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Autophagy, ISSN 1554-8635, 10/2014, Volume 7, Issue 11, pp. 1359 - 1370
Cystic fibrosis (CF) is the most common inherited lethal disease of Caucasians which results in multi organ dysfunction. However, 85% of the deaths are due to... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Burkholderia cenocepacia | Cystic fibrosis | Inflammation | Rapamycin | Macrophages | Autophagy | Host-pathogen interaction | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Phagosomes - microbiology | Burkholderia Infections - complications | Microtubule-Associated Proteins - metabolism | Cystic Fibrosis - pathology | Vacuoles - drug effects | Autophagy - drug effects | Burkholderia Infections - pathology | Burkholderia cenocepacia - ultrastructure | Phagosomes - ultrastructure | Interleukin-1beta - biosynthesis | Autophagy - genetics | Burkholderia Infections - microbiology | Macrophages - microbiology | Disease Models, Animal | Phagosomes - drug effects | Burkholderia cenocepacia - growth & development | Lysosomes - drug effects | Macrophages - ultrastructure | Sirolimus - therapeutic use | Vacuoles - microbiology | Intracellular Space - drug effects | Microbial Viability - drug effects | Cystic Fibrosis - microbiology | Mice, Inbred C57BL | Burkholderia cenocepacia - physiology | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Pneumonia - microbiology | Cystic Fibrosis - complications | Mutation - genetics | Sirolimus - pharmacology | Down-Regulation - genetics | Macrophages - metabolism | Animals | Lysosomes - microbiology | Pneumonia - drug therapy | Intracellular Space - microbiology | Burkholderia cenocepacia - drug effects | Burkholderia Infections - drug therapy | Pneumonia - complications | Macrophages - drug effects | Mice | Cystic Fibrosis - drug therapy | RNA, Small Interfering - metabolism | Index Medicus | host-pathogen interaction | macrophages | autophagy | inflammation | cystic fibrosis | Translational Research Paper | rapamycin
Journal Article
The FEBS journal, ISSN 1742-4658, 04/2018, Volume 285, Issue 12, pp. 2146 - 2160
The finding that the Warburg effect observed in proliferating cancer cells is also observed during immune responses renewed the interest in the study of metabolic reprogramming of immune cells... 
OXPHOS | Warburg‐like metabolism | bacterial infection | mitochondria | glycolysis | Warburg-like metabolism | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Cell Proliferation | Humans | Mitochondria - immunology | Glycolysis - genetics | Oxidative Phosphorylation - drug effects | Bacteria - growth & development | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Cell Differentiation | Interleukin-1beta - biosynthesis | Macrophages - immunology | Macrophages - microbiology | T-Lymphocytes - microbiology | Interleukin-1beta - immunology | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - microbiology | Cellular Reprogramming - drug effects | Host-Pathogen Interactions | Macrophages - metabolism | Animals | Lipopolysaccharides - pharmacology | Macrophages - drug effects | T-Lymphocytes - immunology | Bacteria - pathogenicity | Physiological aspects | Glucose metabolism | Bacterial infections | Health aspects | Cells | Immune response | Bacteria | Infections | Intracellular | Metabolism | Cancer | Mammalian cells | Immune system | Index Medicus | Bacteria/growth & development | Macrophages/microbiology | Mitochondria/metabolism | Interleukin-1beta/biosynthesis | Macrophages/metabolism | Glycolysis/genetics | Macrophages/immunology | Cellular Biology | Bacteria/pathogenicity | Life Sciences | Cellular Reprogramming/drug effects | Mitochondria/drug effects | T-Lymphocytes/metabolism | T-Lymphocytes/drug effects | Oxidative Phosphorylation/drug effects | T-Lymphocytes/microbiology | Interleukin-1beta/immunology | T-Lymphocytes/immunology | Lipopolysaccharides/pharmacology | Mitochondria/microbiology | Mitochondria/immunology | Macrophages/drug effects
Journal Article
Psychopharmacology (Berlin, Germany), ISSN 1432-2072, 10/2018, Volume 236, Issue 2, pp. 591 - 602
.... However, the upstream signaling pathway that regulates cytokines and apoptosis in the antidepressant effect of resveratrol remains... 
Neurosciences | Biomedicine | Resveratrol | Chronic unpredictable mild stress | Proinflammatory cytokines | Pharmacology/Toxicology | Psychiatry | Apoptosis | Interleukin-1beta - drug effects | Tumor Necrosis Factor-alpha - metabolism | Male | Hippocampus - drug effects | Stress, Psychological - psychology | Depression - metabolism | Prefrontal Cortex - drug effects | Interleukin-1beta - metabolism | Stress, Psychological - metabolism | Behavior, Animal - drug effects | Antidepressive Agents - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Fluoxetine - pharmacology | Cytokines - metabolism | Resveratrol - pharmacology | Rats | Antioxidants - pharmacology | Glycogen Synthase Kinase 3 beta - metabolism | Rats, Sprague-Dawley | Hippocampus - metabolism | Animals | Tumor Necrosis Factor-alpha - drug effects | Cytokines - drug effects | Signal Transduction - drug effects | Prefrontal Cortex - metabolism | Depression - psychology | Glycogen Synthase Kinase 3 beta - drug effects | Proto-Oncogene Proteins c-akt - drug effects | Normalizing | Sucrose | Bax protein | Bcl-2 protein | Ventricles (cerebral) | Inflammatory response | AKT protein | mRNA | Mental depression | Neurogenesis | Interleukin 6 | Proteins | Signal transduction | Antidepressants | Rodents | Animal tissues | Field tests | Behavior | Sugars | Open-field behavior | Cytokines | Fluoxetine | Inflammation | Tumor necrosis factor-α | Prefrontal cortex | Gene expression | IL-1β | Signaling | Microinjection | Protein expression | Ventricle | Hippocampus | Index Medicus
Journal Article
Molecular neurobiology, ISSN 0893-7648, 8/2015, Volume 52, Issue 1, pp. 783 - 791
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 2016, Volume 311, Issue 4, pp. H871 - H880
We previously reported that endoplasmic reticulum (ER) stress is induced in the subfornical organ (SFO) and the hypothalamic paraventricular nucleus (PVN) of... 
