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1. Full Text KLF4α stimulates breast cancer cell proliferation by acting as a KLF4 antagonist
by Ferralli, Jacqueline and Chiquet-Ehrismann, Ruth and Degen, Martin
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 29, pp. 45608 - 45621
Kruppel-like factor 4 (KLF4), a transcription factor involved in both tumor suppression and oncogenesis in various human tumors, is subject to alternative... 
Alternative splicing | KLF4α/KLF4(FL) ratio | Proliferation | KLF4 | Tumors | METASTASIS | alternative splicing | proliferation | TUMOR-SUPPRESSOR GENE | DOWN-REGULATION | CELL BIOLOGY | KRUPPEL-LIKE FACTORS | OVEREXPRESSION | tumors | MESSENGER-RNA | GROWTH | KLF4 alpha/KLF4(FL) ratio | TRANSCRIPTION FACTOR | EXPRESSION | PROGRESSION | Protein Isoforms - metabolism | Alternative Splicing | Breast Neoplasms - pathology | Kruppel-Like Transcription Factors - metabolism | Humans | Cell Proliferation - physiology | Cell Line, Tumor | Female | Cell Transformation, Neoplastic - metabolism | Breast Neoplasms - metabolism | Index Medicus
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2. Full Text Frequent AKT1 E17K mutations in skull base meningiomas are associated with mTOR and ERK1/2 activation and reduced time to tumor recurrence
by Yesilöz, Ümmügülsüm and Kirches, Elmar and Hartmann, Christian and Scholz, Johannes and Kropf, Siegfried and Sahm, Felix and Nakamura, Makoto and Mawrin, Christian
Neuro-Oncology, ISSN 1522-8517, 08/2017, Volume 19, Issue 8, pp. 1088 - 1096
meningioma | AKT1 | skull base | KLF4
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3. Full Text Adriamycin inhibits embryonic development in zebrafish through downregulation of Kruppel‐like factor4
by Akthar, I. Syeda Thabassum and Pichiah, P.B. Tirupathi and Arunachalam, Sankarganesh and Raja, Sudhakaran
Journal of Biochemical and Molecular Toxicology, ISSN 1095-6670, 01/2019, Volume 33, Issue 1, pp. e22235 - n/a
Adriamycin is an effective anticancer drug used in a wide range of cancers. Anticancer drugs modulate oncogenes and nodal regulatory molecules that affect cell... 
embryogenesis | Kruppel‐like factor4 | Klf4 | hatching | adriamycin | Kruppel-like factor4 (Klf4) | FACTOR 4 KLF4 | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOXORUBICIN | TOXICITY | MODEL | FAMILY | CELL MOVEMENTS | GENE | MESENDODERM | DNA | TOXICOLOGY | EXPRESSION | Cathepsin L | Hatching | Differentiation (biology) | Zebrafish | Cell differentiation | Gene expression | Embryos | Anticancer properties | Embryonic growth stage | Embryogenesis | Antitumor agents | KLF4 protein | Mesendoderm | Pluripotency | Cancer | Kruppel protein
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4. Full Text Correction: Klf4 Is a Transcriptional Regulator of Genes Critical for EMT, Including Jnk1 (Mapk8)
by Tiwari, Neha and Meyer-Schaller, Nathalie and Arnold, Phil and Antoniadis, Helena and Pachkov, Mikhail and van Nimwegen, Erik and Christofori, Gerhard
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10
KLF4 protein | Transcription
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5. cDNA cloning and sequence analysis of pluripotency genes in tree shrews ( Tupaia belangeri )
by WANG, Cai-Yun and MA, Yun-Han and HE, Da-Jian and YANG, Shi-Hua
04/2013
In this paper, partial sequences of the tree shrew ( Tupaia belangeri ) Klf4, Sox2, and c-Myc genes were cloned and sequenced, which were 382, 612, and 485 bp... 
Sox2 | Tree shrew | c-Myc | Klf4 | Sequence analysis
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6. Full Text LINC01210 accelerates proliferation, invasion and migration in ovarian cancer through epigenetically downregulating KLF4
by Zhang, Chu and Zhang, Rongrong and Zhang, Yang and Liu, Jie and Luo, Chengyan and Zhu, Tong and Yao, Ruiqin
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 11/2019, Volume 119
In recent years, a large number of studies have shown that differentially expressed lncRNAs in tumors are capable of inducing tumorigenesis and promoting tumor... 
