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1. Full Text Potassium transport and plant salt tolerance
by Shabala, Sergey and Cuin, Tracey A
Physiologia Plantarum, ISSN 0031-9317, 08/2008, Volume 133, Issue 4, pp. 651 - 669
Salinity is a major abiotic stress affecting approximately 7% of the world’s total land area resulting in billion dollar losses in crop production around the... 
ORYZA-SATIVA L | ARABIDOPSIS-THALIANA | GLUTAMATE RECEPTORS | COMPATIBLE SOLUTES | COMMON ICE PLANT | TRANSGENIC PLANTS | VACUOLAR NA+/H+ ANTIPORTER | K+ CHANNEL | PLASMA-MEMBRANE | NONSELECTIVE CATION CHANNELS | PLANT SCIENCES | Potassium - metabolism | Plants - metabolism | Plants - genetics | Sodium Chloride - pharmacology | Adaptation, Physiological - drug effects | Ion Transport - drug effects | Homeostasis - drug effects | Plants - drug effects | Homeostasis - genetics | Oxides | Plant genetics
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2. Full Text The Arabidopsis Nitrate Transporter NRT1.8 Functions in Nitrate Removal from the Xylem Sap and Mediates Cadmium Tolerance
by Jian-Yong Li and Yan-Lei Fu and Sharon M. Pike and Juan Bao and Wang Tian and Yu Zhang and Chun-Zhu Chen and Yi Zhang and Hong-Mei Li and Jing Huang and Le-Gong Li and Julian I. Schroeder and Walter Gassmann and Ji-Ming Gong
The Plant Cell, ISSN 1040-4651, 5/2010, Volume 22, Issue 5, pp. 1633 - 1646
Long-distance transport of nitrate requires xylem loading and unloading, a successive process that determines nitrate distribution and subsequent assimilation... 
Cadmium | Xylem | Reverse transcriptase polymerase chain reaction | Epidermal cells | Plant roots | Parenchyma | Nitrates | Plants | Oocytes | Plant cells | ENCODES | ROOT | PHYTOCHELATINS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SHOOT | PLANT SCIENCES | CELL BIOLOGY | HIGHER-PLANTS | CHL1 | GENE-EXPRESSION | ACCUMULATION | LONG-DISTANCE TRANSPORT | THIOL-PEPTIDES | Arabidopsis - enzymology | Xylem - enzymology | Adaptation, Physiological - drug effects | Xylem - drug effects | Gene Expression Profiling | Protein Transport - drug effects | Xylem - genetics | Arabidopsis Proteins - metabolism | Xylem - cytology | Adaptation, Physiological - genetics | Hydrogen-Ion Concentration - drug effects | Stress, Physiological - drug effects | Cell Membrane - drug effects | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Cadmium - toxicity | Subcellular Fractions - drug effects | Stress, Physiological - genetics | Arabidopsis - cytology | Plant Exudates - metabolism | Mutation - genetics | Anion Transport Proteins - metabolism | Arabidopsis - genetics | Up-Regulation - drug effects | Cell Membrane - enzymology | Nitrates - metabolism | Gene Expression Regulation, Plant - drug effects | Plant Exudates - genetics | Subcellular Fractions - enzymology | Anion Transport Proteins - genetics | Arabidopsis thaliana | Biological transport | Genetic aspects | Health aspects
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3. Full Text An orally active TRPV4 channel blocker prevents and resolves pulmonary edema induced by heart failure
by Thorneloe, Kevin S and Cheung, Mui and Bao, Weike and Alsaid, Hasan and Lenhard, Stephen and Jian, Ming-Yuan and Costell, Melissa and Maniscalco-Hauk, Kristeen and Krawiec, John A and Olzinski, Alan and Gordon, Earl and Lozinskaya, Irina and Elefante, Lou and Qin, Pu and Matasic, Daniel S and James, Chris and Tunstead, James and Donovan, Brian and Kallal, Lorena and Waszkiewicz, Anna and Vaidya, Kalindi and Davenport, Elizabeth A and Larkin, Jonathan and Burgert, Mark and Casillas, Linda N and Marquis, Robert W and Ye, Guosen and Eidam, Hilary S and Goodman, Krista B and Toomey, John R and Roethke, Theresa J and Jucker, Beat M and Schnackenberg, Christine G and Townsley, Mary I and Lepore, John J and Willette, Robert N
Science Translational Medicine, ISSN 1946-6234, 11/2012, Volume 4, Issue 159, pp. 159ra148 - 159ra148
Pulmonary edema resulting from high pulmonary venous pressure (PVP) is a major cause of morbidity and mortality in heart failure (HF) patients, but current... 
