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European Heart Journal, ISSN 0195-668X, 08/2014, Volume 35, Issue 29, pp. 1917 - 1924
Epidemiological research over the last 50 years has discovered a plethora of biomarkers (including molecules, traits or other diseases) that associate with... 
Coronary artery disease | Mendelian randomization | C-REACTIVE PROTEIN | FUTURE MYOCARDIAL-INFARCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | ESTER TRANSFER PROTEIN | CARDIOVASCULAR EVENTS | SUBTILISIN/KEXIN TYPE 9 | ISCHEMIC-HEART-DISEASE | PHOSPHOLIPASE A | DENSITY-LIPOPROTEIN CHOLESTEROL | LAPLACE-TIMI 57 | GENOME-WIDE ASSOCIATION | Triglycerides - physiology | Cholesterol Ester Transfer Proteins - physiology | Humans | Obesity - genetics | Cholesterol, HDL - physiology | C-Reactive Protein - metabolism | Cholesterol Ester Transfer Proteins - genetics | Mendelian Randomization Analysis | Diabetes Complications - genetics | Genetic Pleiotropy - genetics | Hypertension - genetics | Phospholipases A2 - physiology | Telomere - genetics | Fibrinogen - physiology | Obesity - complications | Receptors, Interleukin-6 - genetics | Risk Factors | Linkage Disequilibrium - genetics | Lipoprotein(a) - physiology | C-Reactive Protein - genetics | Cholesterol, LDL - physiology | Cholesterol, HDL - genetics | Randomized Controlled Trials as Topic | Cholesterol, LDL - genetics | Serum Amyloid P-Component - genetics | Genetic Markers - genetics | Coronary Artery Disease - genetics | Hypertension - complications | Polymorphism, Single Nucleotide - genetics | Triglycerides - genetics | Lipoprotein(a) - genetics | Phospholipases A2 - genetics
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 3660 - 3663
in this issue of the JCI, two reports provide intriguing new information on the role of the inflammatory cytokine IL-6 in breast and lung cancer. The study by... 
BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | LUNG-CANCER | PATHWAY | STAT3 | NOTCH | STEM/PROGENITOR CELLS | GROWTH-FACTOR | INTERLEUKIN-6 | IDENTIFICATION | TUMORIGENESIS | Receptor, Epidermal Growth Factor - genetics | Transcription, Genetic - drug effects | Interleukin-6 - antagonists & inhibitors | Carbonic Anhydrases - biosynthesis | Cytokine Receptor gp130 - genetics | Receptors, Notch - metabolism | Humans | Lung Neoplasms - metabolism | Antigens, Neoplasm - biosynthesis | Spheroids, Cellular - pathology | Neoplasm Proteins - pharmacology | Receptors, Notch - genetics | Janus Kinases - metabolism | RNA, Messenger - metabolism | Serrate-Jagged Proteins | Phosphorylation - drug effects | Neoplasm Proteins - genetics | Interleukin-6 - metabolism | Gene Expression Regulation, Neoplastic - genetics | STAT3 Transcription Factor - genetics | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | Membrane Proteins - genetics | Spheroids, Cellular - metabolism | Enzyme Inhibitors - pharmacology | Mammary Glands, Human - pathology | Lung Neoplasms - therapy | Signal Transduction - genetics | Autocrine Communication - genetics | Breast Neoplasms - genetics | RNA, Neoplasm - biosynthesis | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | RNA, Neoplasm - genetics | Stem Cells - pathology | Carcinoma - genetics | Mice | Mutation | Transcription, Genetic - genetics | Cell Hypoxia - genetics | Calcium-Binding Proteins - genetics | Neoplasm Transplantation | Carbonic Anhydrases - genetics | Lung Neoplasms - pathology | Autocrine Communication - drug effects | Mammary Glands, Human - metabolism | Neoplasm Proteins - metabolism | Stem Cells - metabolism | Breast Neoplasms - metabolism | Breast Neoplasms - therapy | Intercellular Signaling Peptides and Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | Female | Membrane Proteins - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Carcinoma - pathology | STAT3 Transcription Factor - metabolism | Carbonic Anhydrase IX | Calcium-Binding Proteins - metabolism | Lung Neoplasms - genetics | Antigens, Neoplasm - genetics | Cell Hypoxia - drug effects | Janus Kinases - genetics | Neoplasm Invasiveness | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Intercellular Signaling Peptides and Proteins - genetics | Up-Regulation - genetics | Up-Regulation - drug effects | Animals | Interleukin-6 - pharmacology | Breast Neoplasms - pathology | Interleukin-6 - biosynthesis | Carcinoma - therapy | Receptor, Notch3 | Carcinoma - metabolism | Antagonists (Biochemistry) | Epithelial tumors | Dosage and administration | Diagnosis | Research | Drug therapy | Health aspects | Interleukin-6
Journal Article
Blood, ISSN 0006-4971, 06/2011, Volume 117, Issue 23, pp. 6287 - 6296
B-cell receptor (BCR) signaling is aberrantly activated in chronic lymphocytic leukemia (CLL). Bruton tyrosine kinase (BTK) is essential to BCR signaling and... 