LINC01210 | Migration | Proliferation | KLF4 | Invasion | Ovarian cancer
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7. Full Text CD34+KLF4+ Stromal Stem Cells Contribute to Endometrial Regeneration and Repair
by Yin, Mingzhu and Zhou, Huanjiao Jenny and Lin, Caixia and Long, Lingli and Yang, Xiaolei and Zhang, Haifeng and Taylor, Hugh and Min, Wang
Cell Reports, ISSN 2211-1247, 05/2019, Volume 27, Issue 9, pp. 2709 - 2724.e3
The regenerative capacity of the human endometrium requires a population of local stem cells. However, the phenotypes, locations, and origin of these cells are... 
endometrium | SUMOylation | stem cell | KLF4 | CD34 | SENP1
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8. Full Text Context-dependent functions of KLF4 in cancers: Could alternative splicing isoforms be the key?
by Wang, Liang and Shen, Feng and Stroehlein, John R and Wei, Daoyan
Cancer Letters, ISSN 0304-3835, 12/2018, Volume 438, pp. 10 - 16
Krüppel-like factor 4 (KLF4) is an important transcription factor that is expressed in a variety of tissues and regulates many critical physiologic and... 
Alternative splicing | KLF4 | Context-dependent function | Cancer | STEM-CELLS | PANCREATIC-CANCER | TUMOR-SUPPRESSOR GENE | FACTOR 4 EXPRESSION | BREAST-CANCER | HEPATOCELLULAR-CARCINOMA | MESSENGER-RNA | ONCOLOGY | SIGNALING PATHWAY | KRUPPEL-LIKE FACTOR-4 | TRANSCRIPTION FACTOR | Prevention | Cell differentiation | Stem cells | Cell proliferation | Genes | Colorectal cancer | Homeostasis | Metastasis | Neuroblastoma | Stomach cancer | KLF4 gene | Skin cancer | Proteins | Cell growth | KLF4 protein | Cell cycle | Tumorigenesis | Deoxyribonucleic acid--DNA | Medical research | Splicing | Breast cancer | Esophagus | Molecular modelling | Medical prognosis | Isoforms | Apoptosis | Tumors
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9. Full Text UDP-α-D-glucose 6-dehydrogenase: a promising target for glioblastoma
by Goodwin, C Rory and Ahmed, A Karim and Xia, Shuli
Oncotarget, ISSN 1949-2553, 02/2019, Volume 10, Issue 16, pp. 1542 - 1543
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10. Full Text The Intra-nuclear SphK2-S1P Axis Facilitates M1-to-M2 Shift of Microglia via Suppressing HDAC1-Mediated KLF4 Deacetylation
by Ji, Juan and Wang, Juan and Yang, Jin and Wang, Xi-Peng and Huang, Jing-Jing and Xue, Teng-Fei and Sun, Xiu-Lan
Frontiers in immunology, ISSN 1664-3224, 2019, Volume 10, pp. 1241 - 1241
Sphingosine 1-phosphate (S1P) is involved in a variety of cellular responses including microglial activation and polarization. However, the impacts of S1P on... 
microglial polarization | S1P | Sphk2 | KLF4 | ischemia
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11. Full Text Cellular miR-2909 RNomics governs the genes that ensure immune checkpoint regulation
by Kaul, Deepak and Malik, Deepti and Wani, Sameena
Molecular and Cellular Biochemistry, ISSN 0300-8177, 1/2019, Volume 451, Issue 1, pp. 37 - 42
Cross-talk between coding RNAs and regulatory non-coding microRNAs, within human genome, has provided compelling evidence for the existence of flexible... 
