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Publication DateGastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 3, pp. 882 - 892.e3
Background & Aims Mucosal natural killer (NK) cells that produce interleukin (IL)-22 mediate intestinal homeostasis and inflammation in mice. However, their...
Gastroenterology and Hepatology | Intestinal Macrophages | Intestinal NK Cells | ACTIVATION | STIMULATION | INTERLEUKIN 22 | ULCERATIVE-COLITIS | INNATE | MECHANISMS | PATHOGENESIS | LAMINA-PROPRIA MACROPHAGES | INFLAMMATORY-BOWEL-DISEASE | NK CELLS | GASTROENTEROLOGY & HEPATOLOGY | Coculture Techniques | Humans | Interferon-gamma - metabolism | Case-Control Studies | Interleukins - metabolism | Interleukin-23 - metabolism | Intestinal Mucosa - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Killer Cells, Natural - immunology | Macrophages - immunology | Killer Cells, Natural - microbiology | Enterococcus faecalis - immunology | CD56 Antigen - metabolism | Cells, Cultured | Cell Communication | Escherichia coli - immunology | Immunophenotyping | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | CD3 Complex - metabolism | Crohn Disease - immunology | Intestine, Large - immunology | Crohn Disease - pathology | Intestine, Large - pathology | Interleukin-7 Receptor alpha Subunit - metabolism | Natural Cytotoxicity Triggering Receptor 2 - metabolism | Natural Cytotoxicity Triggering Receptor 1 - metabolism | Intestinal Mucosa - pathology
Gastroenterology and Hepatology | Intestinal Macrophages | Intestinal NK Cells | ACTIVATION | STIMULATION | INTERLEUKIN 22 | ULCERATIVE-COLITIS | INNATE | MECHANISMS | PATHOGENESIS | LAMINA-PROPRIA MACROPHAGES | INFLAMMATORY-BOWEL-DISEASE | NK CELLS | GASTROENTEROLOGY & HEPATOLOGY | Coculture Techniques | Humans | Interferon-gamma - metabolism | Case-Control Studies | Interleukins - metabolism | Interleukin-23 - metabolism | Intestinal Mucosa - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Killer Cells, Natural - immunology | Macrophages - immunology | Killer Cells, Natural - microbiology | Enterococcus faecalis - immunology | CD56 Antigen - metabolism | Cells, Cultured | Cell Communication | Escherichia coli - immunology | Immunophenotyping | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | CD3 Complex - metabolism | Crohn Disease - immunology | Intestine, Large - immunology | Crohn Disease - pathology | Intestine, Large - pathology | Interleukin-7 Receptor alpha Subunit - metabolism | Natural Cytotoxicity Triggering Receptor 2 - metabolism | Natural Cytotoxicity Triggering Receptor 1 - metabolism | Intestinal Mucosa - pathology
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Loading RightsJournal of Controlled Release, ISSN 0168-3659, 01/2013, Volume 165, Issue 2, pp. 139 - 145
Most of the drugs used in the treatment of inflammatory bowel disease (IBD) become systemically bioavailable and potentially bear strong adverse effects....
