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BMC genomics, ISSN 1471-2164, 2019, Volume 20, Issue 1, pp. 878 - 18
Background The function of Toll-like receptor 2 (TLR2) in host defense against pathogens, especially Mycobacterium tuberculosis (Mtb) is poorly understood. To... 
ACYLATION STATE | Mycobacterium | IMMUNE-RESPONSE | ACTIVATED PROTEIN-KINASE | INNATE | Zebrafish | PROINFLAMMATORY CYTOKINES | RNAseq | C-MAF | CUTTING EDGE | Tuberculosis | Transcriptomics | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | HOST-DEFENSE | RNA deep sequencing | TLR2 | Chemokine CXCL11 - genetics | Toll-Like Receptor 2 - genetics | Zebrafish - immunology | Interleukin-1beta - genetics | Host-Pathogen Interactions - immunology | Lymphotoxin-alpha - genetics | Toll-Like Receptor 2 - deficiency | Larva - immunology | Zebrafish Proteins - immunology | Chemokine CXCL11 - immunology | Mycobacterium Infections, Nontuberculous - immunology | Disease Resistance - genetics | Macrophages - microbiology | CCAAT-Enhancer-Binding Protein-beta - genetics | Fish Diseases - genetics | Zebrafish - genetics | Metabolic Networks and Pathways - genetics | Host-Pathogen Interactions - genetics | Mycobacterium marinum - immunology | CCAAT-Enhancer-Binding Protein-beta - immunology | Maf Transcription Factors - immunology | Receptors, CXCR3 - genetics | Embryo, Nonmammalian | Receptors, CXCR3 - immunology | Toll-Like Receptor 2 - immunology | Proto-Oncogene Proteins c-fos - genetics | Larva - microbiology | Zebrafish Proteins - genetics | Receptors, Immunologic - genetics | Maf Transcription Factors - genetics | Transcriptome - immunology | Zebrafish - microbiology | Mycobacterium marinum - pathogenicity | Lymphotoxin-alpha - immunology | Metabolic Networks and Pathways - immunology | Gene Expression Regulation, Developmental | Mycobacterium Infections, Nontuberculous - microbiology | Larva - growth & development | Fish Diseases - immunology | Larva - genetics | Neutrophils - microbiology | Receptors, Immunologic - immunology | Macrophages - immunology | Fish Diseases - microbiology | Neutrophils - immunology | Interleukin-1beta - immunology | Proto-Oncogene Proteins c-fos - immunology | Immunity, Innate | Zebrafish - growth & development | Animals | Mycobacterium Infections, Nontuberculous - veterinary | Mycobacterium Infections, Nontuberculous - genetics | RNA sequencing | Bacterial infections | RNA | Calcifediol | DNA binding proteins | Genetic transcription | Infection | Vitamin D | Physiological aspects | Genetic research | Genetic aspects | Alfacalcidol | Comparative analysis | Health aspects | Larvae | Granuloma | Transcription factors | Genes | Microbiomes | Infections | Macrophages | Protein turnover | Gene sequencing | Proteins | Receptors | Pathways | Interleukin 1 | Toll-like receptors | Pathogens | Antigens | Phenotypes | Immune response | Cytokines | Neutrophils | Leukocytes (neutrophilic) | CXCR3 protein | Ribonucleic acid--RNA | Mammals | Gene expression | Metabolism | Embryos | Defense | Mutants | Infectious diseases | TLR2 protein | Metabolic pathways | CXCL11 protein | Mutation | Vitamin D receptors | Chemokines
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0175153
IL-1 beta is a potent player in cutaneous inflammation and central for the development of a Th17 micro-milieu in autoinflammatory diseases including psoriasis.... 
