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Journal of Clinical Immunology, ISSN 0271-9142, 6/2011, Volume 31, Issue 3, pp. 472 - 478
Leptin, one of the adipokines, functions as a hormone and a cytokine. In this investigation, we show for the first time that leptin, in a... 
Medical Microbiology | Biomedicine | Immunology | Internal Medicine | Infectious Diseases | activation markers | cytokines | B cells | PROTEIN-KINASE | TRANSDUCER | RECEPTOR | PROLIFERATION | IMMUNOLOGY | BLOOD MONONUCLEAR-CELLS | DEFICIENCY | PI3K/AKT | IMMUNE-RESPONSES | T-CELLS | Autoimmunity | Interleukin-6 - analysis | Janus Kinase 2 - biosynthesis | Mitogen-Activated Protein Kinase 3 - analysis | Humans | p38 Mitogen-Activated Protein Kinases - biosynthesis | p38 Mitogen-Activated Protein Kinases - analysis | Dose-Response Relationship, Drug | Signal Transduction - immunology | Inflammation - metabolism | Mitogen-Activated Protein Kinases - analysis | Mitogen-Activated Protein Kinase 3 - biosynthesis | Mitogen-Activated Protein Kinase 6 - analysis | Leptin - pharmacology | Phosphorylation - drug effects | Interleukin-10 - analysis | B-Lymphocytes - metabolism | Janus Kinase 2 - analysis | Enzyme-Linked Immunosorbent Assay | Lymphocyte Activation | Cells, Cultured | Mitogen-Activated Protein Kinases - biosynthesis | Receptors, Leptin - immunology | STAT3 Transcription Factor - analysis | Inflammation - immunology | Tumor Necrosis Factor-alpha - analysis | STAT3 Transcription Factor - biosynthesis | B-Lymphocytes - drug effects | B-Lymphocytes - immunology | Mitogen-Activated Protein Kinase 6 - biosynthesis | Interleukin-6 - biosynthesis | Interleukin-10 - biosynthesis | Receptors, Leptin - metabolism | Tumor Necrosis Factor-alpha - biosynthesis | Index Medicus
Journal Article
The Journal of Immunology, ISSN 0022-1767, 01/2004, Volume 172, Issue 1, pp. 567 - 576
The signaling mechanism by which the anti-inflammatory cytokine IL-10 mediates suppression of proinflammatory cytokine synthesis remains largely unknown.... 
RHEUMATOID-ARTHRITIS | TUMOR-NECROSIS-FACTOR | DNA-BINDING | HUMAN NEUTROPHILS | TYROSINE PHOSPHORYLATION | INTERLEUKIN-10 RECEPTOR | GENE-EXPRESSION | KAPPA-B-ALPHA | IMMUNOLOGY | HUMAN MONOCYTES | MONONUCLEAR PHAGOCYTES | Protein Binding - genetics | Protein Biosynthesis | Interleukin-6 - antagonists & inhibitors | Humans | Tumor Necrosis Factor-alpha - genetics | Immunoglobulins - genetics | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Repressor Proteins - antagonists & inhibitors | Antigens, CD - metabolism | Trans-Activators - physiology | Protein Tyrosine Phosphatases - antagonists & inhibitors | RNA, Messenger - biosynthesis | Protein Tyrosine Phosphatases - genetics | Inflammation Mediators - physiology | Glycoproteins - genetics | DNA-Binding Proteins - physiology | Protein Tyrosine Phosphatases - biosynthesis | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction - genetics | DNA - metabolism | Down-Regulation - genetics | Macrophages - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 2 | Repressor Proteins - biosynthesis | Up-Regulation - immunology | Interleukin-10 - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Adenoviruses, Human - genetics | Interleukin-10 - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Phosphorylation | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Antigens, CD - biosynthesis | Receptors, Cell Surface | Receptors, IgG - biosynthesis | Receptors, IgG - antagonists & inhibitors | Interleukin-10 - physiology | Signal Transduction - immunology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Signaling Lymphocytic Activation Molecule Family Member 1 | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | RNA, Messenger - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type II | Trans-Activators - genetics | Inflammation Mediators - antagonists & inhibitors | Trans-Activators - biosynthesis | Immunoglobulins - biosynthesis | Macrophages - immunology | Inflammation Mediators - immunology | Receptors, Tumor Necrosis Factor - metabolism | Immune Sera - pharmacology | Proteins - physiology | Cells, Cultured | Glycoproteins - antagonists & inhibitors | Histocompatibility Antigens Class II - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - antagonists & inhibitors | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Up-Regulation - genetics | DNA-Binding Proteins - genetics | DNA - antagonists & inhibitors | Glycoproteins - biosynthesis | Suppressor of Cytokine Signaling 3 Protein | Down-Regulation - immunology | Interleukin-6 - biosynthesis | Receptors, Tumor Necrosis Factor - biosynthesis | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Genetic Vectors | DNA-Binding Proteins - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | SOCS-3 protein | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 2007, Volume 194, Issue 1, pp. 54 - 61
Abstract Interleukin (IL)-17-producing helper T cells may play a pivotal role in the pathogenesis of multiple sclerosis. Here, we examined the effects of IL-17... 