Human | Nanoparticles | In vivo | Crohn's disease | Microparticles | Ulcerative colitis | CROHNS-DISEASE | ULCERATIVE-COLITIS | BARRIER | CHEMISTRY, MULTIDISCIPLINARY | DELIVERY | INFLAMMATORY-BOWEL-DISEASE | CHARGED LIPOSOMES | THERAPY | EXPERIMENTAL COLITIS | PHARMACOLOGY & PHARMACY | COLONIC-MUCOSA | Inflammatory Bowel Diseases - drug therapy | Nanoparticles - analysis | Colon - pathology | Rectum - pathology | Humans | Middle Aged | Male | Colitis, Ulcerative - pathology | Drug Carriers - analysis | Drug Delivery Systems | Young Adult | Inflammatory Bowel Diseases - pathology | Adult | Female | Aged | Colitis, Ulcerative - drug therapy | Intestinal Mucosa - pathology | Drugs | Medical research | Drug delivery systems | Medicine, Experimental | Vehicles
Human | Nanoparticles | In vivo | Crohn's disease | Microparticles | Ulcerative colitis | CROHNS-DISEASE | ULCERATIVE-COLITIS | BARRIER | CHEMISTRY, MULTIDISCIPLINARY | DELIVERY | INFLAMMATORY-BOWEL-DISEASE | CHARGED LIPOSOMES | THERAPY | EXPERIMENTAL COLITIS | PHARMACOLOGY & PHARMACY | COLONIC-MUCOSA | Inflammatory Bowel Diseases - drug therapy | Nanoparticles - analysis | Colon - pathology | Rectum - pathology | Humans | Middle Aged | Male | Colitis, Ulcerative - pathology | Drug Carriers - analysis | Drug Delivery Systems | Young Adult | Inflammatory Bowel Diseases - pathology | Adult | Female | Aged | Colitis, Ulcerative - drug therapy | Intestinal Mucosa - pathology | Drugs | Medical research | Drug delivery systems | Medicine, Experimental | Vehicles
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Loading Rights1992, Annals of the New York Academy of Sciences, ISBN 9780897667586, Volume 664., ix, 464
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Grass pollen immunotherapy induces Foxp3-expressing CD4+ CD25+ cells in the nasal mucosa
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2008, Volume 121, Issue 6, pp. 1467 - 1472.e1
Background Regulatory T (Treg) cells play an important role in controlling allergic inflammation. The transcription factor Foxp3 regulates the development and...
Allergy and Immunology | grass pollen immunotherapy | CD4 +CD25 + Treg cells | Foxp3 | IL-10 | hay fever/allergic rhinitis | CD4 | Treg cells | CD25 | TGF-BETA | IMMUNOLOGY | INTESTINAL INFLAMMATION | RHINITIS | CD4(+)CD25(+) Treg cells | MESSENGER-RNA | ALLERGY | EXPANSION | REGULATORY T-CELLS | EXPRESSION | MOLECULAR-MECHANISMS | Immunohistochemistry | T-Lymphocyte Subsets - immunology | Forkhead Transcription Factors - biosynthesis | Poaceae - immunology | T-Lymphocytes, Regulatory - metabolism | Humans | Palatine Tonsil - immunology | Palatine Tonsil - cytology | Randomized Controlled Trials as Topic | T-Lymphocytes, Regulatory - immunology | Rhinitis, Allergic, Seasonal - prevention & control | Nasal Mucosa - immunology | T-Lymphocyte Subsets - metabolism | Nasal Mucosa - cytology | Interleukin-10 - biosynthesis | Desensitization, Immunologic | Transcription factors | Peptides | Lymphocytes | Pollen | Grasses | Seasons | Allergies | Asthma
Allergy and Immunology | grass pollen immunotherapy | CD4 +CD25 + Treg cells | Foxp3 | IL-10 | hay fever/allergic rhinitis | CD4 | Treg cells | CD25 | TGF-BETA | IMMUNOLOGY | INTESTINAL INFLAMMATION | RHINITIS | CD4(+)CD25(+) Treg cells | MESSENGER-RNA | ALLERGY | EXPANSION | REGULATORY T-CELLS | EXPRESSION | MOLECULAR-MECHANISMS | Immunohistochemistry | T-Lymphocyte Subsets - immunology | Forkhead Transcription Factors - biosynthesis | Poaceae - immunology | T-Lymphocytes, Regulatory - metabolism | Humans | Palatine Tonsil - immunology | Palatine Tonsil - cytology | Randomized Controlled Trials as Topic | T-Lymphocytes, Regulatory - immunology | Rhinitis, Allergic, Seasonal - prevention & control | Nasal Mucosa - immunology | T-Lymphocyte Subsets - metabolism | Nasal Mucosa - cytology | Interleukin-10 - biosynthesis | Desensitization, Immunologic | Transcription factors | Peptides | Lymphocytes | Pollen | Grasses | Seasons | Allergies | Asthma
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Loading RightsJournal of Virology, ISSN 0022-538X, 10/2010, Volume 84, Issue 19, pp. 10354 - 10365
Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley...