Heart failure | Brain | Sympathetic activity | Hypothalamic paraventricular nucleus | Mitogen-activated protein kinase | Subfornical organ | Endoplasmic reticulum stress | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cholagogues and Choleretics - pharmacology | Tumor Necrosis Factor-alpha - genetics | Heart Failure - physiopathology | Male | NF-KappaB Inhibitor alpha - genetics | Peptidyl-Dipeptidase A - drug effects | Interleukin-1beta - genetics | Sympathetic Nervous System - physiopathology | RNA, Messenger - metabolism | Activating Transcription Factor 6 - genetics | Subfornical Organ - drug effects | Brain - metabolism | Heat-Shock Proteins - genetics | Inflammation - metabolism | Receptor, Angiotensin, Type 1 - genetics | Cyclooxygenase 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Real-Time Polymerase Chain Reaction | Echocardiography | Signal Transduction | Rats | Cyclooxygenase 2 - drug effects | Heart Failure - metabolism | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | Tumor Necrosis Factor-alpha - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Endoplasmic Reticulum Stress | Paraventricular Hypothalamic Nucleus - metabolism | Infusions, Intraventricular | Mitogen-Activated Protein Kinases - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Interleukin-1beta - drug effects | Sympathetic Nervous System - drug effects | Mitogen-Activated Protein Kinase 1 - drug effects | X-Box Binding Protein 1 - drug effects | Sympathetic Nervous System - metabolism | Transcription Factor RelA - genetics | Activating Transcription Factor 6 - drug effects | Mitogen-Activated Protein Kinase 3 - drug effects | Taurochenodeoxycholic Acid - pharmacology | Peptidyl-Dipeptidase A - genetics | Renin-Angiotensin System | Receptor, Angiotensin, Type 1 - drug effects | RNA, Messenger - drug effects | Subfornical Organ - metabolism | Heat-Shock Proteins - drug effects | Activating Transcription Factor 4 - genetics | Activating Transcription Factor 4 - drug effects | NF-KappaB Inhibitor alpha - drug effects | Paraventricular Hypothalamic Nucleus - drug effects | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Transcription Factor RelA - drug effects | X-Box Binding Protein 1 - genetics | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Cellular signal transduction | Endoplasmic reticulum | Health aspects | Mitogen-activated protein kinases | Signal transduction | Inflammation | Kinases | Index Medicus | mitogen-activated protein kinase | heart failure | Cardiovascular Neurohormonal Regulation | subfornical organ | sympathetic activity | brain | endoplasmic reticulum stress | hypothalamic paraventricular nucleus
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 06/2014, Volume 146, Issue 7, pp. 1763 - 1774
Background & Aims The NACHT, LRR, and pyrin domain–containing protein 3 (NLRP3) inflammasome induces inflammation in response to organ injury, but little is... 
Gastroenterology and Hepatology | Innate Immune Response | Immune Regulation | Pancreas | Mouse Model | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Liver - pathology | Inflammasomes - metabolism | Receptors, G-Protein-Coupled - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Male | NF-kappa B - metabolism | Monocytes - immunology | Lipopolysaccharides | Liver - immunology | Liver - drug effects | RNA Interference | Interleukin-1beta - metabolism | Toll-Like Receptors - drug effects | Anti-Inflammatory Agents - administration & dosage | Toll-Like Receptors - metabolism | Cytoprotection | Disease Models, Animal | Galactosamine | Chemical and Drug Induced Liver Injury - prevention & control | Anti-Inflammatory Agents - pharmacology | Down-Regulation | Liver - metabolism | Injections, Intraperitoneal | Pancreas - pathology | Pancreas - metabolism | Pancreas - immunology | Toll-Like Receptor 4 - metabolism | Chemical and Drug Induced Liver Injury - immunology | Monocytes - drug effects | Macrophages - metabolism | Signal Transduction - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Sodium Lactate - pharmacology | beta-Arrestins | Mice | Receptors, G-Protein-Coupled - genetics | RNA, Small Interfering - metabolism | Monocytes - metabolism | Pancreatitis - prevention & control | Arrestins - metabolism | Dose-Response Relationship, Drug | Pancreatitis - genetics | Transfection | Inflammasomes - drug effects | Sodium Lactate - administration & dosage | Pancreatitis - immunology | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - etiology | Macrophages - immunology | Cell Line | Immunity, Innate - drug effects | Toll-Like Receptor 4 - drug effects | Mice, Inbred C57BL | Pancreas - drug effects | Chemical and Drug Induced Liver Injury - genetics | Pancreatitis - chemically induced | Animals | Carrier Proteins - metabolism | beta-Arrestin 2 | Inflammasomes - immunology | Macrophages - drug effects | Pancreatitis - pathology | Pancreatitis - metabolism | Ceruletide | Lactates | Gastrointestinal diseases | Inflammation | Index Medicus | Abridged Index Medicus
Journal Article
Nature (London), ISSN 1476-4687, 03/2013, Volume 496, Issue 7444, pp. 238 - 242
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 04/2017, Volume 66, Issue 4, pp. 1052 - 1061
Journal Article