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1. Full Text Prevalence and architecture of de novo mutations in developmental disorders
by McRae, Jeremy F and Clayton, Stephen and Fitzgerald, Tomas W and Kaplanis, Joanna and Prigmore, Elena and Rajan, Diana and Sifrim, Alejandro and Aitken, Stuart and Akawi, Nadia and Alvi, Mohsan and Ambridge, Kirsty and Barrett, Daniel M and Bayzetinova, Tanya and Jones, Philip and Jones, Wendy D and King, Daniel and Krishnappa, Netravathi and Mason, Laura E and Singh, Tarjinder and Tivey, Adrian R and Ahmed, Munaza and Anjum, Uruj and Archer, Hayley and Armstrong, Ruth and Awada, Jana and Balasubramanian, Meena and Banka, Siddharth and Baralle, Diana and Barnicoat, Angela and Batstone, Paul and Baty, David and Bennett, Chris and Berg, Jonathan and Bernhard, Birgitta and Bevan, A. Paul and Bitner-Glindzicz, Maria and Blair, Edward and Blyth, Moira and Bohanna, David and Bourdon, Louise and Bourn, David and Bradley, Lisa and Brady, Angela and Brent, Simon and Brewer, Carole and Brunstrom, Kate and Bunyan, David J and Burn, John and Canham, Natalie and Castle, Bruce and Chandler, Kate and Chatzimichali, Elena and Cilliers, Deirdre and Clarke, Angus and Clasper, Susan and Clayton-Smith, Jill and Clowes, Virginia and Coates, Andrea and Cole, Trevor and Colgiu, Irina and Collins, Amanda and Collinson, Morag N and Connell, Fiona and Cooper, Nicola and Cox, Helen and Cresswell, Lara and Cross, Gareth and Crow, Yanick and D'Alessandro, Mariella and Dabir, Tabib and Davidson, Rosemarie and Davies, Sally and De Vries, Dylan and Dean, John and Deshpande, Charu and Devlin, Gemma and Dixit, Abhijit and Dobbie, Angus and Donaldson, Alan and Donnai, Dian and Donnelly, Deirdre and Donnelly, Carina and Douglas, Angela and Douzgou, Sofia and Duncan, Alexis and Eason, Jacqueline and Ellard, Sian and Ellis, Ian and Elmslie, Frances and Evans, Karenza and Everest, Sarah and Fendick, Tina and Fisher, Richard and Flinter, Frances and Foulds, Nicola and Fry, Andrew and Fryer, Alan and Gardiner, Carol and Gaunt, Lorraine and Ghali, Neeti and ... and Deciphering Developmental Disorders Study
Nature, ISSN 0028-0836, 02/2017, Volume 542, Issue 7642, pp. 433 - 438
The genomes of individuals with severe, undiagnosed developmental disorders are enriched in damaging de novo mutations (DNMs) in developmentally important... 
INTELLECTUAL DISABILITY | METAANALYSIS | VARIANTS | GENETICS | HEART-DEFECTS | MULTIDISCIPLINARY SCIENCES | GENES | SEQUENCE | FRAMEWORK | DISCOVERY | GENOME | Prevalence | Humans | Middle Aged | Parents | Male | Mi-2 Nucleosome Remodeling and Deacetylase Complex - genetics | Developmental Disabilities - genetics | Casein Kinase II - genetics | Autoantigens - genetics | Young Adult | ras GTPase-Activating Proteins - genetics | Adult | Female | Child | CDC2 Protein Kinase - genetics | Histone-Lysine N-Methyltransferase - genetics | Repressor Proteins - genetics | Sex Characteristics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mutation - genetics | Nerve Tissue Proteins - genetics | Sequence Analysis, DNA | Homeodomain Proteins - genetics | DEAD-box RNA Helicases - genetics | Exome - genetics | Phenotype | Myeloid-Lymphoid Leukemia Protein - genetics | Adolescent | Heredity - genetics | Protein Phosphatase 2C - genetics | Cohort Studies | Child development deviations | Genetic aspects | Genetic disorders | Developmental disabilities | Distribution | Genes | Families & family life | Births | Genomes | Mutation | Causality | Estimates | Age | TRIO | MYT1L | EHMT1 | HNRNPU | SUV420H1 | COL4A3BP | SYNGAP1 | PPP2R1A | POGZ | EP300 | KCNH1 | SCN1A | MEF2C | CDKL5 | CSNK2A1 | DYRK1A | CASK | ALG13 | FOXP1 | KAT6B | TBL1XR1 | KAT6A | SCN8A | KCNQ2 | EEF1A2 | KCNQ3 | ADNP | PhenIcons | SET | KMT2A | ANKRD11 | STXBP1 | FOXG1 | ZC4H2 | ITPR1 | De novo mutation | Seizures | ZBTB18 | CREBBP | SMAD4 | PDHA1 | IQSEC2 | AUTS2 | BCL11A | BRAF | SMARCA2 | GRIN2B | MED13L | GNAO1 | CNOT3 | TCF4 | SCN2A | CDK13 | GABRB3 | SETD5 | KDM5B | Developmental Disease | DDX3X | CHD8 | PTEN | CHD4 | TCF20 | CTCF | CHD2 | WDR45 | SLC6A1 | MECP2 | CHAMP1 | KIF1A | Average Faces | MSL3 | PPP2R5D | SMC1A | ARID1B | DNM1 | CNKSR2 | PACS1 | WAC | ZMYND11 | AHDC1 | NFIX | SATB2 | HDAC8 | PPM1D | GNAI1 | PURA | PUF60 | NSD1 | Intellectual Disability | SLC35A2 | DYNC1H1 | NAA10 | USP9X | PTPN11 | GATAD2B | ASXL1 | KANSL1 | ASXL3 | CTNNB1 | QRICH1
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2. Full Text MiR-296-3p regulates cell growth and multi-drug resistance of human glioblastoma by targeting ether-à-go-go (EAG1)
by Bai, Yifeng and Liao, Hongzhan and Liu, Tianzhu and Zeng, Xiangping and Xiao, Faman and Luo, Luqiao and Guo, Hongbo and Guo, Linlang
European Journal of Cancer, ISSN 0959-8049, 2012, Volume 49, Issue 3, pp. 710 - 724
Abstract MicroRNAs (miRNAs) – short non-coding RNA molecules – post-transcriptionally regulate gene expressions and play crucial roles in diverse biological... 
Hematology, Oncology and Palliative Medicine | Ether-à-go-go (EAG1, KCNH1) | MicroRNAs | Multi-drug resistance (MDR) | Glioblastoma | CANCER-CELLS | POTASSIUM CHANNELS | MESENCHYMAL TRANSITION | FACTOR RECEPTOR | TUMORS | LUNG-CANCER | Ether-a-go-go (EAG1, KCNH1) | ONCOLOGY | HUMAN GLIOMAS | EXPRESSION | K+ CHANNELS | MICRORNA | Cell Proliferation | Drug Resistance, Multiple | Neoplasm Invasiveness | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | ERG1 Potassium Channel | Ether-A-Go-Go Potassium Channels - genetics | Glioblastoma - pathology | Cell Line, Tumor | Ether-A-Go-Go Potassium Channels - physiology | MicroRNAs - physiology | Glioblastoma - drug therapy | Immunohistochemistry | MicroRNA | Drug resistance | Gene expression | Glioblastoma multiforme
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3. Full Text PIST (GOPC) modulates the oncogenic voltage-gated potassium channel KV10.1
by Herrmann, Solveig and Ninkovic, Milena and Kohl, Tobias and Pardo, Luis A
Frontiers in Physiology, ISSN 1664-042X, 2013, Volume 4, p. 201
Although crucial for their correct function, the mechanisms controlling surface expression of ion channels are poorly understood. In the case of the... 
