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Journal
Journal Article
Current issues in intestinal microbiology, ISSN 1466-531X, 2000
Journal
Gut, ISSN 0017-5749, 2016, Volume 65, Issue 2, pp. 330 - 339
Journal Article
The Journal of infectious diseases, ISSN 1537-6613, 2015, Volume 211, Issue 1, pp. 156 - 165
Background. A predominantly T-helper type 2 (Th2) immune response is critical in the prognosis of pulmonary Pseudomonas aeruginosa infection. But the mucosal... 
human-milk | cytokine production | butyrate | in-vitro | chain fatty-acids | galacto-oligosaccharides | t-cells | lactobacillus | virus-infection | cystic-fibrosis patients | intestinal microbiota | prebiotics | Th1 and M1 polarization | P. aeruginosa lung infection | INFECTIOUS DISEASES | VIRUS-INFECTION | GALACTO-OLIGOSACCHARIDES | MICROBIOLOGY | IMMUNOLOGY | CYTOKINE PRODUCTION | HUMAN-MILK | METABOLISM | CYSTIC-FIBROSIS PATIENTS | CHAIN FATTY-ACIDS | BUTYRATE | T-CELLS | LACTOBACILLUS | Intestines - drug effects | Lung Diseases - microbiology | GATA3 Transcription Factor - immunology | Pseudomonas aeruginosa - growth & development | Microbiota - drug effects | Male | T-Box Domain Proteins - immunology | Lung Diseases - drug therapy | Th1 Cells - immunology | Intestines - immunology | Leukocytes - immunology | Oligosaccharides - pharmacology | Leukocytes - microbiology | Tumor Necrosis Factor-alpha - immunology | Keratinocytes - microbiology | Macrophages - immunology | Macrophages - microbiology | Chemotactic Factors - immunology | Microbiota - immunology | Pseudomonas Infections - drug therapy | Pseudomonas aeruginosa - drug effects | Pseudomonas Infections - microbiology | Keratinocytes - immunology | Pectins - chemistry | Pseudomonas Infections - immunology | Lung Diseases - immunology | Interleukin-4 - immunology | Th1 Cells - microbiology | Animals | Intestines - microbiology | Interferon-gamma - immunology | Mice | Mice, Inbred BALB C | Interleukin-10 - immunology | Pseudomonas Infections/microbiology | Macrophages/microbiology | Oligosaccharides/pharmacology | Macrophages/immunology | Lung Diseases/immunology | Lung Diseases/microbiology | Interferon-gamma/immunology | Keratinocytes/microbiology | Microbiota/drug effects | Life Sciences | Leukocytes/microbiology | Pseudomonas Infections/immunology | Interleukin-10/immunology | Lung Diseases/drug therapy | T-Box Domain Proteins/immunology | Leukocytes/immunology | Pseudomonas aeruginosa/drug effects | Th1 Cells/microbiology | GATA3 Transcription Factor/immunology | Interleukin-4/immunology | Intestines/drug effects | Intestines/microbiology | Keratinocytes/immunology | Chemotactic Factors/immunology | Tumor Necrosis Factor-alpha/immunology | Microbiota/immunology | Pseudomonas aeruginosa/growth & development | Pectins/chemistry | Pseudomonas Infections/drug therapy | Th1 Cells/immunology | Intestines/immunology
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2007, Volume 2, Issue 7, p. e662
Background. Gut bacteria trigger colitis in animal models and are suspected to aggravate inflammatory bowel diseases. We have recently reported that... 
CELLS | MODELS | GENE | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | LIPOPOLYSACCHARIDE | MUCOSAL FLORA | TLR4 | EXPRESSION | Toll-Like Receptor 2 - genetics | Humans | Colitis - pathology | Colon - immunology | Bacterial Infections - complications | Inflammation - complications | Toll-Like Receptor 2 - deficiency | Bacterial Infections - genetics | Toll-Like Receptor 4 - deficiency | Inflammatory Bowel Diseases - genetics | Inflammatory Bowel Diseases - complications | Acute Disease | Inflammation - microbiology | Signal Transduction | Mice, Inbred C57BL | Neutrophils - physiology | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Bacterial Infections - physiopathology | Toll-Like Receptor 4 - physiology | Animals | Colitis - microbiology | Colitis - physiopathology | Bacterial Infections - pathology | Toll-Like Receptor 2 - physiology | Inflammation - genetics | Toll-Like Receptor 2 - immunology | Inflammatory Bowel Diseases - microbiology | Colon - microbiology | Inflammation - prevention & control | Mice | Colitis - prevention & control | Microbiota (Symbiotic organisms) | Gastrointestinal diseases | Escherichia coli | Bacteria | Inflammation | Colitis | T cells | Comparative analysis | Sulfates | Cell culture | Animal models | Inflammatory bowel diseases | Pathogenesis | Homeostasis | Microflora | Lymphocytes T | Kinases | Lymph nodes | Recruitment | Proteins | Receptors | E coli | Intestine | Rodents | Gastroenterology | Toll-like receptors | Foxp3 protein | Gram-negative bacteria | Colon | Intestinal microflora | Digestive tract | Immune system | Cytokines | Ileitis | Digestive system | Therapeutic applications | Lactobacilli | Leukocytes (neutrophilic) | TLR4 protein | Gene expression | Inflammatory bowel disease | Damage detection | Sodium | Flora | TLR2 protein | Biomarkers | Interferon | Sulfate | Adapter proteins | Hygiene
Journal Article
Expert review of anti-infective therapy, ISSN 1744-8336, 2014, Volume 9, Issue 12, pp. 1149 - 1156
Propionibacterium acnes is a Gram-positive bacterium that forms part of the normal flora of the skin, oral cavity, large intestine, the conjunctiva and the... 
