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Journal Article
1972, Organic chemistry, Volume 25, 594
Book
BIOLOGICAL PSYCHIATRY, ISSN 0006-3223, 10/2011, Volume 70, Issue 8, pp. 712 - 719
Background: Drug-associated cues can elicit stress-like responses in addicted individuals, indicating that cue-and stress-induced drug relapse may share some... 
norepinephrine | ANXIOGENIC DRUG YOHIMBINE | relapse | PSYCHIATRY | NEUROSCIENCES | BED NUCLEUS | corticotropin-releasing factor | CORTICOTROPIN-RELEASING-FACTOR | INDUCED RELAPSE | STRIA TERMINALIS | ALCOHOL-SEEKING | self-administration | STRESS-INDUCED REINSTATEMENT | ALPHA-ADRENERGIC RECEPTORS | Cocaine | PITUITARY-ADRENAL AXIS | I-1-IMIDAZOLINE RECEPTOR | imidazoline | Extinction, Psychological - drug effects | Motor Activity - drug effects | Male | Adrenergic alpha-2 Receptor Agonists - pharmacology | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Drug Interactions | Isoquinolines - pharmacology | Self Administration - psychology | Adrenergic alpha-1 Receptor Antagonists - pharmacology | Prazosin - pharmacology | Clonidine - pharmacology | Cocaine - antagonists & inhibitors | Cues | Guanfacine - pharmacology | Reinforcement (Psychology) | Rats | Brimonidine Tartrate | Imidazoles - pharmacology | Pyrimidines - pharmacology | Adrenergic alpha-2 Receptor Antagonists - pharmacology | Rats, Sprague-Dawley | Drug-Seeking Behavior - drug effects | Clonidine - antagonists & inhibitors | Cocaine - administration & dosage | Cocaine - pharmacology | Pyrroles - pharmacology | Animals | Imidazoles - antagonists & inhibitors | Imidazoline Receptors - agonists | Naphthyridines - pharmacology | Index Medicus
Journal Article
Journal of Pharmacological Sciences, ISSN 1347-8613, 03/2017, Volume 133, Issue 3, pp. 168 - 175
Journal Article
The Journal of Physiology, ISSN 0022-3751, 07/2010, Volume 588, Issue 14, pp. 2605 - 2619
In vertebrate retina, melatonin regulates various physiological functions. In this work we investigated the mechanisms underlying melatonin‐induced... 
CIRCADIAN CLOCK | XENOPUS-LAEVIS | BIPOLAR CELLS | PROTEIN-KINASE-C | PHYSIOLOGY | FISH RETINA | PHOSPHOLIPASE-C | VISUAL PATHWAY | FUNCTIONAL-ROLE | MT2 MELATONIN | RECEPTORS | NEUROSCIENCES | Cyclic GMP - pharmacology | Receptor, Melatonin, MT2 - antagonists & inhibitors | Tetradecanoylphorbol Acetate - pharmacology | Maleimides - pharmacology | Male | Calcium - analysis | Melatonin - physiology | Type C Phospholipases - antagonists & inhibitors | Cyclic GMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic GMP-Dependent Protein Kinases - physiology | Isoquinolines - pharmacology | Cyclic GMP - analogs & derivatives | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Bridged-Ring Compounds - pharmacology | Egtazic Acid - analogs & derivatives | Indoles - pharmacology | Guanosine Diphosphate - pharmacology | Pertussis Toxin - pharmacology | Retinal Ganglion Cells - physiology | Protein Kinase C - physiology | Retinal Ganglion Cells - enzymology | 8-Bromo Cyclic Adenosine Monophosphate - pharmacology | Rats | Protein Kinase C - antagonists & inhibitors | Receptor, Melatonin, MT2 - physiology | Sulfonamides - pharmacology | Rats, Sprague-Dawley | Animals | Egtazic Acid - pharmacology | Glycine - physiology | Guanosine Diphosphate - analogs & derivatives | Signal Transduction - drug effects | Tetrahydronaphthalenes - pharmacology | Thiones - pharmacology | Carbazoles - pharmacology | Type C Phospholipases - physiology | Melatonin | Phospholipids | Glycine | Ganglion | Gross domestic product | Protein kinases | Kinases | Retina | Physiology | Index Medicus | Neuroscience
Journal Article
Journal Article
NEUROPSYCHOPHARMACOLOGY, ISSN 0893-133X, 01/2004, Volume 29, Issue 1, pp. 45 - 58
Intense or chronic stress can produce pathophysiological alterations in the systems involved in the stress response. The amygdala is a key component of the... 
