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1. Full Text Inflammation and Colon Cancer
by Terzić, Janoš and Grivennikov, Sergei and Karin, Eliad and Karin, Michael
Gastroenterology, ISSN 0016-5085, 2010, Volume 138, Issue 6, pp. 2101 - 2114.e5
The connection between inflammation and tumorigenesis is well-established and in the last decade has received a great deal of supporting evidence from genetic,... 
Gastroenterology and Hepatology | Inflammation | Cytokines | Immunity | Colon Cancer | CHRONIC INTESTINAL INFLAMMATION | COLITIS-ASSOCIATED CANCER | SEVERE CROHNS-DISEASE | EPITHELIAL-MESENCHYMAL TRANSITION | TUMOR-NECROSIS-FACTOR | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | KINASE-I-EPSILON | TOLL-LIKE RECEPTORS | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | GENOME-WIDE ASSOCIATION | Colonic Neoplasms - genetics | Colonic Neoplasms - prevention & control | Colonic Neoplasms - drug therapy | Colorectal Neoplasms - genetics | Humans | Inflammatory Bowel Diseases - immunology | Gene Expression Regulation, Neoplastic | Colon - immunology | Cell Transformation, Neoplastic - genetics | Colonic Neoplasms - immunology | Colorectal Neoplasms - drug therapy | Inflammation Mediators - metabolism | Inflammatory Bowel Diseases - genetics | Anti-Inflammatory Agents - therapeutic use | Inflammatory Bowel Diseases - complications | Anticarcinogenic Agents - therapeutic use | Inflammatory Bowel Diseases - drug therapy | Genetic Predisposition to Disease | Cytokines - metabolism | Risk Assessment | Signal Transduction | Neoplasm Invasiveness | Risk Factors | Colorectal Neoplasms - prevention & control | Colonic Neoplasms - secondary | Cell Transformation, Neoplastic - immunology | Animals | Colorectal Neoplasms - immunology | Colorectal Neoplasms - secondary | Colon - microbiology | Dendritic cells | Measles-mumps-rubella vaccine | Biological response modifiers | Sulfates | Risk factors | Dextran | Prostaglandins | Colon cancer | Gastrointestinal diseases | Vascular endothelial growth factor | Growth factors | Tumors
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2. Full Text Akkermansia Muciniphila Protects Against Atherosclerosis by Preventing Metabolic Endotoxemia-Induced Inflammation in Apoe−/− Mice
by Li, Jin and Lin, Shaoqiang and Vanhoutte, Paul M and Woo, Connie W and Xu, Aimin
Circulation, ISSN 0009-7322, 06/2016, Volume 133, Issue 24, pp. 2434 - 2446
BACKGROUND—Altered composition of the gut microbiota is involved in both the onset and progression of obesity and diabetes mellitus. However, the link between... 
atherosclerosis | gut microbiota | endotoxemia | CARDIAC & CARDIOVASCULAR SYSTEMS | INTESTINAL MICROBIOTA | HUMAN GUT MICROBIOTA | FECAL MICROBIOTA | DIET-INDUCED OBESITY | BODY-WEIGHT | INSULIN-RESISTANCE | APOE-DEFICIENT MICE | CARDIOVASCULAR-DISEASE | FAT | PERIPHERAL VASCULAR DISEASE | HEALTH | Caco-2 Cells | Atherosclerosis - pathology | Inflammation - microbiology | Endotoxemia - prevention & control | Humans | Male | Mice, Transgenic | Treatment Outcome | Endotoxemia - etiology | Inflammation - etiology | Verrucomicrobia - physiology | Animals | Intestines - microbiology | Atherosclerosis - microbiology | Inflammation - prevention & control | Mice | Atherosclerosis - prevention & control | Disease Models, Animal | Endotoxemia - microbiology | Obesity | Complications and side effects | Care and treatment | Cardiovascular diseases | Atherosclerosis
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3. Full Text Mast cells and inflammation
by Theoharides, Theoharis C and Alysandratos, Konstantinos-Dionysios and Angelidou, Asimenia and Delivanis, Danae-Anastasia and Sismanopoulos, Nikolaos and Zhang, Bodi and Asadi, Shahrzad and Vasiadi, Magdalini and Weng, Zuyi and Miniati, Alexandra and Kalogeromitros, Dimitrios
BBA - Molecular Basis of Disease, ISSN 0925-4439, 2012, Volume 1822, Issue 1, pp. 21 - 33
Mast cells are well known for their role in allergic and anaphylactic reactions, as well as their involvement in acquired and innate immunity. Increasing... 