Life Sciences | Biochemistry, general | MiR-2909 | Checkpoint regulation | Medical Biochemistry | Oncology | Cardiology | KLF4 | Ectopic expression | Human PBMCs | AUTOIMMUNITY | CANCER | MICRORNA | CELL BIOLOGY | Regulators | CD40 antigen | CD86 antigen | Genes | Crosstalk | MiRNA | Homeostasis | Leukocytes (mononuclear) | CD80 antigen | T cell receptors | Genomes | Lymphocytes T | CD134 antigen | Gene expression | CD28 antigen | KLF4 gene | Interleukin 6 | CTLA-4 protein | Cell activation | Immune checkpoint | KLF4 protein | Interleukin 10 | Peripheral blood mononuclear cells | Index Medicus
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12. Full Text Triptolide inhibits viability and migration while promotes apoptosis in nephroblastoma cells by regulation of miR-193b-3p
by Hang, Shiying and Wang, Xianghong and Li, Hai
Experimental and Molecular Pathology, ISSN 0014-4800, 06/2019, Volume 108, pp. 80 - 88
Triptolide (TPL) is a potential anti-tumor natural compound. However, its role in nephroblastoma is poorly studied. Herein, we aimed to reveal the regulatory... 
Nephroblastoma | KLF4 | miR-193b-3p | PI3K/AKT/ERK | Triptolide
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13. Full Text Elafin treatment rescues EGFR-Klf4 signaling and lung cell survival in ventilated newborn mice
by Alejandre Alcazar, Miguel A and Kaschwich, Mark and Ertsey, Robert and Preuss, Stefanie and Milla, Carlos and Mujahid, Sana and Masumi, Juliet and Khan, Suleman and Mokres, Lucia M and Tian, Lu and Mohr, Jasmine and Hirani, Dharmesh V and Rabinovitch, Marlene and Bland, Richard D
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 11/2018, Volume 59, Issue 5, pp. 623 - 634
Mechanical ventilation with O-2-rich gas (MV-O-2) inhibits alveologenesis and lung growth. We previously showed that MV-O-2 increased elastase activity and... 
Bronchopulmonary dysplasia | Klf4 proteins | Elastase inhibition | Neonatal chronic lung disease | Lung growth arrest | EGFR | lung growth arrest | ALVEOLAR SEPTATION | PULMONARY-HYPERTENSION | PROTEINASE-INHIBITOR IMBALANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | elastase inhibition | MECHANICAL VENTILATION | PRETERM LAMBS | bronchopulmonary dysplasia | FACTOR RECEPTOR | CELL BIOLOGY | EGFR and Klf4 proteins | RESPIRATORY SYSTEM | EPIDERMAL-GROWTH-FACTOR | neonatal chronic lung disease | GENE-EXPRESSION | NEONATAL MICE | Plasma | Pediatrics | Phosphorylation | Transcription factors | Lung | Infants | Proteins | Signal transduction | Epidermal growth factor | KLF4 protein | Newborn babies | Fibroblasts | Elastase | Vascular endothelial growth factor | Kruppel protein | Ventilators | Dysplasia | Cell survival | Risk groups | Epidermal growth factor receptors | Lung diseases | siRNA | Epithelium | Lungs | Ventilation | Lung Kruppel-like factor | Apoptosis
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14. Full Text p22phox promotes Ang-II-induced vascular smooth muscle cell phenotypic switch by regulating KLF4 expression
by Tang, Yixin and Huang, Dan and Huang, Qin and Liu, Changhui and Liu, Chaoyan and Ou, Hongji and Peng, Fengling and Mo, Zhongcheng
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 06/2019, Volume 514, Issue 1, pp. 280 - 286
NADPH oxidase (Nox) is the main source of reactive oxygen species in vascular diseases, which have been implicated in promoting VSMCs phenotypic switch.... 
p22phox | VSMC phenotypic switch | Reactive oxygen species | Ang-II | KLF4
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15. Full Text Smooth muscle-specific Gsα deletion exaggerates angiotensin II-induced abdominal aortic aneurysm formation in mice in vivo
by Qin, Xiaoteng and He, Lifan and Tian, Mi and Hu, Ping and Yang, Jianmin and Lu, Huixia and Chen, Wenqiang and Chen, Min and Jiang, Xiuxin and Zhang, Wencheng and Zhang, Cheng and Zhang, Yun and Gao, Jiangang and Weinstein, Lee S
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 07/2019, Volume 132, pp. 49 - 59
Abdominal aortic aneurysm (AAA) is a life-threatening vascular disease without an effective pharmaceutical treatment. Genetic studies have proved the... 
