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Journal of Biological Chemistry, ISSN 0021-9258, 08/2017, Volume 292, Issue 34, pp. 14250 - 14257
The histidine-phosphorylatable phosphocarrier protein (HPr) is an essential component of the sugar-transporting phosphotransferase system (PTS) in many... 
COMPLEX | DOMAIN | PATHOGEN | glucosamine 6-phosphate deaminase | GLYCOGEN-PHOSPHORYLASE | adenylate kinase (ADK) | BIOCHEMISTRY & MOLECULAR BIOLOGY | glycolysis | phosphofructokinase | isomerase | adenylate kinase | Histidine phosphocarrier protein | GLUCOSAMINE-6-PHOSPHATE DEAMINASE | SUBSTRATE-BINDING | PYRUVATE-KINASE | REPRESSION | pyruvate kinase | protein-protein interaction | PHOSPHOENOLPYRUVATE | ALLOSTERIC ACTIVATION | Phosphorylation | Allosteric Regulation | Bacterial Proteins - chemistry | Pyruvate Kinase - chemistry | Isoenzymes - chemistry | Histidine - metabolism | Adenylate Kinase - chemistry | Recombinant Fusion Proteins - metabolism | Isoenzymes - metabolism | Adenylate Kinase - metabolism | Escherichia coli - metabolism | Escherichia coli Proteins - agonists | Protein Interaction Domains and Motifs | Binding Sites | Aldose-Ketose Isomerases - metabolism | Phosphoenolpyruvate Sugar Phosphotransferase System - chemistry | Phosphoenolpyruvate Sugar Phosphotransferase System - genetics | Recombinant Proteins - metabolism | Aldose-Ketose Isomerases - chemistry | Phosphofructokinase-2 - chemistry | Escherichia coli - enzymology | Pyruvate Kinase - metabolism | Bacterial Proteins - genetics | Models, Molecular | Recombinant Proteins - chemistry | Escherichia coli Proteins - metabolism | Recombinant Fusion Proteins - chemistry | Phosphofructokinase-2 - metabolism | Energy Metabolism | Phosphofructokinase-2 - genetics | Phosphoenolpyruvate Sugar Phosphotransferase System - metabolism | Proteomics | Escherichia coli Proteins - genetics | Glycolysis | Bacterial Proteins - metabolism | Protein Conformation | Protein Processing, Post-Translational | Aldose-Ketose Isomerases - genetics | Enzyme Activation | Escherichia coli Proteins - chemistry | Adenylate Kinase - genetics | Pyruvate Kinase - genetics | Index Medicus | Metabolism
Journal Article
Xenobiotica, ISSN 0049-8254, 10/2012, Volume 42, Issue 10, pp. 957 - 967
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 05/2009, Volume 29, Issue 10, pp. 2704 - 2715
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
OXIDATIVE-STRESS-RESPONSE | NRF2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENES | RESISTANCE | TRANSCRIPTION FACTOR SKN-1 | C-ELEGANS | CHEMOPREVENTION | RIBOSOMAL-RNA | IDENTIFICATION | CANCER | CELL BIOLOGY | Repressor Proteins - classification | Humans | Stress, Physiological | Caenorhabditis elegans Proteins - metabolism | Molecular Sequence Data | Phylogeny | Recombinant Fusion Proteins - metabolism | DNA-Binding Proteins - metabolism | Repetitive Sequences, Amino Acid | Cell Nucleus - metabolism | Glutathione Transferase - genetics | MAP Kinase Kinase 4 - metabolism | Isoenzymes - metabolism | Receptor, Insulin - genetics | Cullin Proteins - metabolism | Repressor Proteins - metabolism | Amino Acid Sequence | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - genetics | Isoenzymes - genetics | Gene Expression Regulation | Glutathione Transferase - metabolism | Repressor Proteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Cullin Proteins - genetics | Transcription Factors - metabolism | Two-Hybrid System Techniques | Caenorhabditis elegans Proteins - classification | Sequence Alignment | Animals | Caenorhabditis elegans - cytology | MAP Kinase Kinase 4 - genetics | Recombinant Fusion Proteins - genetics | Receptor, Insulin - metabolism | Signal Transduction - physiology | Proteasome Endopeptidase Complex - metabolism | Caenorhabditis elegans Proteins - genetics | Forkhead Transcription Factors | Index Medicus
Journal Article
Basic & Clinical Pharmacology & Toxicology, ISSN 1742-7835, 11/2018, Volume 123, Issue 5, pp. 539 - 548
Obesity and insulin resistance are associated with overexpression of retinaldehyde dehydrogenase 1 ( RALDH 1). We aimed to investigate the roles of hepatic... 