LOW-LEVEL VIREMIA | INDIVIDUALS | CHRONIC INFECTION | VIROLOGY | IMMUNE-RESPONSES | MONONUCLEAR-CELLS | ANTIRETROVIRAL THERAPY | PERIPHERAL-BLOOD | HUMAN-IMMUNODEFICIENCY-VIRUS | LYMPHOCYTE RESPONSE | TYPE-1 ELITE CONTROLLERS | env Gene Products, Human Immunodeficiency Virus - immunology | HLA-B Antigens | Humans | Immunity, Mucosal | Male | HIV Long-Term Survivors | Rectum - virology | Viral Load | Host-Pathogen Interactions - immunology | HIV Infections - immunology | Intestinal Mucosa - immunology | HLA-B27 Antigen | Female | HIV Antigens | HIV Infections - blood | HIV Infections - virology | Intestinal Mucosa - virology | Rectum - immunology | Antiretroviral Therapy, Highly Active | HIV-1 - immunology | nef Gene Products, Human Immunodeficiency Virus - immunology | HIV Infections - drug therapy | CD8-Positive T-Lymphocytes - immunology | gag Gene Products, Human Immunodeficiency Virus - immunology | Immunodominant Epitopes | Pathogenesis and Immunity
LOW-LEVEL VIREMIA | INDIVIDUALS | CHRONIC INFECTION | VIROLOGY | IMMUNE-RESPONSES | MONONUCLEAR-CELLS | ANTIRETROVIRAL THERAPY | PERIPHERAL-BLOOD | HUMAN-IMMUNODEFICIENCY-VIRUS | LYMPHOCYTE RESPONSE | TYPE-1 ELITE CONTROLLERS | env Gene Products, Human Immunodeficiency Virus - immunology | HLA-B Antigens | Humans | Immunity, Mucosal | Male | HIV Long-Term Survivors | Rectum - virology | Viral Load | Host-Pathogen Interactions - immunology | HIV Infections - immunology | Intestinal Mucosa - immunology | HLA-B27 Antigen | Female | HIV Antigens | HIV Infections - blood | HIV Infections - virology | Intestinal Mucosa - virology | Rectum - immunology | Antiretroviral Therapy, Highly Active | HIV-1 - immunology | nef Gene Products, Human Immunodeficiency Virus - immunology | HIV Infections - drug therapy | CD8-Positive T-Lymphocytes - immunology | gag Gene Products, Human Immunodeficiency Virus - immunology | Immunodominant Epitopes | Pathogenesis and Immunity
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Loading RightsEnvironmental and Molecular Mutagenesis, ISSN 0893-6692, 08/2015, Volume 56, Issue 7, pp. 621 - 628
Exposure to high concentrations of hexavalent chromium [Cr(VI)] in drinking water was associated with an increased incidence of oral tumors in F344 rats in a...
