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Annals of Neurology, ISSN 0364-5134, 2009, Volume 66, Issue 3, pp. 378 - 389
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, pp. e49023 - e49023
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is... 
INDUCED CELL-DEATH | ISCHEMIC TOLERANCE | MICROGLIAL TOXICITY | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | PERIVENTRICULAR LEUKOMALACIA | DEVELOPING OLIGODENDROCYTES | NECROSIS-FACTOR-ALPHA | EXCITOTOXIC INJURY | INDUCED BRAIN-INJURY | BACTERIAL-ENDOTOXIN SENSITIZES | Inflammation - chemically induced | Inflammation - pathology | Microglia - metabolism | Oligodendroglia - metabolism | Rats, Wistar | Apoptosis - drug effects | Caspase 3 - metabolism | Brain - metabolism | Inflammation - metabolism | Oligodendroglia - drug effects | Nerve Fibers, Myelinated - pathology | Hyperoxia - metabolism | Microglia - pathology | Animals, Newborn | Nerve Fibers, Myelinated - metabolism | Leukoencephalopathies - pathology | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cells, Cultured | Rats | Leukoencephalopathies - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Hyperoxia - pathology | Oligodendroglia - pathology | Animals | Lipopolysaccharides - pharmacology | Brain - pathology | Apoptosis - physiology | Immunohistochemistry | Brain | Cell death | Infants (Premature) | Inflammation | Research | Mitogens | Health aspects | Injuries | Neuroimaging | Neonates | Cell culture | Pediatrics | Transcription factors | Traumatic brain injury | Lung | Premature birth | Interleukin | Superoxide dismutase | Infants | Infections | Birth | Western blotting | Lipopolysaccharides | Head injuries | Newborn babies | Ischemia | Rodents | Oligodendrocytes | Oxygen | Pain perception | Cytokines | Maturation | Incubation | Mortality | Superoxide | Gene expression | Substantia alba | Microglia | Hospitals | Placenta | Magnetic resonance imaging | Magnetic permeability | Hyperoxia | Interleukin 10 | Brain damage | Hypoxia | Oxygen tension | Brain injury | Apoptosis | Index Medicus | pathology | Leukoencephalopathies | Sprague-Dawley | pharmacology | Caspase 3 | Cultured | Basic Medicine | chemically induced | Myelinated | physiology | drug effects | Cells | Newborn | Wistar | Medicinska grundvetenskaper | Oligodendroglia | metabolism | Nerve Fibers
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2018, Volume 72, Issue 18, pp. 2164 - 2165
The pathobiology of limb ischemic events in patients with peripheral artery disease is complex. The severity of arterial stenoses correlates with reduced limb... 
atherosclerosis | amputation | critical limb ischemia | peripheral artery disease | thrombosis | TRIAL | CARDIAC & CARDIOVASCULAR SYSTEMS | ISCHEMIA | Occlusion | Vein & artery diseases | Patients | Thrombosis | Blood clots | Pathology | Vascular diseases | Ischemia | Perfusion | Arteriosclerosis | Atherosclerosis | Amputation | Calcification | Nutrients | Thromboembolism | Cardiovascular diseases | Viability
Journal Article
Journal Article
Pathology, ISSN 0031-3025, 06/2008, Volume 40, Issue 4, pp. 385 - 391
This retrospective study aimed to document and illustrate the histomorphological changes underlying peripheral vascular disease (PVD). More specifically, it... 
atherosclerosis | Lower limb amputations | ischaemia | chronic total occlusion | medial calcification | diabetes | Ischaemia | Diabetes | Chronic total occlusion | Medial calcification | Atherosclerosis | DIABETES-MELLITUS | SERIES | lower limb amputations | CALCIFICATION | COMPLICATIONS | PATHOLOGY | LOWER-EXTREMITY AMPUTATION | Constriction, Pathologic - metabolism | Tunica Media - pathology | Constriction, Pathologic - pathology | Constriction, Pathologic - etiology | Humans | Middle Aged | Male | Diabetic Angiopathies - pathology | Peripheral Vascular Diseases - metabolism | Tunica Media - metabolism | Amputation | Aged, 80 and over | Adult | Diabetic Angiopathies - complications | Female | Retrospective Studies | Calcinosis - metabolism | Diabetes Mellitus, Type 2 - complications | Ischemia - pathology | Atherosclerosis - pathology | Atherosclerosis - complications | Arteries - pathology | Atherosclerosis - metabolism | Leg - blood supply | Peripheral Vascular Diseases - etiology | Aged | Diabetes Mellitus, Type 2 - pathology | Calcinosis - pathology | Peripheral Vascular Diseases - pathology | Constriction, Pathologic, pathology | Diabetic Foot | Arterioles, pathology | Tibial Arteries, pathology | Popliteal Artery, pathology | Coronary Disease | Thrombosis, pathology | Peripheral Vascular Diseases, pathology | Amputation, statistics and numerical data | Hypertension | Pulmonary Disease, Chronic Obstructive | Atherosclerosis, pathology | Arterial Occlusive Diseases, pathology | Calcinosis, pathology | Diabetes Mellitus | Hypercholesterolemia | Lower Extremity | Kidney Failure, Chronic | Femoral Artery, pathology | Smoking | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 412 - 427
Endothelial progenitor cells are known to reverse acute kidney injury by paracrine mechanisms. We previously found that microvesicles released from these... 
