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by White, Gemma and White, Jacqueline K and Gerdin, Anna-Karin and Karp, Natasha A and Ryder, Ed and Buljan, Marija and Bussell, James N and Salisbury, Jennifer and Clare, Simon and Ingham, Neil J and Podrini, Christine and Houghton, Richard and Estabel, Jeanne and Bottomley, Joanna R and Melvin, David G and Sunter, David and Adams, David J and Adams, Niels C and Baker, Lauren and Barnes, Caroline and Beveridge, Ryan and Cambridge, Emma and Carragher, Damian and Chana, Prabhjoat and Clarke, Kay and Hooks, Yvette and Igosheva, Natalia and Ismail, Ozama and Jackson, Hannah and Kane, Leanne and Lacey, Rosalind and Lafont, David Tino and Lucas, Mark and Maguire, Simon and McGill, Katherine and McIntyre, Rebecca E and Messager, Sophie and Mottram, Lynda and Mulderrig, Lee and Pearson, Laila and Pearson, Selina and Protheroe, Hayley J and Roberson, Laura-Anne and Salsbury, Grace and Sanderson, Mark and Sanger, Daniel and Shannon, Carl and Thompson, Paul C and Tuck, Elizabeth and Vancollie, Valerie E and Brackenbury, Lisa and Bushell, Wendy and Cook, Ross and Dalvi, Priya and Gleeson, Diane and Habib, Bishoy and Hardy, Matt and Liakath-Ali, Kifayathullah and Miklejewska, Evelina and Price, Stacey and Sethi, Debarati and Trenchard, Elizabeth and von Schiller, Dominique and Vyas, Sapna and West, Anthony P and Woodward, John and Wynn, Elizabeth and Evans, Arthur and Gannon, David and Griffiths, Mark and Holroyd, Simon and Iyer, Vivek and Kipp, Christian and Lewis, Morag and Li, Wei and Oakley, Darren and Richardson, David and Smedley, Damian and Agu, Chukwuma and Bryant, Jackie and Delaney, Liz and Gueorguieva, Nadia I and Tharagonnet, Helen and Townsend, Anne J and Biggs, Daniel and Brown, Terry and Brown, Ellen and Collinson, Adam and Dumeau, Charles-Etienne and Grau, Evelyn and Harrison, James and Harrison, Sarah and Ingle, Catherine E and Kundi, Helen and Madich, Alla and Mayhew, Danielle and Metcalf, Tom and Newman, Stuart and Pass, Johanna and Reynolds, Helen and ... and Sanger Inst Mouse Genetics Project and Sanger Institute Mouse Genetics Project
Cell, ISSN 0092-8674, 07/2013, Volume 154, Issue 2, pp. 452 - 464
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e62509 - e62509
miR-132 and miR-212 are two closely related miRNAs encoded in the same intron of a small non-coding gene, which have been suggested to play roles in both... 
HIPPOCAMPUS | ACTIVATION | CREB | COCAINE INTAKE | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | MSK1 | GENERATION | EXPRESSION | MICRORNA | Electric Stimulation | Interferon-beta - immunology | Synaptic Transmission - genetics | Male | MicroRNAs - metabolism | Neurons - cytology | Synapses - genetics | Neocortex - metabolism | Neuronal Plasticity - genetics | Synapses - metabolism | Female | Neurons - metabolism | Receptors, AMPA - genetics | Excitatory Postsynaptic Potentials - genetics | Receptors, AMPA - metabolism | Fibroblasts - virology | Sendai virus - physiology | Interferon-beta - biosynthesis | Long-Term Potentiation - genetics | Hippocampus - cytology | Mice, Knockout | Hippocampus - metabolism | Animals | Fibroblasts - immunology | Neocortex - cytology | Mice | MicroRNAs - genetics | Primary Cell Culture | Virus diseases | Infection | MicroRNA | Cytokines | Analysis | Health aspects | Neurophysiology | Potentiation | Phosphorylation | α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptors | Proteins | Receptors | Clonal deletion | Synaptic transmission | α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid | Coding | Rodents | Plasticity (cortical) | Plasticity | Fibroblasts | Deletion | Life sciences | Chromosomes | Immune response | Long-term potentiation | Cortex | Paired-pulse facilitation | Plasticity (synaptic) | Stem cells | Genetic engineering | Receptor mechanisms | Hippocampus | Index Medicus
Journal Article
Development, ISSN 0950-1991, 02/2012, Volume 139, Issue 4, pp. 772 - 782
Journal Article
Genes and Development, ISSN 0890-9369, 03/2010, Volume 24, Issue 6, pp. 549 - 560
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2002, Volume 168, Issue 7, pp. 3195 - 3204
IFN-gamma-inducible protein 10 (IP-10, CXCL10), a chemokine secreted from cells stimulated with type I and II IFNs and LPS, is a chemoattractant for activated... 
