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The EMBO Journal, ISSN 0261-4189, 02/2011, Volume 30, Issue 4, pp. 770 - 782
Notch signalling is important for development and tissue homeostasis and activated in many human cancers. Nevertheless, mutations in Notch pathway components... 
miR‐200 | ZEB1 | EMT | Notch | stemness | miR-200 | STEM-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | PHENOTYPE | E-CADHERIN | MIR-200 FAMILY | REPRESSORS ZEB1 | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | COLORECTAL-CANCER | Receptors, Notch - metabolism | Humans | Receptors, Notch - genetics | Gene Knockdown Techniques | Intercellular Signaling Peptides and Proteins - physiology | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neoplasms - genetics | Membrane Proteins - physiology | Serrate-Jagged Proteins | Base Sequence | Membrane Proteins - metabolism | Nuclear Proteins - genetics | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Transcription Factors - physiology | DNA-Binding Proteins - antagonists & inhibitors | Membrane Proteins - genetics | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Signal Transduction - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Models, Biological | Calcium-Binding Proteins - physiology | Homeodomain Proteins - antagonists & inhibitors | Nuclear Proteins - antagonists & inhibitors | Signal Transduction - physiology | MicroRNAs - genetics | Feedback, Physiological - physiology | MicroRNAs - physiology | Homeodomain Proteins - physiology | Calcium-Binding Proteins - genetics | Zinc Finger E-box-Binding Homeobox 1 | Proteins | Signal transduction | Cellular biology | Molecular biology | Gene expression | Cancer
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2008, Volume 118, Issue 1, pp. 217 - 228
Loss of the tumor suppressor gene von Hippel-Lindau (VHL) plays a key role in the oncogenesis of clear cell renal cell carcinoma (CCRCC). The loss leads to... 
MEDICINE, RESEARCH & EXPERIMENTAL | HIF-1 | TUMOR SUPPRESSION | CYCLIN D1 | PATHWAY | GENE ALTERATIONS | VHL | KIDNEY | FATE | EXPRESSION | CANCER | Neoplasm Transplantation | RNA, Small Interfering - genetics | Humans | Carcinoma, Renal Cell - genetics | Male | Intracellular Signaling Peptides and Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Basic Helix-Loop-Helix Transcription Factors - metabolism | Cell Transformation, Neoplastic - genetics | Serrate-Jagged Proteins | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Membrane Proteins - metabolism | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Carcinoma, Renal Cell - drug therapy | Intracellular Signaling Peptides and Proteins - genetics | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Carcinoma, Renal Cell - pathology | Membrane Proteins - genetics | RNA, Small Interfering - pharmacology | Intercellular Signaling Peptides and Proteins - genetics | Signal Transduction - genetics | Receptor, Notch1 - metabolism | Cell Transformation, Neoplastic - metabolism | Cyclin-Dependent Kinase Inhibitor p27 | Carcinoma, Renal Cell - metabolism | Animals | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Mice | Cell Transformation, Neoplastic - drug effects | Receptor, Notch1 - antagonists & inhibitors | Cell Transformation, Neoplastic - pathology | Receptor, Notch1 - genetics | Calcium-Binding Proteins - genetics | Care and treatment | Enzyme inhibitors | Tumor suppressor genes | Dosage and administration | Genetic aspects | Carcinoma, Renal cell | Research | Health aspects | RNA; Small Interfering/genetics/pharmacology | Membrane Proteins/genetics/metabolism | Mice; Nude | Signal Transduction/drug effects/genetics | Intracellular Signaling Peptides and Proteins/genetics/metabolism | Basic Helix-Loop-Helix Transcription Factors/genetics/metabolism | Receptor; Notch1/antagonists & inhibitors/genetics/metabolism | Von Hippel-Lindau Tumor Suppressor Protein/genetics/metabolism | Cell Transformation; Neoplastic/drug effects/genetics/metabolism/pathology | Cyclin-Dependent Kinase Inhibitor p21/genetics/metabolism | Cell Line; Tumor | Intercellular Signaling Peptides and Proteins/genetics/metabolism | Calcium-Binding Proteins/genetics/metabolism | Carcinoma; Renal Cell/drug therapy/genetics/metabolism/pathology
Journal Article
Nature: international weekly journal of science, ISSN 0028-0836, 01/2014, Volume 506, Issue 7487, pp. 240 - 244
textabstractCells of the osteoblast lineage affect the homing and the number of long-term repopulating haematopoietic stem cells, haematopoietic stem cell... 
