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Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 12/2004, Volume 101, Issue 50, pp. 17468 - 17473
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 37, pp. 15561 - 15576
Preferential expression of the low-activity (dimeric) M2 isoform of pyruvate kinase (PK) over its constitutively active splice variant M1 isoform is considered... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Mitochondrial Dynamics | AMP-Activated Protein Kinases - metabolism | Humans | Lung Neoplasms - metabolism | Pyruvate Kinase - chemistry | Lung Neoplasms - pathology | Neoplasm Proteins - antagonists & inhibitors | Isoenzymes - chemistry | Neoplasm Proteins - metabolism | Autophagy | Recombinant Fusion Proteins - metabolism | Thyroid Hormones - chemistry | Organelle Biogenesis | RNA Interference | Isoenzymes - metabolism | Adenosine Triphosphate - metabolism | Carcinoma - enzymology | Carrier Proteins - chemistry | Membrane Proteins - metabolism | Carcinoma - pathology | Neoplasm Proteins - genetics | Dimerization | A549 Cells | Lung Neoplasms - enzymology | Pyruvate Kinase - metabolism | Isoenzymes - genetics | Membrane Proteins - genetics | AMP-Activated Protein Kinases - antagonists & inhibitors | Carrier Proteins - antagonists & inhibitors | Neoplasm Proteins - chemistry | Recombinant Fusion Proteins - chemistry | Thyroid Hormones - genetics | Protein Transport | Carrier Proteins - genetics | Pyruvate Kinase - antagonists & inhibitors | Carrier Proteins - metabolism | Energy Metabolism | Membrane Proteins - antagonists & inhibitors | Membrane Proteins - chemistry | Thyroid Hormones - metabolism | Cell Line, Tumor | Carcinoma - metabolism | Mutation | Pyruvate Kinase - genetics | AMP-Activated Protein Kinases - genetics | Isoenzymes - antagonists & inhibitors | Amino Acid Substitution | Apoptosis | Index Medicus | cancer metabolism | cancer therapy | pyruvate kinase M1 | mitochondria | pyruvate kinase M2 | cancer biology | Metabolism | mitochondrial oxidative phosphorylation | energy metabolism | Warburg effect
Journal Article
Journal of medicinal chemistry, ISSN 0022-2623, 03/2012, Volume 55, Issue 5, pp. 2311 - 2323
Journal Article
Neoplasia (New York, N.Y.), ISSN 1476-5586, 03/2011, Volume 13, Issue 3, pp. 286 - 298
In the United States, more than 40% of cancer patients develop brain metastasis. The median survival for untreated patients is 1 to 2 months, which may be... 
Oncology | Life Sciences & Biomedicine | Science & Technology | Lung Neoplasms - drug therapy | Up-Regulation | Adenocarcinoma - pathology | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Humans | Astrocytes - pathology | bcl-X Protein - genetics | Lung Neoplasms - pathology | Gene Expression Profiling | Glutathione Transferase - antagonists & inhibitors | Brain Neoplasms - secondary | Glutathione Transferase - genetics | Isoenzymes - metabolism | bcl-X Protein - antagonists & inhibitors | Biomarkers, Tumor - metabolism | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | Nuclear Proteins - genetics | Lung Neoplasms - genetics | Twist-Related Protein 1 - antagonists & inhibitors | Isoenzymes - genetics | RNA, Messenger - genetics | Cells, Cultured | Brain Neoplasms - genetics | Glutathione Transferase - metabolism | Nuclear Proteins - metabolism | Brain Neoplasms - drug therapy | Fibroblasts - pathology | Reverse Transcriptase Polymerase Chain Reaction | Adenocarcinoma - drug therapy | Breast Neoplasms - drug therapy | Blotting, Western | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Fibroblasts - drug effects | Nuclear Proteins - antagonists & inhibitors | Twist-Related Protein 1 - genetics | Biomarkers, Tumor - genetics | Mice | Twist-Related Protein 1 - metabolism | bcl-X Protein - metabolism | Isoenzymes - antagonists & inhibitors | Apoptosis | Astrocytes - metabolism | Index Medicus | Brain | Cell survival | Astrocytes | Tumor cells | Gap junctions | Lung cancer | Data processing | Breast cancer | siRNA | Radiotherapy | Metastases | Chemotherapy | Fibroblasts | Microenvironments
Journal Article
Cancer chemotherapy and pharmacology, ISSN 0344-5704, 8/2011, Volume 68, Issue 2, pp. 405 - 413
Thioredoxin-1 (Trx-1) redox signaling regulates multiple aspects of cell growth and survival, and elevated tumor levels of Trx-1 have been associated with... 
