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Nature Neuroscience, ISSN 1097-6256, 07/2004, Volume 7, Issue 7, pp. 711 - 718
Voltage-dependent Kv2.1 K+ channels, which mediate delayed rectifier Kv currents (I-K), are expressed in large clusters on the somata and dendrites of... 
ALPHA-SUBUNITS | RAT HIPPOCAMPAL-NEURONS | MAMMALIAN BRAIN | ACTIVATION | GATED POTASSIUM CHANNELS | RECEPTOR | NEOCORTICAL PYRAMIDAL NEURONS | IDENTIFICATION | KV2.1 K+ CHANNEL | BINDING | NEUROSCIENCES | Potassium - metabolism | Okadaic Acid - pharmacology | Potassium Channels, Voltage-Gated | Pyramidal Cells - metabolism | Cyclosporine - pharmacology | Cell Count | Neuronal Plasticity - drug effects | Pyrethrins - pharmacology | Translocation, Genetic - drug effects | Patch-Clamp Techniques - methods | Seizures - chemically induced | Dose-Response Relationship, Drug | Potassium Channels - metabolism | Phosphoprotein Phosphatases - pharmacology | Seizures - physiopathology | Drug Interactions | Neuronal Plasticity - physiology | Time Factors | Dendrites - physiology | Potassium Chloride - pharmacology | Nitriles | Pyramidal Cells - drug effects | Phosphorylation - drug effects | Cadmium Chloride - pharmacology | Ionophores - pharmacology | Potassium Channel Blockers - pharmacology | Disease Models, Animal | Animals, Newborn | Membrane Potentials - drug effects | Dendrites - drug effects | Translocation, Genetic - physiology | Glutamic Acid - pharmacology | Kainic Acid - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Excitatory Amino Acid Antagonists - pharmacology | Nitrendipine - pharmacology | Calcium Channel Blockers - pharmacology | Delayed Rectifier Potassium Channels | Hippocampus - cytology | Membrane Potentials - physiology | Shab Potassium Channels | Calcimycin - pharmacology | Blotting, Western | Animals | Ion Channel Gating - drug effects | Physiological aspects | Phosphorylation | Ion channels | Research | Neurons
Journal Article
by Wang, LX and Nick, P
PLANT CELL AND ENVIRONMENT, ISSN 0140-7791, 11/2017, Volume 40, Issue 11, pp. 2844 - 2857
Plants can acquire freezing tolerance in response to cold but non-freezing temperatures. To efficiently activate this cold acclimation, low temperature has to... 
GOSSYPIUM-HIRSUTUM-L | CYTOSKELETON CONTROLS | calcium | RYE SECALE-CEREALE | PHOSPHOLIPASE-D | cold stress | microtubules | LOW-TEMPERATURE | CYTOPLASMIC CALCIUM | PLANT SCIENCES | CORTICAL MICROTUBULES | PHOSPHATIDIC-ACID | ABSCISIC-ACID | grapevine (Vitis rupestris) | signalling | FREEZING TOLERANCE | Gadolinium - pharmacology | Calcium - metabolism | NADPH Oxidases - metabolism | Vitis - physiology | Cytoplasm - metabolism | Dimethyl Sulfoxide - pharmacology | Nitroprusside - pharmacology | Microtubules - metabolism | Biphenyl Compounds - pharmacology | Microtubules - drug effects | Cell Membrane - metabolism | Stress, Physiological - drug effects | Cell Membrane - drug effects | Ionophores - pharmacology | Pertussis Toxin - pharmacology | Pyrazoles - pharmacology | Cold Temperature | Benzyl Alcohol - pharmacology | Polymerization | Vitis - drug effects | Cyclopentanes - pharmacology | Onium Compounds - pharmacology | Calcimycin - pharmacology | Phospholipase D - metabolism | Fluorides - pharmacology | Egtazic Acid - pharmacology | Signal Transduction - drug effects | Oxylipins - pharmacology | Aluminum Compounds - pharmacology | Oxidases | Arabidopsis thaliana | Fluorescence | Calcium-binding proteins | Analysis | Measuring instruments | Climate | Image processing | Calcium | Oxidase | Transgenic | Activation | NAD(P)H oxidase | Calcium influx | Freezing | Cold acclimation | Cash crops | Proteins | Signal transduction | Tubulin | Cold tolerance | Calcium-binding protein | Fusion protein | Calmodulin | Jasmonic acid | Low temperature | Cultivars | Stresses | Cold | Breeding | Interphase | Acclimatization | Pharmacology | Stress | Phospholipase D | Acclimation | NAD | Phospholipase | Signaling | Image analysis | Plant breeding | Microtubules | Green fluorescent protein | Cytoskeleton | Perception
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 12/2002, Volume 137, Issue 7, pp. 1021 - 1030
The mechanism(s) by which vascular endothelial growth factor (VEGF) induces endothelial nitric oxide synthase (eNOS) activation remain(s) unclear up to a... 
