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Cancer discovery, ISSN 2159-8290, 07/2018, Volume 8, Issue 7, pp. 822 - 835
Journal Article
Lung cancer (Amsterdam, Netherlands), ISSN 0169-5002, 09/2010, Volume 69, Issue 3, pp. 279 - 283
Journal Article
Clinical lung cancer, ISSN 1525-7304, 05/2013, Volume 14, Issue 3, pp. 205 - 214
Journal Article
Cancer cell, ISSN 1535-6108, 09/2017, Volume 32, Issue 3, pp. 360 - 376.e6
... these pathways and are associated with smoking-related non-small cell lung cancer. These cells, in the absence of any driver gene mutations, now transform by introducing a single KRAS mutation and form adenosquamous lung carcinomas in mice... 
genetic | lung cancer | epigenetic | polycomb | addiction | DNA methylation | cigarette smoke exposure | Kras | human bronchial epithelial cells | oncogene | HBECs | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Sirtuin 1 - metabolism | Smoking - adverse effects | Chromatin - metabolism | Epithelial Cells - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | Epithelial-Mesenchymal Transition - genetics | Promoter Regions, Genetic - genetics | DNA (Cytosine-5-)-Methyltransferases - metabolism | Cell Transformation, Neoplastic - genetics | Bronchi - pathology | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Cell Proliferation - genetics | Enhancer of Zeste Homolog 2 Protein - metabolism | Epigenomics | Carcinoma, Non-Small-Cell Lung - genetics | DNA (Cytosine-5-)-Methyltransferase 1 | Epithelial Cells - pathology | Smoking - genetics | DNA Methylation - genetics | Signal Transduction - genetics | Mutation - genetics | Phenotype | Animals | Mice | DNA Damage | Cell Transformation, Neoplastic - pathology | Genome, Human | Epigenetic inheritance | Chromatin | Gene mutations | Stem cells | Genetic aspects | Lung cancer, Non-small cell | Methylation | Smoking | Medical colleges | Anopheles | Genes | Cellular signal transduction | Index Medicus
Journal Article
Cell (Cambridge), ISSN 0092-8674, 01/2018, Volume 172, Issue 3, pp. 578 - 589.e17
Journal Article
Cancer letters, ISSN 0304-3835, 02/2015, Volume 357, Issue 2, pp. 520 - 526
Abstract Although mutations of p53 and KRAS and activation of NF-κB signaling have been highly associated with chemoresistance and tumorigenesis of lung cancer, the interactive mechanisms between two of p53, KRAS, and NF-κB are elusive... 
Hematology, Oncology and Palliative Medicine | NF-κB | KRAS | Lung cancer | Chemoresistance | p53 | P53 | Life Sciences & Biomedicine | Oncology | Science & Technology | Cell Cycle - genetics | Lung Neoplasms - drug therapy | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Paclitaxel - pharmacology | Apoptosis - drug effects | Heterocyclic Compounds, 3-Ring - pharmacology | Humans | Lung Neoplasms - metabolism | ras Proteins - metabolism | Apoptosis - genetics | NF-kappa B - metabolism | Tumor Suppressor Protein p53 - genetics | Cell Nucleus - metabolism | I-kappa B Kinase - metabolism | I-kappa B Kinase - antagonists & inhibitors | Tumor Burden - genetics | Antineoplastic Agents - pharmacology | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Carcinogenesis - genetics | Tumor Suppressor Protein p53 - metabolism | Proto-Oncogene Proteins - genetics | Cisplatin - pharmacology | Xenograft Model Antitumor Assays - methods | Blotting, Western | Carcinogenesis - drug effects | Drug Resistance, Neoplasm - genetics | Animals | Transcription Factor RelA - metabolism | Tumor Burden - drug effects | Active Transport, Cell Nucleus - drug effects | Cell Line, Tumor | Mice, Inbred BALB C | Pyridines - pharmacology | Mutation | Cell Cycle - drug effects | Cell Nucleus - drug effects | Drug Resistance, Neoplasm - drug effects | Index Medicus
Journal Article