VITAMIN-D | SYSTEMIC-LUPUS-ERYTHEMATOSUS | INTERFERON-GAMMA | MULTIDISCIPLINARY SCIENCES | PSORIASIN S100A7 | INFLAMMATORY CASPASES | KERATINOCYTES | 1,25-DIHYDROXYVITAMIN D-3 | MOLECULAR-CLONING | EXPRESSION | T-CELLS | Caspases - immunology | Humans | Interleukin-17 - immunology | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | S100 Proteins - immunology | Psoriasis - genetics | S100 Proteins - genetics | Th17 Cells - drug effects | Transfection | Inflammasomes - drug effects | Psoriasis - pathology | Adaptor Proteins, Signal Transducing - immunology | Apoptosis Regulatory Proteins - genetics | Skin - pathology | Skin - immunology | Th17 Cells - pathology | Interleukin-1beta - secretion | Psoriasis - immunology | S100 Calcium Binding Protein A7 | Apoptosis Regulatory Proteins - immunology | DNA - immunology | Signal Transduction | Caspases - genetics | Cells, Cultured | Gene Expression Regulation | Interleukin-17 - genetics | Interleukin-1beta - immunology | Vitamin D - pharmacology | Keratinocytes - cytology | Keratinocytes - immunology | DNA - genetics | Interleukin-17 - antagonists & inhibitors | Keratinocytes - drug effects | Inflammasomes - immunology | Adaptor Proteins, Signal Transducing - genetics | Th17 Cells - immunology | Interferon-gamma - pharmacology | Interleukins | Physiological aspects | Development and progression | Skin diseases | Genetic aspects | Inflammation | Research | Cell culture | Transcription | Interleukin | Helper cells | Lymphocytes T | Inflammatory diseases | Vitamin D | Lymphocytes | Interleukin 1 | Lesions | Deoxyribonucleic acid--DNA | Psoriasis | Cytokines | Dermatology | Keratinocytes | Caspase | Epidermis | Hazards | Gene expression | Caspase-5 | Hospitals | Biopsy | Interferon | Autoimmune diseases | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Nature medicine, ISSN 1546-170X, 2017, Volume 23, Issue 2, pp. 174 - 184
Low-grade, chronic inflammation has been associated with many diseases of aging, but the mechanisms responsible for producing this inflammation remain unclear.... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | INTERLEUKIN-18 LEVELS | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARDIOVASCULAR-DISEASE | RECEPTOR | ESSENTIAL-HYPERTENSION | PROTEINS | T-CELLS | AGE | CELL BIOLOGY | Toll-Like Receptor 5 - genetics | Humans | Middle Aged | Oxidative Stress - immunology | Transcriptome | Immunoblotting | Male | Purinergic P1 Receptor Antagonists - pharmacology | Young Adult | Toll-Like Receptor 8 - immunology | Aging - immunology | Toll-Like Receptor 6 - immunology | Aging - genetics | Calcium-Binding Proteins - immunology | Interleukin-1beta - metabolism | Aged, 80 and over | Caffeine - pharmacology | Hypertension - genetics | Intracellular Signaling Peptides and Proteins - genetics | Interleukin 1 Receptor Antagonist Protein - immunology | Carotid Intima-Media Thickness | Mortality | Neutrophils - drug effects | Hypertension - immunology | Nucleotides - metabolism | Toll-Like Receptor 5 - immunology | Monocytes - drug effects | Regression Analysis | Phenotype | Mice | Calcium-Binding Proteins - genetics | Metabolomics | Pulse Wave Analysis | Neutrophil Activation - drug effects | Cytidine - pharmacology | Interleukin 1 Receptor Antagonist Protein - genetics | Intracellular Signaling Peptides and Proteins - immunology | Cytidine - analogs & derivatives | CARD Signaling Adaptor Proteins - genetics | Toll-Like Receptor 6 - genetics | Adult | Female | Blood Pressure - drug effects | Blood Platelets - drug effects | Macrophages - immunology | Cytokines - immunology | CARD Signaling Adaptor Proteins - immunology | Cell Line | Vascular Stiffness - genetics | Cytokines - metabolism | Oxidative Stress - genetics | Interleukin-1beta - immunology | Adenine - pharmacology | Inflammation - immunology | Inflammasomes - genetics | Vascular Stiffness - immunology | Animals | Inflammasomes - immunology | Toll-Like Receptor 8 - genetics | Inflammation - genetics | Aged | Platelet Activation - drug effects | Genetic aspects | Immune response | Gene expression | Health aspects | Aging | Inflammation | Immune system
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 7/2014, Volume 111, Issue 30, pp. 11181 - 11186
Acute glaucoma is a sight-threatening condition characterized by a sudden and substantial rise in intraocular pressure (IOP) and consequent retinal ganglion... 
