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by Zhou, Q and Wang, X and Yu, Z and Wu, X and Chen, X and Li, J and Zhu, Z and Liu, B and Su, L
Oncogene, ISSN 0950-9232, 03/2017, Volume 36, Issue 13, pp. 1873 - 1886
Journal Article
by Wan, M and Liu, J and Ouyang, X
Journal of Periodontal Research, ISSN 0022-3484, 04/2015, Volume 50, Issue 2, pp. 189 - 196
Journal Article
Journal of Medicinal Chemistry, ISSN 0022-2623, 06/2016, Volume 59, Issue 12, pp. 5650 - 5660
Journal Article
Journal of Nuclear Medicine, ISSN 0161-5505, 02/2012, Volume 53, Issue 2, pp. 275 - 280
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 23, pp. 9554 - 9559
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2014, Volume 34, Issue 3, pp. 552 - 564
OBJECTIVE—Interleukin 1 Receptor 1 (IL1R1) and its ligand, IL1β, are upregulated in cardiovascular disease, obesity, and infection. Previously, we reported a... 
Infection | Diet, high-fat | Megakaryocytes | Blood platelets | IL1R1 protein, human | THROMBOPOIESIS | CELLS | blood platelets | infection | megakaryocytes | HIGH-FAT DIET | CANAKINUMAB | ATHEROSCLEROSIS | IL1R1 protein | ARTERIES | CYTOKINE PRODUCTION | INHIBITION | high-fat | PERIPHERAL VASCULAR DISEASE | IL-1-BETA | diet | HEMATOLOGY | human | EXPRESSION | Inflammation - pathology | Transcription, Genetic - drug effects | Interleukin-1beta - pharmacology | Receptors, Interleukin-1 Type I - physiology | Humans | Platelet Activation - physiology | Platelet Adhesiveness - physiology | NF-kappa B - metabolism | Gene Expression Profiling | Obesity - blood | Obesity - genetics | Atherosclerosis - etiology | Bacteroidaceae Infections - blood | Thrombin - pharmacology | Protein Processing, Post-Translational - drug effects | Collagen - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Megakaryocytes - cytology | Proto-Oncogene Proteins c-akt - metabolism | Bacteroidaceae Infections - pathology | Disease Models, Animal | Receptors, Interleukin-1 Type I - deficiency | Porphyromonas gingivalis | Cell Line | Interleukin-1beta - physiology | Obesity - complications | Imidazoles - pharmacology | Inflammation - etiology | Mice, Knockout | Platelet Adhesiveness - drug effects | Transcription, Genetic - physiology | Animals | MAP Kinase Signaling System - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Receptors, Interleukin-1 Type I - genetics | Inflammation - genetics | Mice | Platelet Activation - drug effects | Pyridines - pharmacology | Dietary Fats - toxicity
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 3050 - 3059
DJ‐1/Park7 is a redox‐sensitive chaperone protein counteracting oxidation and presumably contributing to the control of oxidative stress responses and thus... 
HEART | IMMUNITY | PHYSIOLOGY | PROTEIN | DENDRITIC CELLS | GENE | ROS | DJ-1 | EXPRESSION | PARKINSONS-DISEASE | PLASTICITY | CELL BIOLOGY | Up-Regulation | Cation Transport Proteins - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Protein-Tyrosine Kinases - metabolism | Protein Deglycase DJ-1 - genetics | RNA, Messenger - metabolism | Sodium-Hydrogen Exchangers - metabolism | Time Factors | Cation Transport Proteins - metabolism | Cation Transport Proteins - genetics | Sodium-Hydrogen Exchangers - genetics | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Protein Deglycase DJ-1 - deficiency | Protein Deglycase DJ-1 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Genotype | Antioxidants - pharmacology | Mice, Knockout | Phenotype | Animals | Protein Kinase Inhibitors - pharmacology | Sodium-Hydrogen Exchanger 1 | CD4-Positive T-Lymphocytes - drug effects | Hydrogen-Ion Concentration | Protein-Tyrosine Kinases - antagonists & inhibitors | T cells | Cysteine | Oxidative stress | Ammonium | Parkinson's disease | Staurosporine | Acetylcysteine | Fluorescence | Lymphocytes T | Gene deletion | Kinases | Western blotting | Proteins | Clonal deletion | Lymphocytes | Rodents | Deletion | Oxidation | Protein-tyrosine kinase | Movement disorders | Tyrosine | Neurodegenerative diseases | Na+/H+-exchanging ATPase | Inflammation | CD4 antigen | Polymerase chain reaction | PARK7 protein | Mice
Journal Article
Journal Article