PHOSPHOENOLPYRUVATE CARBOXYKINASE | IN-VITRO | OBESITY | LIPID-METABOLISM | INSULIN-RESISTANCE | ISOTRETINOIN | RATS | 13-CIS-RETINOIC ACID | PHARMACOLOGY & PHARMACY | TOXICOLOGY | TRANS-RETINOIC ACID | ALL-TRANS | Humans | Retinal Dehydrogenase - metabolism | Glucose Tolerance Test - methods | Phosphoenolpyruvate Carboxykinase (GTP) - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Hepatocytes - metabolism | Insulin - blood | Monoterpenes - pharmacology | Isoenzymes - metabolism | Diabetes Mellitus, Experimental - metabolism | Diet, High-Fat - methods | Hepatocytes - drug effects | Hyperlipidemias - metabolism | Mice, Inbred C57BL | Insulin Resistance | Rats | Glucokinase - metabolism | Hep G2 Cells | Animals | Diabetes Mellitus, Experimental - etiology | Hyperlipidemias - etiology | Lipid Metabolism - drug effects | Glucose - metabolism | Retinal Dehydrogenase - antagonists & inhibitors | Mice | Isoenzymes - antagonists & inhibitors | Isoenzymes | Physiological aspects | Insulin resistance | Glucose | Glucose tolerance tests | Insulin | Dextrose | Dehydrogenases | Fat metabolism | Liver | Impairment | Pyruvic acid | Lipids | Dehydrogenase | High fat diet | Glucose metabolism | Rodents | Hyperlipidemia | Diabetes mellitus | Retinaldehyde | Metabolism | Glucose tolerance | Hepatocytes | Diet | Glucokinase | Retinaldehyde dehydrogenase | Diabetes | Citral | Index Medicus
Journal Article
Cancer Biology & Therapy, ISSN 1538-4047, 11/2012, Volume 13, Issue 13, pp. 1284 - 1289
Journal Article
Circulation, ISSN 0009-7322, 03/2002, Volume 105, Issue 12, pp. 1429 - 1435
Background-NAD(P)H oxidases are important sources of superoxide in the vasculature, the activity of which is associated with risk factors for human... 
Enzymes | Coronary disease | Arteries | Atherosclerosis | atherosclerosis | GP91PHOX | NADPH OXIDASE | CARDIAC & CARDIOVASCULAR SYSTEMS | arteries | enzymes | LESIONS | coronary disease | CYTOCHROME-B | NAD(P)H OXIDASE | PERIPHERAL VASCULAR DISEASE | STRESS | HEMATOLOGY | ASSOCIATION | Endothelium, Vascular - cytology | Membrane Glycoproteins - metabolism | Muscle, Smooth, Vascular - metabolism | NADPH Dehydrogenase - metabolism | Coronary Vessels - chemistry | Humans | Middle Aged | NADPH Oxidases - metabolism | Superoxides - analysis | Male | NADPH Dehydrogenase - genetics | Phosphoproteins - metabolism | Coronary Vessels - metabolism | RNA, Messenger - biosynthesis | Isoenzymes - metabolism | Coronary Artery Disease - pathology | Polymerase Chain Reaction | Superoxides - metabolism | NADPH Oxidases - genetics | Female | NADH, NADPH Oxidoreductases - biosynthesis | Fibroblasts - metabolism | NADH, NADPH Oxidoreductases - genetics | Coronary Vessels - pathology | Isoenzymes - genetics | Coronary Artery Disease - metabolism | Cells, Cultured | Heart Failure - metabolism | NADPH Oxidase 4 | Phosphoproteins - genetics | Muscle, Smooth, Vascular - cytology | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Endothelium, Vascular - metabolism | Membrane Transport Proteins | Fluorescent Antibody Technique | Arteriosclerosis - metabolism | Fibroblasts - cytology | Arteriosclerosis - pathology | Fluorescent Dyes | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Biology of Reproduction, ISSN 0006-3363, 2013, Volume 89, Issue 4, pp. 93 - 93
JmjC domain-containing proteins are a class of enzymes responsible for histone demethylation. Previous studies revealed that the JmjC domain-containing protein... 