Glaucoma | Reperfusion | Messenger RNA | Ischemia | Toll like receptors | Central nervous system | Retina | Brain damage | Intraocular pressure | Microglia | Retinal ischemia/reperfusion injury | Cell apoptosis | BRAIN-INJURY | MICROGLIA ACTIVATION | INTERLEUKIN-1-BETA | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | POPULATIONS | retinal ischemia/reperfusion injury | ANGLE-CLOSURE GLAUCOMA | cell apoptosis | TOLL-LIKE RECEPTORS | CENTRAL-NERVOUS-SYSTEM | MICE | CEREBRAL-ISCHEMIA | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Caspase 8 - metabolism | Caspase 1 - metabolism | Interleukin-1beta - genetics | Caspase 8 - genetics | Interleukin-1beta - metabolism | Apoptosis Regulatory Proteins - genetics | Eye Proteins - genetics | Glaucoma - metabolism | Disease Models, Animal | Acute Disease | Rats | Toll-Like Receptor 4 - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Toll-Like Receptor 4 - metabolism | Inflammasomes - genetics | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Eye Proteins - metabolism | Caspase 1 - genetics | Adaptor Proteins, Signal Transducing - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Enzyme Activation - genetics | Glaucoma - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Biological Sciences | reperfusion injury | retinal ischemia
Journal Article
Journal Article
Archives of Oral Biology, ISSN 0003-9969, 2013, Volume 58, Issue 8, pp. 912 - 929
Abstract Objective An analysis of the genes involved in both osteoporosis and modifications of the jawbone, through text mining, using a web search tool, of... 
Advanced Basic Science | Dentistry | Bone resorption | Osteoporosis | Mandible | Bone remodelling | Genomics | Bone repair | Maxilla | Computational biology | BONE-MINERAL DENSITY | HUMAN OSTEOBLASTS | TOMOGRAPHIC FOLLOW-UP | SECONDARY HYPERPARATHYROIDISM | FACTOR-I | DENTISTRY, ORAL SURGERY & MEDICINE | CELL-CYCLE | HUMAN T-LYMPHOCYTES | EXPRESSION | ALVEOLAR RIDGE AUGMENTATION | GENOME-WIDE ASSOCIATION | Osteopontin - genetics | Bone Regeneration - genetics | Bone Morphogenetic Protein 7 - genetics | Genes, jun - genetics | Humans | Data Mining | Insulin-Like Growth Factor I - genetics | Interleukin-1beta - genetics | NF-kappa B p50 Subunit - genetics | Vascular Endothelial Growth Factor A - genetics | Collagen Type I - genetics | Bone Resorption - genetics | Jaw Diseases - genetics | Matrix Metalloproteinase 9 - genetics | Osteoporosis - genetics | Multigene Family - genetics | Bone Morphogenetic Protein 2 - genetics | Interleukin-6 - genetics | Computational Biology | Transforming Growth Factor beta1 - genetics | beta Catenin - genetics | RANK Ligand - genetics | Intercellular Adhesion Molecule-1 - genetics | Interleukin-10 - genetics | Bone Remodeling - genetics | Core Binding Factor Alpha 1 Subunit - genetics | Medical colleges | Analysis | Genes | Online searching | Genetic research | Internet/Web search services | Database searching | Data mining
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2015, Volume 125, Issue 2, pp. 665 - 680
