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PLoS pathogens, ISSN 1553-7366, 09/2013, Volume 9, Issue 9, pp. e1003615 - e1003615
Respiratory virus infections, such as influenza, typically induce a robust type I (pro-inflammatory cytokine) immune response, however, the production of type... 
Parasitology | Life Sciences & Biomedicine | Microbiology | Science & Technology | Virology | Influenza A Virus, H1N1 Subtype - immunology | Up-Regulation | Eosinophils - metabolism | Macrophages, Alveolar - pathology | Killer Cells, Natural - pathology | Eosinophils - immunology | Interleukins - metabolism | Protein Subunits - metabolism | Receptors, Interleukin - metabolism | Lung - virology | Killer Cells, Natural - immunology | Eosinophils - pathology | Lung - metabolism | Respiratory Tract Infections - virology | Macrophages, Alveolar - immunology | Respiratory Tract Infections - pathology | Macrophages, Alveolar - virology | Eosinophilia - etiology | Lung - pathology | Respiratory Tract Infections - metabolism | Specific Pathogen-Free Organisms | Interleukin-33 | Interleukin-5 - metabolism | Killer Cells, Natural - virology | Orthomyxoviridae Infections - metabolism | Eosinophils - virology | Mice, Inbred C57BL | Orthomyxoviridae Infections - pathology | Cells, Cultured | Immunity, Innate | Mice, Knockout | Animals | Respiratory Tract Infections - immunology | Mice, Inbred BALB C | Killer Cells, Natural - metabolism | Macrophages, Alveolar - metabolism | Orthomyxoviridae Infections - virology | Lung - immunology | Orthomyxoviridae Infections - immunology | Killer cells | Interleukins | Influenza | Physiological aspects | Host-parasite relationships | Research | Health aspects | Index Medicus | T cell receptors | Immunology | Lungs | Rodents | Immune system
Journal Article
PloS one, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, pp. e0137699 - e0137699
The transcription factor PU.1 is predominantly expressed in dendritic cells (DCs) and is essential for DC differentiation. Although there are several reports... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | GATA3 Transcription Factor - genetics | RNA, Small Interfering - genetics | Exons | Interleukin-5 - genetics | Gene Knockdown Techniques | Interleukin-13 - genetics | Nucleotide Motifs | Trans-Activators - genetics | Acetylation | Cytokines - genetics | Dendritic Cells - metabolism | GATA2 Transcription Factor - genetics | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | Cytokines - metabolism | Interleukin-5 - metabolism | Gene Expression Regulation | Gene Silencing | Histone Deacetylases - metabolism | Proto-Oncogene Proteins - genetics | Interleukin-13 - metabolism | GATA1 Transcription Factor - metabolism | Th2 Cells - metabolism | Amino Acid Motifs | Animals | GATA1 Transcription Factor - genetics | GATA2 Transcription Factor - metabolism | Protein Binding | Trans-Activators - metabolism | Mice | Histones - metabolism | Promoters (Genetics) | Research | Cytokines | Dendritic cells | Neutrophils | Histone deacetylase | Chromatin | Transcription factors | Immunoprecipitation | Laboratories | Science | Lymphocytes T | Lipopolysaccharides | Recruitment | PU.1 protein | Immunology | Interleukin 5 | Rodents | DNA methylation | Bone marrow | Trichostatin A | Interleukin 13 | siRNA | Gene expression | Medicine | Gene silencing | GATA-3 protein | Molecular biology | Histone H3 | Index Medicus
Journal Article
Journal of Applied Physiology, ISSN 1522-1601, 10/2009, Volume 107, Issue 4, pp. 1285 - 1292
We showed recently that IL-4 causes mitochondrial dysfunction in allergic asthma. IL-4 is also known to induce 12/15-lipoxygenase (12/15-LOX), a potent... 