Neurology | Allergy and Immunology | EAE | Cytokine | IL-17 | Microglia | cytokine | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MULTIPLE-SCLEROSIS LESIONS | MESSENGER-RNA EXPRESSION | NEUROTROPHIC FACTOR | IMMUNOLOGY | NEUROSCIENCES | NERVE GROWTH-FACTOR | INTERLEUKIN-17 | NEURONAL DEATH | NITRIC-OXIDE | TNF-ALPHA | microglia | T-CELLS | Brain-Derived Neurotrophic Factor - genetics | Microglia - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | Interleukin-17 - physiology | Receptors, Interleukin-17 - genetics | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | Interleukin-23 - genetics | RNA, Messenger - biosynthesis | Chemokine CXCL2 - genetics | Brain-Derived Neurotrophic Factor - biosynthesis | Interleukin-1beta - biosynthesis | Multiple Sclerosis - metabolism | Autocrine Communication | Nitric Oxide - biosynthesis | Cells, Cultured - drug effects | Interleukin-6 - genetics | Microglia - drug effects | Receptors, Interleukin-17 - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - genetics | Interleukin-17 - biosynthesis | Recombinant Proteins - pharmacology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Interleukin-23 - biosynthesis | Interleukin-6 - biosynthesis | Cells, Cultured - metabolism | Chemokine CXCL2 - biosynthesis | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Multiple sclerosis | Interleukins | T cells | Nitric oxide | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 03/2011, Volume 2, Issue 1, pp. 240 - 240
The immune system can both promote and suppress cancer. Chronic inflammation and proinflammatory cytokines such as interleukin (IL)-1 and IL-6 are considered... 
LUNG-CANCER | INTERFERON-INDUCIBLE PROTEIN-10 | NECROSIS IN-VIVO | PROGNOSTIC-FACTORS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | CD4(+) T-CELLS | ADJUVANT THERAPY | CARCINOMA | EXPRESSION | Angiostatic Proteins - immunology | Immunohistochemistry | Neoplasms - metabolism | Interleukin-1alpha - immunology | Chemokine CXCL9 - biosynthesis | Tumor Microenvironment | Multiple Myeloma - immunology | Th2 Cells - immunology | Th1 Cells - immunology | CD4-Positive T-Lymphocytes - immunology | Chemokine CXCL10 - biosynthesis | Inflammation - metabolism | Interleukin-2 - immunology | Chemokine CXCL10 - immunology | Lymphoma, B-Cell - immunology | Interleukin-1beta - biosynthesis | Macrophages - immunology | Interleukin-12 - biosynthesis | Lymphoma, B-Cell - metabolism | Signal Transduction | Angiostatic Proteins - biosynthesis | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Interleukin-1beta - immunology | Chemokine CXCL9 - immunology | Inflammation - immunology | Macrophages - cytology | Mice, SCID | Multiple Myeloma - metabolism | Macrophages - metabolism | Multiple Myeloma - pathology | Animals | Neoplasms - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Interleukin-6 - immunology | Lymphoma, B-Cell - pathology | Cell Line, Tumor | Interleukin-6 - biosynthesis | Interleukin-1alpha - biosynthesis | Mice | Interleukin-2 - biosynthesis | Neoplasms - pathology | Interferon-gamma - biosynthesis | Index Medicus
Journal Article
Hypertension, ISSN 0194-911X, 09/2004, Volume 44, Issue 3, pp. 264 - 270
Angiotensin II (Ang II) upregulates vascular endothelial growth factor (VEGF) and activates vascular inflammation. However, the decisive role of VEGF in Ang... 