eNOS | PAF | PI3K | VEGF | PLC | PLA2 | PKC | PLA | PROTEIN-KINASE-C | ANGIOGENESIS | PERMEABILITY | PHOSPHORYLATION | NITRIC-OXIDE SYNTHASE | CALMODULIN | CALCIUM | PAF SYNTHESIS | PHARMACOLOGY & PHARMACY | PLATELET-ACTIVATING-FACTOR | GROWTH-FACTOR | Endothelium, Vascular - cytology | Vascular Endothelial Growth Factors | Platelet Activating Factor - pharmacology | Protein-Serine-Threonine Kinases | Type C Phospholipases - metabolism | Endothelium, Vascular - drug effects | Endothelial Growth Factors - pharmacology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Receptors, Cell Surface - antagonists & inhibitors | Lactones - pharmacology | Platelet Membrane Glycoproteins - antagonists & inhibitors | Calcium - antagonists & inhibitors | Time Factors | Cattle | Protein Kinase C - metabolism | Phosphorylation - drug effects | Chromones - pharmacology | Enzyme Inhibitors - pharmacology | Nitric Oxide Synthase Type III | Receptors, G-Protein-Coupled | Chelating Agents - pharmacology | Serine - metabolism | 1-Methyl-3-isobutylxanthine - pharmacology | Ginkgolides | Platelet Activating Factor - metabolism | Cyclic GMP - metabolism | Endothelium, Vascular - metabolism | Intercellular Signaling Peptides and Proteins - pharmacology | Nitric Oxide Synthase - metabolism | Vascular Endothelial Growth Factor A | Maleimides - pharmacology | Nitric Oxide Synthase - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - metabolism | Nitroprusside - pharmacology | Type C Phospholipases - antagonists & inhibitors | Dose-Response Relationship, Drug | Egtazic Acid - analogs & derivatives | Indoles - pharmacology | Lymphokines - pharmacology | Nitric Oxide Donors - pharmacology | Ionophores - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Proto-Oncogene Proteins - metabolism | Cells, Cultured | Morpholines - pharmacology | Protein Kinase C - antagonists & inhibitors | Calcimycin - pharmacology | Proto-Oncogene Proteins c-akt | Animals | Egtazic Acid - pharmacology | Androstadienes - pharmacology | Diterpenes | Signal Transduction - physiology | Nitric Oxide - metabolism | Papers
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 08/2012, Volume 32, Issue 33, pp. 11356 - 11364
Journal Article
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 11/2014, Volume 58, Issue 11, pp. 6413 - 6423
Journal Article
European Journal of Immunology, ISSN 0014-2980, 12/2012, Volume 42, Issue 12, pp. 3334 - 3345
Extracellular ATP, released upon microbial infection, cell damage, or inflammation, acts as an alert signal toward immune cells by activating P2 receptors. The... 