B-CELLS | SURVIVAL | APOPTOSIS | ACTIVATION | FLUDARABINE PLUS CYCLOPHOSPHAMIDE | RITUXIMAB | LYMPHOMA | PROLIFERATION | INHIBITOR | HEMATOLOGY | EXPRESSION | T-Lymphocytes - enzymology | Apoptosis - drug effects | Humans | Tumor Necrosis Factor-alpha - genetics | Cell Survival - genetics | Apoptosis - genetics | Male | NF-kappa B - metabolism | Neoplasm Proteins - antagonists & inhibitors | Receptors, Antigen, B-Cell - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | Neoplasm Proteins - genetics | Interleukin-6 - metabolism | Cell Survival - drug effects | Drug Screening Assays, Antitumor - methods | Interleukin-6 - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Gene Expression Regulation, Leukemic - drug effects | Pyrimidines - pharmacology | Leukemia, Lymphocytic, Chronic, B-Cell - enzymology | B-Cell Activating Factor - metabolism | CD40 Ligand - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Gene Expression Regulation, Enzymologic - genetics | Cell Line, Tumor | Receptors, Antigen, B-Cell - genetics | Mice | Leukemia, Lymphocytic, Chronic, B-Cell - drug therapy | Mitogen-Activated Protein Kinase 1 - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - metabolism | Gene Expression Regulation, Enzymologic - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Gene Expression Regulation, Leukemic - genetics | Protein-Tyrosine Kinases - genetics | CD40 Ligand - metabolism | MAP Kinase Signaling System - genetics | Female | Interleukin-4 - genetics | Protein-Tyrosine Kinases - biosynthesis | Pyrazoles - pharmacology | B-Lymphocytes - enzymology | Neoplasm Proteins - biosynthesis | Interleukin-4 - metabolism | Phosphatidylinositol 3-Kinases - genetics | Animals | MAP Kinase Signaling System - drug effects | NF-kappa B - genetics | B-Cell Activating Factor - genetics | Cell Proliferation - drug effects | Lymphoid Neoplasia
Journal Article
PLoS Genetics, ISSN 1553-7390, 10/2014, Volume 10, Issue 10, p. e1004758
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 2007, Volume 117, Issue 12, pp. 3988 - 4002
High serum levels of IL-6 correlate with poor outcome in breast cancer patients. However, no data are available on the relationship between IL-6 and mammary... 