infection | Propionibacterium acnes | inflammation | opportunistic pathogen | device related | skin commensal | INFECTIOUS DISEASES | PROSTHETIC VALVE ENDOCARDITIS | LYMPH-NODES | MICROBIOLOGY | SAPHO SYNDROME | QUANTITATIVE-ANALYSIS | GRANULOMA | COMPLETE GENOME SEQUENCE | DNA | LUMBAR SPONDYLODISCITIS | PHARMACOLOGY & PHARMACY | SARCOIDOSIS | BIOFILM FORMATION | Bone and Bones - pathology | Inflammation - pathology | Brain - microbiology | Skin - microbiology | Mouth - drug effects | Humans | Male | Bone and Bones - drug effects | Anti-Bacterial Agents - therapeutic use | Inflammation - complications | Prostate - pathology | Acquired Hyperostosis Syndrome - drug therapy | Inflammation - drug therapy | Acquired Hyperostosis Syndrome - pathology | Gram-Positive Bacterial Infections - microbiology | Prostate - drug effects | Gram-Positive Bacterial Infections - complications | Skin - pathology | Inflammation - microbiology | Acquired Hyperostosis Syndrome - complications | Propionibacterium acnes - physiology | Prostate - microbiology | Sarcoidosis - drug therapy | Bone and Bones - microbiology | Brain - drug effects | Gram-Positive Bacterial Infections - pathology | Sarcoidosis - complications | Mouth - microbiology | Prostatic Neoplasms - microbiology | Brain - pathology | Mouth - pathology | Sarcoidosis - microbiology | Acquired Hyperostosis Syndrome - microbiology | Gram-Positive Bacterial Infections - drug therapy | Propionibacterium acnes - drug effects | Anti-Bacterial Agents - administration & dosage | Skin - drug effects | Sarcoidosis - pathology | Infection | Implants, Artificial | Prosthesis | Genomics | Skin | Inflammation | Conjunctivitis | Toy industry | Heart | Brain | Synovitis | Virulence | Opportunist infection | Genomes | Eye | Mouth | Osteitis | Ear canal | Prosthetics | Shunts | Pathogens | Sarcoidosis | pustulosis | Acne | Conjunctiva | Joint diseases | Oral cavity | Reviews | Large intestine | Prostate | Prostate cancer | Cancer
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2008, Volume 320, Issue 5883, pp. 1647 - 1651
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 4, pp. 1220 - 1230.e3
Background & Aims Chronic infection with hepatitis B or C virus (HBV or HCV) is a leading cause of cirrhosis by unknown mechanisms of pathogenesis.... 
Gastroenterology and Hepatology | Hepatitis | Soluble CD14 | Intestinal Fatty Acid Binding Protein | Microbial Translocation | CELLS | ENDOTOXIN | LIVER-DISEASE | BACTEROIDES-FRAGILIS | ACID-BINDING-PROTEIN | C VIRUS-INFECTION | LIPOPOLYSACCHARIDE | GASTROENTEROLOGY & HEPATOLOGY | HEPATITIS-C | EXPRESSION | Liver Cirrhosis - immunology | Hepatitis B, Chronic - immunology | Humans | Middle Aged | Male | Monocytes - immunology | End Stage Liver Disease - microbiology | Intestines - immunology | Fatty Acid-Binding Proteins - blood | Enterocytes - pathology | Odds Ratio | Monocytes - microbiology | Hypertension, Portal - microbiology | Logistic Models | Biomarkers - blood | Hepatitis C, Chronic - diagnosis | Disease Progression | Lipopolysaccharide Receptors - blood | Host-Pathogen Interactions | Monocytes - virology | Intestines - microbiology | Hepatitis C, Chronic - microbiology | Maryland | Enterocytes - virology | Hepatitis B, Chronic - diagnosis | Hepatitis C, Chronic - complications | Kupffer Cells - virology | Lipopolysaccharides - blood | Interleukin-6 - blood | Cell Death | Intestines - virology | Female | Retrospective Studies | Hypertension, Portal - virology | Severity of Illness Index | Liver Cirrhosis - diagnosis | Intestines - pathology | Enzyme-Linked Immunosorbent Assay | Hepatitis B, Chronic - microbiology | Kupffer Cells - microbiology | Bacterial Translocation | Enterocytes - microbiology | Hepatitis B, Chronic - complications | Liver Cirrhosis - virology | Biopsy | Hepatitis C, Chronic - immunology | Liver Cirrhosis - microbiology | Limulus Test | End Stage Liver Disease - virology | Medical research | Phosphatases | Patient outcomes | Aspartate | Permeability | HIV (Viruses) | Transforming growth factors | Fatty acids | Medicine, Experimental | Kidney diseases | Hepatitis C virus | Mitogens | Hepatitis B | Protein binding | microbiota | histology | immune response | soluble CD14 | intestinal fatty acid binding protein | chronic liver disease | Microbial translocation | hepatitis
Journal Article