MEDICINE, RESEARCH & EXPERIMENTAL | HIGH VASCULAR PRESSURE | ACTIVATION | PERMEABILITY | SHEAR-STRESS | INDUCED LUNG INJURY | RESISTANCE | RECEPTOR POTENTIAL VANILLOID-4 | MICE | EXPRESSION | INDUCED VASODILATATION | CELL BIOLOGY | Membrane Transport Modulators - chemistry | Calcium - metabolism | Humans | Heart Failure - physiopathology | Water-Electrolyte Balance - drug effects | Protein Transport - drug effects | Pulmonary Edema - etiology | Heart Rate - drug effects | TRPV Cation Channels - metabolism | TRPV Cation Channels - antagonists & inhibitors | Membrane Transport Modulators - pharmacology | Pulmonary Edema - prevention & control | Pulmonary Edema - pathology | Blood Pressure - drug effects | Lung - metabolism | Disease Models, Animal | Endothelium - pathology | Heart Failure - complications | Lung - pathology | Permeability - drug effects | Administration, Oral | Diuretics - pharmacology | Rats | Membrane Transport Modulators - administration & dosage | Heart Failure - pathology | Pulmonary Edema - drug therapy | Mice, Knockout | Endothelium - drug effects | Animals | Endothelium - metabolism | Lung - drug effects | Membrane Transport Modulators - therapeutic use | Mice | In Vitro Techniques | Ion Channel Gating - drug effects
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4. Full Text SLAC1 is required for plant guard cell S-type anion channel function in stomatal signalling
by Vahisalu, Triin and Kollist, Hannes and Wang, Yong-Fei and Nishimura, Noriyuki and Chan, Wai-Yin and Valerio, Gabriel and Lamminmäki, Airi and Brosché, Mikael and Moldau, Heino and Desikan, Radhika and Schroeder, Julian I and Kangasjärvi, Jaakko
Nature, ISSN 0028-0836, 03/2008, Volume 452, Issue 7186, pp. 487 - 491
Stomatal pores, formed by two surrounding guard cells in the epidermis of plant leaves, allow influx of atmospheric carbon dioxide in exchange for... 
MUTANT | ARABIDOPSIS-THALIANA | VICIA-FABA | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | OZONE | CLOSURE | TRANSDUCTION | ION CHANNELS | ABSCISIC-ACID | PLASMA-MEMBRANE | Xenopus | Darkness | Calcium - metabolism | Ozone - metabolism | Ion Channel Gating - radiation effects | Plant Stomata - metabolism | Oocytes | Arabidopsis Proteins - metabolism | Arabidopsis - radiation effects | Light | Humidity | Plant Stomata - drug effects | Ion Transport - drug effects | Ion Transport - radiation effects | Abscisic Acid - metabolism | Membrane Proteins - metabolism | Plant Leaves - drug effects | Signal Transduction - radiation effects | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Calcium - pharmacology | Membrane Proteins - genetics | Tobacco - metabolism | Arabidopsis - cytology | Hydrogen Peroxide - pharmacology | Water - metabolism | Onions - metabolism | Anions - metabolism | Arabidopsis - metabolism | Hydrogen Peroxide - metabolism | Tobacco - cytology | Abscisic Acid - pharmacology | Animals | Ion Channels - metabolism | Ozone - pharmacology | Plant Leaves - metabolism | Plant Stomata - radiation effects | Signal Transduction - drug effects | Environment | Nitric Oxide - metabolism | Ion Channel Gating - drug effects | Signal transduction | Ozone | Genetics | Cellular biology | Botany | Carbon dioxide
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5. Full Text Structure–Activity Relationships in Tripodal Transmembrane Anion Transporters: The Effect of Fluorination
by Busschaert, Nathalie and Wenzel, Marco and Light, Mark E and Iglesias-Hernández, Paulina and Pérez-Tomás, Ricardo and Gale, Philip A
Journal of the American Chemical Society, ISSN 0002-7863, 09/2011, Volume 133, Issue 35, pp. 14136 - 14148
A series of easy-to-make fluorinated tripodal anion transporters containing urea and thiourea groups have been prepared and their anion transport properties... 
