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Clinical cancer research, ISSN 1557-3265, 2014, Volume 20, Issue 22, pp. 5745 - 5755
Purpose: KIT is the major oncogenic driver of gastrointestinal stromal tumors (GIST). Imatinib, sunitinib, and regorafenib are approved therapies; however,... 
EFFICACY | ONCOLOGY | SAFETY | SUNITINIB | C-KIT | MYELOID-LEUKEMIA | MECHANISMS | MUTATIONS | DASATINIB | IMATINIB RESISTANCE | NILOTINIB | Exons | Humans | Molecular Conformation | Imidazoles - chemistry | Tomography, X-Ray Computed | Antineoplastic Agents - therapeutic use | Pyridazines - pharmacology | Proto-Oncogene Proteins c-kit - antagonists & inhibitors | Dose-Response Relationship, Drug | Protein Kinase Inhibitors - chemistry | Gastrointestinal Stromal Tumors - diagnosis | Gastrointestinal Stromal Tumors - pathology | Inhibitory Concentration 50 | Female | Indoles - pharmacology | Proto-Oncogene Proteins c-kit - genetics | Tumor Burden - genetics | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Imidazoles - therapeutic use | Gastrointestinal Stromal Tumors - genetics | Pyridazines - therapeutic use | Disease Models, Animal | Neoplasm Recurrence, Local | Models, Molecular | Imidazoles - pharmacology | Pyrimidines - pharmacology | Antineoplastic Agents - chemistry | Imatinib Mesylate | Piperazines - pharmacology | Pyridazines - chemistry | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Pyrroles - pharmacology | Animals | Tumor Burden - drug effects | Gastrointestinal Stromal Tumors - drug therapy | Protein Kinase Inhibitors - therapeutic use | Cell Line, Tumor | Protein Binding | Protein Kinase Inhibitors - pharmacology | Mutation | Proto-Oncogene Proteins c-kit - chemistry | Index Medicus | GIST | ponatinib | tyrosine kinase inhibitor | resistance | KIT
Journal Article
Human reproduction update, ISSN 1460-2369, 2015, Volume 21, Issue 6, pp. 779 - 786
BACKGROUND: The first small follicles to appear in the mammalian ovaries are primordial follicles. The initial pool of primordial follicles serves as the... 
KIT ligand | Primordial follicle activation | Mechanistic target of rapamycin complex 1 | KIT | Primordial follicle granulosa cells | PI3 kinase signaling | OOCYTE GROWTH | primordial follicle activation | FACTOR RECEPTOR/C-KIT | C-KIT | TRANSCRIPTION FACTOR FOXL2 | ANTI-MULLERIAN HORMONE | RAT OVARIES | primordial follicle granulosa cells | OBSTETRICS & GYNECOLOGY | REPRODUCTIVE BIOLOGY | mechanistic target of rapamycin complex 1 | FEMALE RATS | CLINICAL-IMPLICATIONS | PHYSIOLOGICAL FUNCTIONS | MOUSE OVARY | TOR Serine-Threonine Kinases - metabolism | Humans | Multiprotein Complexes - genetics | Phosphatidylinositol 3-Kinases - metabolism | Stem Cell Factor - genetics | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | TOR Serine-Threonine Kinases - genetics | Mammals - physiology | Ovarian Follicle - physiology | Female | TOR Serine-Threonine Kinases - physiology | Ovarian Follicle - cytology | Ovarian Follicle - metabolism | Stem Cell Factor - physiology | Ovary - metabolism | Stem Cell Factor - metabolism | Signal Transduction | Multiprotein Complexes - physiology | Phosphatidylinositol 3-Kinases - genetics | Animals | Oocytes - physiology | Models, Biological | Phosphatidylinositol 3-Kinases - physiology | Granulosa Cells - cytology | female rats | c-kit | mechanistic target of rapamycin | rat ovaries | v110 | Obstetrics & Gynecology | mouse ovary | p17474 | anti-mullerian hormone | Biokemi och molekylärbiologi | receptor/c-kit | Biochemistry and Molecular Biology | physiological functions | clinical-implications | transcription factor foxl2 | factor | oocyte growth | Reproductive Biology | ates of america
Journal Article
Cancer, ISSN 0008-543X, 04/2017, Volume 123, Issue 8, pp. 1333 - 1344
BACKGROUND Optimal treatments for vulvar and vaginal melanomas (VVMs) have not been identified. Herein, the authors compare molecular profiles between VVM and... 
