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PLoS pathogens, ISSN 1553-7374, 2013, Volume 9, Issue 11, p. e1003773
Interferons (IFNs) are a group of cytokines with a well-established antiviral function. They can be induced by viral infection, are secreted and bind to... 
CELLS | DEFENSE | VIRUS-INFECTION | DISTINCT | RIG-I | MICROBIOLOGY | IRF-7 | ANTIVIRAL RESPONSE | VIROLOGY | GENE | SIGNALING PATHWAY | ADAPTER PROTEIN | PARASITOLOGY | Epithelial Cells - metabolism | Influenza A virus - genetics | Respiratory Mucosa - virology | Interferon Regulatory Factor-7 - genetics | Interferon Type I - immunology | Interferon Regulatory Factor-3 - genetics | Interleukins - metabolism | Orthomyxoviridae Infections - genetics | Respiratory Mucosa - pathology | Interleukins - genetics | Interleukins - immunology | Respiratory Mucosa - immunology | Adaptor Proteins, Signal Transducing - immunology | Interferon Type I - metabolism | Influenza A virus - immunology | Membrane Proteins - metabolism | Interferon Regulatory Factor-3 - immunology | Nerve Tissue Proteins - immunology | Membrane Proteins - genetics | Orthomyxoviridae Infections - metabolism | Epithelial Cells - pathology | Membrane Proteins - immunology | Interferon Regulatory Factor-7 - immunology | Interferon Regulatory Factor-7 - metabolism | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Influenza A virus - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | Adaptor Proteins, Signal Transducing - genetics | Interferon Regulatory Factor-3 - metabolism | Interferon Type I - genetics | Mice | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Orthomyxoviridae Infections - immunology | Epithelial cells | Influenza | Physiological aspects | Host-parasite relationships | Interferon | Genetic aspects | Genetic transcription | Research | Health aspects | Airway (Medicine) | Interleukins/metabolism | Nerve Tissue Proteins/immunology | Orthomyxoviridae Infections/genetics | Orthomyxoviridae Infections/metabolism | Membrane Proteins/genetics | Adaptor Proteins, Signal Transducing/genetics | Interferon Regulatory Factor-7/genetics | Interleukins/immunology | Membrane Proteins/immunology | Life Sciences | Orthomyxoviridae Infections/immunology | Influenza A virus/genetics | Nerve Tissue Proteins/metabolism | Respiratory Mucosa/immunology | Immunology | Interferon Type I/immunology | Epithelial Cells/immunology | Epithelial Cells/virology | Interferon Regulatory Factor-7/immunology | Epithelial Cells/metabolism | Interferon Regulatory Factor-3/immunology | Respiratory Mucosa/pathology | Influenza A virus/immunology | Interleukins/genetics | Interferon Regulatory Factor-3/genetics | Influenza A virus/metabolism | Adaptor Proteins, Signal Transducing/metabolism | Epithelial Cells/pathology | Adaptor Proteins, Signal Transducing/immunology | Interferon Type I/genetics | Interferon Regulatory Factor-3/metabolism | Membrane Proteins/metabolism | Nerve Tissue Proteins/genetics | Interferon Regulatory Factor-7/metabolism | Interferon Type I/metabolism | Respiratory Mucosa/metabolism | Respiratory Mucosa/virology | Cytokines | Genes | Rodents | Genomics | Genomes | Kinases | Experiments | Viral infections
Journal Article
Nature genetics, ISSN 1061-4036, 06/2013, Volume 45, Issue 6, pp. 664 - 669
Journal Article
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2014, Volume 134, Issue 6, pp. 1422 - 1432.e11
Background The initiation and regulation of pulmonary fibrosis are not well understood. IL-33, an important cytokine for respiratory diseases, is overexpressed... 
Allergy and Immunology | type 2 innate lymphoid cells | alternatively activated macrophages | IL-33 | lung fibrosis | IMMUNITY | RECRUITMENT | NEUTROPHIL | MOUSE | AIRWAY INFLAMMATION | BLEOMYCIN | CYTOKINE | MECHANISMS | IMMUNOLOGY | ALLERGY | IN-VIVO | PULMONARY-FIBROSIS | Interleukin-1 Receptor-Like 1 Protein | Lung - pathology | Receptors, Interleukin - immunology | Interleukin-33 | Mice, Inbred C57BL | Interleukin-13 - immunology | Transforming Growth Factor beta1 - genetics | Receptors, Interleukin - genetics | Mice, Knockout | Interleukins - genetics | Transforming Growth Factor beta1 - immunology | Interleukin-13 - genetics | Animals | Lymphocytes - immunology | Interleukins - immunology | Fibrosis | Lung - immunology | Macrophages, Alveolar - immunology | Macrophages | Transforming growth factors | Studies | Pulmonary fibrosis | Cytokines | Transgenic animals | Lymphocytes | Rodents | Collagen | Extracellular matrix | Software | Life Sciences | CT, Computed tomography | flIL-33, Full-length IL-33 | ICOS, Inducible costimulator | M2 macrophage, Alternatively activated macrophage | BAL, Bronchoalveolar lavage | Nos2, Inducible nitric oxide synthase 2 gene | WT, Wild-type | APC, Allophycocyanin | Arg1, Arginase 1 | IPF, Idiopathic pulmonary fibrosis | qPCR, Quantitative PCR | M1 macrophage, Classically activated macrophage | PE, Phycoerythrin | FITC, Fluorescein isothiocyanate | ILC2, Type 2 innate lymphoid cell | mIL-33, Mature IL-33 | BMDM, Bone marrow–derived macrophage | Mechanisms of Allergy and Clinical Immunology
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2009, Volume 206, Issue 1, pp. 35 - 41
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2015, Volume 136, Issue 2, pp. 312 - 322.e7
Background The mechanism underlying severe asthma with fungal sensitization (SAFS) is unknown. IL-33 is important in fungus-induced asthma exacerbations, but... 