MEDICINAL CHEMISTRY | MEMBRANE TRANSPORTERS | PHOSPHOLIPID-VESICLES | CHLORIDE TRANSPORT | BACTERIAL PRODIGININES | INTRACELLULAR PH | PI SLIDES | CELL-MEMBRANES | SULFATE RECEPTORS | CYSTIC-FIBROSIS | CHEMISTRY, MULTIDISCIPLINARY | Cell Survival - drug effects | Humans | Models, Molecular | Crystallography, X-Ray | Structure-Activity Relationship | Anions - chemistry | Anions - metabolism | Urea - analogs & derivatives | Halogenation | Cell Line, Tumor | Ion Transport - drug effects | Binding Sites | Urea - pharmacology | Composition | Analysis | Cancer cells | Physiological aspects | Ion transport | Cell membranes | Research | Fluorination | Apoptosis
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6. Full Text Rapid ammonia gas transport accounts for futile transmembrane cycling under NH3/NH4 + toxicity in plant roots
by Coskun, Devrim and Britto, Dev T and Li, Mingyuan and Becker, Alexander and Kronzucker, Herbert J
Plant Physiology, ISSN 0032-0889, 12/2013, Volume 163, Issue 4, pp. 1859 - 1867
Futile transmembrane NH3/NH4+ cycling in plant root cells, characterized by extremely rapid fluxes and high efflux to influx ratios, has been successfully... 
GLOBAL NITROGEN-CYCLE | RICE ROOTS | COMPARTMENTAL ANALYSIS | POTASSIUM HOMEOSTASIS | SODIUM-TRANSPORT | SUBCELLULAR-DISTRIBUTION | HORDEUM-VULGARE L | BARLEY ROOTS | PLASMA-MEMBRANE | METHIONINE SULFOXIMINE | PLANT SCIENCES | Hordeum - drug effects | Plant Roots - metabolism | Ammonium Compounds - toxicity | Ammonia - metabolism | Hordeum - metabolism | Ammonia - toxicity | Seedlings - drug effects | Plant Roots - drug effects | Models, Biological | Hydrogen-Ion Concentration - drug effects | Cell Respiration - drug effects | Biological Transport - drug effects | Cell Membrane - metabolism | Seedlings - metabolism | Index Medicus
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7. Full Text Central role of cyclic electron transport around photosystem I in the regulation of photosynthesis
by Shikanai, Toshiharu
Current Opinion in Biotechnology, ISSN 0958-1669, 2013, Volume 26, pp. 25 - 30
Graphical abstract 
Internal Medicine | PHOTOPROTECTION | DEHYDROGENASE-LIKE COMPLEX | FERREDOXIN | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | NAD(P)H DEHYDROGENASE | BIOCHEMICAL RESEARCH METHODS | STATE TRANSITIONS | REQUIRES | EFFICIENT OPERATION | SUPERCOMPLEX FORMATION | FLOW | PGR5 | Protons | Chloroplasts - enzymology | Cytochrome b6f Complex - metabolism | Photosynthetic Reaction Center Complex Proteins - metabolism | Electron Transport - drug effects | Electron Transport - radiation effects | Homeostasis | NADH Dehydrogenase - genetics | Plastoquinone - metabolism | Antimycin A - pharmacology | Magnoliopsida - metabolism | Arabidopsis Proteins - metabolism | Photosynthetic Reaction Center Complex Proteins - genetics | Adenosine Triphosphate - metabolism | Light | Chloroplasts - radiation effects | Photosystem I Protein Complex - metabolism | Membrane Proteins - metabolism | Photosynthesis - radiation effects | Arabidopsis Proteins - genetics | Oxidation-Reduction | Ferredoxins - metabolism | Adenosine Triphosphatases - metabolism | NADH Dehydrogenase - chemistry | NADH Dehydrogenase - metabolism | Chloroplasts - metabolism | Hydrogen-Ion Concentration | Photosystem I Protein Complex - drug effects | Photosynthesis | Electron transport | Laws, regulations and rules | Phytochemistry | Transportation
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8. Full Text A synthetic ion transporter that disrupts autophagy and induces apoptosis by perturbing cellular chloride concentrations
by Busschaert, Nathalie and Park, Seong-Hyun and Baek, Kyung-Hwa and Choi, Yoon Pyo and Park, Jinhong and Howe, Ethan N. W and Hiscock, Jennifer R and Karagiannidis, Louise E and Marques, Igor and Félix, Vítor and Namkung, Wan and Sessler, Jonathan L and Gale, Philip A and Shin, Injae
Nature Chemistry, ISSN 1755-4330, 07/2017, Volume 9, Issue 7, pp. 667 - 675
Perturbations in cellular chloride concentrations can affect cellular pH and autophagy and lead to the onset of apoptosis. With this in mind, synthetic ion... 