genitourinary melanoma | gene profiling | vulvar melanoma | KIT proto‐oncogene receptor tyrosine kinase (KIT) mutation | mucosal melanoma | B‐Raf proto‐oncogene serine/threonine kinase (BRAF) mutation | molecular targets | vaginal melanoma | neuroblastoma rat sarcoma (NRAS) mutation | KIT proto-oncogene receptor tyrosine kinase (KIT) mutation | B-Raf proto-oncogene serine/threonine kinase (BRAF) mutation | threonine kinase (BRAF) mutation | B-Raf proto-oncogene serine | MALIGNANT-MELANOMA | DACARBAZINE | TUMORS | ONCOLOGY | KIT GENE-MUTATIONS | BRAF MUTATIONS | UNCOMMON | EXPRESSION | IPILIMUMAB | Immunohistochemistry | Melanoma - diagnosis | Humans | Middle Aged | Male | Gene Expression Profiling | Biomarkers, Tumor | Vaginal Neoplasms - genetics | Young Adult | Melanoma - genetics | Aged, 80 and over | Adult | Female | Proto-Oncogene Proteins c-kit - genetics | Vulvar Neoplasms - diagnosis | In Situ Hybridization, Fluorescence | Mucous Membrane - metabolism | Mucous Membrane - pathology | Vulvar Neoplasms - genetics | Proto-Oncogene Proteins B-raf - genetics | Aged | High-Throughput Nucleotide Sequencing | Mutation | Neoplasm Staging | Vaginal Neoplasms - diagnosis | Usage | Care and treatment | Gene mutations | Vulvar cancer | Genetic aspects | Research | Biological markers | Drugs | PD-1 protein | Identification methods | Estrogens | Mucosa | Clinical trials | Raf protein | Alkylation | Skin cancer | Platinum | Immunotherapy | Medical research | Threonine | Melanoma | Vagina | Gene expression | Sensitivity | Reviews | Anthracycline | Valine | PD-L1 protein | Biomarkers | ERCC1 protein | Protein-serine/threonine kinase | Progesterone | Tumors | Cancer
Journal Article
Clinical cancer research, ISSN 1557-3265, 2008, Volume 14, Issue 21, pp. 6821 - 6828
Journal Article
Cancer Research, ISSN 0008-5472, 04/2009, Volume 69, Issue 8, pp. 3563 - 3569
Journal Article
by Shen, Hui and Shih, Juliann and Hollern, Daniel P and Wang, Linghua and Bowlby, Reanne and Tickoo, Satish K and Thorsson, Vésteinn and Mungall, Andrew J and Newton, Yulia and Hegde, Apurva M and Armenia, Joshua and Sánchez-Vega, Francisco and Pluta, John and Pyle, Louise C and Mehra, Rohit and Reuter, Victor E and Godoy, Guilherme and Jones, Jeffrey and Shelley, Carl S and Feldman, Darren R and Vidal, Daniel O and Lessel, Davor and Kulis, Tomislav and Cárcano, Flavio M and Leraas, Kristen M and Lichtenberg, Tara M and Brooks, Denise and Cherniack, Andrew D and Cho, Juok and Heiman, David I and Kasaian, Katayoon and Liu, Minwei and Noble, Michael S and Xi, Liu and Zhang, Hailei and Zhou, Wanding and ZenKlusen, Jean C and Hutter, Carolyn M and Felau, Ina and Zhang, Jiashan and Schultz, Nikolaus and Getz, Gad and Meyerson, Matthew and Stuart, Joshua M and Akbani, Rehan and Wheeler, David A and Laird, Peter W and Nathanson, Katherine L and Cortessis, Victoria K and Hoadley, Katherine