PROGENITOR CELLS | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | BONE-MARROW NICHE | NOTCH ACTIVATION | STEM-CELL NICHE | MYELOID-LEUKEMIA | MICE | DIFFERENTIATION | EXPRESSION | MYELODYSPLASTIC SYNDROMES | Osteoblasts - secretion | Receptors, Notch - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Hematopoietic Stem Cells - pathology | Anemia - pathology | Male | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Membrane Proteins - deficiency | Anemia - genetics | Cell Nucleus - metabolism | Tumor Microenvironment - genetics | Cell Transformation, Neoplastic - genetics | Serrate-Jagged Proteins | Base Sequence | Female | Membrane Proteins - metabolism | Intercellular Signaling Peptides and Proteins - deficiency | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Myelodysplastic Syndromes - metabolism | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Calcium-Binding Proteins - deficiency | Hematopoietic Stem Cells - metabolism | Mutation - genetics | beta Catenin - metabolism | beta Catenin - genetics | Anemia - metabolism | Cell Lineage | Osteoblasts - pathology | Animals | Chromosome Aberrations | Myeloid Cells - metabolism | Ligands | Mice | Myelodysplastic Syndromes - genetics | Myeloid Cells - pathology | Cell Transformation, Neoplastic - pathology | Myelodysplastic Syndromes - pathology | Osteoblasts - metabolism | Calcium-Binding Proteins - genetics | Leukemia, Myeloid, Acute - genetics
Journal Article
Cancer Research, ISSN 0008-5472, 03/2009, Volume 69, Issue 6, pp. 2400 - 2407
Despite rapid advances in many fronts, pancreatic cancer (PC) remains one of the most difficult human malignancies to treat due, in part, to de novo and... 
TARGET | STEM-CELLS | GROWTH INHIBITION | INVASION | TUMOR PROGRESSION | ONCOLOGY | PROSTATE-CANCER | TAMOXIFEN RESISTANCE | DOWN-REGULATION | E-CADHERIN | NF-KAPPA-B | RNA, Small Interfering - genetics | Pancreatic Neoplasms - metabolism | Receptors, Notch - metabolism | Humans | Deoxycytidine - pharmacology | Drug Resistance, Neoplasm | Intercellular Signaling Peptides and Proteins - biosynthesis | NF-kappa B - metabolism | Receptor, Notch2 - genetics | Receptors, Notch - genetics | Proto-Oncogene Proteins - biosynthesis | Cell Movement - physiology | Pancreatic Neoplasms - drug therapy | Intercellular Signaling Peptides and Proteins - metabolism | Transfection | Receptors, Notch - biosynthesis | Serrate-Jagged Proteins | Antimetabolites, Antineoplastic - pharmacology | Membrane Proteins - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Calcium-Binding Proteins - biosynthesis | Signal Transduction | Membrane Proteins - genetics | Down-Regulation | Pancreatic Neoplasms - pathology | Intercellular Signaling Peptides and Proteins - genetics | Receptor, Notch2 - metabolism | Epithelial Cells - pathology | Pancreatic Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Membrane Proteins - biosynthesis | Phenotype | Receptor, Notch2 - biosynthesis | Mesoderm - pathology | RNA, Messenger | Deoxycytidine - analogs & derivatives | Receptor, Notch4 | Calcium-Binding Proteins - genetics | Index Medicus
Journal Article
Stem Cells, ISSN 1549-4918, 2012, Volume 30, Issue 10, pp. 2320 - 2329
Adult neurogenesis is regulated by a number of cellular players within the neurogenic niche. Astrocytes participate actively in brain development, regulation... 