transgenic rat | mode of action | oral cavity | hexavalent chromium [Cr(VI)] | Big Blue | 4‐nitroquinoline‐1‐oxide | in vivo mutation | 4-nitroquinoline-1-oxide | In vivo mutation | Oral cavity | Transgenic rat | Hexavalent chromium [Cr(VI)] | Mode of action | INTESTINAL EPITHELIA | Big Blue((R)) | MODEL | CANCER | HEXAVALENT CHROMIUM | INDUCED MUTATIONS | SALIVA | ENVIRONMENTAL SCIENCES | CARCINOGENESIS | GENETICS & HEREDITY | TOXICOLOGY | DRINKING-WATER | B6C3F1 MICE | EXPOSURE | Gene Frequency | Rats, Inbred F344 | Male | Carcinogens, Environmental - toxicity | Mouth Mucosa - drug effects | Mouth Neoplasms - chemically induced | Animals | Water Pollutants, Chemical - toxicity | DNA Mutational Analysis | Chromium - toxicity | Female | Mouth Mucosa - pathology | Mouth Neoplasms - genetics | Chromium | Genetic engineering
transgenic rat | mode of action | oral cavity | hexavalent chromium [Cr(VI)] | Big Blue | 4‐nitroquinoline‐1‐oxide | in vivo mutation | 4-nitroquinoline-1-oxide | In vivo mutation | Oral cavity | Transgenic rat | Hexavalent chromium [Cr(VI)] | Mode of action | INTESTINAL EPITHELIA | Big Blue((R)) | MODEL | CANCER | HEXAVALENT CHROMIUM | INDUCED MUTATIONS | SALIVA | ENVIRONMENTAL SCIENCES | CARCINOGENESIS | GENETICS & HEREDITY | TOXICOLOGY | DRINKING-WATER | B6C3F1 MICE | EXPOSURE | Gene Frequency | Rats, Inbred F344 | Male | Carcinogens, Environmental - toxicity | Mouth Mucosa - drug effects | Mouth Neoplasms - chemically induced | Animals | Water Pollutants, Chemical - toxicity | DNA Mutational Analysis | Chromium - toxicity | Female | Mouth Mucosa - pathology | Mouth Neoplasms - genetics | Chromium | Genetic engineering
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Loading Rights1983, ISBN 9780387117324, xii, 163
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Journal of Immunology Research, ISSN 2314-8861, 2015, Volume 2015, pp. 673815 - 7
Human and simian immunodeficiency virus (HIV and SIV) infections are characterized by manifestation of numerous opportunistic infections and inflammatory...
MEMORY | LYMPHOID-TISSUE | ACUTE SIV INFECTION | GASTROINTESTINAL-TRACT | IMMUNE ACTIVATION | ANIMAL-MODELS | HAIRY LEUKOPLAKIA | IMMUNOLOGY | HIV-1 INFECTION | MICROBIAL TRANSLOCATION | VIRAL REPLICATION | Immunity, Mucosal - immunology | HIV Infections - virology | Humans | Mouth Mucosa - immunology | CD4-Positive T-Lymphocytes - immunology | DNA, Viral - immunology | HIV Infections - immunology | Animals | Simian Acquired Immunodeficiency Syndrome - drug therapy | Simian Acquired Immunodeficiency Syndrome - immunology | Intestinal Mucosa - immunology | Mouth Mucosa - virology | Antiretroviral Therapy, Highly Active - methods | Simian Immunodeficiency Virus - drug effects | Immunologic Memory - immunology | Simian Immunodeficiency Virus - immunology
MEMORY | LYMPHOID-TISSUE | ACUTE SIV INFECTION | GASTROINTESTINAL-TRACT | IMMUNE ACTIVATION | ANIMAL-MODELS | HAIRY LEUKOPLAKIA | IMMUNOLOGY | HIV-1 INFECTION | MICROBIAL TRANSLOCATION | VIRAL REPLICATION | Immunity, Mucosal - immunology | HIV Infections - virology | Humans | Mouth Mucosa - immunology | CD4-Positive T-Lymphocytes - immunology | DNA, Viral - immunology | HIV Infections - immunology | Animals | Simian Acquired Immunodeficiency Syndrome - drug therapy | Simian Acquired Immunodeficiency Syndrome - immunology | Intestinal Mucosa - immunology | Mouth Mucosa - virology | Antiretroviral Therapy, Highly Active - methods | Simian Immunodeficiency Virus - drug effects | Immunologic Memory - immunology | Simian Immunodeficiency Virus - immunology
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Loading Rights1998, Old Herborn University seminar monograph, ISBN 3923022220, Volume 11., xvii, 129
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Inflammatory Bowel Diseases, ISSN 1078-0998, 04/2010, Volume 16, Issue 4, pp. 684 - 695
The short‐chain fatty acid butyrate, which is mainly produced in the lumen of the large intestine by the fermentation of dietary fibers, plays a major role in...