FIG | 10.1 | Trafficking | GOPC | Potassium channels | KCNH1 | PIST | Potassium Channels | KV10.1 | trafficking
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4. Approaches targeting KV10.1 open a novel window for cancer diagnosis and therapy
by Pardo, L.A and Gómez-Varela, D and Major, F and Sansuk, K and Leurs, R and Downie, B.R and Tietze, L.F and Stühmer, W
Current Medicinal Chemistry, ISSN 0929-8673, 02/2012, Volume 19, Issue 5, pp. 675 - 682
KV10.1 | Eag1 | Cancer diagnosis | Ether à go-go | Tumor therapy | Astemizole | Imipramine | ADEPT | Cancer prognosis | Immunotherapy | Potassium channel | Immunotargeted therapy | KCNH1
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5. Full Text Neurodevelopmental disease genes implicated by de novo mutation and copy number variation morbidity
by Coe, Bradley P and Stessman, Holly A. F and Sulovari, Arvis and Geisheker, Madeleine R and Bakken, Trygve E and Lake, Allison M and Dougherty, Joseph D and Lein, Ed S and Hormozdiari, Fereydoun and Bernier, Raphael A and Eichler, Evan E
Nature Genetics, ISSN 1061-4036, 01/2019, Volume 51, Issue 1, pp. 106 - 116
We combined de novo mutation (DNM) data from 10,927 individuals with developmental delay and autism to identify 253 candidate neurodevelopmental disease genes... 
INTELLECTUAL DISABILITY | AUTISM | MOUSE MODEL | GENETICS & HEREDITY | MISSENSE MUTATION | SEGMENTAL DUPLICATIONS | DEVELOPMENTAL DELAY | SPECTRUM | TRUNCATING MUTATIONS | CLINICAL-SIGNIFICANCE | HAPLOINSUFFICIENCY | Autistic Disorder - genetics | Humans | Mi-2 Nucleosome Remodeling and Deacetylase Complex - genetics | DNA Copy Number Variations - genetics | Developmental Disabilities - genetics | Neurodevelopmental Disorders - genetics | Mutation - genetics | Intellectual Disability - genetics | Exome - genetics | Phenotype | Animals | Chromosome Aberrations | Polymorphism, Single Nucleotide - genetics | Mice | Intracellular Signaling Peptides and Proteins - genetics | Gene mutations | Copy number variations | Analysis | Nervous system | Degeneration | Genetic aspects | Research | Morbidity | Neurosciences | Intellectual disabilities | Copy number | Genes | Disorders | Likelihood ratio | Genomes | Gene deletion | Neurodevelopmental disorders | Spiny neurons | Autism | Missense mutation | Clonal deletion | Reproduction (copying) | Neostriatum | Deletion | Gene duplication | Mutation | Chromosome 16 | TRIO | MYT1L | HNRNPU | EHMT1 | ENO3 | CAPN15 | SUV420H1 | PPP1CB | EFTUD2 | COL4A3BP | SYNGAP1 | PPP2R1A | POGZ | EP300 | KCNH1 | SOX5 | MAP2K1 | MEF2C | CSNK2A1 | TMEM178A | DYRK1A | CASK | FOXP1 | KAT6B | TBL1XR1 | KAT6A | SCN8A | HECW2 | KCNQ2 | EEF1A2 | KCNQ3 | ADNP | Genetics of Developmental Delay | DLX3 | SET | KMT2A | SRCAP | ANP32A | STXBP1 | CUL3 | FOXG1 | ANKRD11 | ITPR1 | de novo Mutation | HIVEP3 | SNX5 | ZBTB18 | CREBBP | IQSEC2 | BCL11A | NONO | UPF3B | BRAF | ADAP1 | GRIN2B | SMARCA2 | SMARCA4 | MED13L | GNAO1 | AGO4 | DLG4 | CAPRIN1 | Neurodevelopmental Disorders | TCF4 | CDK13 | SCN2A | GABRB2 | KDM5B | SETD5 | DDX3X | CHD8 | PTEN | TCF20 | CTCF | CHD3 | CHD2 | WDR45 | SLC6A1 | MECP2 | SNAPC5 | SYNCRIP | TLK2 | CHAMP1 | KIF1A | RAC1 | TNPO2 | DNMT3A | MSL3 | PPP2R5D | ASH1L | SMC1A | ARID1B | CNKSR2 | PACS2 | SETBP1 | PIK3CA | PACS1 | WAC | ZMYND11 | SHANK3 | AHDC1 | SMARCD1 | WDR26 | NFIX | SATB2 | KIAA2022 | HDAC8 | PPM1D | GNAI1 | PURA | PUF60 | NSD1 | LEO1 | DYNC1H1 | MBD5 | NAA10 | USP9X | PTPN11 | MEIS2 | GATAD2B | KANSL1 | ASXL3 | CTNNB1 | TRIP12 | QRICH1 | NAA15 | TAF1
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6. Full Text Alternatively spliced isoforms of KV10.1 potassium channels modulate channel properties and can activate cyclin-dependent kinase in Xenopus oocytes
by Gomes, Fernanda Ramos and Romaniello, Vincenzo and Sánchez, Araceli and Weber, Claudia and Narayanan, Pratibha and Psol, Maryna and Pardo, Luis A
Journal of Biological Chemistry, ISSN 0021-9258, 12/2015, Volume 290, Issue 51, pp. 30351 - 30365
KV10.1 is a voltage-gated potassium channel expressed selectively in the mammalian brain but also aberrantly in cancer cells. In this study we identified short... 
