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PLoS ONE, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, pp. e42404 - e42404
Fungi are major causes of human, animal and plant disease. Human fungal infections can be fatal, but there are limited options for therapy, and resistance to... 
RECEPTOR POTENTIAL CHANNELS | CANDIDA-ALBICANS | K-ATP CHANNELS | 2 PORE DOMAINS | MULTIDISCIPLINARY SCIENCES | CA2+ CHANNEL | RECTIFYING POTASSIUM CHANNEL | ION CHANNELS | ANTIFUNGAL ANTIBIOTIC CLOTRIMAZOLE | PLASMA-MEMBRANE | NEUROSPORA-CRASSA | Amino Acid Sequence | Calcium Channels - metabolism | Transient Receptor Potential Channels - chemistry | Cations - metabolism | Humans | Ion Channels - genetics | Molecular Sequence Data | Mitochondria - metabolism | Phylogeny | Fungi - metabolism | Fungi - genetics | Potassium Channels - genetics | Potassium Channels - metabolism | Sequence Alignment | Ion Channels - metabolism | Transient Receptor Potential Channels - genetics | Calcium Channels - chemistry | Potassium Channels - chemistry | Transient Receptor Potential Channels - metabolism | Calcium Channels - genetics | Ion Channels - chemistry | Infection | Proteins | Fungi | Analysis | Genes | Genomics | Antifungal agents | Mycoses | Genomes | Health aspects | Drugs | Fungicides | Disease | Calcium | Homeostasis | Homology | Infections | Identification | Saccharomyces | Channels | Transient receptor potential proteins | Mitochondria | Cryptococcus | Physiology | Potassium channels (voltage-gated) | Tennis | Calcium channels | Pharmacology | Mammals | Disease control | Calcium (mitochondrial) | Plant diseases | Sodium | Cations | Potassium | Saccharomyces cerevisiae | Calcium ions | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 10, pp. 3546 - 3561
Inwardly rectifying potassium (Kir) channels establish and regulate the resting membrane potential of excitable cells in the heart, brain, and other peripheral... 
ISCHEMIA-REPERFUSION INJURY | RECTIFYING POTASSIUM CHANNELS | ACTIVATION | SULFHYDRATION | MECHANISM | K-ATP CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | G-PROTEINS | H2S | CARDIOPROTECTION | PHOSPHOINOSITIDES | Hydrogen Sulfide - metabolism | Cricetulus | Sulfides - chemistry | Cystathionine gamma-Lyase - metabolism | Potassium Channels, Inwardly Rectifying - chemistry | Recombinant Fusion Proteins - metabolism | Oocytes - cytology | Phosphatidylinositol 4,5-Diphosphate - metabolism | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Potassium Channels, Inwardly Rectifying - antagonists & inhibitors | Sulfides - metabolism | Cystathionine gamma-Lyase - genetics | Phosphatidylinositol 4,5-Diphosphate - chemistry | G Protein-Coupled Inwardly-Rectifying Potassium Channels - antagonists & inhibitors | CHO Cells | Sulfides - pharmacology | Allosteric Regulation - drug effects | Recombinant Proteins - metabolism | Hydrogen Sulfide - pharmacology | Mutagenesis, Site-Directed | Oocytes - metabolism | Hydrogen Sulfide - chemistry | Xenopus laevis | G Protein-Coupled Inwardly-Rectifying Potassium Channels - chemistry | Models, Molecular | Recombinant Proteins - chemistry | Potassium Channels, Inwardly Rectifying - genetics | Recombinant Fusion Proteins - chemistry | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Molecular Dynamics Simulation | Patch-Clamp Techniques | Animals | Protein Conformation | Mice | Mutation | Potassium Channels, Inwardly Rectifying - metabolism | Amino Acid Substitution | Index Medicus | GIRK channels | Signal Transduction | phosphatidylinositol 4,5-bisphosphate (PIP2) | stroke | phosphoinositide | ischemia | KATP channels | gasotransmitters | hydrogen sulfide | potassium channel | Kir3 or GIRK channels | inwardly rectifying K+ (Kir) channels | Kir3
Journal Article
CIRCULATION RESEARCH, ISSN 0009-7330, 08/2012, Volume 111, Issue 4, pp. 446 - 454
Rationale: Activation of the mitochondrial ATP-sensitive potassium channel (mitoK(ATP)) has been implicated in the mechanism of cardiac ischemic... 
SENSITIVE POTASSIUM CHANNELS | CARDIAC & CARDIOVASCULAR SYSTEMS | cytoprotection | mitochondria | K-ATP CHANNELS | apoptosis | IDENTIFICATION | ATP-sensitive potassium channel | preconditioning | ischemia | INFARCT SIZE | ACTION-POTENTIAL DURATION | HIGH-AFFINITY INHIBITOR | DISEASE | renal outer medullary potassium channel | PERIPHERAL VASCULAR DISEASE | MATRIX VOLUME | CARDIOPROTECTION | HEMATOLOGY | EXPRESSION | Diazoxide - pharmacology | Mitochondria, Heart - metabolism | Cricetulus | Mitochondria, Heart - pathology | Humans | Thallium - metabolism | Mitochondria, Heart - drug effects | RNA, Messenger - metabolism | Necrosis | Mitochondrial Membranes - drug effects | Potassium Channels - metabolism | Transfection | RNA Interference | Time Factors | Cattle | Adenosine Triphosphate - metabolism | Mass Spectrometry | Proteomics - methods | Potassium Channel Blockers - pharmacology | CHO Cells | Cytoprotection | Animals, Newborn | Cricetinae | Potassium Channels, Inwardly Rectifying - drug effects | Bee Venoms - pharmacology | Gene Expression Regulation | Rats | Potassium Channels, Inwardly Rectifying - genetics | Potassium Channels - drug effects | Mitochondrial Proton-Translocating ATPases - metabolism | Mitochondrial Membranes - metabolism | Potassium Channels - genetics | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Potassium Channels, Inwardly Rectifying - metabolism | Apoptosis | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 11/2011, Volume 109, Issue 11, pp. 1259 - 1268
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2017, Volume 12, Issue 9, pp. e0184261 - e0184261
Objectives Assess direct versus indirect action(s) of ghrelin on hypothalamic NPY neurons. Materials and methods Electrophysiology was used to measure ion... 