exosome | ischemia–reperfusion | acute kidney injury | ischemia-reperfusion | HORIZONTAL TRANSFER | STEM-CELLS | CONTRIBUTE | REGENERATION | FAILURE | EXPERIMENTAL GLOMERULONEPHRITIS | REPAIR | EPITHELIAL-CELLS | MESSENGER-RNA | UROLOGY & NEPHROLOGY | PLATELET-ACTIVATING-FACTOR | Chemotaxis, Leukocyte | Cell Proliferation | Epithelial Cells - metabolism | Kidney - blood supply | Capillaries - pathology | Kidney - pathology | Rats, Wistar | Endothelial Cells - transplantation | Cell-Derived Microparticles - transplantation | Male | MicroRNAs - metabolism | Acute Kidney Injury - genetics | Stem Cells - metabolism | Cell Hypoxia | Stem Cell Transplantation | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Cell-Derived Microparticles - pathology | Cell-Derived Microparticles - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Capillaries - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Ribonuclease III - genetics | Ribonuclease III - metabolism | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Endothelial Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Rats | Epithelial Cells - pathology | Acute Kidney Injury - prevention & control | Regeneration | Oligonucleotides - metabolism | Animals | Reperfusion Injury - prevention & control | Fibrosis | Stem Cells - pathology | Acute Kidney Injury - metabolism | Endothelial Cells - pathology | Apoptosis | Index Medicus | Cell proliferation | Intravenous administration | Paracrine signalling | mRNA | Leukocytes | Ribonuclease | Endothelial cells | Kidney | Angiogenesis | Ischemia | Stem cells | Hypoxia | miRNA | Capillaries | Injuries
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 12/2007, Volume 114, Issue 6, pp. 619 - 631
Neuroimaging studies indicate reduced volumes of certain gray matter regions in survivors of prematurity with periventricular leukomalacia (PVL). We... 
Pathology | Perinatal hypoxia–ischemia | Basal ganglia | Medicine & Public Health | Neurodevelopmental disability | Brainstem | Thalamus | Perinatal panencephalopathy | White matter gliosis | Perinatal hypoxia-ischemia | perinatal panencephalopathy | THALAMOCORTICAL CONNECTIONS | basal ganglia | LOW-BIRTH-WEIGHT | BRAIN-INJURY | ABNORMALITIES | COMPLICATION | PRETERM INFANTS | SUBPLATE NEURONS | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | MEMORY | neurodevelopmental disability | CEREBELLUM | brainstem | white matter gliosis | perinatal hypoxia-ischemia | thalamus | PLASTICITY | Neurons - pathology | Prevalence | Humans | Nerve Degeneration - physiopathology | Cerebellar Nuclei - growth & development | Infant | Male | Gliosis - physiopathology | Leukomalacia, Periventricular - pathology | Thalamus - pathology | Brain - growth & development | Gliosis - pathology | Cerebellar Nuclei - physiopathology | Leukomalacia, Periventricular - physiopathology | Gliosis - epidemiology | Premature Birth - physiopathology | Nerve Fibers, Myelinated - pathology | Female | Globus Pallidus - physiopathology | Brain Damage, Chronic - pathology | Globus Pallidus - pathology | Infant, Newborn | Leukomalacia, Periventricular - epidemiology | Globus Pallidus - growth & development | Comorbidity | Brain - physiopathology | Cerebellar Nuclei - pathology | Nerve Degeneration - epidemiology | Brain Damage, Chronic - physiopathology | Nerve Degeneration - pathology | Thalamus - growth & development | Thalamus - physiopathology | Brain - pathology | Brain Damage, Chronic - epidemiology | Index Medicus | Original Paper
Journal Article
Science Translational Medicine, ISSN 1946-6234, 01/2014, Volume 6, Issue 219, pp. 219ra7 - 219ra7
Journal Article