RESPONSE GENE | ALPHA-CHEMOATTRACTANT | DIFFERENTIAL EXPRESSION | MULTIPLE-SCLEROSIS | DENDRITIC CELLS | LYMPHOID ORGANS | CHEMOKINE RECEPTOR CXCR3 | ENDOTHELIAL-CELLS | CONTACT HYPERSENSITIVITY | CENTRAL-NERVOUS-SYSTEM | IMMUNOLOGY | CD8-Positive T-Lymphocytes - cytology | Encephalomyelitis - genetics | Spleen - immunology | Ovalbumin - immunology | Demyelinating Diseases - genetics | Demyelinating Diseases - virology | Interferon-gamma - metabolism | Lymphocyte Depletion | Cell Movement - genetics | Lymphocyte Culture Test, Mixed | CD4-Positive T-Lymphocytes - immunology | Cell Movement - immunology | Murine hepatitis virus - immunology | Lymphocyte Activation - genetics | Lymphocyte Activation - immunology | Dermatitis, Contact - prevention & control | Isoantigens - immunology | Demyelinating Diseases - immunology | Interferon-gamma - antagonists & inhibitors | Spleen - pathology | Growth Inhibitors - pharmacology | Dermatitis, Contact - genetics | Mutagenesis, Site-Directed | CD4-Positive T-Lymphocytes - cytology | Mice, Inbred C57BL | Coronavirus Infections - genetics | Chemokines, CXC - genetics | Demyelinating Diseases - prevention & control | Ovalbumin - pharmacology | Chemokines, CXC - deficiency | Coronavirus Infections - immunology | Down-Regulation - genetics | Chemokine CXCL10 | Mice, Knockout | Animals | Down-Regulation - immunology | Encephalomyelitis - immunology | Chemokines, CXC - physiology | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | Antigens - pharmacology | IP-10 protein | g-Interferon-inducible protein 10 | g-Interferon | CXCL10 protein | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e58156 - e58156
Otitis media is a common reason for hearing loss, especially in children. Otitis media is a multifactorial disease and environmental factors, anatomic... 
INNER-EAR | MITOTIC ENTRY | MIDDLE-EAR MUCOSA | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | ULTRASTRUCTURE | SUSCEPTIBILITY | DYSFUNCTION | EUSTACHIAN-TUBE | HEARING | HERITABILITY | Genomic Instability | Hearing Loss - diagnosis | Humans | Otitis Media - etiology | Ear, Inner - pathology | Otitis Media - physiopathology | Skull - pathology | Ear, Inner - ultrastructure | Otitis Media - genetics | Ear, Middle - pathology | Gene Order | Disease Models, Animal | Gene Targeting | Gene Expression | Chromosomal Proteins, Non-Histone - metabolism | Otitis Media - pathology | Genotype | Eye Abnormalities - genetics | Chromosomal Proteins, Non-Histone - deficiency | Genetic Vectors - genetics | Chromosomal Proteins, Non-Histone - genetics | Mice, Knockout | Hearing Loss - genetics | Animals | B-Lymphocytes - immunology | Eye Abnormalities - pathology | Antibodies, Bacterial - immunology | Mice | Mutation | Otitis media | B cells | Embryonic stem cells | Analysis | Animal models | Pathogenesis | Epithelial cells | Brain stem | Genes | Embryo cells | Erythrocytes | Impairment | Ear diseases | Stem cell transplantation | Genomes | Microcephaly | Proteins | Red blood cells | Allografts | Cell cycle | Genetics | Children | Middle ear | Deoxyribonucleic acid--DNA | Age | Immune system | Effusion | Abnormalities | Blood cells | Cell division | Media | Environmental factors | Hearing impairment | Medical screening | Patients | Hearing loss | Studies | Hearing | Microencephaly | Lymphocytes B | Micronuclei | Stem cells | Ear | Auditory defects | Cancer | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2009, Volume 106, Issue 47, pp. 19860 - 19865
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 2014, Volume 51, Issue 2, pp. 210 - 222
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 2009, Volume 18, Issue 18, pp. 3484 - 3495
The Fanconi anemia (FA) core complex member FANCM remodels synthetic replication forks and recombination intermediates. Thus far, only one FA patient with... 
TARGETED DISRUPTION | REPLICATION FORKS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | MOUSE MODEL | GENETICS & HEREDITY | HOMOLOGOUS RECOMBINATION | GROUP-A GENE | KNOCKOUT MICE | CORE COMPLEX | REDUCED FERTILITY | <