short‐chain fatty acid | monocarboxylate transporter 1 | colorectal cancer | butyrate | inflammatory bowel diseases | Short-chain fatty acid | Inflammatory bowel diseases | Monocarboxylate transporter 1 | Colorectal cancer | Butyrate | short-chain fatty acid | MONOCARBOXYLATE TRANSPORTER | CROHNS-DISEASE | NECROSIS-FACTOR-ALPHA | INTESTINAL EPITHELIAL-CELLS | QUIESCENT ULCERATIVE-COLITIS | CHAIN FATTY-ACIDS | ISOLATED RAT COLONOCYTES | TUMOR-SUPPRESSOR | PLACEBO-CONTROLLED TRIAL | GASTROENTEROLOGY & HEPATOLOGY | GASTROINTESTINAL PH PROFILES | Butyrates - metabolism | Intestinal Mucosa - metabolism | Animals | Biological Transport | Colon - pathology | Humans | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - metabolism | Colon - metabolism | Intestinal Mucosa - pathology | Life Sciences
short‐chain fatty acid | monocarboxylate transporter 1 | colorectal cancer | butyrate | inflammatory bowel diseases | Short-chain fatty acid | Inflammatory bowel diseases | Monocarboxylate transporter 1 | Colorectal cancer | Butyrate | short-chain fatty acid | MONOCARBOXYLATE TRANSPORTER | CROHNS-DISEASE | NECROSIS-FACTOR-ALPHA | INTESTINAL EPITHELIAL-CELLS | QUIESCENT ULCERATIVE-COLITIS | CHAIN FATTY-ACIDS | ISOLATED RAT COLONOCYTES | TUMOR-SUPPRESSOR | PLACEBO-CONTROLLED TRIAL | GASTROENTEROLOGY & HEPATOLOGY | GASTROINTESTINAL PH PROFILES | Butyrates - metabolism | Intestinal Mucosa - metabolism | Animals | Biological Transport | Colon - pathology | Humans | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - metabolism | Colon - metabolism | Intestinal Mucosa - pathology | Life Sciences
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Loading RightsJournal of Immunology, ISSN 0022-1767, 07/2018, Volume 201, Issue 2, pp. 725 - 733
The Th2-biased inflammation and immune deregulation play a critical role in the pathogenesis of ulcerative colitis (UC). Recent studies indicate that the...
B-CELLS | CYTOKINES | CROHNS-DISEASE | FOOD ALLERGY | ULCERATIVE-COLITIS | GENES | IMMUNOLOGY | TRANSCRIPTION FACTOR | EXPRESSION | GATA-3 | BOWEL-DISEASE | RNA, Small Interfering - genetics | Cytokines - metabolism | Lymphocyte Activation | Humans | Inflammatory Bowel Diseases - immunology | Cells, Cultured | Male | Inflammation - immunology | Th2 Cells - immunology | Young Adult | Proto-Oncogene Proteins c-bcl-2 - metabolism | Animals | Intestinal Mucosa - immunology | Muscle Proteins - metabolism | Adult | Female | Mice | Mice, Inbred BALB C | Flow cytometry | Deregulation | Inflammatory bowel diseases | Cytokines | Therapeutic applications | Pathogenesis | Mucosa | Lymphocytes T | Inflammation | Patients | Western blotting | CD4 antigen | Polymerase chain reaction | Proteins | Interleukin 4 | Lymphocytes | Intestine | Peripheral blood | GATA-3 protein | Colon | Ulcerative colitis | Apoptosis