Aging | Epigenetics | Histone modifications | Male infertility | SPERMATOGENESIS | STEM-CELLS | METHYLATION | MOUSE | SELF-RENEWAL | HISTONE DEMETHYLASES | histone modifications | male infertility | PLZF | epigenetics | REPRODUCTIVE BIOLOGY | GENE | DYNAMICS | aging | DIFFERENTIATION | Infertility, Male - enzymology | Testis - metabolism | Alternative Splicing | Spermatogenesis | Androstenedione - blood | Male | Spermatogonia - metabolism | Isoenzymes - metabolism | Mice, Mutant Strains | Jumonji Domain-Containing Histone Demethylases - antagonists & inhibitors | Adult Stem Cells - pathology | Spermatogonia - cytology | Testosterone - blood | Spermatogonia - pathology | Infertility, Male - blood | Animals, Newborn | Adult Stem Cells - cytology | Jumonji Domain-Containing Histone Demethylases - deficiency | Testis - cytology | Isoenzymes - genetics | Mice, Inbred C57BL | Mutant Proteins - metabolism | Isoenzymes - deficiency | Infertility, Male - pathology | Testis - growth & development | Jumonji Domain-Containing Histone Demethylases - genetics | Gene Expression Regulation, Enzymologic | Adult Stem Cells - metabolism | Animals | Testis - pathology | Infertility, Male - metabolism | Mice | Jumonji Domain-Containing Histone Demethylases - metabolism | Isoenzymes - antagonists & inhibitors | Apoptosis | Index Medicus
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 12/2016, Volume 101, pp. 334 - 347
AMPK dysregulation contributes to the onset and development of type 2 diabetes (T2DM). AMPK is known to be activated by reactive oxygen species (ROS) and... 
Type 2 diabetes | Glucose metabolism | AMPK | Glutathionylation | Glutaredoxins | ROS | OXIDATIVE STRESS | RAT | MECHANISM | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PREVENTS | INHIBITION | OXIDASE | IN-VIVO | ENDOCRINOLOGY & METABOLISM | RESISTANCE | ENERGY-METABOLISM | AMP-Activated Protein Kinases - metabolism | Liver - pathology | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Streptozocin | Humans | Diet, High-Fat - adverse effects | Male | Diabetes Mellitus, Type 2 - metabolism | Hepatocytes - metabolism | Protein Subunits - metabolism | Reactive Oxygen Species - agonists | Hepatocytes - cytology | Diabetes Mellitus, Experimental - prevention & control | Glycogen - metabolism | HEK293 Cells | Glutaredoxins - genetics | Diabetes Mellitus, Experimental - metabolism | Epithelial Cells - cytology | Protein Subunits - genetics | Cell Line | Glutaredoxins - metabolism | Oxidation-Reduction | Signal Transduction | Liver - metabolism | Gene Expression Regulation | Rats | Rats, Sprague-Dawley | Reactive Oxygen Species - antagonists & inhibitors | Animals | Diabetes Mellitus, Experimental - etiology | Diabetes Mellitus, Experimental - pathology | Glucose - metabolism | Diabetes Mellitus, Type 2 - pathology | AMP-Activated Protein Kinases - genetics | Antioxidants | Synthesis | Glycogen | Physiological aspects | Development and progression | Hypoglycemic agents | Glucose | Dextrose | Oxidases | Cysteine | Isoenzymes | Transmission electron microscopes | Superoxide | Glucose tolerance tests | Hyperglycemia | High performance liquid chromatography | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2005, Volume 280, Issue 35, pp. 31036 - 31044
Journal Article
Cellular Signalling, ISSN 0898-6568, 02/2015, Volume 27, Issue 2, pp. 228 - 235
The Warburg effect is an oncogenic metabolic switch that allows cancer cells to take up more glucose than normal cells and favors anaerobic glycolysis.... 
