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The New England Journal of Medicine, ISSN 0028-4793, 08/2011, Volume 365, Issue 7, pp. 620 - 628
Journal Article
Journal Article
Medicine (United States), ISSN 0025-7974, 03/2013, Volume 92, Issue 2, pp. 109 - 122
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 06/2009, Volume 360, Issue 23, pp. 2426 - 2437
Journal Article
Science, ISSN 0036-8075, 02/2017, Volume 355, Issue 6327, pp. 842 - 847
Journal Article
PLOS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0169681 - e0169681
Serum levels of choline and its derivatives are lower in patients with inflammatory bowel disease (IBD) than in healthy individuals. However, the effect of... 
DEXTRAN SULFATE SODIUM | INFLAMMATORY-BOWEL-DISEASE | CROHNS-DISEASE | FATTY LIVER-DISEASE | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | PARENTERAL-NUTRITION | NONALCOHOLIC STEATOHEPATITIS | KUPFFER CELL | HEPATIC STEATOSIS | PROSTAGLANDIN E-2 | Cytokines - metabolism | Mice, Inbred C57BL | Choline Deficiency - complications | Male | Interferon-gamma - metabolism | Inflammation - etiology | Dextran Sulfate - toxicity | Animals | Colitis - chemically induced | Inflammation - prevention & control | Mice | Colitis - immunology | Colitis - prevention & control | Disease Models, Animal | Natural Killer T-Cells - immunology | Choline | Analysis | Colitis | Research | Biological response modifiers | Health aspects | Risk factors | Health sciences | Inflammatory bowel diseases | Liver | Interleukin | Lymphocytes T | Lymph nodes | Interleukin 4 | Immunology | Lymphocytes | Intestine | Rodents | Hepatology | Surgery | Gastroenterology | Dextran sulfate | Physiology | Life sciences | Colon | Natural killer cells | Endoscopy | Pathogens | Liver diseases | Secretion | Methionine | Lamina propria | T cell receptors | Metabolism | Gene expression | Serum levels | CD1d antigen | Inflammatory bowel disease | Dextran | Nutrition research | Hospitals | Diet | Sodium | γ-Interferon | Interferon | Sulfate | Laboratory animals | Chemokines | Peritoneum | Index Medicus
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 02/2017, Volume 101, Issue 2, pp. 367 - 376
PI3Ks have been identified as key signaling proteins involved in many basic biologic processes in health and disease. Transgenic animals have been essential... 
influenza | mucin 2 | PI3K | GM‐CSF receptor | Mucin 2 | GM-CSF receptor | Influenza | ACTIVATION | PROTEIN | PATHOLOGY | IMMUNOLOGY | REGION | CELL BIOLOGY | GENE | GM-CSF | INFLAMMATION | FETAL MONOCYTES | HEMATOLOGY | SUBUNIT | DEVELOP | Disease Susceptibility | Macrophages, Alveolar - pathology | Signal Transduction | Mice, Inbred C57BL | Orthomyxoviridae Infections - pathology | Respiratory Tract Diseases - pathology | Male | Mutation - genetics | Respiratory Tract Diseases - genetics | Mice, Knockout | Animals | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Class Ib Phosphatidylinositol 3-Kinase - metabolism | Class Ib Phosphatidylinositol 3-Kinase - deficiency | Female | Macrophages, Alveolar - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor | Orthomyxoviridae Infections - virology | Orthomyxoviridae - physiology | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Respiratory Tract Diseases - virology | Crosses, Genetic | Colorectal carcinoma | Animal diseases | Viruses | Chains | Genomes | Infections | Macrophages | Inflammatory diseases | Proteins | Transgenic animals | Alveoli | Granulocyte-macrophage colony-stimulating factor | Colonies | Hemopoiesis | Signaling | Mucin | Molecular modelling | Mice | In vivo methods and tests | Interleukin 5 receptors | Mutation | Hematopoietic system | Auditory defects | Tumors | Cancer | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 12/2012, Volume 13, Issue 12, pp. 1178 - 1186
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, pp. e33747 - e33747
To evaluate the relevance of C-C chemokine receptor type 5 (CCR5) expression and tumor development, we compared melanoma growth in CCR5 knockout (CCR5(-/-))... 
INTERLEUKIN-1 | ANTAGONIST | INHIBITION | ADENOCARCINOMA | IN-VIVO | BIOLOGY | SKIN CARCINOGENESIS | HUMAN BREAST-CANCER | CELL GROWTH | EXPRESSION | CARCINOMA | Immunohistochemistry | Melanoma - metabolism | Receptors, CCR5 - deficiency | Receptors, CCR5 - genetics | Apoptosis - genetics | Interleukin 1 Receptor Antagonist Protein - genetics | NF-kappa B - metabolism | Melanoma - pathology | Blotting, Western | Mice, Knockout | Animals | NF-kappa B - genetics | Melanoma - genetics | Interleukin 1 Receptor Antagonist Protein - metabolism | Cell Line, Tumor | Mice | Apoptosis - physiology | CD57 antigen | Transcription factors | Bcl-2 protein | CD8 antigen | Cytotoxicity | Metastasis | Inactivation | Caspase-3 | Metastases | Skin cancer | Signal transduction | Cell growth | Animal tissues | Inhibition | Natural killer cells | Deoxyribonucleic acid--DNA | Carbon-carbon composites | Spleen | Medical research | NF-κB protein | CCR5 protein | Cytokines | Melanoma | Gene expression | Survival | Studies | Chemotherapy | Pharmacy | Weight reduction | Ligands | Infiltration | Lymphomas | Binding sites | Phosphorylation | Bax protein | Gene regulation | Lymphocytes T | Interleukin 1 receptors | Kinases | Killer cells | Rodents | Localization | Translocation | Deactivation | Interleukin 1 receptor antagonist | Poly(ADP-ribose) polymerase | Caspase | Breast cancer | Cell death | Adenoviruses | Prostate cancer | Chemokines | Apoptosis | Tumors | Index Medicus | CC chemokine receptors | Animal models | Transcription | Nuclear transport | NF- Kappa B protein | DNA | Deoxyribonucleic acid
Journal Article