Skin inflammation | Multiple sclerosis | Mast cell | Selective release | CORTICOTROPIN-RELEASING HORMONE | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | FC-EPSILON-RI | MULTIPLE-SCLEROSIS PATIENTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | BLOOD-BRAIN-BARRIER | BIOPHYSICS | TOLL-LIKE RECEPTORS | CENTRAL-NERVOUS-SYSTEM | ANTIGEN-PRESENTING CELLS | ENDOTHELIAL GROWTH-FACTOR | Inflammation - pathology | Mast Cells - immunology | Humans | Stress, Physiological | Skin Diseases - immunology | Inflammation - immunology | Skin Diseases - pathology | Corticotropin-Releasing Hormone - metabolism | Inflammation - metabolism | Animals | Mast Cells - pathology | Inflammation Mediators - metabolism | Mast Cells - metabolism | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Phosphatases | Psoriasis | Leukemia | Immunoglobulin E | ACTH | Nerve growth factor | Inflammation | Neurotensin | Transforming growth factors | Biological response modifiers | T cells | Endothelium | Corticotropin releasing hormone | Protease inhibitors | Atopic dermatitis | Proteases | Stem cells | Lymphomas | Macrophage colony stimulating factor | Vasoactive intestinal peptides | Vascular endothelial growth factor | Mitogens | Myelin proteins
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4. From inflammation to cancer
: advances in diagnosis and therapy for gastrointestinal and hepatological diseases
by Cho, Chi Hin and Yu, Jun
2012, ISBN 9814343595, xv, 361
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5. Full Text Quercetin, inflammation and immunity
by Li, Yao and Yao, Jiaying and Han, Chunyan and Yang, Jiaxin and Chaudhry, Maria Tabassum and Wang, Shengnan and Liu, Hongnan and Yin, Yulong
Nutrients, ISSN 2072-6643, 03/2016, Volume 8, Issue 3, p. 167
In vitro and some animal models have shown that quercetin, a polyphenol derived from plants, has a wide range of biological actions including... 
Inflammation | Immune function | Dietary sources | Metabolism | Quercetin | MAJOR FOOD SOURCES | MITOCHONDRIAL BIOGENESIS | RATS | ADULTS | BIOAVAILABILITY | POLYPHENOLS | NUTRITION & DIETETICS | inflammation | immune function | IN-VIVO | dietary sources | GENE-EXPRESSION | quercetin | metabolism | NATURALLY-OCCURRING FLAVONOIDS | HUMAN MAST-CELLS | Humans | Biological Availability | Intestinal Absorption | Inflammation - metabolism | Anti-Inflammatory Agents - adverse effects | Quercetin - pharmacokinetics | Anti-Inflammatory Agents - therapeutic use | Immune System - immunology | Quercetin - administration & dosage | Anti-Inflammatory Agents - administration & dosage | Anti-Inflammatory Agents - pharmacokinetics | Dietary Supplements - adverse effects | Administration, Oral | Quercetin - therapeutic use | Treatment Outcome | Inflammation - immunology | Animals | Immune System - physiopathology | Diet | Immune System - metabolism | Immune System - drug effects | Quercetin - adverse effects | Inflammation - prevention & control | Recommended Dietary Allowances | Inflammation - physiopathology
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6. Full Text CD4+ T‐cell subsets in intestinal inflammation
by Shale, Matthew and Schiering, Chris and Powrie, Fiona
Immunological Reviews, ISSN 0105-2896, 03/2013, Volume 252, Issue 1, pp. 164 - 182
Summary Intestinal CD4+ T cells are essential mediators of immune homeostasis and inflammation. Multiple subsets of CD4+ T cells have been described in the... 
intestinal inflammation | Th1 | T‐cell differentiation | Th17 | Treg | Intestinal inflammation | T-cell differentiation | PATHOGENIC T(H)17 CELLS | IMMUNOLOGY | ACTIVE CROHNS-DISEASE | CHRONIC MUCOCUTANEOUS CANDIDIASIS | SEGMENTED FILAMENTOUS BACTERIA | BOWEL-DISEASE | HYPER-IGE SYNDROME | IN-VIVO | LAMINA PROPRIA LYMPHOCYTES | CYTOKINE GM-CSF | TH17 CELLS | T-Lymphocyte Subsets - immunology | Intestines - pathology | Humans | Inflammatory Bowel Diseases - immunology | Immunity, Mucosal | Homeostasis - immunology | CD4-Positive T-Lymphocytes - pathology | Intestines - immunology | CD4-Positive T-Lymphocytes - immunology | T-Lymphocyte Subsets - pathology | Intestinal Mucosa - immunology | Inflammatory Bowel Diseases - pathology | Metagenome - immunology | Cytokines - immunology | Intestinal Mucosa - pathology | T cells | Gastrointestinal system | Inflammation | Inflammatory bowel diseases | Translation | Intestine | Homeostasis | Gastrointestinal tract | Lymphocytes T | CD4 antigen | Invited Reviews
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7. Full Text Ginsenoside Rb1 and its metabolite compound K inhibit IRAK-1 activation—The key step of inflammation
by Joh, Eun-Ha and Lee, In-Ah and Jung, Il-Hoon and Kim, Dong-Hyun
Biochemical Pharmacology, ISSN 0006-2952, 08/2011, Volume 82, Issue 3, pp. 278 - 286
In the preliminary study, ginsenoside Rb1, a main constituent of the root of (family Araliaceae), and its metabolite compound K inhibited a key factor of... 
Interleukin-1 receptor-associated kinase-1 | Ginsenoside Rb1 | Compound K | CELLS | RECEPTOR | INTESTINAL BACTERIA | IN-VITRO | RB-1 | DISEASE | LIPOPOLYSACCHARIDE | PHARMACOLOGY & PHARMACY | MICE | NF-KAPPA-B | RAT PLASMA | Enzyme Inhibitors - pharmacology | Colitis - pathology | Male | Ginsenosides - metabolism | Ginsenosides - pharmacology | Mice, Inbred ICR | Inflammation - metabolism | Animals | Panax - chemistry | Colitis - chemically induced | Interleukin-1 Receptor-Associated Kinases - antagonists & inhibitors | Colitis - drug therapy | Mice | Molecular Structure | Trinitrobenzenesulfonic Acid - toxicity | Ginsenosides - chemistry | Metabolites
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