Cyclin-Dependent Kinases - metabolism | Oocytes - metabolism | Humans | Xenopus laevis | Ether-A-Go-Go Potassium Channels - metabolism | Molecular Sequence Data | Alternative Splicing - physiology | Ether-A-Go-Go Potassium Channels - genetics | Oocytes - cytology | Animals | Protein Isoforms - metabolism | Base Sequence | HEK293 Cells | Cell Cycle - physiology | Enzyme Activation | HeLa Cells | Cyclin-Dependent Kinases - genetics | Protein Isoforms - genetics | KV10.1 | cell cycle | post-transcriptional regulation | glycosylation | potassium channel | Cell Biology | RNA splicing | KCNH1
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7. De novo KCNH1 mutations in four patients with syndromic developmental delay, hypotonia and seizures
by Fukai, Ryoko and Saitsu, Hirotomo and Tsurusaki, Yoshinori and Sakai, Yasunari and Haginoya, Kazuhiro and Takahashi, Kazumasa and Hubshman, Monika Weisz and Okamoto, Nobuhiko and Nakashima, Mitsuko and Tanaka, Fumiaki and Miyake, Noriko and Matsumoto, Naomichi
Journal of Human Genetics, ISSN 1434-5161, 05/2016, Volume 61, Issue 5, pp. 381 - 387
The voltage-gated Kv10.1 potassium channel, also known as ether-a-go-go-related gene 1, encoded by KCNH1 (potassium voltage-gated channel, subfamily H (eag... 
GLYCINE | PHARMACOLOGY | GENETICS & HEREDITY | GATED POTASSIUM CHANNELS | ION CHANNELS | EPILEPTIC ENCEPHALOPATHY | KCNH1 | Ether-A-Go-Go Potassium Channels - chemistry | Muscle Hypotonia - genetics | Seizures - genetics | Humans | Male | Seizures - diagnosis | Developmental Disabilities - genetics | Ether-A-Go-Go Potassium Channels - genetics | Syndrome | Muscle Hypotonia - diagnosis | Exome | Phenotype | Protein Interaction Domains and Motifs - genetics | Alleles | Facies | Heterozygote | High-Throughput Nucleotide Sequencing | Mutation | Child | Developmental Disabilities - diagnosis | Amino Acid Substitution
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8. Full Text Dynamics of the EAG1 K+ channel selectivity filter assessed by molecular dynamics simulations
by Bernsteiner, Harald and Bründl, Michael and Stary-Weinzinger, Anna
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 02/2017, Volume 484, Issue 1, pp. 107 - 112
EAG1 channels belong to the KCNH family of voltage gated potassium channels. They are expressed in several brain regions and increased expression is linked to... 
Ion binding sites | KCNH1 channel | Na+ binding sites | Molecular dynamics simulation | Y464A mutant | C-type inactivation | binding sites | Na | ACTIVATION | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | PARAMETERS | INSIGHT | HYDRATION | KIRBAC1.1 | BIOPHYSICS | HERG POTASSIUM CHANNEL | ION PERMEATION | MUTATIONS | Cryoelectron Microscopy | Ether-A-Go-Go Potassium Channels - metabolism | Ether-A-Go-Go Potassium Channels - chemistry | Humans | Molecular Dynamics Simulation | Index Medicus | MOLECULAR DYNAMICS METHOD | ELECTRIC POTENTIAL | FILTERS | CONDENSED MATTER PHYSICS, SUPERCONDUCTIVITY AND SUPERFLUIDITY | SIMULATION | KAONS PLUS | POTASSIUM IONS | SODIUM IONS | CONFORMATIONAL CHANGES
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9. Approaches Targeting K(V)10.1 Open a Novel Window for Cancer Diagnosis and Therapy
by Pardo, LA and Gomez-Varela, D and Major, F and Sansuk, K and Leurs, R and Downie, BR and Tietze, LF and Stuhmer, W
CURRENT MEDICINAL CHEMISTRY, ISSN 0929-8673, 02/2012, Volume 19, Issue 5, pp. 675 - 682
K(V)10.1 has recently become generally accepted as a promising cancer target, as it is ectopically expressed in the majority of solid tumors. Due to its... 