Phenotype switch | Gsα | HuR | KLF4 | Abdominal aortic aneurysm
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16. Full Text Kruppel-like factor 4 (KLF4) is required for maintenance of breast cancer stem cells and for cell migration and invasion
by Yu, F and Li, J and Chen, H and Fu, J and Ray, S and Huang, S and Zheng, H and Ai, W
Oncogene, ISSN 0950-9232, 05/2011, Volume 30, Issue 18, pp. 2161 - 2172
Kruppel-like factor 4 (KLF4) is highly expressed in more than 70% of breast cancers and functions as an oncogene. However, an exact mechanism by which KLF4... 
migration | breast cancer | cancer stem cell | invasion | KLF4 | TRANSFORMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | SELF-RENEWAL | IDENTIFICATION | TUMORIGENICITY | P53 | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | TUMOR-SUPPRESSOR | DIFFERENTIATION | TRANSCRIPTION FACTOR | Neoplasm Metastasis | Neoplastic Stem Cells - cytology | Signal Transduction | Breast Neoplasms - pathology | Neoplasm Invasiveness | Receptors, Notch - metabolism | Humans | Cell Line, Tumor | Kruppel-Like Transcription Factors - physiology | Breast Neoplasms - physiopathology | Kruppel-Like Transcription Factors - genetics | Gene Knockdown Techniques | Transcription factors | Cancer cells | Physiological aspects | Development and progression | Breast cancer | Research | Risk factors | Cell migration | Proteins | Signal transduction | Gene expression | Stem cells | Cell adhesion & migration | Severe combined immunodeficiency | Colonies | Diabetes mellitus | Tumorigenicity | Tumor cell lines | Aldehyde dehydrogenase | KLF4 protein | Notch protein | Tumorigenesis | Mammary gland | Surface markers | Oncogenes | Tumors | Index Medicus
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17. Full Text KLF4 regulates corneal epithelial cell cycle progression by suppressing canonical TGF-b signaling and upregulating cdk inhibitors P16 and P27
by Tiwari, Anil and Swamynathan, Sudha and Alexander, Nicholas and Gnalian, John and Tian, Shenghe and Kinchington, Paul R and Swamynathan, Shivalingappa K and Swamynathan, Shivalingappa K
Investigative ophthalmology & visual science, ISSN 0146-0404, 02/2019, Volume 60, Issue 2, pp. 731 - 740
TGF-β | Corneal epithelium | SMAD | KLF4 | EMT | Squamous neoplasia
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18. Full Text EGR3 Is a Late Epidermal Differentiation Regulator that Establishes the Skin-Specific Gene Network
by Kim, Kyu-Han and Kim, Hyoung-June and Son, Eui Dong and Lee, Sung Hoon and Lee, Tae Ryong and Bae, Il-Hong
Journal of Investigative Dermatology, ISSN 0022-202X, 03/2019, Volume 139, Issue 3, pp. 615 - 625
Late epidermal differentiation is a key step of skin barrier formation; however, the specific genetic factors that distinguish late differentiation from early... 
ELEMENTS | KERATINOCYTE DIFFERENTIATION | COMPLEX | KLF4 | P63 | EXPRESSION | ZNF750 | EZH2 | DERMATOLOGY
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19. Full Text Correction: Biologically anchored knowledge expansion approach uncovers KLF4 as a novel insulin signaling regulator
by Muthiah, Annamalai and Angulo, Morgan S and Walker, Natalie N and Keller, Susanna R and Lee, Jae K
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 11, pp. e0207325 - e0207325
[This corrects the article DOI: 10.1371/journal.pone.0204100.]. 
Insulin | Proteins | Adenosine | Dehydrogenases | KLF4 protein | Genes | Adipocytes | Phosphatase | Kinases
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20. The Mechanism of a BMP-Driven Mesenchymal-to-Epithelial Transition in the Reprogramming of Induced Pluripotent Stem Cells
by Liu, Da
03/2014
Somatic cells can be reprogrammed to induced pluripotent stem cells (iPSCs) by the ectopic expression of defined factors. iPSCs hold great promise for... 
Smad1,5,8 | BMP | RNA-Seq | iPSC | Mesenchymal-to-Epithelial Transition | ChIP-Seq | 0307 | Klf4 | MET
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