CANCER-CELLS | INITIATING CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | GENE SIGNATURE | CARBONIC-ANHYDRASE-IX | STEM-CELLS | STEM/PROGENITOR CELLS | SELF-RENEWAL | GROWTH-FACTOR | EXTRACELLULAR PH | IN-VITRO PROPAGATION | Carbonic Anhydrases - biosynthesis | Carcinoma, Ductal - genetics | Receptors, Notch - metabolism | Carbonic Anhydrases - genetics | Carcinoma, Ductal - pathology | Humans | Antigens, Neoplasm - biosynthesis | Spheroids, Cellular - pathology | Neoplasm Proteins - pharmacology | Receptors, Notch - genetics | Autocrine Communication - drug effects | Mammary Glands, Human - metabolism | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Breast Neoplasms - metabolism | Serrate-Jagged Proteins | Female | Carcinoma, Ductal - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Interleukin-6 - metabolism | Gene Expression Regulation, Neoplastic - genetics | Carbonic Anhydrase IX | Jagged-1 Protein | Antigens, Neoplasm - genetics | Cell Hypoxia - drug effects | Calcium-Binding Proteins | Interleukin-6 - genetics | Neoplasm Invasiveness | Intercellular Signaling Peptides and Proteins | RNA, Messenger - genetics | Spheroids, Cellular - metabolism | Mammary Glands, Human - pathology | Up-Regulation - genetics | Autocrine Communication - genetics | Membrane Proteins | Up-Regulation - drug effects | Breast Neoplasms - genetics | RNA, Neoplasm - biosynthesis | Interleukin-6 - pharmacology | Breast Neoplasms - pathology | Cell Line, Tumor | RNA, Neoplasm - genetics | Stem Cells - pathology | Receptor, Notch3 | Cell Hypoxia - genetics | Breast cancer | Genetic aspects | Research | Gene expression | Health aspects | Interleukin-6
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e68434
Glycoprotein gp43 is an immunodominant diagnostic antigen for paracoccidioidomycosis caused by Paracoccidioides brasiliensis. It is abundantly secreted in... 
PROTEOLYTIC ACTIVITY | FUNGUS | MAJOR DIAGNOSTIC ANTIGEN | MULTIDISCIPLINARY SCIENCES | IMMUNODIFFUSION TESTS | 43-KILODALTON GLYCOPROTEIN | EXOCELLULAR COMPONENTS | 43 KDA GLYCOPROTEIN | INFLAMMATORY RESPONSE | BALB/C MICE | YEAST PHASE | Paracoccidioides - immunology | Tumor Necrosis Factor-alpha - metabolism | Lung - microbiology | Virulence Factors - genetics | Gene Expression - genetics | Tumor Necrosis Factor-alpha - genetics | Glycoproteins - metabolism | Paracoccidioidomycosis - metabolism | Virulence Factors - immunology | Mutation - immunology | Paracoccidioidomycosis - genetics | Fungal Proteins - immunology | Paracoccidioides - metabolism | Antigens, Fungal - genetics | Gene Expression - immunology | Paracoccidioidomycosis - immunology | Tumor Necrosis Factor-alpha - immunology | Paracoccidioides - genetics | Lung - metabolism | Interleukin-6 - metabolism | Glycoproteins - genetics | Genetic Vectors - immunology | Interleukin-6 - genetics | Antigens, Fungal - metabolism | Genetic Vectors - metabolism | RNA, Antisense - immunology | Antigens, Fungal - immunology | Fungal Proteins - genetics | Mutation - genetics | Genetic Vectors - genetics | Glycoproteins - immunology | Animals | Paracoccidioidomycosis - microbiology | Interleukin-6 - immunology | Virulence Factors - metabolism | Mice | Mice, Inbred BALB C | Lung - immunology | Fungal Proteins - metabolism | RNA, Antisense - genetics | Infection | Viral antibodies | Fibronectins | Antigens | Genetically modified organisms | Antibodies | Antisense RNA | B cells | Health aspects | Protein binding | Cell culture | Transformation | Yeast | Virulence | Cytology | Immunoblotting | Infections | Biology | Macrophages | Fibronectin | Fungi | Interleukin 6 | Proteins | Laminin | Extracellular matrix | Genetic modification | Killing | Pathogens | Glycoprotein gp43 | Immune response | Cytokines | Glycoprotein | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Mutants | Pathogenicity | Chemotherapy | Lungs | γ-Interferon | Monoclonal antibodies | Interferon | Diagnostic systems | South American blastomycosis | RNA | Ribonucleic acid
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 09/2012, Volume 342, Issue 3, pp. 654 - 664
Nodakenin, a coumarin isolated from the roots of Angelicae gigas, has been reported to possess neuroprotective, antiaggregatory, antibacterial, and... 