Extracellular ATP | IL‐18 | P2X7 | P2 receptors | Human macrophages | IL-18 | DENDRITIC CELLS | CASPASE-1 | IFN-GAMMA | GAMMA-INDUCING FACTOR | IMMUNOLOGY | POLYMYXIN-B | HUMAN BLOOD | IL-1-BETA | TNF-ALPHA | SECRETION | ATP | Polymyxin B - pharmacology | Interleukin-18 - immunology | Tetrazoles - pharmacology | Humans | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Acetamides - pharmacology | Purinergic P2X Receptor Antagonists - pharmacology | Quinolines - pharmacology | Adenosine Triphosphate - pharmacology | Calcium Ionophores - pharmacology | Cell-Derived Microparticles - metabolism | Receptors, Purinergic P2X7 - immunology | Macrophages - immunology | Receptors, Purinergic P2X4 - immunology | Protein Precursors - immunology | Toll-Like Receptor 4 - immunology | Sulfonamides - pharmacology | Calcimycin - pharmacology | Purinergic P2X Receptor Agonists - pharmacology | Toll-Like Receptor 4 - metabolism | Adenosine Triphosphate - analogs & derivatives | Protein Precursors - metabolism | Macrophages - metabolism | Cell-Derived Microparticles - immunology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Lipopolysaccharides - pharmacology | Receptors, Purinergic P2X7 - metabolism | Anti-Bacterial Agents - pharmacology | Pyridines - pharmacology | Receptors, Purinergic P2X4 - metabolism | Interleukin-18 - metabolism | Macrophages | Ionophores | Rodents
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2009, Volume 29, Issue 41, pp. 12994 - 13005
In Alzheimer's disease (AD), rod-like cofilin aggregates (cofilin-actin rods) and thread-like inclusions containing phosphorylated microtubule-associated... 
F-ACTIN | OXIDATIVE STRESS | ROD FORMATION | AMYLOID-BETA | DISEASE | NEURONS | CHICK BRAIN | TAU | FILAMENT TURNOVER | NEUROSCIENCES | MITOCHONDRIAL FISSION | Phosphorylation - physiology | Neurons - pathology | RNA, Small Interfering - genetics | Oxidants - pharmacology | Actin Depolymerizing Factors - genetics | Amyloid beta-Peptides - pharmacology | Humans | Actins - metabolism | tau Proteins - metabolism | Neurons - cytology | Peptide Fragments - pharmacology | Depsipeptides - pharmacology | Green Fluorescent Proteins - genetics | Antimycin A - pharmacology | Alzheimer Disease - pathology | Actin Depolymerizing Factors - metabolism | Adenosine Triphosphate - pharmacology | Chick Embryo - cytology | Transfection - methods | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Neurons - drug effects | Thiazolidines - pharmacology | Ionophores - pharmacology | Neurites - drug effects | Organ Culture Techniques | Animals, Newborn | p21-Activated Kinases - genetics | Antimycin A - analogs & derivatives | Cells, Cultured | Enzyme Inhibitors - pharmacology | Hydrogen Peroxide - pharmacology | Rats | Neurites - metabolism | p21-Activated Kinases - metabolism | Serine - metabolism | Amino Acid Motifs - physiology | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Animals | Brain - pathology | Fluorescence Resonance Energy Transfer - methods | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2012, Volume 122, Issue 5, pp. 941 - 951
J. Neurochem. (2012) 122, 941951. Abstract In vitro and in vivo models of Parkinsons disease (PD) suggest that increased oxidant production leads to... 