Growth substances | Endothelial growth factors | Angiotensin II | Remodeling | Arteriosclerosis | MACROPHAGE INFILTRATION | FACTOR VEGF | remodeling | NITRIC-OXIDE SYNTHESIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | endothelial growth factors | FACTOR-KAPPA-B | TUMOR ANGIOGENESIS | CHRONIC BLOCKADE | TYPE-1 RECEPTOR | arteriosclerosis | growth substances | angiotensin II | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | TISSUE ANGIOTENSIN | Genetic Therapy | Tetrazoles - pharmacology | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Male | Gene Expression Profiling | Vasculitis - prevention & control | Vascular Endothelial Growth Factor A - genetics | Angiotensin II Type 1 Receptor Blockers - pharmacology | Transforming Growth Factor beta - biosynthesis | Hypoxia-Inducible Factor 1, alpha Subunit | Interleukin-1 - biosynthesis | Vascular Endothelial Growth Factor Receptor-2 - genetics | Imidazoles - therapeutic use | Receptors, Chemokine - genetics | Vasculitis - chemically induced | Coronary Vessels - pathology | Natriuretic Peptide, Brain - genetics | Transforming Growth Factor beta1 | Hypoxia-Inducible Factor 1 | Interleukin-6 - genetics | Extracellular Matrix Proteins - genetics | Imidazoles - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Aorta - pathology | Macrophages - metabolism | Intercellular Adhesion Molecule-1 - genetics | Chemokine CCL2 - biosynthesis | Nonmuscle Myosin Type IIB | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Vascular Endothelial Growth Factor A - physiology | Hypertrophy | Interleukin-1 - genetics | Tunica Media - pathology | Vascular Endothelial Growth Factor A - biosynthesis | Extracellular Matrix Proteins - biosynthesis | Tunica Media - drug effects | Vasculitis - physiopathology | Recombinant Fusion Proteins - physiology | Ventricular Remodeling | Nuclear Proteins - biosynthesis | Cell Division | Intercellular Adhesion Molecule-1 - biosynthesis | Receptors, Chemokine - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Vascular Endothelial Growth Factor Receptor-2 - biosynthesis | Angiotensin II - toxicity | Nuclear Proteins - genetics | Olmesartan Medoxomil | Hypertrophy, Left Ventricular - etiology | Mice, Inbred C57BL | Extracellular Matrix Proteins - physiology | Natriuretic Peptide, Brain - biosynthesis | Chemokine CCL2 - genetics | Renin-Angiotensin System - physiology | Transcription Factors - biosynthesis | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Animals | Transforming Growth Factor beta - genetics | Interleukin-6 - biosynthesis | Myosin Heavy Chains | Tetrazoles - therapeutic use | DNA-Binding Proteins - biosynthesis | Receptors, CCR2 | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2011, Volume 300, Issue 4, pp. 1418 - 1426
Post-myocardial infarction (MI), chemokine homing of inflammatory cells into the injured left ventricle (LV) regulates ventricular remodeling, in part by... 
CC chemokines | Inflammation | Matrix metalloproteinases | RECRUITMENT | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ANGIOGENESIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INCREASES | ISLET XENOGRAFTS | HEART | inflammation | PERIPHERAL VASCULAR DISEASE | MICE | matrix metalloproteinases | EXPRESSION | LEFT-VENTRICULAR FUNCTION | Myocardial Infarction - genetics | Macrophage Activation - genetics | Antigens, CD - biosynthesis | Mannose-Binding Lectins - biosynthesis | Receptors, CCR5 - genetics | Arginase - biosynthesis | Male | Lectins, C-Type - biosynthesis | Gene Deletion | Myocardial Infarction - pathology | Ventricular Remodeling - genetics | Female | Interleukin-1beta - biosynthesis | Receptors, Cell Surface - biosynthesis | Receptors, CCR5 - physiology | Procollagen - biosynthesis | Transforming Growth Factor beta1 - biosynthesis | HSP47 Heat-Shock Proteins - biosynthesis | Antigens, Differentiation, Myelomonocytic - biosynthesis | Macrophages - pathology | Ventricular Remodeling - physiology | Collagen Type I - biosynthesis | Macrophages - metabolism | Animals | Interleukin-6 - biosynthesis | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Gene mutations | Physiological aspects | Genetic aspects | Research | Macrophages | Health aspects | Chemokine receptors | Heart attack | Risk factors | Proteins | Heart attacks | Collagen | Rodents | Metabolism | Cells | Tumors | Index Medicus | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2016, Volume 196, Issue 8, pp. 3421 - 3428
Journal Article
Journal Article
Biochemical Journal, ISSN 0264-6021, 08/2010, Volume 430, Issue 1, pp. 141 - 149
A paradoxical but common finding in the obesity clinic is the identification of individuals who can be considered 'inappropriately' healthy for their degree of... 