GLUT1 | ECM1 | LDHA | PKM2 | Warburg effect | HYPOXIA | IDENTIFICATION | CELL BIOLOGY | HEPATOCELLULAR-CARCINOMA | PYRUVATE-KINASE | METABOLISM | HUMAN CANCERS | TUMOR-GROWTH | CELL | GLUCOSE-TRANSPORTER EXPRESSION | L-Lactate Dehydrogenase - metabolism | Up-Regulation | Phosphorylation | Humans | Transcriptional Activation | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Glucose Transporter Type 1 - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Tandem Mass Spectrometry | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Isoenzymes - metabolism | Extracellular Matrix Proteins - metabolism | Pyruvate Kinase - metabolism | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Isoenzymes - genetics | Extracellular Matrix Proteins - antagonists & inhibitors | Extracellular Matrix Proteins - genetics | Epidermal Growth Factor - metabolism | Pyruvate Kinase - antagonists & inhibitors | L-Lactate Dehydrogenase - genetics | Glucose Transporter Type 1 - genetics | Cell Line, Tumor | Glucose - metabolism | Glycolysis | Pyruvate Kinase - genetics | Early Growth Response Protein 1 - metabolism | RNA, Small Interfering - metabolism | Glucose metabolism | Epidermal growth factor | Glucose | Gene expression | Mass spectrometry | Dextrose | Index Medicus | Proteins | Cellular | Genes | Activation | Transporter
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2008, Volume 283, Issue 41, pp. 27653 - 27667
Protein kinase B (PKB)/Akt has been strongly implicated in the insulin-dependent stimulation of GLUT4 translocation and glucose transport in skeletal muscle... 
GLYCOGEN-SYNTHASE | TRANSPORT | ACTIVATION | SKELETAL-MUSCLE CELLS | PLECKSTRIN-HOMOLOGY-DOMAIN | PHOSPHATIDYLINOSITOL 3,4,5-TRISPHOSPHATE | MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUT4 VESICLES | B-ALPHA | GROWTH-FACTOR-I | Glucose Transporter Type 4 - metabolism | Ribosomal Protein S6 Kinases - metabolism | Glycogen - genetics | Adipocytes - cytology | Muscle, Skeletal - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | GTPase-Activating Proteins - metabolism | Mutation, Missense | Amino Acids - genetics | Muscle, Skeletal - cytology | Extracellular Signal-Regulated MAP Kinases - genetics | Proto-Oncogene Proteins c-akt - genetics | Amino Acids - metabolism | Glycogen - metabolism | Isoenzymes - metabolism | Insulin Receptor Substrate Proteins | Biological Transport - genetics | Amino Acid Transport System A - genetics | Proto-Oncogene Proteins c-akt - metabolism | Insulin - pharmacology | Glucose Transporter Type 4 - genetics | Isoenzymes - genetics | Rats | 3T3-L1 Cells | Glycogen Synthase Kinase 3 - metabolism | Hypoglycemic Agents - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance - genetics | Animals | Glycogen Synthase Kinase 3 - genetics | Adipocytes - metabolism | Adaptor Proteins, Signal Transducing - genetics | Amino Acid Transport System A - metabolism | Glucose - metabolism | Mice | GTPase-Activating Proteins - genetics | Adaptor Proteins, Signal Transducing - metabolism | Enzyme Activation - genetics | Ribosomal Protein S6 Kinases - genetics | Isoenzymes - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Index Medicus | Clinical Medicine | Endokrinologi och diabetes | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
Journal Article
The Journal of Physiology, ISSN 0022-3751, 07/2007, Volume 582, Issue 1, pp. 379 - 392
Journal Article
Blood, ISSN 0006-4971, 12/2011, Volume 118, Issue 24, pp. 6332 - 6341
Journal Article