hydroethidine | mitochondria | glycolysis | reactive oxygen species | NADPH OXIDASE | OXYGEN SPECIES PRODUCTION | OXIDATIVE STRESS | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOPAMINERGIC-NEURONS | PARAQUAT | NEUROSCIENCES | BRAIN MITOCHONDRIA | REACTIVE OXYGEN | ANIMAL-MODELS | CELL-CULTURE | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Insecticides - pharmacology | Rats | Herbicides - pharmacology | Adrenergic Agents - pharmacology | Dose-Response Relationship, Drug | Neurotoxins - pharmacology | Animals | Oligomycins - pharmacology | Oxidopamine - pharmacology | Time Factors | Dopamine Agents - pharmacology | Oxygen Consumption - drug effects | Adenosine Triphosphate - metabolism | Dopaminergic Neurons - metabolism | Proton Ionophores - pharmacology | Rotenone - pharmacology | Superoxides - metabolism | Dopaminergic Neurons - drug effects | Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone - pharmacology | Paraquat - pharmacology | Cell Line, Transformed | Energy Metabolism - drug effects | Glucose metabolism | Herbicides | Parkinson's disease | Cell death | Analysis | Superoxide | Mitochondrial DNA | Universities and colleges | Cells | Neurochemistry | Mitochondria | Neurotransmitters | Bioenergetics | Parkinsons disease | Apoptosis | Index Medicus | Dopamine | Neurodegenerative diseases | 6-Hydroxydopamine | Acidification | Data processing | Oxygen consumption | Rotenone | Toxicants | MPP super(+) | Oxidants | Glycolysis | Paraquat | Oxidation | Movement disorders
Journal Article
NATURE NEUROSCIENCE, ISSN 1097-6256, 09/2005, Volume 8, Issue 9, pp. 1160 - 1168
Exocytosis proceeds by either full fusion or 'kiss-and-run' between vesicle and plasma membrane. Switching between these two modes permits the cell to regulate... 
TRANSMITTER RELEASE | VESICLE FUSION | PROTEIN-KINASE-C | PRESYNAPTIC INHIBITION | EXOCYTOSIS | NEUROTRANSMITTER RELEASE | RECEPTOR | BETA-GAMMA-SUBUNITS | NEUROSCIENCES | FUSION PORE KINETICS | SECRETORY GRANULES | GTP-Binding Protein beta Subunits - pharmacology | Receptors, G-Protein-Coupled - metabolism | Recombinant Fusion Proteins - pharmacology | Calcium - metabolism | Analgesics, Opioid - pharmacology | Membrane Potentials - radiation effects | Patch-Clamp Techniques - methods | Somatostatin - pharmacology | Dynamins - pharmacology | Adenosine Triphosphate - pharmacology | Neural Inhibition - physiology | Drug Interactions | Protein Kinase C - metabolism | Potassium Chloride - pharmacology | Action Potentials - radiation effects | GTP-Binding Protein beta Subunits - metabolism | Chromaffin Cells - drug effects | Electric Stimulation - methods | Action Potentials - drug effects | Ionophores - pharmacology | Pertussis Toxin - pharmacology | Membrane Potentials - drug effects | Protein Kinases - pharmacology | GTP-Binding Protein gamma Subunits - metabolism | Enkephalin, Ala-MePhe-Gly- - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Thionucleotides - pharmacology | Enzyme Activation - drug effects | Adenosine Triphosphate - analogs & derivatives | Adrenal Medulla - cytology | Action Potentials - physiology | Chromaffin Cells - metabolism | Animals | Ionomycin - pharmacology | Muscarine - pharmacology | Electrochemistry - methods | Dose-Response Relationship, Radiation | Enzyme Activation - physiology | Neural Inhibition - drug effects | Chromaffin cells | Physiological aspects | Cell receptors | Research | G proteins | Protein kinases
Journal Article
FASEB Journal, ISSN 0892-6638, 03/2006, Volume 20, Issue 3, pp. 542 - 544
It is widely believed that the potencies of nonsteroid anti-inflammatory drugs ( NSAIDs) as inhibitors of cyclooxygenase ( COX) are influenced by protein... 