Vimentin | Astrocytes | Neurogenesis | Glial fibrillary acidic protein | Intermediate filaments | PROGENITOR CELLS | MOUSE-BRAIN | MICE DEFICIENT | ADULT HIPPOCAMPAL NEUROGENESIS | SUBVENTRICULAR ZONE | CELL & TISSUE ENGINEERING | CELL BIOLOGY | NEURAL STEM-CELLS | IN-VITRO | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | FIBRILLARY ACIDIC PROTEIN | DENTATE GYRUS | HEMATOLOGY | INTERMEDIATE-FILAMENTS | Amyloid Precursor Protein Secretases - genetics | Receptors, Notch - metabolism | Coculture Techniques | Nerve Tissue Proteins - deficiency | Male | Receptors, Notch - genetics | Stem Cells - cytology | Vimentin - deficiency | Stem Cells - metabolism | Wnt Proteins - metabolism | Glial Fibrillary Acidic Protein | Neurogenesis - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Endocytosis | Wnt Proteins - genetics | Gene Expression Regulation, Developmental | Serrate-Jagged Proteins | Vimentin - genetics | Cell Differentiation | Membrane Proteins - metabolism | Jagged-1 Protein | Astrocytes - cytology | Calcium-Binding Proteins - metabolism | Signal Transduction | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Cell Communication - genetics | Nerve Tissue Proteins - genetics | Mice, Knockout | Amyloid Precursor Protein Secretases - metabolism | Animals | Mice | Primary Cell Culture | Astrocytes - metabolism | Calcium-Binding Proteins - genetics | Neurosciences | Neurons | Analysis | Physiological aspects | Universities and colleges | Information management | Intermediate filament proteins | Ligands | Cells | Basic Medicine | Medicinska grundvetenskaper | Neurovetenskaper
Journal Article
Cancer Cell, ISSN 1535-6108, 02/2013, Volume 23, Issue 2, pp. 171 - 185
We report a paracrine effect whereby endothelial cells (ECs) promote the cancer stem cell (CSC) phenotype of human colorectal cancer (CRC) cells. We showed... 
COLON-CANCER | METASTASIS | ONCOLOGY | NOTCH | NICHE | SELF-RENEWAL | RECEPTOR | GROWTH-FACTOR | TUMOR ANGIOGENESIS | DIFFERENTIATION | ANGIOCRINE FACTORS | CELL BIOLOGY | ADAM17 Protein | RNA, Small Interfering - genetics | Immunoprecipitation | Receptors, Notch - metabolism | Colorectal Neoplasms - genetics | Humans | Calcium-Binding Proteins - antagonists & inhibitors | Culture Media, Conditioned - pharmacology | Drug Resistance, Neoplasm | Immunoblotting | Peptide Fragments - pharmacology | Intercellular Signaling Peptides and Proteins - metabolism | Neoplastic Stem Cells - metabolism | Serrate-Jagged Proteins | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Membrane Proteins - metabolism | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization | Tumor Cells, Cultured | Liver Neoplasms - secondary | Colorectal Neoplasms - metabolism | Jagged-1 Protein | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | ADAM Proteins - antagonists & inhibitors | Liver Neoplasms - genetics | Signal Transduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Cell Communication | Xenograft Model Antitumor Assays | ADAM Proteins - metabolism | Phenotype | Animals | Membrane Proteins - antagonists & inhibitors | Mice, Nude | Liver Neoplasms - metabolism | Mice | ADAM Proteins - genetics | Colorectal Neoplasms - pathology | Endothelial Cells - pathology | Calcium-Binding Proteins - genetics | Genetic aspects | Colorectal cancer | Stem cells | Endothelium
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2013, Volume 110, Issue 5, pp. 1875 - 1880
Here we examined the involvement of Notch signaling in the endochondral ossification process, which is crucial for osteoarthritis (OA) development.... 