Medicine & Public Health | Nrf2 | PX-12 | SSAT1 | SSAT2 | Cancer Research | Oncology | HIF-1α | Trx-1 | Pharmacology/Toxicology | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Science & Technology | Neoplasms - metabolism | Acetyltransferases - metabolism | NF-E2-Related Factor 2 - antagonists & inhibitors | Antioxidants - metabolism | Humans | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Acetyltransferases - genetics | DNA-Binding Proteins - metabolism | Thioredoxins - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Isoenzymes - metabolism | Acetyltransferases - antagonists & inhibitors | Disulfides - pharmacology | NF-E2-Related Factor 2 - genetics | Antineoplastic Agents - pharmacology | Thioredoxins - antagonists & inhibitors | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Cell Hypoxia - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Isoenzymes - genetics | Gene Silencing | Transcription Factors - antagonists & inhibitors | Imidazoles - pharmacology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Neoplasms - drug therapy | Response Elements - drug effects | Transcription Factors - metabolism | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Cell Line, Tumor | RNA, Small Interfering | Cell Line, Transformed | Isoenzymes - antagonists & inhibitors | Index Medicus
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 08/2012, Volume 61, Issue 11, pp. 2842 - 2850
In insulin-secreting cells, expression of NADPH oxidase (NOX), a potent source of ROS, has been reported, along with controversial findings regarding its... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Islets of Langerhans - drug effects | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Humans | NADPH Oxidases - metabolism | Secretory Vesicles - metabolism | Insulin-Secreting Cells - metabolism | Membrane Glycoproteins - antagonists & inhibitors | Islets of Langerhans - metabolism | Isoenzymes - metabolism | Islets of Langerhans - cytology | NADPH Oxidases - genetics | Insulin-Secreting Cells - cytology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic AMP - metabolism | Insulin Secretion | Cyclic AMP-Dependent Protein Kinases - metabolism | Second Messenger Systems - drug effects | Glucagon-Like Peptide 1 - metabolism | Isoenzymes - genetics | Tissue Culture Techniques | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Gene Silencing | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Mice, Knockout | Cyclic AMP - antagonists & inhibitors | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Secretory Vesicles - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | RNA, Small Interfering | Cyclic AMP - agonists | Isoenzymes - antagonists & inhibitors | Oxidases | Physiological aspects | Pancreatic beta cells | Research | Analysis | NADP (Coenzyme) | Index Medicus | Abridged Index Medicus | Islet Studies
Journal Article
Breast cancer research : BCR, ISSN 1465-542X, 10/2008, Volume 10, Issue 5, pp. R84 - R84
Introduction Glycolysis is increased in breast adenocarcinoma cells relative to adjacent normal cells in order to produce the ATP and anabolic precursors... 
Life Sciences & Biomedicine | Oncology | Science & Technology | Aspartate Aminotransferases - genetics | Adenocarcinoma - pathology | Humans | Neoplasm Proteins - physiology | Neoplasm Proteins - antagonists & inhibitors | Antineoplastic Agents - therapeutic use | Cell Line, Tumor - drug effects | Glycolysis - drug effects | Oxamic Acid - pharmacology | Cytostatic Agents - therapeutic use | Drug Delivery Systems | Aspartate Aminotransferases - antagonists & inhibitors | Aspartate Aminotransferases - physiology | Recombinant Fusion Proteins - antagonists & inhibitors | Breast Neoplasms - enzymology | RNA Interference | Oxamic Acid - therapeutic use | Aminooxyacetic Acid - therapeutic use | Female | Antineoplastic Agents - pharmacology | Cytostatic Agents - pharmacology | Neoplasm Proteins - genetics | Tumor Stem Cell Assay | Cell Line, Tumor - enzymology | Isoenzymes - genetics | RNA, Small Interfering - pharmacology | Adenocarcinoma - enzymology | Citric Acid Cycle - drug effects | Adenocarcinoma - drug therapy | Breast Neoplasms - drug therapy | Cell Division - drug effects | Xenograft Model Antitumor Assays | Animals | L-Lactate Dehydrogenase - genetics | Breast Neoplasms - pathology | Mice, Nude | Aminooxyacetic Acid - pharmacology | Mice | Mice, Inbred BALB C | L-Lactate Dehydrogenase - antagonists & inhibitors | Isoenzymes - antagonists & inhibitors | Care and treatment | Cancer cells | Physiological aspects | Aspartate aminotransferase | Breast cancer | Research | Health aspects | Index Medicus
Journal Article
Cancer research (Chicago, Ill.), ISSN 0008-5472, 07/2011, Volume 71, Issue 13, pp. 4720 - 4731
Journal Article