GLUCOCORTICOID RECEPTOR | stress | amygdala | alpha adrenoceptors | NUCLEUS-TRACTUS-SOLITARIUS | BENZODIAZEPINE RECEPTORS | PSYCHIATRY | inhibitory synaptic transmission | ALPHA-ADRENOCEPTOR SUBTYPES | RECEPTOR MESSENGER-RNA | CYCLIC-AMP RESPONSE | GABA release | NOREPINEPHRINE RELEASE | NEUROSCIENCES | TRANSDUCTION PATHWAYS | RAT SENSORIMOTOR CORTEX | norepinelphrine | SYSTEM IN-VITRO | PHARMACOLOGY & PHARMACY | Receptors, Adrenergic, alpha-1 - metabolism | Amygdala - physiopathology | Norepinephrine - pharmacology | Amygdala - drug effects | Body Weight | gamma-Aminobutyric Acid - metabolism | 2-Amino-5-phosphonovalerate - pharmacology | Male | Adrenergic alpha-Antagonists - pharmacology | Anesthetics, Local - pharmacology | Patch-Clamp Techniques - methods | Dose-Response Relationship, Drug | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Drug Interactions | Time Factors | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Restraint, Physical - methods | Stress, Physiological - metabolism | Neurons - physiology | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Aging | Tetrodotoxin - pharmacology | Neurons - drug effects | Animals, Newborn | Isoquinolines - metabolism | Membrane Potentials - drug effects | Amygdala - metabolism | Morpholines - pharmacology | Phosphodiesterase Inhibitors - pharmacology | Rats | Amygdala - cytology | Imidazoles - pharmacology | Bicuculline - pharmacology | Rats, Sprague-Dawley | GABA Antagonists - pharmacology | Animals | Tetrahydronaphthalenes - pharmacology | In Vitro Techniques | Excitatory Amino Acid Antagonists | Neural Inhibition - drug effects | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 09/2006, Volume 114, Issue 13, pp. 1372 - 1379
Background - At birth, the increase in oxygen causes contraction of the ductus arteriosus, thus diverting blood flow to the lungs. Although this contraction is... 
Oxygen | Ion channels | Vasoconstriction | Ductus arteriosus, patent | Heart defects, congenital | heart defects | CARDIAC & CARDIOVASCULAR SYSTEMS | ductus arteriosus, patent | TRPC1 | RECEPTOR | CLOSURE | congenital | CAPACITATIVE CA2+ ENTRY | PULMONARY VASOCONSTRICTION | CYTOSOLIC CALCIUM | ion channels | CONSTRICTION | PERIPHERAL VASCULAR DISEASE | vasoconstriction | SMOOTH-MUSCLE-CELLS | RABBIT | oxygen | Oxygen - pharmacology | Tetraethylammonium - pharmacology | Calcium Signaling - physiology | Maleimides - pharmacology | Potassium Channels - physiology | Thiourea - pharmacology | Polyunsaturated Alkamides | Isoquinolines - pharmacology | rho-Associated Kinases | Niflumic Acid - pharmacology | Indoles - pharmacology | Endocannabinoids | Ductus Arteriosus - physiology | Boron Compounds - pharmacology | Mibefradil - pharmacology | Nifedipine - pharmacology | Calcium - pharmacology | Oxidation-Reduction | Protein-Serine-Threonine Kinases - physiology | Rabbits - embryology | Calcium Channels, L-Type - physiology | Imidazoles - pharmacology | Calcium Channel Blockers - pharmacology | Potassium Channels - drug effects | Sulfonamides - pharmacology | Arachidonic Acids - pharmacology | Ductus Arteriosus - embryology | Patch-Clamp Techniques | Animals | Menthol - pharmacology | Ruthenium Red - pharmacology | Muscle Contraction - drug effects | Calcium Channels, L-Type - drug effects | Muscle Contraction - physiology | Thapsigargin - pharmacology | Cytosol - metabolism | In Vitro Techniques | Intracellular Signaling Peptides and Proteins - physiology | Thiourea - analogs & derivatives | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 07/2017, Volume 313, Issue 1, pp. C118 - C129
Parathyroid hormone (PTH), a pleiotropic hormone that maintains mineral homeostasis, is also essential for controlling pH balance and ion transport across... 