ACTIVATION | CYTOKINES | CYCLOOXYGENASE-2 | RAW264.7 CELLS | NITRIC-OXIDE SYNTHASE | KINASE | SEPTIC SHOCK | PHARMACOLOGY & PHARMACY | MEDIATORS | SEPSIS | ANGELICA-GIGAS | Transcription, Genetic - drug effects | Interleukin-6 - antagonists & inhibitors | Tumor Necrosis Factor-alpha - genetics | Male | NF-kappa B - metabolism | TNF Receptor-Associated Factor 6 - antagonists & inhibitors | Interleukin-1beta - genetics | Promoter Regions, Genetic - drug effects | Mitogen-Activated Protein Kinase Kinases - metabolism | Inflammation - metabolism | Phosphorylation - genetics | Drug Interactions | Ubiquitination - drug effects | Cyclooxygenase 2 - genetics | Interleukin-1beta - metabolism | Phosphorylation - drug effects | TNF Receptor-Associated Factor 6 - genetics | Interleukin-6 - metabolism | Interleukin-1beta - antagonists & inhibitors | NF-KappaB Inhibitor alpha | NF-kappa B - antagonists & inhibitors | Interleukin-6 - genetics | Anti-Inflammatory Agents - pharmacology | Dinoprostone - genetics | MAP Kinase Kinase Kinases - genetics | MAP Kinase Kinase Kinases - metabolism | Signal Transduction - genetics | Down-Regulation - genetics | Coumarins - pharmacology | Dinoprostone - metabolism | Macrophages - metabolism | Signal Transduction - drug effects | Nitric Oxide - genetics | Lipopolysaccharides - pharmacology | Mice | Transcription, Genetic - genetics | Dinoprostone - antagonists & inhibitors | Nitric Oxide Synthase Type II - metabolism | Tumor Necrosis Factor-alpha - metabolism | Mitogen-Activated Protein Kinase Kinases - genetics | Shock, Septic - prevention & control | I-kappa B Proteins - metabolism | I-kappa B Proteins - genetics | Shock, Septic - metabolism | I-kappa B Kinase - metabolism | I-kappa B Kinase - antagonists & inhibitors | Inflammation - drug therapy | Nitric Oxide Synthase Type II - antagonists & inhibitors | I-kappa B Proteins - antagonists & inhibitors | Shock, Septic - drug therapy | Glucosides - pharmacology | Nitric Oxide - antagonists & inhibitors | Mice, Inbred C57BL | Cells, Cultured | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | I-kappa B Kinase - genetics | Down-Regulation - drug effects | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Cyclooxygenase 2 - metabolism | TNF Receptor-Associated Factor 6 - metabolism | Inflammation - genetics | Macrophages - drug effects | Nitric Oxide - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Ubiquitination - genetics
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2010, Volume 126, Issue 2, pp. 338 - 346.e10
Background Regulatory T–cell dysfunction is associated with development of the complex genetic conditions atopy and asthma. Therefore, we hypothesized that... 
Allergy and Immunology | T regulatory cells | birth cohort | IgE | atopy | interaction | asthma | MDR | polymorphism | SERUM IGE | AIRWAY INFLAMMATION | UMCG Approved | CHILDREN | MULTIFACTOR-DIMENSIONALITY REDUCTION | PROMOTER POLYMORPHISMS | TRANSFORMING GROWTH-FACTOR-BETA-1 | ALLERGIC DISEASE | ASSOCIATION | IMMUNOGLOBULIN-E | Interaction | Birth cohort | Asthma | Atopy | Polymorphism | IMMUNOLOGY | ALLERGY | Forkhead Transcription Factors - immunology | Allergens - adverse effects | Receptors, Transforming Growth Factor beta - genetics | Prospective Studies | Receptors, Transforming Growth Factor beta - immunology | Toll-Like Receptor 2 - genetics | Humans | Genetic Loci - genetics | Child, Preschool | Infant | Male | Genetic Loci - immunology | Heme Oxygenase-1 - genetics | Transforming Growth Factor beta1 - immunology | Immunoglobulin E - immunology | Interleukin-2 - immunology | Interleukin-2 Receptor alpha Subunit - genetics | Asthma - immunology | Female | Receptors, Interleukin-6 - immunology | T-Lymphocytes - pathology | Child | Interleukin-2 Receptor alpha Subunit - immunology | Genetic Predisposition to Disease | Interleukin-6 - genetics | Receptors, Interleukin-6 - genetics | Protein-Serine-Threonine Kinases - genetics | Logistic Models | Transforming Growth Factor beta1 - genetics | Forkhead Transcription Factors - genetics | Asthma - genetics | Polymorphism, Genetic | Interleukin-2 - genetics | Interleukin-10 - genetics | Epistasis, Genetic - immunology | Heme Oxygenase-1 - immunology | Interleukin-6 - immunology | Toll-Like Receptor 2 - immunology | Protein-Serine-Threonine Kinases - immunology | T-Lymphocytes - immunology | Models, Genetic | Asthma - pathology | Interleukin-10 - immunology | Allergy | Genetic disorders | Child development | Asthma in children | Immunoglobulin E | Family medicine | Children | Transforming growth factors | T cells | Health aspects | Allergic reaction | Public health | Studies | Food allergies | Genes | Statistical methods | Allergies | Deoxyribonucleic acid--DNA | Milk
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2012, Volume 131, Issue 11, pp. 2553 - 2561
Estrogen receptor‐beta (ERβ) has been suggested to exert anti‐inflammatory and anti‐tumorigenic effects in the colon, providing a translational potential to... 