Cartilage | Knee joint | Articular cartilage | Chondrocytes | Ligands | Bones | Mice | Skeletal development | Gene expression regulation | Osteoarthritis | Cartilage degradation | CELLS | OSTEOBLAST DIFFERENTIATION | MULTIDISCIPLINARY SCIENCES | skeletal development | BONE-DEVELOPMENT | KNEE-JOINT INSTABILITY | LIGANDS | ARTICULAR-CARTILAGE | PATHWAY | GROWTH | cartilage degradation | EXPERIMENTAL MOUSE MODELS | EXPRESSION | Cartilage - pathology | Homeodomain Proteins - metabolism | Humans | Cartilage - drug effects | Collagen Type II - metabolism | Knee Joint - pathology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Serrate-Jagged Proteins | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | Jagged-1 Protein | SOX9 Transcription Factor - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Membrane Proteins - genetics | Osteoarthritis - metabolism | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Cell Line, Tumor | HeLa Cells | Receptor, Notch1 - antagonists & inhibitors | Receptor, Notch1 - genetics | Calcium-Binding Proteins - genetics | Transcription Factor HES-1 | Receptor, Notch2 - antagonists & inhibitors | Receptor, Notch2 - genetics | Chondrocytes - drug effects | Intercellular Signaling Peptides and Proteins - metabolism | Membrane Proteins - metabolism | Chondrocytes - metabolism | Calcium-Binding Proteins - metabolism | Knee Joint - metabolism | Cell Line | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Osteoarthritis - prevention & control | Dipeptides - pharmacology | Knee Joint - drug effects | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Receptor, Notch2 - metabolism | Receptor, Notch1 - metabolism | Cartilage - metabolism | Collagen Type II - genetics | Homeodomain Proteins - genetics | Osteoarthritis - genetics | Animals | Fluorescent Antibody Technique | SOX9 Transcription Factor - genetics | Osteogenesis | Endochondral ossification | Physiological aspects | Development and progression | Cellular signal transduction | Health aspects | Membrane proteins | Biological Sciences
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 2007, Volume 117, Issue 12, pp. 3988 - 4002
High serum levels of IL-6 correlate with poor outcome in breast cancer patients. However, no data are available on the relationship between IL-6 and mammary... 
CANCER-CELLS | INITIATING CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | GENE SIGNATURE | CARBONIC-ANHYDRASE-IX | STEM-CELLS | STEM/PROGENITOR CELLS | SELF-RENEWAL | GROWTH-FACTOR | EXTRACELLULAR PH | IN-VITRO PROPAGATION | Carbonic Anhydrases - biosynthesis | Carcinoma, Ductal - genetics | Receptors, Notch - metabolism | Carbonic Anhydrases - genetics | Carcinoma, Ductal - pathology | Humans | Antigens, Neoplasm - biosynthesis | Spheroids, Cellular - pathology | Neoplasm Proteins - pharmacology | Receptors, Notch - genetics | Autocrine Communication - drug effects | Mammary Glands, Human - metabolism | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Breast Neoplasms - metabolism | Serrate-Jagged Proteins | Female | Carcinoma, Ductal - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Interleukin-6 - metabolism | Gene Expression Regulation, Neoplastic - genetics | Carbonic Anhydrase IX | Jagged-1 Protein | Antigens, Neoplasm - genetics | Cell Hypoxia - drug effects | Calcium-Binding Proteins | Interleukin-6 - genetics | Neoplasm Invasiveness | Intercellular Signaling Peptides and Proteins | RNA, Messenger - genetics | Spheroids, Cellular - metabolism | Mammary Glands, Human - pathology | Up-Regulation - genetics | Autocrine Communication - genetics | Membrane Proteins | Up-Regulation - drug effects | Breast Neoplasms - genetics | RNA, Neoplasm - biosynthesis | Interleukin-6 - pharmacology | Breast Neoplasms - pathology | Cell Line, Tumor | RNA, Neoplasm - genetics | Stem Cells - pathology | Receptor, Notch3 | Cell Hypoxia - genetics | Breast cancer | Genetic aspects | Research | Gene expression | Health aspects | Interleukin-6
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e60244
Endocardial to mesenchymal transformation (EMT) is a fundamental cellular process required for heart valve formation. Notch, Wnt and Bmp pathways are known to... 