Mitochondria | 12/15-lipoxygenase | Physiology | Sport Sciences | Life Sciences & Biomedicine | Science & Technology | Bronchial Hyperreactivity - immunology | Hyperplasia | Transforming Growth Factor beta1 - metabolism | Hypersensitivity - immunology | Male | Mitochondria - immunology | Hypersensitivity - drug therapy | Immunoglobulin E - blood | Electron Transport Complex IV - metabolism | Asthma - immunology | Disease Models, Animal | 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid - metabolism | Anti-Asthmatic Agents - pharmacology | Asthma - physiopathology | Lung - physiopathology | Pulmonary Fibrosis - pathology | Interleukin-13 - metabolism | Linoleic Acids - metabolism | Bronchial Hyperreactivity - pathology | Hypersensitivity - physiopathology | Hypersensitivity - pathology | Lung - drug effects | Mice | Mice, Inbred BALB C | Oxidative Stress - drug effects | Asthma - pathology | Vitamin E - pharmacology | Asthma - metabolism | Arachidonate 12-Lipoxygenase - metabolism | Bronchial Hyperreactivity - drug therapy | Bronchial Provocation Tests | Goblet Cells - drug effects | Mitochondria - ultrastructure | Hypersensitivity - metabolism | Bronchial Hyperreactivity - physiopathology | Lung - ultrastructure | Bronchial Hyperreactivity - metabolism | Lung - metabolism | Ovalbumin | Interleukin-5 - metabolism | Cytochromes c - metabolism | Interleukin-4 - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Arachidonate 15-Lipoxygenase - metabolism | Asthma - drug therapy | Animals | Airway Remodeling - drug effects | Goblet Cells - pathology | Pulmonary Fibrosis - drug therapy | Lung - immunology | Airway management | Cytokines | Vitamin E | Rodents | Gene expression | Allergies | Asthma | Index Medicus | Innovative Methodologies | mitochondria | 15-lipoxygenase
Journal Article
PloS one, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, pp. e24092 - e24092
We present immunological data from two clinical trials where the effect of experimental human hookworm (Necator americanus) infection on the pathology of... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Forkhead Transcription Factors - immunology | Immunohistochemistry | Leukocytes, Mononuclear - metabolism | Intestinal Mucosa - metabolism | Humans | Interleukin-17 - immunology | Interferon-gamma - metabolism | Interleukin-2 - immunology | Time Factors | Celiac Disease - immunology | Forkhead Transcription Factors - metabolism | Intestinal Mucosa - immunology | Leukocytes, Mononuclear - immunology | Duodenum - metabolism | Celiac Disease - parasitology | Necator americanus - immunology | Interleukin-2 Receptor alpha Subunit - immunology | Duodenum - immunology | Interleukin-2 - metabolism | CD4 Antigens - immunology | Necatoriasis - parasitology | Gliadin - immunology | Necatoriasis - pathology | Interleukin-5 - metabolism | Necatoriasis - immunology | Cells, Cultured | Necator americanus - physiology | Clinical Trials as Topic | Host-Parasite Interactions - immunology | Interleukin-5 - immunology | Celiac Disease - pathology | Interleukin-17 - metabolism | Animals | Interleukin-2 Receptor alpha Subunit - metabolism | Biopsy | Duodenum - pathology | Interferon-gamma - immunology | Leukocytes, Mononuclear - cytology | CD4 Antigens - metabolism | Infection | Celiac disease | Immune response | Interleukins | Interferon | Biological response modifiers | Comparative analysis | Health aspects | Gluten | Cell culture | Mucosa | Interleukin | Helper cells | Clinical trials | Inflammatory response | Lymphocytes T | Infections | Parasites | Small intestine | Interleukin 5 | Immunology | Interleukin 2 | Lymphocytes | Bowel disease | Gastroenterology | Gliadin | CD25 antigen | Foxp3 protein | Medical research | Cytokines | Inflammation | Crohns disease | CD4 antigen | Whooping cough | Studies | Pathology | Hospitals | Diet | γ-Interferon | Interleukin 10 | Autoimmune diseases | Index Medicus
Journal Article
Allergy (Copenhagen), ISSN 0105-4538, 07/2017, Volume 72, Issue 7, pp. 1105 - 1113
Background Bullous pemphigoid (BP) is an autoimmune bullous disease of the skin characterized by subepidermal blister formation due to tissue‐bound and... 
blister formation | eosinophils | bullous pemphigoid | degranulation | interleukin‐5 | interleukin-5 | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Eosinophils - metabolism | Reactive Oxygen Species - metabolism | Cytokines - metabolism | Eosinophils - drug effects | Interleukin-5 - metabolism | Skin - metabolism | Humans | Cell Adhesion - drug effects | Cell Degranulation - immunology | Eosinophils - immunology | Pemphigoid, Bullous - pathology | Receptors, IgG - metabolism | CD18 Antigens - metabolism | Cell Adhesion - immunology | Autoantibodies - immunology | Biomarkers | Blister - etiology | Blister - pathology | Eosinophils - pathology | Pemphigoid, Bullous - metabolism | Pemphigoid, Bullous - immunology | Skin - drug effects | Skin - pathology | Skin - immunology | Autoimmunity | Autoantibodies | Interleukins | Collagen | Analysis | Bullous pemphigoid | Skin | Immunity | Pemphigoid | Reactive oxygen species | Separation | Pathogenesis | Brefeldin A | Blood | Fragmentation | Cell activation | Interleukin 5 | Peripheral blood | Skin diseases | Lesions | Activation analysis | Binding | Antigens | Deoxyribonuclease | Oxygen | Leukocytes (eosinophilic) | Fragments | Adhesion | Allergies | Computer programs | Fc receptors | CD16 antigen | Microscopy | Degranulation | Light microscopy | CD18 antigen | Blistering | Software | Receptor mechanisms | Autoimmune diseases | Eosinophils | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2005, Volume 280, Issue 24, pp. 23243 - 23250
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2004, Volume 31, Issue 4, pp. 413 - 422
Journal Article