Allergy and Immunology | steroid resistance | Severe asthma | innate immunity | IL-33 | fungal sensitization | pediatric | VORICONAZOLE | IMMUNITY | ALLERGENS | INNATE | ITRACONAZOLE | IMMUNOLOGY | CHILDREN | ALLERGY | EXACERBATIONS | MOLDS | LUNG INFLAMMATION | EXPOSURE | Interleukin-1 Receptor-Like 1 Protein | Anti-Asthmatic Agents - therapeutic use | Humans | Receptors, Interleukin - deficiency | Interleukin-13 - immunology | Omalizumab | Male | Th2 Cells - immunology | Interleukins - genetics | Interleukin-13 - genetics | Immunoglobulin E - immunology | Immunoglobulin E - genetics | Interleukins - immunology | Asthma - immunology | Female | Antibodies, Anti-Idiotypic - therapeutic use | Child | Th2 Cells - pathology | Disease Models, Animal | Animals, Newborn | Severity of Illness Index | Pyroglyphidae - chemistry | Antibodies, Monoclonal, Humanized - therapeutic use | Skin Tests | Receptors, Interleukin - immunology | Interleukin-33 | Asthma - drug therapy | Receptors, Interleukin - genetics | Alternaria - immunology | Cladosporium - immunology | Animals | Asthma - complications | Aspergillus fumigatus - immunology | Budesonide - therapeutic use | Mycoses - pathology | Adolescent | Mycoses - drug therapy | Mice | Mycoses - complications | Asthma - pathology | Mycoses - immunology | Pyroglyphidae - immunology | Pediatrics | Asthma in children | Fungi | Cytokines | Mortality | Smooth muscle | Inflammation | Allergies | Morbidity | Asthma | Milk | ABPA, Allergic bronchopulmonary aspergillosis | ICOS, Inducible costimulator | SPT, Skin prick test | sIgE, Specific IgE | BAL, Bronchoalveolar lavage | HDM, House dust mite | SAFS, Severe asthma with fungal sensitization | MMP-9, Matrix metalloproteinase 9 | Asthma and Lower Airway Disease | STRA, Severe therapy-resistant asthma | ILC, Innate lymphoid cell | AHR, Airway hyperresponsiveness
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 8, p. e11944
Background: IL-33 is a recently characterized IL-1 family cytokine and found to be expressed in inflammatory diseases, including severe asthma and inflammatory... 
IL-1-LIKE CYTOKINE IL-33 | HUMAN BASOPHILS | FAMILY-MEMBER | ACTIVATION | FC-EPSILON-RI | ST2 RECEPTOR | MULTIDISCIPLINARY SCIENCES | IN-VIVO | NECROSIS-FACTOR-ALPHA | INTERLEUKIN-33 | MICE | Interleukin-1 Receptor-Like 1 Protein | Calcium - metabolism | Humans | Interleukins - metabolism | Interleukins - genetics | Inflammation - metabolism | Immunoglobulin E - immunology | Mast Cells - metabolism | Female | Interleukins - biosynthesis | Mast Cells - immunology | Bone Marrow Cells - cytology | Gene Expression Regulation - immunology | Interleukin-33 | Rats | Inflammation - immunology | Mast Cells - drug effects | Receptors, Interleukin - genetics | Gene Expression Regulation - drug effects | Animals | Inflammation - genetics | Ionomycin - pharmacology | Mast Cells - cytology | Antigens - immunology | Mice | Immunoglobulin E | Skin | Inflammation | Cytokines | Analysis | Cell proliferation | Ionomycin | Transcription | Calcium | Epithelial cells | Inflammatory response | Lymphocytes T | Activation | Kinases | Inflammatory diseases | Blockage | Recruitment | Interleukin 6 | Proteins | Interleukin 4 | Anaphylaxis | Interleukin 5 | Immunology | Rodents | Interleukin 1 | Bone marrow | Fibroblasts | Trends | Edema | Antigens | Immunoglobulins | Immune response | Neutrophils | Gene expression | Allergies | Asthma | Studies | Mast cells | Passive cutaneous anaphylaxis | Chemokines | Peritoneum
Journal Article
Journal Article