Na | Anion secretion | Impedance analysis | exchanger | H | Ussing chamber | Potassium channel | anion secretion | PHYSIOLOGY | Na+/H+ exchanger | RAT | EXCHANGER NHE-3 | PROTEIN-KINASE | MEMBRANE REPAIR | CELL BIOLOGY | TRANSPORT | impedance analysis | HCO3-SECRETION | potassium channel | PARATHYROID-HORMONE | CL-SECRETION | CELL-LINE | BICARBONATE SECRETION | Potassium - metabolism | Sodium-Potassium-Exchanging ATPase - genetics | Calcium - metabolism | Humans | Parathyroid Hormone - pharmacology | Electric Conductivity | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Potassium Channels, Calcium-Activated - genetics | Isoquinolines - pharmacology | Chlorides - metabolism | Potassium Channels, Calcium-Activated - metabolism | Ion Transport - drug effects | Chromones - pharmacology | Action Potentials - drug effects | Pyrazoles - pharmacology | Caco-2 Cells | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Acetazolamide - pharmacology | Chlorides - pharmacology | Colforsin - pharmacology | Clotrimazole - pharmacology | Morpholines - pharmacology | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Carbonic Anhydrase Inhibitors - pharmacology | Sulfonamides - pharmacology | Barium Compounds - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Sodium-Potassium-Exchanging ATPase - metabolism | Androstadienes - pharmacology | Phosphorus - metabolism | Wortmannin | Bicarbonates - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Potassium Channels, Calcium-Activated - antagonists & inhibitors | Hydrogen-Ion Concentration | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 06/2007, Volume 74, Issue 3, pp. 406 - 415
Objectives: Canine atrial cardiomyocytes display a constitutively active, acetylcholine-regulated, time-dependent K current (I ) that contributes to atrial... 
Second-messengers | Signal transduction | Ion-channels | PROTEIN-KINASE-C | CARDIAC & CARDIOVASCULAR SYSTEMS | PHOSPHATIDYLINOSITOL 4,5-BISPHOSPHATE | signal transduction | RECTIFYING K+ CHANNELS | FORM STABLE COMPLEXES | VENTRICULAR MYOCYTES | GUINEA-PIG | second-messengers | SIGNALING COMPLEX | TRANSIENT OUTWARD CURRENT | ion-channels | ADENYLYL-CYCLASE | XENOPUS-OOCYTES | Maleimides - pharmacology | Adrenergic alpha-Antagonists - pharmacology | Phorbol 12,13-Dibutyrate - pharmacology | Type C Phospholipases - antagonists & inhibitors | Dose-Response Relationship, Drug | Propranolol - pharmacology | Isoquinolines - pharmacology | Phenylephrine - pharmacology | Prazosin - pharmacology | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Clonidine - pharmacology | Indoles - pharmacology | Receptors, Adrenergic, beta-1 - drug effects | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Action Potentials - drug effects | Acetylcholine - pharmacology | Heart Atria | Receptors, Adrenergic, alpha-1 - drug effects | Bee Venoms - pharmacology | Imidazoles - pharmacology | Protein Kinase C - antagonists & inhibitors | Potassium Channels - drug effects | Sulfonamides - pharmacology | Adrenergic Agents - pharmacology | Piperazines - pharmacology | Clonidine - analogs & derivatives | Patch-Clamp Techniques | Animals | Myocytes, Cardiac - drug effects | Isoproterenol - pharmacology | Dogs | Dihydropyridines - pharmacology | Myocytes, Cardiac - metabolism | Adrenergic alpha-Agonists - pharmacology | Propanolamines - pharmacology | Heart cells | Index Medicus
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 01/2005, Volume 21, Issue 1, pp. 151 - 160
The mechanisms by which uridine triphosphate (UTP) stimulates ATP release from Schwann cells cultured from the sciatic nerve were investigated using online... 