dextran sodium sulfate | estrogen receptor‐beta | colon cancer | azoxymethane | inflammatory bowel disease | estrogen receptor-beta | Estradiol - blood | Mucin-2 - genetics | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Colitis - genetics | Colorectal Neoplasms - genetics | Tumor Necrosis Factor-alpha - genetics | Colitis - pathology | NF-kappa B - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Estrogen Receptor beta - metabolism | Cell Differentiation - genetics | Inflammation - metabolism | Estrogen Receptor beta - genetics | Proliferating Cell Nuclear Antigen - genetics | Neoplasms - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Female | Interferon-gamma - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Mucin-2 - metabolism | Mucin-1 - metabolism | Interleukin-6 - genetics | Colon - pathology | Mice, Inbred C57BL | Caveolin 1 - genetics | Interleukin-17 - genetics | beta Catenin - metabolism | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Caveolin 1 - metabolism | Interleukin-17 - metabolism | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Colitis - metabolism | Inflammation - genetics | Mice | Colorectal Neoplasms - pathology | Neoplasms - pathology | Proliferating Cell Nuclear Antigen - metabolism | Mucin-1 - genetics | Nitric Oxide Synthase Type II - metabolism | Estrogen receptor-beta
Journal Article
Breast Cancer Research, ISSN 1465-5411, 08/2015, Volume 17, Issue 1, p. 112
Introduction: The steady increase in the incidence of obesity among adults has been paralleled with higher levels of obesity-associated breast cancer. While... 
STEM-CELLS | INVASION | ACTIVATION | ONCOLOGY | TISSUE | GROWTH | BODY-MASS INDEX | ALPHA | RISK | DIFFERENTIATION | EPIDEMIOLOGY | RNA, Small Interfering - genetics | Stromal Cells - pathology | Humans | Leptin - metabolism | Epithelial-Mesenchymal Transition - genetics | Stem Cells - metabolism | Breast - metabolism | Obesity - genetics | Cell Movement - genetics | Breast Neoplasms - metabolism | Adipose Tissue - metabolism | MCF-7 Cells | Plasminogen Activator Inhibitor 1 - metabolism | Female | Plasminogen Activator Inhibitor 1 - genetics | Breast - pathology | Gene Expression Regulation, Neoplastic - physiology | Interleukin-6 - metabolism | Gene Expression Regulation, Neoplastic - genetics | Cell Proliferation - genetics | Receptors, Estrogen - genetics | Interleukin-6 - genetics | Matrix Metalloproteinase 2 - metabolism | Stromal Cells - metabolism | Adipose Tissue - pathology | Carcinogenesis - genetics | Coculture Techniques - methods | Mice, SCID | Adipocytes - pathology | Adiposity - genetics | Obesity - metabolism | Obesity - pathology | Carcinogenesis - pathology | Matrix Metalloproteinase 2 - genetics | Neoplasm Metastasis - genetics | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Neoplasm Metastasis - pathology | Adipocytes - metabolism | Cell Line, Tumor | Stem Cells - pathology | Mice | Adipose tissues | Obesity | Care and treatment | Leptin | Genes | Estrogen | Stem cells | Phenols | Breast cancer | Metastasis | Gene expression | Cancer
Journal Article