Cellular lipid metabolism has been linked to immune responses; however, the precise mechanisms by which de novo fatty acid synthesis can regulate inflammatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | GLUCOSE-METABOLISM | ATP-CITRATE LYASE | UCP2 | SEPTIC SHOCK | CELL-GROWTH | CRITICALLY-ILL PATIENTS | INTENSIVE INSULIN THERAPY | CANCER | MITOCHONDRIAL UNCOUPLING PROTEINS | Fatty Acid Synthase, Type I - genetics | Fatty Acid Synthase, Type I - biosynthesis | Lipids - genetics | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Ion Channels - genetics | Caspase 1 - metabolism | Mitochondrial Proteins - genetics | Interleukin-1beta - genetics | Lipids - biosynthesis | Sepsis - pathology | Interleukin-1beta - metabolism | Mitochondrial Proteins - metabolism | Sepsis - metabolism | Macrophages - pathology | Down-Regulation - genetics | Inflammasomes - genetics | Sepsis - genetics | Mice, Knockout | Carrier Proteins - genetics | Enzyme Induction - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Sepsis - chemically induced | Ion Channels - metabolism | Caspase 1 - genetics | Sepsis - therapy | Mice | Uncoupling Protein 2 | Interleukin-18 - genetics | Interleukin-18 - metabolism | Cellular proteins | Immune response | Synthesis | Sepsis | Development and progression | Lipids | Genetic aspects | Properties | Enzymes | Cytokines | Pathogenesis | Mortality | Glucose | Metabolism | Gene expression | Fatty acids | Proteins | Studies | Metabolites | Consent | Rodents
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e89019
...), which are critical events in AD pathology. Although genetic studies have focused on the association between the miR146a gene and susceptibility to several diseases, no association study of miR146a variability with AD has been conducted... 
CELLS | DIFFERENTIAL EXPRESSION | MULTIDISCIPLINARY SCIENCES | CANCER SUSCEPTIBILITY | PRE-MIR-146A GENE | RS2910164 | MIR-146A | NF-KAPPA-B | THAN-C POLYMORPHISM | TRANSCRIPTION FACTOR | BRAIN | Gene Frequency - genetics | Genetic Predisposition to Disease - genetics | Interleukin-6 - genetics | Humans | Asian Continental Ancestry Group - genetics | Genotype | Male | Risk | Cognition | Cognition Disorders - genetics | Interleukin-1beta - genetics | Alzheimer Disease | Promoter Regions, Genetic - genetics | Case-Control Studies | Alleles | Polymorphism, Single Nucleotide - genetics | Female | Aged | MicroRNAs - genetics | Advertising executives | Medical research | MicroRNA | Genes | Physiological aspects | Medicine, Experimental | Disease susceptibility | Genetic aspects | Inflammation | Alzheimer's disease | Risk factors | Genetic polymorphisms | Brain | Intensive care | Laboratories | Pathogenesis | Cognitive ability | Cardiovascular disease | Nervous system | Family medical history | Gene polymorphism | Skin cancer | Lipopolysaccharides | Interleukin 6 | Peripheral blood | Population | miRNA | Amyloid | Age | Genotypes | Chronic illnesses | Immune system | Immune response | Neurodegenerative diseases | Cytokines | Blood cells | Melanoma | Health risks | Metabolism | Gene expression | Ribonucleic acid--RNA | Patients | Disease control | Studies | Hospitals | Gene frequency | MicroRNAs | Mutation | Polymorphism | RNA | Ribonucleic acid
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2009, Volume 284, Issue 36, pp. 24035 - 24048
The role of endogenous inducers of inflammation is poorly understood. To produce the proinflammatory master cytokine interleukin (IL)-1β, macrophages need... 