Bioluminescence | Protein kinase C | Purinergic receptors | Exocytosis | purinergic receptors | NERVE | MECHANISMS | RELEASE | ASTROCYTES | NEUROSCIENCES | UTP | NUCLEOTIDE RECEPTORS | bioluminescence | GLUTAMATE | CALCIUM | NEURONS | exocytosis | SMOOTH-MUSCLE-CELLS | protein kinase C | Guanosine Triphosphate - pharmacology | Nucleic Acid Synthesis Inhibitors - pharmacology | Calcium - metabolism | Adenosine Triphosphate - secretion | Brefeldin A - pharmacology | Phorbol 12,13-Dibutyrate - pharmacology | Receptors, Purinergic P2Y2 | Cytochalasin D - pharmacology | Phenanthridines - pharmacology | Type C Phospholipases - antagonists & inhibitors | Diagnostic Imaging - methods | Dose-Response Relationship, Drug | Alkaloids | Botulinum Toxins - pharmacology | Glycyrrhetinic Acid - pharmacology | Drug Interactions | Isoquinolines - pharmacology | Glyburide - pharmacology | Time Factors | Botulinum Toxins, Type A | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Sciatic Nerve - cytology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Protein Synthesis Inhibitors - pharmacology | Pyridoxal Phosphate - analogs & derivatives | Schwann Cells - secretion | Suramin - pharmacology | Schwann Cells - drug effects | Animals, Newborn | Pyridoxal Phosphate - pharmacology | Uridine Triphosphate - pharmacology | Purinergic P2 Receptor Antagonists | Rats | Protein Kinase C - antagonists & inhibitors | Purinergic P2 Receptor Agonists | Sulfonamides - pharmacology | Microscopy, Confocal - methods | Rats, Sprague-Dawley | Immunohistochemistry - methods | Animals | Benzophenanthridines | Receptors, Purinergic P2 - metabolism | Thapsigargin - pharmacology | Furosemide - pharmacology | Index Medicus
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 2013, Volume 304, Issue 6, pp. C508 - C518
Younce CW, Burmeister MA, Ayala JE. Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and... 
Heart | Glucagon-like peptide-1 | Hyperglycemia | CARDIAC-FUNCTION | PEPTIDE-1 RECEPTOR | OXIDATIVE STRESS | PHYSIOLOGY | GLP-1 | CONTRACTILE DYSFUNCTION | ER STRESS | glucagon-like peptide-1 | DIABETIC CARDIOMYOPATHY | heart | CELL BIOLOGY | hyperglycemia | ENDOTHELIAL-CELLS | ZINC-FINGER PROTEIN | Receptors, Glucagon - antagonists & inhibitors | Diabetic Cardiomyopathies - metabolism | Apoptosis - drug effects | Peptide Fragments - pharmacology | Exenatide | RNA, Messenger - biosynthesis | Isoquinolines - pharmacology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - antagonists & inhibitors | Phosphorylation - drug effects | Transcription Factor CHOP - biosynthesis | Calcium-Binding Proteins - metabolism | Receptors, Glucagon - agonists | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Endoplasmic Reticulum Stress - drug effects | Colforsin - pharmacology | Heat-Shock Proteins - metabolism | Cells, Cultured | Hydrogen Peroxide - pharmacology | Rats | Receptors, Glucagon - metabolism | Sulfonamides - pharmacology | Unfolded Protein Response | Hypoglycemic Agents - pharmacology | Peptides - pharmacology | Hyperglycemia - metabolism | Membrane Proteins | Animals | Glucagon-Like Peptide-1 Receptor | Myocytes, Cardiac - physiology | Tunicamycin - pharmacology | Glucose - metabolism | Thapsigargin - pharmacology | Protein Kinase Inhibitors - pharmacology | Venoms - pharmacology | Enzyme Activation | Oxidative Stress - drug effects | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Transcription Factor CHOP - metabolism | HSP70 Heat-Shock Proteins | Care and treatment | Heart cells | Physiological aspects | Stress (Physiology) | Dosage and administration | Health aspects | Endoplasmic reticulum | Apoptosis | Molecules | Phosphorylation | Glucose | Enzyme kinetics | Rodents | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 06/1998, Volume 20, Issue 6, pp. 1211 - 1218
The mechanism through which kainate receptors downregulate the release of GABA in the hippocampus is not known. We have found that the action of kainate on the... 
PROTEIN-KINASE-C | GLUTAMATE RECEPTORS | INHIBITION | CA2+ CHANNELS | RAT HIPPOCAMPUS | SYNAPTIC TRANSMISSION | CURRENTS | HIPPOCAMPAL-NEURONS | EXPRESSION | NEUROSCIENCES | BRAIN | Tetraethylammonium - pharmacology | Rats, Wistar | Hippocampus - chemistry | gamma-Aminobutyric Acid - metabolism | Receptors, Metabotropic Glutamate - physiology | Type C Phospholipases - metabolism | Phorbol 12,13-Dibutyrate - pharmacology | Calcium - physiology | Carcinogens - pharmacology | Isoquinolines - pharmacology | Cyclic AMP - analogs & derivatives | Protein Kinase C - metabolism | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Hippocampus - enzymology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Tetrodotoxin - pharmacology | Virulence Factors, Bordetella | Membrane Potentials - drug effects | Sodium - pharmacology | Calcium - pharmacology | Kainic Acid - pharmacology | Enzyme Inhibitors - pharmacology | Pertussis Toxin | Phosphodiesterase Inhibitors - pharmacology | Rats | Excitatory Amino Acid Agonists - pharmacology | Thionucleotides - pharmacology | Enzyme Activation - drug effects | Cyclic AMP - pharmacology | Receptors, Kainic Acid - metabolism | Animals | Sulfonamides | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - pharmacology | Second Messenger Systems - physiology | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Staurosporine - pharmacology | Phorbol esters | GABA | Protein kinases | calcium (extracellular) | kainic acid | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2010, Volume 30, Issue 21, pp. 7236 - 7248
Similar to dopamine (DA), cannabinoids strongly influence prefrontal cortical functions, such as working memory, emotional learning, and sensory perception.... 