B-CELLS | CYTOKINES | CROHNS-DISEASE | FOOD ALLERGY | ULCERATIVE-COLITIS | GENES | IMMUNOLOGY | TRANSCRIPTION FACTOR | EXPRESSION | GATA-3 | BOWEL-DISEASE | RNA, Small Interfering - genetics | Cytokines - metabolism | Lymphocyte Activation | Humans | Inflammatory Bowel Diseases - immunology | Cells, Cultured | Male | Inflammation - immunology | Th2 Cells - immunology | Young Adult | Proto-Oncogene Proteins c-bcl-2 - metabolism | Animals | Intestinal Mucosa - immunology | Muscle Proteins - metabolism | Adult | Female | Mice | Mice, Inbred BALB C | Flow cytometry | Deregulation | Inflammatory bowel diseases | Cytokines | Therapeutic applications | Pathogenesis | Mucosa | Lymphocytes T | Inflammation | Patients | Western blotting | CD4 antigen | Polymerase chain reaction | Proteins | Interleukin 4 | Lymphocytes | Intestine | Peripheral blood | GATA-3 protein | Colon | Ulcerative colitis | Apoptosis
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Loading Rights1990, ISBN 9780387970592, x, 217
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Gastrointestinal Endoscopy, ISSN 0016-5107, 2008, Volume 68, Issue 4, pp. S3 - S47
Gastroenterology and Hepatology | K-RAS MUTATIONS | DISTINCTIVE BIOLOGIC FEATURES | GASTROINTESTINAL EPITHELIAL NEOPLASIA | ULCERATIVE-COLITIS | NUCLEAR ANTIGEN PCNA | LATERALLY SPREADING TUMORS | RANDOMIZED CONTROLLED-TRIAL | SESSILE SERRATED ADENOMAS | NORTH-AMERICAN POPULATION | GASTROENTEROLOGY & HEPATOLOGY | ABERRANT CRYPT FOCI | Immunohistochemistry | Diagnosis, Differential | Adenocarcinoma - pathology | Prognosis | Adenomatous Polyps - pathology | Humans | Male | Colonoscopy - methods | Colorectal Neoplasms - physiopathology | Adenoma - physiopathology | Adenocarcinoma - physiopathology | Adenoma - pathology | Adenomatous Polyps - physiopathology | Female | Precancerous Conditions - pathology | Biopsy, Needle | Colorectal Neoplasms - pathology | Intestinal Mucosa - pathology | Antimitotic agents | Antineoplastic agents | Colorectal cancer
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Loading RightsClinical Cancer Research, ISSN 1078-0432, 09/2012, Volume 18, Issue 18, pp. 4919 - 4930
Purpose: miRNAs play a prominent role in a variety of physiologic and pathologic biologic processes, including cancer. For rectal cancers, only limited data...
EPITHELIAL-MESENCHYMAL TRANSITION | REAL-TIME PCR | BREAST-CANCER | PROFILES | ONCOLOGY | CARCINOMAS | SUPPRESSES TUMORIGENICITY | COLON | IDENTIFICATION | HUMAN COLORECTAL-CANCER | PROMOTES APOPTOSIS | Colonic Neoplasms - genetics | Intestinal Mucosa - metabolism | Reproducibility of Results | Prognosis | Rectum - pathology | Humans | Rectum - metabolism | Gene Expression Regulation, Neoplastic | Gene Expression Profiling | Gene Regulatory Networks | Rectal Neoplasms - mortality | Rectal Neoplasms - genetics | MicroRNAs - genetics | Rectal Neoplasms - pathology | Cluster Analysis | Intestinal Mucosa - pathology | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
EPITHELIAL-MESENCHYMAL TRANSITION | REAL-TIME PCR | BREAST-CANCER | PROFILES | ONCOLOGY | CARCINOMAS | SUPPRESSES TUMORIGENICITY | COLON | IDENTIFICATION | HUMAN COLORECTAL-CANCER | PROMOTES APOPTOSIS | Colonic Neoplasms - genetics | Intestinal Mucosa - metabolism | Reproducibility of Results | Prognosis | Rectum - pathology | Humans | Rectum - metabolism | Gene Expression Regulation, Neoplastic | Gene Expression Profiling | Gene Regulatory Networks | Rectal Neoplasms - mortality | Rectal Neoplasms - genetics | MicroRNAs - genetics | Rectal Neoplasms - pathology | Cluster Analysis | Intestinal Mucosa - pathology | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
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