis | Interleukin-1beta - genetics | RNA, Messenger - metabolism | TNF-Related Apoptosis-Inducing Ligand - biosynthesis | Cytomegalovirus Retinitis - immunology | Murine Acquired Immunodeficiency Syndrome - pathology | Murine Acquired Immunodeficiency Syndrome - virology | Caspase 3 - biosynthesis | Female | Interleukin-1beta - biosynthesis | Fas Ligand Protein - biosynthesis | Granulocytes - metabolism | Cytomegalovirus Retinitis - complications | Mice, Inbred C57BL | RNA, Messenger - genetics | Cytomegalovirus Retinitis - pathology | Muromegalovirus | Mice, Transgenic | Receptor-Interacting Protein Serine-Threonine Kinases - biosynthesis | Retina - virology | Interleukin-18 - biosynthesis | Macrophages - metabolism | Animals | Receptors, Tumor Necrosis Factor - biosynthesis | Mice | Mice, Inbred BALB C | Fas Ligand Protein - genetics | Interleukin-18 - genetics | Retina - pathology | Tumor Necrosis Factor-alpha - biosynthesis | Apoptosis | Caspase 1 - biosynthesis | Index Medicus | Pathogenesis and Immunity
Journal Article
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