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Molecular Cancer, ISSN 1476-4598, 12/2014, Volume 13, Issue 1, p. 259
Background: Lung cancer is the major cause of cancer-related deaths and many cases of Non Small Cell Lung Cancer (NSCLC), a common type of lung cancer, have... 
Tumor xenograft | NSCLC | PI3K | STAT3 | Targeted therapeutics | EGFR-TKI | mTORC2 | PK-PD | mTORC1 | GEFITINIB | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ANTITUMOR-ACTIVITY | EGFR | CHEMOTHERAPY | KRAS MUTATIONS | STAT3 ACTIVATION | K-RAS | ONCOLOGY | RESISTANCE | TUMOR-GROWTH | Erlotinib Hydrochloride | Lung Neoplasms - drug therapy | Proto-Oncogene Proteins p21(ras) | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Activin Receptors, Type II - antagonists & inhibitors | Male | Mechanistic Target of Rapamycin Complex 2 | Quinolines - pharmacology | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | TOR Serine-Threonine Kinases - antagonists & inhibitors | ras Proteins - pharmacology | Phosphatidylinositol 3-Kinases - pharmacology | Antineoplastic Agents - pharmacology | Receptor, Epidermal Growth Factor - pharmacology | Phosphorylation - drug effects | Proto-Oncogene Proteins - pharmacology | Carcinoma, Non-Small-Cell Lung - metabolism | Imidazoles - pharmacology | Animals | Class I Phosphatidylinositol 3-Kinases | Mice, Nude | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | HeLa Cells | Quinazolines - pharmacology | Drug Resistance, Neoplasm - drug effects | Medical equipment and supplies industry | Growth | Health aspects | Medical test kit industry | Mutation | Kinases | Lung cancer | Rodents
Journal Article
Microcirculation, ISSN 1073-9688, 02/2014, Volume 21, Issue 2, pp. 93 - 103
Journal Article
Cardiovascular Research, ISSN 0008-6363, 05/2007, Volume 74, Issue 2, pp. 279 - 289
Objective: Activin-like kinase receptor 1 (ALK1) is a transforming growth factor (TGF)-β type I receptor expressed in vascular mesenchyme, yet its function in... 
Matrix GLA protein | Smooth muscle cell differentiation | Bone morphogenetic protein | Vascular mesenchymal cells | Activin-like kinase receptor 1 | activin-like kinase receptor 1 | smooth muscle cell differentiation | CARDIAC & CARDIOVASCULAR SYSTEMS | TGF-BETA | BONE MORPHOGENETIC PROTEIN-2 | KINASE | RECEPTOR | bone morphogenetic protein | vascular mesenchymal cells | matrix GLA protein | ENDOTHELIAL-CELLS | GROWTH | CALCIFICATION | SMOOTH-MUSCLE-CELLS | EXPRESSION | Immunohistochemistry | Activin Receptors, Type II - metabolism | Cell Proliferation | Humans | Coronary Vessels | Activin Receptors, Type II - analysis | Bone Morphogenetic Proteins - metabolism | Cattle | Coronary Artery Disease - pathology | Cell Differentiation | Bone Morphogenetic Proteins - genetics | Myocytes, Smooth Muscle - metabolism | Transduction, Genetic - methods | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Cell Line | Bone Morphogenetic Protein 2 | Coronary Artery Disease - metabolism | Extracellular Matrix Proteins - genetics | Cells, Cultured | Mesoderm - chemistry | Activin Receptors, Type I - metabolism | Myocytes, Smooth Muscle - chemistry | Activin Receptors, Type I - genetics | Animals | Transforming Growth Factor beta - genetics | Activin Receptors, Type II - genetics | Mesoderm - metabolism | Transforming Growth Factor beta - metabolism | Calcium-Binding Proteins - genetics
Journal Article
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 05/2018, Volume 63, Issue 4, pp. 1433 - 1443
The pathophysiology of Alzheimer’s disease (AD) includes signaling defects mediated by the transforming growth factor β—bone morphogenetic protein—growth and... 
Immunohistochemistry | Animal models | Neurons | Therapeutic applications | Memory | Neuropil | Kinases | Proteins | Fornix | Axons | Bone growth | Neurofibrillary tangles | Pyramidal cells | Rodents | Amyloidosis | Activin | Bone morphogenetic protein 9 | Alzheimer's disease | Hippocampus | ACVRL1 | CA1 | CA3 | hippocampus | immunohistochemistry | GAD67 | ALK1
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 11/2016, Volume 7, Issue 1, p. 13247
Functional blood vessel growth depends on generation of distinct but coordinated responses from endothelial cells. Bone morphogenetic proteins (BMP), part of... 
PATHWAYS | PLATFORM | ZEBRAFISH | ANGIOGENESIS | RNA | BONE MORPHOGENETIC PROTEIN | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | GENE-EXPRESSION | KINASE 1 ALK1 | GROWTH-FACTOR
Journal Article
Cancer, ISSN 0008-543X, 12/2016, Volume 122, Issue 23, pp. 3641 - 3649
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 07/2019, Volume 116, Issue 31, pp. 15570 - 15579
The type I TGF beta receptor TGF beta RI (encoded by Tgfbr1) was ablated in cartilage. The resulting Tgfbr1(Col2) mice exhibited lethal chondrodysplasia.... 
ACTIVATION | PROTEIN | BMP | CHONDROCYTES | MULTIDISCIPLINARY SCIENCES | cartilage | COMPONENTS | ALK5 | ALK1 | GROWTH-FACTOR-BETA | ARTICULAR-CARTILAGE | KINASE 1 ALK1 | DIFFERENTIATION | TGF beta | EXPRESSION | CHONDROGENESIS | Cartilage | Signaling | Growth plate | Intracellular signalling | Mice | Bone morphogenetic protein 9 | Ablation | Chondrodystrophy | Biological Sciences | PNAS Plus | TGFβ
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 09/2019, Volume 116, Issue 36, pp. 17800 - 17808
Journal Article
Journal Article