GENE | LEUCINE-RICH PROTEOGLYCANS | CASPASE-1 | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE IMMUNE-RESPONSE | HYALURONAN | IL-1-BETA | SECRETION | ATP | NALP3 INFLAMMASOME | Toll-Like Receptor 2 - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Immunity, Innate - genetics | Extracellular Matrix - metabolism | Caspase 1 - metabolism | Male | Ureteral Obstruction - metabolism | Interleukin-1beta - genetics | Inflammation - metabolism | Kidney - metabolism | Carrier Proteins - immunology | Signal Transduction | Proteoglycans - immunology | Extracellular Matrix Proteins - genetics | Proteoglycans - metabolism | Toll-Like Receptor 4 - genetics | Biglycan | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Ureteral Obstruction - immunology | Macrophages - metabolism | Receptors, Purinergic P2 - metabolism | Toll-Like Receptor 2 - immunology | Mice | Proteoglycans - genetics | Extracellular Matrix - immunology | Kidney - immunology | Caspase 1 - immunology | Extracellular Matrix - genetics | Receptors, Purinergic P2 - genetics | Ureteral Obstruction - genetics | Interleukin-1beta - biosynthesis | Lung - metabolism | Receptors, Purinergic P2 - immunology | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Carrier Proteins - biosynthesis | Interleukin-1beta - immunology | Inflammation - immunology | Protein Structure, Tertiary - genetics | Receptors, Purinergic P2X7 | Carrier Proteins - genetics | Immunity, Innate - immunology | Animals | Caspase 1 - genetics | Extracellular Matrix Proteins - immunology | Receptors, Purinergic P2X4 | Inflammation - genetics | Lung - immunology | Animal models | Reactive oxygen species | Oligomerization | Proteoglycans | Transcription | Lung | Inflammation | Macrophages | Kidney | Stress | Caspase-1 | Purine P2X receptors | Signal transduction | Cell activation | TLR2 protein | Interleukin 1 | Toll-like receptors | Extracellular matrix | Sepsis | Cooperativity | Injuries | Glycobiology and Extracellular Matrices
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 02/2014, Volume 53, Issue 2, pp. 85 - 97
... periods of time may induce DNA damage . This DNA damage includes point mutations in cancer‐associated genes and may result in the genetic alteration of essential... 
androgen | loss of p53 | inflammation | DNA damage | ROS | prostate | NFkB | Loss of p53 | Androgen | Inflammation | Prostate | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTATHIONE-PEROXIDASE | CANCER | ONCOLOGY | GENES | NKX3.1 | LINK | EXPRESSION | Proto-Oncogene Proteins c-mdm2 - genetics | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Antioxidants - metabolism | Homeodomain Proteins - metabolism | Humans | Transcriptional Activation - drug effects | Tumor Necrosis Factor-alpha - genetics | Apoptosis - genetics | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Prostatitis - metabolism | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | Phosphorylation - genetics | Acetylcysteine - analogs & derivatives | Interleukin-1beta - metabolism | Matrix Metalloproteinase 9 - genetics | U937 Cells | Prostate-Specific Antigen - metabolism | Phosphorylation - drug effects | Interleukin-6 - metabolism | Prostate-Specific Antigen - genetics | Glutathione Peroxidase - metabolism | Lysine - analogs & derivatives | Interleukin-6 - genetics | Down-Regulation - genetics | Glutathione Peroxidase - genetics | Prostatitis - drug therapy | Androgens - genetics | Macrophages - metabolism | Receptors, Androgen - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Androgens - metabolism | Tumor Necrosis Factor-alpha - metabolism | Prostatic Neoplasms - metabolism | Receptors, Androgen - metabolism | I-kappa B Proteins - metabolism | I-kappa B Proteins - genetics | Prostatitis - pathology | Tumor Suppressor Protein p53 - genetics | Cell Differentiation - genetics | Prostatic Neoplasms - genetics | Inflammation Mediators - metabolism | Proto-Oncogene Proteins c-mdm2 - metabolism | Prostatic Neoplasms - pathology | Kallikreins - genetics | Tumor Suppressor Protein p53 - metabolism | Antioxidants - pharmacology | Transcription Factors - genetics | Down-Regulation - drug effects | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Up-Regulation - drug effects | Lysine - pharmacology | NF-kappa B - genetics | Cell Differentiation - drug effects | Acetylcysteine - pharmacology | Inflammation - genetics | Macrophages - drug effects | Kallikreins - metabolism | Ubiquitin | Analysis | Kallikrein | Hormones, Sex | Tumor proteins | Mitogens | Androgens | Cytokines | Molecular biology | Gene expression | Prostate cancer | Index Medicus
Journal Article