RAT ARCUATE NUCLEUS | UNION-OF-PHARMACOLOGY | NONSELECTIVE CATION CHANNEL | ACTIVATED PROTEIN-KINASE | RECEPTOR POTENTIAL CHANNELS | GROWTH-HORMONE SECRETAGOGUE | CALCIUM-CHANNELS | K-ATP CHANNELS | MULTIDISCIPLINARY SCIENCES | EXPRESSING NEURONS | NEUROPEPTIDE-Y NEURONS | Animals | Ghrelin - physiology | Calcium - metabolism | Fluorescent Antibody Technique | Neurons - physiology | Male | Signal Transduction - physiology | Mice | Hypothalamus, Middle - physiology | Membrane Potentials - physiology | Neuropeptide Y - physiology | Hypothalamus, Middle - cytology | Neuropeptide Y | Physiological aspects | Genetic aspects | Research | Ghrelin | Neurons | Molecular machines | Brain | Pathogenesis | Brain slice preparation | Nervous system | Neuropeptides | Glucose | Neuromodulation | Adenylate cyclase | γ-Aminobutyric acid | Physiology | Membrane potential | Inhibition | Glibenclamide | Obesity | AMP | Channel gating | Calcium channels (voltage-gated) | Cyclic AMP | Resistance | Conductance | Growth hormone | Endocrinology | Tetrodotoxin | Neuroimaging | Neurosciences | Calcium channels (N-type) | Electrophysiology | Antagonists | Activation | Neurosurgery | Hypothalamus | AMP-activated protein kinase | Machinery | Modulators | Depolarization | Calcium channels (R-type) | Rodents | Ion channels | Food | Calcium channels | Potassium channels (inwardly-rectifying) | Calcium imaging | Pharmacology | Calcium channels (T-type) | Phospholipase | Cations | Diabetes | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2012, Volume 287, Issue 12, pp. 8746 - 8756
Journal Article
by Zhang, Q and Bai, Y and Yang, Z and Tian, J and Meng, Z
Human & Experimental Toxicology, ISSN 0960-3271, 4/2016, Volume 35, Issue 4, pp. 418 - 427
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2017, Volume 313, Issue 4, pp. 732 - 743
The actions of hydrogen sulfide (H2S) on the heart and vasculature have been extensively reported. However, the mechanisms underlying the effects of H2S are... 
Bradycardia | ATP-sensitive potassium channels | L-type calcium channels | Hypotension | S donors | hypotension | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | H2S donors | K-ATP CHANNELS | NITRIC-OXIDE SYNTHASE | MYOCARDIAL-ISCHEMIA | H2S CONCENTRATIONS | CARDIOPULMONARY-RESUSCITATION | ISCHEMIA-REPERFUSION INJURY | SMOOTH-MUSCLE | CARDIAC-ARREST | HYPERPOLARIZING FACTOR | VERTEBRATE BLOOD | PERIPHERAL VASCULAR DISEASE | bradycardia | Rats, Inbred SHR | Hydrogen Sulfide - pharmacology | Rats | Electrocardiography - drug effects | Male | Cardiac Pacing, Artificial | Rats, Sprague-Dawley | Respiration, Artificial | Hypoglycemic Agents - pharmacology | Heart Rate - drug effects | Animals | KATP Channels - drug effects | Glyburide - pharmacology | Calcium Channels, L-Type - drug effects | Arterial Pressure - drug effects | Potassium Channel Blockers - pharmacology | Sulfides - pharmacology | Hydrogen sulfide | Dosage and administration | Blood pressure | Heart beat | Health aspects | Patient outcomes | Sulfide | Respiratory function | Intravenous administration | Experiments | Channels | Rodents | Heart diseases | Adenosine triphosphate | Hydrogen ion concentration | Ion channels (cyclic nucleotide-gated) | EKG | Cardiovascular system | Calcium channels | Calcium channels (voltage-gated) | Diltiazem | Methylene blue | Heart rate | Sodium | Calcium channels (L-type) | Ventilation | Respiration | Hyperpolarization | Sodium sulfide | Veins & arteries | Index Medicus
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 09/2018, Volume 472, pp. 97 - 106
The antimalarial agent, mefloquine, inhibits the function of connexin Cx36 gap junctions and hemichannels and has thus become a tool to investigate their... 
Mefloquine | KATP channels | Insulin secretion | Connexin hemichannels | L-type Ca2+ channels | L-type Ca | channels | GAP-JUNCTION CHANNELS | RELEASE | CURRENTS | CELL BIOLOGY | B-CELLS | DEPOLARIZATION | CALCIUM-CHANNELS | GLUCOSE | HEMICHANNELS | ENDOCRINOLOGY & METABOLISM | K-ATP channels | HYPOGLYCEMIA | PANCREATIC BETA-CELLS | Index Medicus
Journal Article