Obesity | Signal transduction | Insulin resistance | Inflammation | 3T3-L1 ADIPOCYTES | SER | PROINFLAMMATORY STATE | signal transduction | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MONONUCLEAR-CELLS | ALPHA | inflammation | RESISTANCE | insulin resistance | INSULIN-RECEPTOR SUBSTRATE-1 | obesity | Insulin - physiology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Obesity, Morbid - immunology | Male | Interleukin-1beta - genetics | I-kappa B Proteins - biosynthesis | I-kappa B Proteins - genetics | Proto-Oncogene Proteins c-akt - genetics | RNA, Messenger - biosynthesis | Inflammation - metabolism | Insulin Receptor Substrate Proteins - biosynthesis | Adult | Female | Interleukin-1beta - biosynthesis | Insulin Receptor Substrate Proteins - genetics | Obesity, Morbid - metabolism | Macrophages - immunology | Biomarkers - metabolism | Intra-Abdominal Fat - immunology | NF-KappaB Inhibitor alpha | Interleukin-6 - genetics | Insulin Resistance | Mitogen-Activated Protein Kinases - biosynthesis | Inflammation - immunology | Intra-Abdominal Fat - metabolism | Proto-Oncogene Proteins c-akt - biosynthesis | NF-kappa B - genetics | NF-kappa B - biosynthesis | Interleukin-6 - biosynthesis | Mitogen-Activated Protein Kinases - genetics | Tumor Necrosis Factor-alpha - biosynthesis | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 09/2014, Volume 135, Issue 5, pp. 1165 - 1177
Recent studies have identified molecular events characteristic of immunogenic cell death (ICD), including surface exposure of calreticulin (CRT), the heat... 
high hydrostatic pressure | dendritic cells | heat shock proteins | immunogenic cell death | cancer immunotherapy | APOPTOSIS | PHOTODYNAMIC THERAPY | OVARIAN-CANCER | CANCER-THERAPY | CHEMOTHERAPY | CALRETICULIN EXPOSURE | ADAPTIVE IMMUNITY | ONCOLOGY | ATP RELEASE | T-CELLS | HSP90 Heat-Shock Proteins - biosynthesis | Phosphorylation | Reactive Oxygen Species - metabolism | Antigens, CD - biosynthesis | Dendritic Cells - immunology | Humans | Membrane Glycoproteins - biosynthesis | Adenosine Triphosphate - secretion | Caspase 8 - metabolism | HMGB1 Protein - immunology | Endoplasmic Reticulum Stress - immunology | Enzyme Activation - immunology | HMGB1 Protein - secretion | T-Lymphocytes, Regulatory - immunology | HSP90 Heat-Shock Proteins - immunology | Eukaryotic Initiation Factor-2 - metabolism | Immunoglobulins - biosynthesis | Interleukin-12 - secretion | Calreticulin - biosynthesis | HSP70 Heat-Shock Proteins - biosynthesis | HSP70 Heat-Shock Proteins - immunology | Tumor Necrosis Factor-alpha - secretion | Phagocytosis - immunology | Hydrostatic Pressure | Interleukin-6 - secretion | Membrane Proteins - biosynthesis | Apoptosis - immunology | B7-2 Antigen - biosynthesis | Neoplasms - immunology | Cell Line, Tumor | Calreticulin - immunology | HLA-DR Antigens - biosynthesis | Proteins | Immune response | Dendritic cells | Cell death | Immunotherapy | Heat shock proteins | Biochemistry | T cells | Genetic translation | Tumors | Rodents | Immune system | Cancer | Apoptosis | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2015, Volume 195, Issue 3, pp. 944 - 952
Journal Article
Cancer Research, ISSN 0008-5472, 03/2010, Volume 70, Issue 6, pp. 2235 - 2244
Journal Article