CONCURRENT STIMULATION | CATECHOL-O-METHYLTRANSFERASE | PYRAMIDAL NEURONS | GLUTAMATERGIC TRANSMISSION | NEURONS IN-VITRO | RECEPTOR KNOCKOUT MICE | LONG-TERM DEPRESSION | SYNAPTIC-TRANSMISSION | CANNABINOID CB1 | NEUROSCIENCES | NUCLEUS-ACCUMBENS | Silver Staining - methods | Rats, Wistar | Microscopy, Electron, Transmission - methods | gamma-Aminobutyric Acid - metabolism | Receptors, Dopamine D2 - metabolism | Cannabinoid Receptor Modulators | Neuronal Plasticity - genetics | Isoquinolines - pharmacology | Neuronal Plasticity - physiology | Receptor, Cannabinoid, CB1 - agonists | Inhibitory Postsynaptic Potentials - genetics | Piperidines - pharmacology | Dopamine Agents - pharmacology | Egtazic Acid - analogs & derivatives | Female | Dopamine - metabolism | Endocannabinoids | Pyrazoles - pharmacology | Animals, Newborn | Membrane Potentials - drug effects | Synapses - drug effects | Synapses - physiology | Mice, Inbred C57BL | Morpholines - pharmacology | Rats | Synapses - ultrastructure | Chelating Agents - pharmacology | Prefrontal Cortex - cytology | Sulfonamides - pharmacology | Prefrontal Cortex - ultrastructure | Mice, Knockout | Naphthalenes - pharmacology | Receptor, Cannabinoid, CB1 - metabolism | Patch-Clamp Techniques | Animals | Egtazic Acid - pharmacology | Benzoxazines - pharmacology | Mice | Protein Kinase Inhibitors - pharmacology | Receptor, Cannabinoid, CB1 - deficiency | In Vitro Techniques | Inhibitory Postsynaptic Potentials - drug effects | Index Medicus | transmitter release | CB1 receptor | presynaptic | inhibition | schizophrenia | Neocortex | LTD | dopamine receptor
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 08/2002, Volume 174, Issue 2, pp. 233 - 246
Glucose-dependent insulinotropic polypeptide (GIP) acts as a glucose-dependent growth factor for beta-cells. Here we show that GIP and glucose also act... 
PATHWAYS | IGF-I | RECEPTOR TYROSINE KINASES | ACTIVATED PROTEIN-KINASE | PHOSPHATIDYLINOSITOL 3-KINASE | MAP KINASE | ENDOCRINOLOGY & METABOLISM | ELEMENT-BINDING PROTEIN | INS-1 CELLS | GROWTH-FACTOR | FACTOR-I | Apoptosis - drug effects | Calcium - metabolism | Humans | Maleimides - pharmacology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | MAP Kinase Kinase 1 | Isoquinolines - pharmacology | Deoxyglucose - pharmacology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Indoles - pharmacology | Flavonoids - pharmacology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Gastric Inhibitory Polypeptide - pharmacology | B-Lymphocytes - metabolism | Cell Line | Enzyme Inhibitors - pharmacology | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Arginase - genetics | Protein Kinase C - antagonists & inhibitors | Chelating Agents - pharmacology | Calcium-Calmodulin-Dependent Protein Kinases - antagonists & inhibitors | Sulfonamides - pharmacology | Egtazic Acid - pharmacology | MAP Kinase Signaling System - drug effects | Mitosis - drug effects | Androstadienes - pharmacology | Signal Transduction - drug effects | Glucokinase - antagonists & inhibitors | Plasmids | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Wortmannin | Genistein - pharmacology | Glucose - metabolism | Alloxan - pharmacology | Benzylamines - pharmacology | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus
Journal Article