A and Wheeler, David and Hughes, Daniel and Covington, Kyle and Jayaseelan, Joy C and Korchina, Viktoriya and Lewis, Lora and Hu, Jianhong and Doddapaneni, HarshaVardhan and Muzny, Donna and Gibbs, Richard and Hollern, Daniel and Vincent, Benjamin G and Chai, Shengjie and Smith, Christof C and Auman, J. Todd and Shi, Yan and Meng, Shaowu and Skelly, Tara and Tan, Donghui and Veluvolu, Umadevi and Mieczkowski, Piotr A and Jones, Corbin D and Wilkerson, Matthew D and Balu, Saianand and Bodenheimer, Tom and Hoyle, Alan P and Jefferys, Stuart R and Mose, Lisle E and Simons, Janae V and Soloway, Matthew G and Roach, Jeffrey and Parker, Joel S and Hayes, D. Neil and Perou, Charles M and Saksena, Gordon and Cibulskis, Carrie and Schumacher, Steven E and Beroukhim, Rameen and Gabriel, Stacey B and Ally, Adrian and Balasundaram, Miruna and Carlsen, Rebecca and Cheung, Dorothy and Chuah, Eric and Dhalla, Noreen and Holt, Robert A and Jones, Steven J.M and Ma, Yussanne and Mayo, Michael and Moore, Richard A and ... and The Cancer Genome Atlas Research Network and Canc Genome Atlas Res Network and Cancer Genome Atlas Research Network
Cell reports (Cambridge), ISSN 2211-1247, 2018, Volume 23, Issue 11, pp. 3392 - 3406
Journal Article
Journal Article
Drug design, development and therapy, ISSN 1177-8881, 2016, Volume 10, pp. 2443 - 2459
c-Kit, a receptor tyrosine kinase, is involved in intracellular signaling, and the mutated form of c-Kit plays a crucial role in occurrence of some cancers.... 
c-Kit | Cancer therapy | Oncogene | Cancer | GENE-MUTATIONS | GASTROINTESTINAL STROMAL TUMOR | CHEMISTRY, MEDICINAL | POINT MUTATION | CATALYTIC DOMAIN | ACUTE MYELOID-LEUKEMIA | THYMIC CARCINOMA | oncogene | INTERSTITIAL-CELLS | SIGNAL-TRANSDUCTION | cancer therapy | PHARMACOLOGY & PHARMACY | cancer | GROWTH-FACTOR | SRC FAMILY KINASES | Humans | Leukemia, Myeloid, Acute - metabolism | Antineoplastic Agents - chemistry | Proto-Oncogene Proteins c-kit | Receptor Protein-Tyrosine Kinases - metabolism | Signal Transduction - drug effects | Leukemia, Myeloid, Acute - drug therapy | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Mutation | Receptor Protein-Tyrosine Kinases - chemistry | Leukemia, Myeloid, Acute - genetics | Molecular targeted therapy | Innovations | Cellular signal transduction | Genetic aspects | Phosphotransferases | Health aspects | Oncogenes | Pictures | Therapy | Transformation | Leukemia | Lung cancer | Intracellular signalling | Metastasis | Kinases | Cancer therapies | Defects | Cell adhesion & migration | Signal transduction | Cell growth | Protein-tyrosine kinase receptors | Inhibition | Protein-tyrosine kinase | Tyrosine | Therapeutic applications | Myeloid leukemia | Melanoma | c-Kit protein | Inhibitors | Infertility | Stem cells | KIT protein | Ligands | Acute myeloid leukemia | Protein structure | Tumors | Apoptosis
Journal Article