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Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 486, Issue 7404, pp. 532 - 536
Journal Article
Cancer research (Chicago, Ill.), ISSN 1538-7445, 2012, Volume 72, Issue 10, pp. 2457 - 2467
.... Although there is a high degree of similarity among the isoforms, K-Ras mutations are far more frequently observed in cancer, and each isoform displays preferential coupling to particular cancer types... 
ONCOGENES | LUNG-CANCER | H-RAS | K-RAS | ONCOLOGY | N-RAS | ACTIVATE RAF-1 | KRAS | PROTEINS | STEM-CELL EXPANSION | HUMAN P53 GENE | Catalytic Domain | Signal Transduction | Epigenesis, Genetic | Humans | Molecular Conformation | Databases, Genetic | Models, Molecular | Protein Isoforms - physiology | Point Mutation | Codon | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | ras Proteins - chemistry | Genes, ras | NRAS | mutation hotspot | HRAS | GTPase | mutagenesis
Journal Article
Nature genetics, ISSN 1546-1718, 2015, Volume 47, Issue 4, pp. 330 - 337
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e55793
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 7, p. e22769
Background: Oncogenic mutations of PIK3CA, RAS (KRAS, NRAS), and BRAF have been identified in various malignancies, and activate the PI3K/AKT/mTOR and RAS/RAF/MEK pathways, respectively... 
CELL LUNG-CANCER | KRAS MUTATIONS | NRAS | EFFICACY | GENE | PROGNOSIS | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | PREVALENCE | INHIBITOR | ACTIVATING MUTATIONS | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Prognosis | Humans | Middle Aged | Male | Proto-Oncogene Proteins - genetics | Mutation - genetics | Phosphatidylinositol 3-Kinases - genetics | Young Adult | Class I Phosphatidylinositol 3-Kinases | Neoplasms - genetics | DNA Mutational Analysis | Proto-Oncogene Proteins B-raf - genetics | Adolescent | Polymerase Chain Reaction | Aged, 80 and over | Adult | Female | Aged | DNA, Neoplasm - genetics | Neoplasms - pathology | Genes, ras - genetics | Care and treatment | Cancer patients | Gene mutations | Analysis | Melanoma | Colorectal cancer | Genetic aspects | Nucleotide sequencing | Health aspects | Cancer | DNA sequencing | TOR protein | Laboratories | Colorectal carcinoma | Clinical trials | Raf protein | AKT protein | Metastasis | Kinases | Tissues | K-Ras protein | Gene sequencing | Pathways | Tumorigenesis | Pancreas | Deoxyribonucleic acid--DNA | Histology | Cervix | Patients | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Studies | Thyroid cancer | Biopsy | Medical prognosis | Mutation | Uterine cancer | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Journal of clinical oncology, ISSN 1527-7755, 2013, Volume 31, Issue 17, pp. 2173 - 2181
Journal Article
Cancer research (Chicago, Ill.), ISSN 1538-7445, 2017, Volume 77, Issue 14, pp. 3922 - 3930
Journal Article