HUMAN-PAPILLOMAVIRUS TYPE-16 | DIFFERENTIAL LOCALIZATION | tumor therapy | FUNCTIONAL EXPRESSION | immunotherapy | TETRAMERIZATION DOMAIN | PHARMACOLOGY & PHARMACY | astemizole | KCNH1 | CHEMISTRY, MEDICINAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | imipramine | cancer diagnosis | eag1 | immunotargeted therapy | cancer prognosis | A-GO-GO | ether a go-go | ADEPT | GATED K+ CHANNELS | EAG1 POTASSIUM CHANNELS | K(V)10.1 | HUMAN ETHER | potassium channel | ION CHANNELS | ABERRANT EXPRESSION | Neoplasms - therapy | Neoplasms - diagnosis | Ether-A-Go-Go Potassium Channels - therapeutic use | Molecular Targeted Therapy | Humans
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10. Full Text Temple-Baraitser Syndrome and Zimmermann-Laband Syndrome: One clinical entity?
by Mégarbané, André and Al-Ali, Rashid and Choucair, Nancy and Lek, Monko and Wang, Ena and Ladjimi, Moncef and Rose, Catherine M and Hobeika, Remy and Macary, Yvette and Temanni, Ramzi and Jithesh, Puthen V and Chouchane, Aouatef and Sastry, Konduru S and Thomas, Remy and Tomei, Sara and Liu, Wei and Marincola, Francesco M and MacArthur, Daniel and Chouchane, Lotfi
BMC Medical Genetics, ISSN 1471-2350, 06/2016, Volume 17, Issue 1, pp. 42 - 42
Background: KCNH1 encodes a voltage-gated potassium channel that is predominantly expressed in the central nervous system. Mutations in this gene were recently... 
Zimmermann-Laband syndrome | Temple-Baraitser syndrome | Whole genome sequencing | KCNH1 | CELLS | POLLEX | SEVERE MENTAL-RETARDATION | DNA | GENETICS & HEREDITY | ABSENT NAILS | HALLUX | MUTATIONS | Hallux - abnormalities | Humans | Fibromatosis, Gingival - genetics | Infant | Male | Mutation, Missense | Hand Deformities, Congenital - diagnosis | Intellectual Disability - genetics | Toes - diagnostic imaging | DNA Mutational Analysis | Abnormalities, Multiple - genetics | Craniofacial Abnormalities - genetics | Thumb - diagnostic imaging | Fibromatosis, Gingival - diagnosis | Protein-Serine-Threonine Kinases - genetics | DNA - metabolism | Ether-A-Go-Go Potassium Channels - genetics | Nails, Malformed - genetics | DNA - isolation & purification | DNA - chemistry | Craniofacial Abnormalities - diagnosis | Hand Deformities, Congenital - genetics | Abnormalities, Multiple - diagnosis | Intellectual Disability - diagnosis | Thumb - abnormalities | Nails, Malformed - diagnosis | Genetic disorders | Gene mutations | Physiological aspects | Development and progression | Genetic aspects | Potassium channels | Research | Risk factors | Index Medicus
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11. Full Text PIST (GOPC) modulates the oncogenic voltage-gated potassium channel K(v)10.1
by Herrmann, S and Ninkovic, M and Kohl, T and Pardo, LA
FRONTIERS IN PHYSIOLOGY, ISSN 1664-042X, 2013, Volume 4
Although crucial for their correct function, the mechanisms controlling surface expression of ion channels are poorly understood. In the case of the... 