CYTOCHROME-C | LYSOSOMES | SMALL-MOLECULE | DISSECTION | TRANSMEMBRANE ANION TRANSPORTERS | ATPASE | TRAFFICKING | MEMBRANES | ACIDIFICATION | DEATH | CHEMISTRY, MULTIDISCIPLINARY | Quinine - analogs & derivatives | Apoptosis - drug effects | Chlorides - pharmacology | Humans | Structure-Activity Relationship | Chlorides - chemistry | Autophagy - drug effects | Quinine - chemistry | Dose-Response Relationship, Drug | Quinine - pharmacology | Ion Transport - drug effects | Molecular Structure | HeLa Cells | Hydrogen-Ion Concentration | Enzyme activity | Chloride transport | Homeostasis | Caspase | Chloride currents | Sodium chloride | pH effects | Cytosol | Autophagy | Anions | Chloride | Cell death | pH | Disruption | Transport | Transporter | Phagocytosis | Apoptosis
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9. Full Text SLC25A23 augments mitochondrial Ca2+ uptake, interacts with MCU, and induces oxidative stress-mediated cell death
by Hoffman, Nicholas E and Chandramoorthy, Harish C and Shanmughapriya, Santhanam and Zhang, Xueqian Q and Vallem, Sandhya and Doonan, Patrick J and Malliankaraman, Karthik and Guo, Shuchi and Rajan, Sudarsan and Elrod, John W and Koch, Walter J and Cheung, Joseph Y and Madesh, Muniswamy
Molecular Biology of the Cell, ISSN 1059-1524, 03/2014, Volume 25, Issue 6, pp. 936 - 947
Emerging findings suggest that two lineages of mitochondrial Ca2+ uptake participate during active and resting states: 1) the major eukaryotic membrane... 
TRANSPORTER FAMILY SLC25 | CONFOCAL MICROSCOPY | ESSENTIAL COMPONENT | LIVER-MITOCHONDRIA | MG/P-I CARRIER | CALCIUM UNIPORTER | ENDOPLASMIC-RETICULUM | NEURONAL MITOCHONDRIA | VENTRICULAR MYOCYTES | PERMEABILITY TRANSITION | CELL BIOLOGY | Amino Acid Transport Systems, Acidic - antagonists & inhibitors | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Calcium Channels - metabolism | Oxidative Stress | Calcium - metabolism | Humans | Amino Acid Transport Systems, Acidic - metabolism | Calcium-Binding Proteins - antagonists & inhibitors | Mitochondrial Proteins - genetics | Membrane Potential, Mitochondrial - drug effects | Mitochondrial Membrane Transport Proteins - genetics | Cation Transport Proteins - metabolism | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Antiporters - genetics | Cell Death | Cation Transport Proteins - genetics | Ion Transport - drug effects | Clone Cells | Calcium Channels - genetics | Calcium Signaling | Calcium-Binding Proteins - metabolism | Protein Structure, Tertiary | Mitochondrial Membrane Transport Proteins - metabolism | Histamine - pharmacology | Mitochondrial Proteins - antagonists & inhibitors | Gene Expression Regulation | Antiporters - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Oxygen Consumption - drug effects | Amino Acid Transport Systems, Acidic - genetics | HeLa Cells | Antiporters - antagonists & inhibitors | Calcium-Binding Proteins - genetics | RNA, Small Interfering - metabolism
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10. Full Text Abscisic acid alleviates iron deficiency by promoting root iron reutilization and transport from root to shoot in Arabidopsis
by LEI, GUI JIE and ZHU, XIAO FANG and WANG, ZHI WEI and DONG, FANG and DONG, NING YU and ZHENG, SHAO JIAN
Plant, Cell & Environment, ISSN 0140-7791, 04/2014, Volume 37, Issue 4, pp. 852 - 863
Abscisic acid (ABA) has been demonstrated to be involved in iron (Fe) homeostasis, but the underlying mechanism is largely unknown. Here, we found that Fe... 
abi5 | ABA‐responsive elements | apoplast | cell wall | long‐distance transport | phenolics | Long-distance transport | Apoplast | ABA-responsive elements | Phenolics | Cell wall | ACTS DOWNSTREAM | CELL-WALL POLYSACCHARIDES | long-distance transport | PLANT SCIENCES | ALUMINUM | RESPONSES | FERRIC-CHELATE REDUCTASE | GROWTH | NITRIC-OXIDE | DEVELOPING SEEDS | METAL-IONS | TRANSCRIPTION FACTOR | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Plant Roots - metabolism | Solubility | Chlorophyll - metabolism | Iron - metabolism | Vacuoles - drug effects | Arabidopsis - metabolism | Plant Roots - drug effects | Arabidopsis - genetics | Arabidopsis Proteins - metabolism | Abscisic Acid - pharmacology | Gene Expression Regulation, Plant - drug effects | Cation Transport Proteins - metabolism | Plant Shoots - metabolism | Iron - deficiency | Plant Shoots - drug effects | Vacuoles - metabolism | Cation Transport Proteins - genetics | Abscisic Acid - metabolism | Biological Transport - drug effects | Arabidopsis thaliana | Abscisic acid
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