SITE | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | nonsteroid anti-inflammatory drugs | coxibs | PROSTAGLANDIN H-2 SYNTHASE | CELL BIOLOGY | CYCLO-OXYGENASE-2 | THERAPY | FLUID | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | BIOLOGY | prostaglandins | SELECTIVITY | Organic Chemicals - cerebrospinal fluid | Humans | Naproxen - pharmacology | Synovial Fluid - metabolism | Pyrazoles - cerebrospinal fluid | Sodium Salicylate - pharmacology | Lactones - pharmacology | Aspirin - cerebrospinal fluid | Cell Line - drug effects | Calcium - physiology | Pyrazoles - blood | Organic Chemicals - pharmacology | Dinoprostone - cerebrospinal fluid | Diclofenac - cerebrospinal fluid | Aspirin - pharmacology | Sulfones - cerebrospinal fluid | Aspirin - blood | Thiazines - blood | Sulfonamides - pharmacology | Indomethacin - cerebrospinal fluid | Indomethacin - pharmacology | Cyclooxygenase 1 - drug effects | Cyclooxygenase Inhibitors - adverse effects | Cyclooxygenase Inhibitors - blood | Diclofenac - pharmacology | Thiazoles - blood | Thromboxane A2 - blood | Cerebrospinal Fluid Proteins - pharmacology | Dinoprostone - biosynthesis | Lactones - cerebrospinal fluid | Blood Platelets - enzymology | Cyclooxygenase 2 Inhibitors - cerebrospinal fluid | Sodium Salicylate - cerebrospinal fluid | Sulfones - pharmacology | Blood Proteins - pharmacology | Cyclooxygenase 2 Inhibitors - adverse effects | Sulfonamides - blood | Lactones - blood | Naproxen - cerebrospinal fluid | Sodium Salicylate - blood | Thiazines - pharmacology | Cyclooxygenase 2 Inhibitors - blood | Cyclooxygenase Inhibitors - cerebrospinal fluid | Blood Platelets - drug effects | Sulfones - blood | Organic Chemicals - blood | Ionophores - pharmacology | Pyrazoles - pharmacology | Sulfonamides - cerebrospinal fluid | Cyclooxygenase 2 Inhibitors - pharmacology | Thiazoles - cerebrospinal fluid | Celecoxib | Diclofenac - blood | Dinoprostone - blood | Naproxen - blood | Organ Specificity | Calcimycin - pharmacology | Cyclooxygenase Inhibitors - pharmacology | Indomethacin - blood | Protein Binding | Thiazines - cerebrospinal fluid | Thiazoles - pharmacology
Journal Article
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 01/2019, Volume 20, Issue 2, p. 338
Journal Article
Nature Neuroscience, ISSN 1097-6256, 05/2010, Volume 13, Issue 5, pp. 622 - 629
Stress and anxiety disorders are risk factors for depression and these behaviors are modulated by corticotrophin-releasing factor receptor 1 (CRFR1) and... 
INTERNALIZATION | CORTICOTROPIN-RELEASING-FACTOR | PDZ PROTEINS | CORTICAL PYRAMIDAL NEURONS | DESENSITIZATION | SEROTONIN RECEPTORS | TRAFFICKING | CENTRAL-NERVOUS-SYSTEM | ENDOCYTOSIS | NEUROSCIENCES | RAB GTPASES | Embryo, Mammalian | Amphetamines - pharmacology | Humans | Receptors, Corticotropin-Releasing Hormone - genetics | Male | Anxiety - drug therapy | Receptors, Serotonin, 5-HT2 - metabolism | Serotonin - pharmacology | Dose-Response Relationship, Drug | Fluorobenzenes - pharmacology | Serotonin 5-HT2 Receptor Antagonists | Transfection | Piperidines - pharmacology | Reaction Time - drug effects | Behavior, Animal - drug effects | Inositol Phosphates - metabolism | Anxiety - metabolism | Cyclic AMP - metabolism | Ionophores - pharmacology | Disease Models, Animal | Hormones - pharmacology | Maze Learning - physiology | Neurons | Cells, Cultured | Anxiety - physiopathology | Monensin - pharmacology | Rats | Behavior, Animal - physiology | Maze Learning - drug effects | Mutation - genetics | Prefrontal Cortex - cytology | Corticotropin-Releasing Hormone - pharmacology | Receptors, Corticotropin-Releasing Hormone - physiology | Animals | Receptors, Serotonin, 5-HT2 - genetics | Serotonin Agents - pharmacology | Signal Transduction - drug effects | Signal Transduction - physiology | Biotinylation - methods | Mice | Serotonin 5-HT2 Receptor Agonists | Endocytosis | Physiological aspects | Cellular signal transduction | Anxiety | Genetic aspects | Research | Genetic regulation
Journal Article