CANCER PATIENTS | PHYSIOLOGY | SOMATOSTATIN RECEPTOR SUBTYPE-5 | SMALL GTPASE RAB5 | FIG | SURFACE EXPRESSION | CELL-LINES | LURCHER MICE | K(v)10.1 | potassium channels | PLASMA-MEMBRANE EXPRESSION | GOPC | COIL DOMAIN | GOLGI PROTEIN | trafficking | ABERRANT EXPRESSION | KCNH1 | PIST
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12. Full Text Epilepsy in KCNH1-related syndromes
by Mastrangelo, Mario and Scheffer, Ingrid E and Bramswig, Nuria C and Nair, Lal. D. V and Myers, Candace T and Dentici, Maria Lisa and Korenke, Georg C and Schoch, Kelly and Campeau, Philippe M and White, Susan M and Shashi, Vandana and Kansagra, Sujay and Van Essen, Anthonie J and Leuzzi, Vincenzo
Epileptic disorders, ISSN 1294-9361, 06/2016, Volume 18, Issue 2, pp. 123 - 136
Aim. KCNH1 mutations have been identified in patients with Zimmermann-Laband syndrome and Temple-Baraitser syndrome, as well as patients with uncharacterized... 
Temple‐Baraitser syndrome | related encephalopathy | genetic epilepsy | undefined intellectual disability | Zimmermann‐Laband syndrome | KCNH1 | KCNH1-Related encephalopathy | Zimmermann-laband syndrome | Temple-baraitser syndrome | Genetic epilepsy | Undefined intellectual disability | KCNH1-related encephalopathy | Zimmermann-Laband syndrome | MICE | Temple-Baraitser syndrome | MUTATIONS | CLINICAL NEUROLOGY | Hallux - abnormalities | Humans | Thumb - physiopathology | Child, Preschool | Fibromatosis, Gingival - genetics | Infant | Male | Abnormalities, Multiple - drug therapy | Electroencephalography | Craniofacial Abnormalities - physiopathology | Epilepsy - physiopathology | Fibromatosis, Gingival - drug therapy | Nails, Malformed - physiopathology | Intellectual Disability - genetics | Young Adult | Adult | Epilepsy - genetics | Female | Child | Abnormalities, Multiple - genetics | Craniofacial Abnormalities - genetics | Brain - physiopathology | Anticonvulsants - therapeutic use | Craniofacial Abnormalities - drug therapy | Nails, Malformed - drug therapy | Ether-A-Go-Go Potassium Channels - genetics | Abnormalities, Multiple - physiopathology | Syndrome | Nails, Malformed - genetics | Hand Deformities, Congenital - drug therapy | Intellectual Disability - drug therapy | Intellectual Disability - physiopathology | Fibromatosis, Gingival - physiopathology | Hand Deformities, Congenital - genetics | Hand Deformities, Congenital - physiopathology | Adolescent | Epilepsy - drug therapy | Hallux - physiopathology | Thumb - abnormalities | Convulsions & seizures | Mutation | Intellectual disabilities | Epilepsy
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13. Full Text Differences Between Ion Binding to eag and HERG voltage sensors Contribute to Differential Regulation of Activation and Deactivation Gating
by Lin, Meng-chin and Papazian, Diane
Channels, ISSN 1933-6950, 11/2007, Volume 1, Issue 6, pp. 429 - 437
HERG (KCNH2) and ether-à-go-go (eag) (KCNH1) are members of the same subfamily of voltage-gated K+ channels. In eag, voltage-dependent activation is... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | KCNH2 | IKr | Cardiac ion channel | Deactivation | KCNE2 | Voltage-dependent | Activation | Magnesium | KCNH1 | SHAKER K+ CHANNEL | BIOCHEMISTRY & MOLECULAR BIOLOGY | magnesium | I-Kr | ARRHYTHMIA | voltage-dependent | CARDIAC POTASSIUM CHANNEL | STRUCTURAL BASIS | cardiac ion channel | MG2 | LONG QT SYNDROME | XENOPUS OOCYTES | activation | DEPENDENT ACTIVATION | MOLECULAR-BASIS | deactivation | Xenopus | Amino Acid Sequence | Ether-A-Go-Go Potassium Channels - chemistry | Ions - chemistry | Potassium Channels, Voltage-Gated - chemistry | Oocytes - metabolism | Ether-A-Go-Go Potassium Channels - metabolism | Molecular Sequence Data | ERG1 Potassium Channel | Sequence Homology, Amino Acid | Magnesium - chemistry | Animals | Protein Binding | Cations | Kinetics | Binding Sites
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