EPITHELIAL-MESENCHYMAL TRANSITION | TRANSFORMATION | DEVELOPING CHICKEN HEART | MORPHOGENESIS | CUSHION | CELLS | NF-ATC | MULTIDISCIPLINARY SCIENCES | GROWTH-FACTOR | TRANSCRIPTION FACTOR | CARDIAC DEVELOPMENT | Wnt4 Protein - metabolism | Embryo, Mammalian | Heart Valves - metabolism | Epithelial-Mesenchymal Transition - genetics | Endocardium - metabolism | Wnt4 Protein - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Gene Expression Regulation, Developmental | Serrate-Jagged Proteins | Bone Morphogenetic Protein 2 - metabolism | Myocardium - metabolism | Membrane Proteins - metabolism | Heart Valves - embryology | Extracellular Matrix Proteins - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Bone Morphogenetic Protein 2 - genetics | Signal Transduction | Endocardium - embryology | Membrane Proteins - genetics | Extracellular Matrix Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Mice, Transgenic | Receptor, Notch1 - metabolism | Animals | Organogenesis - genetics | Mice | Receptor, Notch1 - genetics | Calcium-Binding Proteins - genetics | Cellular signal transduction | Research | Wnt proteins | Heart valves | Heart | Transformation | Wnt protein | Mesenchyme | Laboratories | Paracrine signalling | Defects | Morphogenesis | Signal transduction | Cell growth | Pathways | Rodents | Jagged1 protein | Cardiology | Immunoglobulins | Bone morphogenetic protein 2 | Cultures | Histology | Ribonucleic acid--RNA | Embryos | Medicine | Hospitals | Myocardium | Notch protein | RNA | Bone morphogenetic proteins | Ribonucleic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166984
Birth defects are among the leading causes of infant mortality and contribute substantially to illness and long-term disability. Defects in Bone Morphogenetic... 
BRANCHIAL ARCH | DLX GENES | HEAD SKELETON | ALAGILLE-SYNDROME | DOWNS-SYNDROME | MULTIDISCIPLINARY SCIENCES | AURICULOCONDYLAR SYNDROME | LEFT-RIGHT ASYMMETRY | REPEAT PROTEIN | HUMAN JAGGED1 | NEURAL CREST CELLS | Jagged-1 Protein - metabolism | Zebrafish - embryology | Intercellular Signaling Peptides and Proteins - metabolism | Somites - embryology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Nuclear Proteins - biosynthesis | Smad5 Protein - metabolism | Smad1 Protein - genetics | Smad5 Protein - genetics | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | Zebrafish Proteins - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - genetics | Endothelin-1 - genetics | Endothelin-1 - metabolism | Zebrafish Proteins - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Repressor Proteins - genetics | Jagged-1 Protein - genetics | Zebrafish - genetics | Body Patterning - physiology | Animals | Transforming Growth Factor beta - genetics | Facial Bones - enzymology | Smad1 Protein - metabolism | Signal Transduction - physiology | Zebrafish Proteins - genetics | Transforming Growth Factor beta - metabolism | Bone morphogenetic proteins | Genetic aspects | Research | Craniofacial abnormalities | Neural crest | Pattern formation | Jaw | Transcription factors | Endothelin | Arches | Congenital defects | Alagille syndrome | Genomics | Transforming growth factor-b | Genomes | Somites | Kinases | Defects | Proteins | Cartilage | Signal transduction | Pathways | Growth factors | Territory | Patterning | Mandible | Interference | Craniofacial syndromes | Zebrafish | Infant mortality | Birth defects | Endothelin 1 | Gene expression | Embryos | Mutants | Cleft lip/palate | Craniofacial growth | Signaling | Scaffolding | In vivo methods and tests | Notch protein
Journal Article