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MOLECULAR CELL, ISSN 1097-2765, 06/2008, Volume 30, Issue 6, pp. 678 - 688
Starvation induces autophagy to preserve cellular homeostasis in virtually all eukaryotic organisms. However, the mechanisms by which starvation induces... 
SIGNAL-TRANSDUCTION | BCL-2 PHOSPHORYLATION | PROTEIN | BECLIN-1 | PATHWAY | JNK | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL KINASE | STRESS | T-CELLS | CELL-DEATH | CELL BIOLOGY
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, pp. e23999 - e23999
Systemic inflammation is often associated with impaired glucose metabolism. We therefore studied the activation of inflammatory pathway intermediates that... 
HUMAN ENDOTOXEMIA | METABOLIC SYNDROME | OBESITY | PROTEIN-KINASE | INFLAMMATION | BIOLOGY | RESISTANCE | NECROSIS-FACTOR-ALPHA | GLUCOSE-TRANSPORT | INSULIN-RECEPTOR SUBSTRATE-1 | EXPRESSION | AMP-Activated Protein Kinases - metabolism | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | RNA, Messenger - metabolism | Transcription Factor RelA - genetics | Muscle, Skeletal - drug effects | Protein Binding - drug effects | Adult | Phosphorylation - drug effects | Muscle, Skeletal - enzymology | RNA, Messenger - genetics | Insulin Resistance | DNA - metabolism | Gene Expression Regulation - drug effects | Diabetes Mellitus, Type 2 - physiopathology | Transcription Factor RelA - metabolism | Lipopolysaccharides - pharmacology | Aged | Diabetes Mellitus, Type 2 - pathology | Muscle, Skeletal - pathology | Acetyl-CoA Carboxylase - metabolism | Transcription factors | Intravenous administration | Escherichia coli | Glucose | Family medical history | Lipopolysaccharides | Proteins | Signal transduction | Hyperglycemia | Acetyl-CoA carboxylase | Deoxyribonucleic acid--DNA | Binding | NF-κB protein | Cytokines | AMP | Tumor necrosis factor-α | Metabolism | Gene expression | Patients | Fatty acids | Polymerase chain reaction | Infectious diseases | Sepsis | TNF inhibitors | Plasma | Phosphorylation | Drug delivery systems | Intensive care | Interleukin | Reverse transcription | Kinases | AMP-activated protein kinase | Western blotting | Interleukin 6 | Glucose metabolism | Ethics | Pathways | Tumor necrosis factor-TNF | Oxidation | Enzyme-linked immunosorbent assay | Age | Diabetes mellitus | c-Jun protein | Inflammation | Skeletal muscle | Intermediates | Glucose tolerance | Musculoskeletal system | Ethical standards | Biopsy | Protein expression | Informed consent | Insulin resistance | Diabetes | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Behavioural Brain Research, ISSN 0166-4328, 03/2011, Volume 217, Issue 2, pp. 424 - 431
▶ SP600125 pretreatment of the rats protected them against impairment of spatial learning induced by Aβ infusion. ▶ SP600125 pretreatment fortified the treated... 
Morris water maze | Rat | JNK | Alzheimer | β-amyloid | Apoptosis | SP600125 | INDUCED NEURONAL APOPTOSIS | SIGNALING PATHWAYS | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | ALZHEIMERS-DISEASE | MAP KINASE | N-TERMINAL KINASE | NEUROSCIENCES | beta-amyloid | A-BETA | BEHAVIORAL SCIENCES | RAT HIPPOCAMPUS
Journal Article
The EMBO Journal, ISSN 0261-4189, 04/2004, Volume 23, Issue 8, pp. 1889 - 1899
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 08/2008, Volume 28, Issue 34, pp. 8430 - 8441
17 beta-Estradiol (E2) has been implicated to be neuroprotective in a variety of neurodegenerative disorders, although the mechanism remains poorly understood.... 
Cerebral ischemia | 17β-estradiol | Survivin | Alzheimer's disease | Neuronal cell death | Hippocampus | survivin | GLOBAL-ISCHEMIA | 17 beta-estradiol | hippocampus | ALZHEIMERS-DISEASE | NEUROSCIENCES | CELL-DEATH | neuronal cell death | WNT PATHWAY | cerebral ischemia | NEURONAL DEATH | NEGATIVE MODULATOR | ACUTE STROKE | ISCHEMIC BRAIN-INJURY | SEX-DIFFERENCES | CEREBRAL-ISCHEMIA | Neurons - pathology | Up-Regulation | Microtubule-Associated Proteins - metabolism | Injections, Intraventricular | Brain Ischemia - metabolism | tau Proteins - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Wnt Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neuroprotective Agents - pharmacology | Female | Cell Death - drug effects | Phosphorylation - drug effects | Intercellular Signaling Peptides and Proteins - administration & dosage | Neurons - drug effects | Estradiol - pharmacology | Rats | Brain Ischemia - physiopathology | Hippocampus - pathology | Recombinant Proteins - pharmacology | Down-Regulation - drug effects | Rats, Sprague-Dawley | beta Catenin - metabolism | Recombinant Proteins - administration & dosage | Hippocampus - metabolism | Animals | Signal Transduction - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | Proto-Oncogene Proteins c-jun - metabolism | Brain Ischemia - pathology | Hippocampus - physiopathology | Index Medicus | 17β-Estradiol | Alzheimer’s disease | surviving
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2009, Volume 29, Issue 28, pp. 9078 - 9089
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 10/2001, Volume 176, Issue 2, pp. 110 - 117
Exposure of normal human epidermal keratinocytes (NHEK) to UVB radiation induces intracellular release of hydrogen peroxide (oxidative stress) and... 
mitogen-activated protein kinase | epigallocatechin-3-gallate | keratinocytes | oxidative stress | ultraviolet light | Keratinocytes | Mitogen-activated protein kinase | Oxidative stress | Ultraviolet light | Epigallocatechin-3-gallate | TRANSDUCTION | MAP KINASES | CELL-SURVIVAL | GENE-EXPRESSION | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | HYDROGEN-PEROXIDE | TOXICOLOGY | GROWTH-FACTOR | C-JUN | ULTRAVIOLET-RADIATION | SKIN-CANCER | MAP Kinase Signaling System - physiology | Oxidants - pharmacology | Phosphorylation | Humans | Oxidative Stress - physiology | Mitogen-Activated Protein Kinase Kinases - metabolism | MAP Kinase Kinase 4 | Ultraviolet Rays | JNK Mitogen-Activated Protein Kinases | Oxidative Stress - radiation effects | Catechin - pharmacology | p38 Mitogen-Activated Protein Kinases | Epidermis - metabolism | MAP Kinase Signaling System - radiation effects | Epidermis - drug effects | Hydrogen Peroxide - pharmacology | Antioxidants - pharmacology | Blotting, Western | Keratinocytes - drug effects | Keratinocytes - metabolism | Ascorbic Acid - pharmacology | Catechin - analogs & derivatives | Mitogen-Activated Protein Kinase 3 | Epidermis - cytology | Mitogen-Activated Protein Kinases - metabolism | Antioxidants | Catechin | Hydrogen peroxide | Cellular signal transduction | Skin | Mitogens | Green tea | Protein kinases | EGCG | Index Medicus
Journal Article